Search results for "reactivation"

showing 10 items of 27 documents

EGFP Reporters for Direct and Sensitive Detection of Mutagenic Bypass of DNA Lesions

2020

The sustainment of replication and transcription of damaged DNA is essential for cell survival under genotoxic stress

DNA RepairTranscription GeneticDNA damageMutantGenetic VectorsGreen Fluorescent Proteinslcsh:QR1-502host cell reactivation (HCR)BiochemistryArticlelcsh:Microbiology03 medical and health scienceschemistry.chemical_compoundmutation assay0302 clinical medicinetranslesion synthesis (TLS)transcriptional mutagenesisTranscription (biology)Genes ReporterHumansCloning MolecularMolecular Biologyenhanced green fluorescent protein (EGFP)PolymeraseCells CulturedDNA damage tolerance030304 developmental biology0303 health sciencesbiologyDNA synthesisChemistryPoint mutationreporter assayRNACell biologyAmino Acid SubstitutionMutagenesis030220 oncology & carcinogenesisMutationbiology.proteinDNA damageDNAHeLa Cellsdamage bypassBiomolecules
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Cockayne syndrome: varied requirement of transcription-coupled nucleotide excision repair for the removal of three structurally different adducts fro…

2014

Hereditary defects in the transcription-coupled nucleotide excision repair (TC-NER) pathway of damaged DNA cause severe neurodegenerative disease Cockayne syndrome (CS), however the origin and chemical nature of the underlying DNA damage had remained unknown. To find out, to which degree the structural properties of DNA lesions determine the extent of transcription arrest in human CS cells, we performed quantitative host cell reactivation analyses of expression vectors containing various synthetic adducts. We found that a single 3-(deoxyguanosin-N 2-yl)-2-acetylaminofluorene adduct (dG(N 2)-AAF) constitutes an unsurmountable obstacle to transcription in both CS-A and CS-B cells and is remov…

DNA RepairTranscription GeneticGenetic ToxicologyDNA damagelcsh:MedicineBiologyToxicologyHost-Cell ReactivationBiochemistryCockayne syndromeCell LineDNA Adductschemistry.chemical_compoundGenes ReporterTranscription (biology)Nucleic AcidsMolecular Cell BiologyGene expressionmedicineHumansGene SilencingCockayne SyndromePoly-ADP-Ribose Binding Proteinslcsh:ScienceFluorenesMultidisciplinaryBiology and life sciencesOligonucleotidelcsh:RDNA HelicasesDeoxyguanosineDNACell Biologymedicine.diseaseMolecular biologyDNA Repair EnzymesGene Expression RegulationchemistryBiochemistrylcsh:QDNAResearch ArticleNucleotide excision repairPLoS ONE
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Alterations of DNA Repair in Melanoma Cell Lines Resistant to Cisplatin, Fotemustine, or Etoposide

2000

Resistance to chemotherapy is a common phenomenon in malignant melanoma. In order to assess the role of altered DNA repair in chemoresistant melanoma, we investigated different DNA repair pathways in one parental human melanoma line (MeWo) and in sublines of MeWo selected in vitro for drug resistance against four commonly used drugs (cisplatin, fotemustine, etoposide, and vindesine). Host cell reactivation assays with the plasmid pRSVcat were used to assess processing of different DNA lesions. With ultraviolet-irradiated plasmids, no significant differences were found, indicating a normal (nucleotide excision) repair of DNA photoproducts. With singlet oxygen-treated plasmid, the fotemustine…

DNA RepairUltraviolet RaysDNA repairDNA damageDrug ResistanceAntineoplastic AgentsDermatologyBiologyHost-Cell Reactivationbase excision repairBiochemistryNitrosourea Compounds03 medical and health sciencesOrganophosphorus Compounds0302 clinical medicineTumor Cells CulturedmedicineHumansMelanomaMolecular BiologyEtoposide030304 developmental biology0303 health scienceschemoresistanceMicrosatellite instabilityDNA NeoplasmBase excision repairCell BiologyDNA repair protein XRCC4nucleotide excision repairmedicine.diseaseAntineoplastic Agents PhytogenicMolecular biology3. Good healthOxygenmismatch repair030220 oncology & carcinogenesisDNA mismatch repairCisplatinMicrosatellite RepeatsNucleotide excision repairJournal of Investigative Dermatology
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Reactivación participativa del espacio público. Estudios de caso en Valencia y Madrid

2017

[EN] This work aims to understand the phenomenon of what we have propose to call "participatory reactivation of public space", which is characterized by the direct participation of citizens in the creation and management of social and cultural activities in public spaces, as part of a general will to reclaim that space as truly "public", where the citizens have power in the decision-making processes. This movement has been reinforced by the establishment of professional collectives and initiatives from public institutions. Seven case studies of organizations from Valencia and Madridwere conducted, in the form of semi-structured in-depth interviews,in order to define the shared characteristi…

Espacio público05 social sciences0211 other engineering and technologies0507 social and economic geography021107 urban & regional planning02 engineering and technologyReactivación participativaGeneral Earth and Planetary SciencesParticipatory reactivationUrban reactivationPublic spaceReactivación urbanaParticipación ciudadanaCitizen participation050703 geographyGeneral Environmental Science
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8-Oxoguanine DNA glycosylase (Ogg1) causes a transcriptional inactivation of damaged DNA in the absence of functional Cockayne syndrome B (Csb) prote…

2008

We have analysed the effect of oxidative guanine lesions on the expression of a transfected reporter gene in mouse embryonic fibroblasts deficient in Cockayne syndrome B protein (Csb) and/or the 8-oxoguanine DNA glycosylase (Ogg1). We used a highly sensitive flow cytometry-based approach and quantitative real-time PCR to measure the changes in gene expression caused by the presence of oxidised guanine residues generated by photosensitisation in the vector DNA. In wild-type cells, small numbers (one or three) of oxidised guanines did not affect gene expression at short times after transfections, whereas progressive reduction of the transgene expression was observed at later time points. Alth…

GuanineGuanineGreen Fluorescent ProteinsGene ExpressionBiologyHost-Cell ReactivationBiochemistryCell LineDNA GlycosylasesMicechemistry.chemical_compoundGenes ReporterGene expressionAnimalsHumansGene SilencingPoly-ADP-Ribose Binding ProteinsMolecular BiologyGeneReporter genePhotosensitizing AgentsReverse Transcriptase Polymerase Chain ReactionDNA HelicasesCell BiologyBase excision repairFlow CytometryMolecular biologyDNA Repair EnzymeschemistryDNA glycosylaseDNADNA DamageDNA Repair
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Hepatitis Virus Reactivation in Patients with Psoriasis Treated with Secukinumab in a Real-World Setting of Hepatitis B or Hepatitis C Infection

2022

Background and Objective Biologics for psoriasis, especially anti-tumor necrosis factor-alpha therapies, may reactivate hepatitis B virus (HBV) or hepatitis C virus (HCV) infections, as well in inactive carriers or patients with occult infection. However, some biologics, including anti-interleukin-17 therapies such as secukinumab, seem to be less likely to cause hepatitis reactivation. This study assessed the safety of secukinumab treatment in patients with psoriasis with HBV or HBC infection. Methods This was a retrospective cohort study of patients with moderate-to-severe psoriasis treated with secukinumab at seven Italian centers. Patients serologically positive for one or more of the fo…

Hepatitis B virusSecukinumab.HepacivirusAntibodies Monoclonal HumanizedAntiviral AgentsAntibodiesHepatitis B ChronicMonoclonalHumansPsoriasisPharmacology (medical)ViralChronicHumanizedRetrospective StudiesPsoriasiBiological ProductsHepatitis B Surface AntigensHepatitis ReactivationGeneral MedicineDNAAntibodies Monoclonal Humanized; Antiviral Agents; DNA Viral; Hepacivirus; Hepatitis B Surface Antigens; Hepatitis B virus; Humans; RNA; Retrospective Studies; Virus Activation; Biological Products; Hepatitis B; Hepatitis B Chronic; Hepatitis C; Psoriasispsoriasis treatment secukinumab hepatitis B hepatitis CHepatitis BHepatitis CDNA ViralRNAVirus Activation
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Corrigendum to “Premature ovarian senescence and a high miscarriage rate impair fertility in women with HCV” [J Hepatol 68 (2018) 33–41](S01688278173…

2018

It has come to our attention that the PITER framework investigator, Alessandro Federico, was incorrectly listed as F. Alessandro in the original manuscript. Please note that the correct name of this author is Alessandro Federico (2nd University of Naples). The correct list of PITER investigators is in the footnote below.

HepatologyHepatitis B; EASL guidelines; Treatment; Interferon; Entecavir; Tenofovir; TAF; HBsAg; Hepatocellular carcinoma; HBV DNA; HBV reactivation; Mother to child transmissionHepatocellular carcinomaHBV reactivationEASL guidelinesHepatitis BEntecavirTreatmentHBsAgTAFHBV DNAMother to child transmissionInterferonTenofovirEASL guideline
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Trans-Reactivation: A New Epigenetic Phenomenon Underlying Transcriptional Reactivation of Silenced Genes

2015

In order to study the role played by cellular RNA pools produced by homologous genomic loci in defining the transcriptional state of a silenced gene, we tested the effect of non-functional alleles of the white gene in the presence of a functional copy of white, silenced by heterochromatin. We found that non-functional alleles of white, unable to produce a coding transcript, could reactivate in trans the expression of a wild type copy of the same gene silenced by heterochromatin. This new epigenetic phenomenon of transcriptional trans-reactivation is heritable, relies on the presence of homologous RNA’s and is affected by mutations in genes involved in post-transcriptional gene silencing. Ou…

MaleCancer ResearchPEV white Trans-reactivation Epigenetics Gynogenesis ncRNAsRNA Untranslatedlcsh:QH426-470Transcription GeneticHeterochromatinSettore BIO/11 - Biologia MolecolareGenes InsectBiologySettore MED/13 - EndocrinologiaRNA interferenceSettore BIO/10 - BiochimicaHeterochromatinGene clusterGene expressionGeneticsGene silencingAnimalsDrosophila ProteinsEpigeneticsCompound Eye ArthropodEye ProteinsMolecular BiologyGeneGenetics (clinical)Ecology Evolution Behavior and SystematicsAllelesGeneticsEye ColorRNAlcsh:GeneticsSettore BIO/18 - GeneticaDrosophila melanogasterATP-Binding Cassette TransportersFemaleRNA InterferenceResearch ArticlePLoS Genetics
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Viral Sequence Analysis of Occult HBV Infection and Its Reactivation in Immunosuppressed Patients

2012

Mechanisms associated with reactivation of hepatitis B virus (HBV) in patients with occult HBV infection (OBI) remain unclear. In some cases immunosuppression is an enhancer of viral replication. However, not all patients with OBI who undergo immunosuppression experience reactivation. This study explores the role of viral heterogeneity as a determinant of occult HBV reactivation. HBV genotype, mutation patterns and quasispecies were assessed by sequencing the PreS/S region of 16 patients with OBI undergoing chemotherapy, 3 of whom experienced a OBI reactivation. The latter were also assessed at the time of reactivation. Phylogenetic analysis identified low nucleotide and amino acid diversit…

MaleHepatitis B virusSettore MED/07 - Microbiologia E Microbiologia ClinicaSettore MED/12 - GastroenterologiaGenotypeoccult HBV HBV reactivation phylogenetic analysismolecular epidemiologyGenetic VariationSequence Analysis DNAHepatitis BImmunocompromised HostDNA ViralHumansFemaleRetrospective Studies
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Isolated Nodal TBC Reactivation in a Patient with Post-Thrombocythemia Myelofibrosis Treated with Ruxolitinib: Case Report and Review of the Literatu…

2021

Ruxolitinib side effects include the most frequent hematological toxicity along with a more recently evidenced immunosuppressive activity, interfering both with the innate and adaptive immunity, and several cases of reactivation of latent infections by opportunistic agents in patients in treatment with ruxolitinib have been published in the last years. Several pathophysiological mechanisms may explain an association between ruxolitinib and opportunistic infections. From what we know, the only case of an isolated lymph node TBC reactivation in a ruxolitinib-treated myelofibrosis (MF) patient was reported by Patil et al. in 2016 [Int J Med Sci Public Health. 2017;6(3):1]. Other 10 cases descr…

Oncologymedicine.medical_specialtyRuxolitinibConstitutional symptomsSettore MED/15 - Malattie Del SangueHematological toxicityInternal medicineDrug DiscoverymedicinePharmacology (medical)In patientMyelofibrosisProspective cohort studyLymph nodePharmacologybusiness.industryInfection Myelofibrosis Reactivation Tuberculosis Ruxolitinib TBCGeneral Medicinemedicine.diseaseInfectious Diseasesmedicine.anatomical_structureOncologySputummedicine.symptombusinessmedicine.drug
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