Search results for "regulator"

showing 10 items of 1009 documents

Fumarate regulation of gene expression in Escherichia coli by the DcuSR (dcuSR genes) two-component regulatory system.

1998

ABSTRACT In Escherichia coli the genes encoding the anaerobic fumarate respiratory system are transcriptionally regulated by C 4 -dicarboxylates. The regulation is effected by a two-component regulatory system, DcuSR, consisting of a sensory histidine kinase (DcuS) and a response regulator (DcuR). DcuS and DcuR are encoded by the dcuSR genes (previously yjdHG ) at 93.7 min on the calculated E. coli map. Inactivation of the dcuR and dcuS genes caused the loss of C 4 -dicarboxylate-stimulated synthesis of fumarate reductase ( frdABCD genes) and of the anaerobic fumarate-succinate antiporter DcuB ( dcuB gene). DcuS is predicted to contain a large periplasmic domain as the supposed site for C 4…

Histidine KinaseGenetics and Molecular Biologymedicine.disease_causeMicrobiologyAntiportersBacterial ProteinsFumaratesmedicineEscherichia coliDicarboxylic AcidsMolecular BiologyEscherichia coliRegulation of gene expressionDicarboxylic Acid TransportersbiologySuccinate dehydrogenaseEscherichia coli ProteinsHistidine kinaseMembrane ProteinsPeriplasmic spaceGene Expression Regulation BacterialFumarate reductaseTwo-component regulatory systemDNA-Binding ProteinsSuccinate DehydrogenaseResponse regulatorMutagenesis InsertionalBiochemistryGenes Bacterialbiology.proteinCarrier ProteinsProtein KinasesSignal TransductionTranscription FactorsJournal of bacteriology
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Immunohistochemical expression of apoptotic factors, cytokeratins, and metalloproteinase-9 in periapical and epithelialized gingival lesions

2012

Bellmann K, 2010, CELL STRESS CHAPERON, V15, P101, DOI 10.1007-s12192-009-0126-9; Cappello Francesco, 2011, Front Biosci (Schol Ed), V3, P341, DOI 10.2741-s155; Cappello F, 2006, CANCER, V107, P2417, DOI 10.1002-cncr.22265; Cappello F, 2002, EUR J HISTOCHEM, V46, P199; Carneiro E, 2009, ORAL SURG ORAL MED O, V107, P127, DOI 10.1016-j.tripleo.2008.07.030; Chandra D, 2007, J BIOL CHEM, V282, P31289, DOI 10.1074-jbc.M702777200; Fujita Y, 2011, ODONTOLOGY, V100, P215; Garcia Celia Carrillo, 2007, Med Oral Patol Oral Cir Bucal, V12, pE585; Garcia CC, 2009, ORAL SURG ORAL MED O, V107, pE43, DOI 10.1016-j.tripleo.2008.12.002; Gregory CD, 2011, J PATHOL, V223, P177, DOI 10.1002-path.2792; Gupta S, …

Histologybusiness.industryCaspase 3GingivaApoptosisGeneral MedicineMolecular biologyImmunohistochemistryCaspase 9EpitheliumPathology and Forensic MedicineCell stressMatrix Metalloproteinase 9cytokeratins MMP-9 caspase-3 caspase-9 perapical lesions epithelial gingival lesions apoptosisIHC PCNA TUNELProliferating Cell Nuclear AntigenMedicineHumansKeratinsbusinessApoptosis Regulatory ProteinsPeriapical Granuloma
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Tackling Market Failure or Building a Cartel? Creation of an Investment Regulation System in Finnish Forest Industries

2015

Government intervention in the economy is often justified by the need to correct market failures. This study analyzes one case, the investments of Finnish forest industries, in which, in the late 1960s and early 1970s, both policy makers and the trade association representing the sector reasoned that intervention was particularly necessary because otherwise, the only substantial natural resource in the small country would be overexploited. In the long run, however, the growth of forest resources turned out to be higher, and the demand for wood lower, than expected. Furthermore, the most influential industrialists managed to “capture” the regulatory system and make it a component of their ne…

Historyforest industryRegulatory capture060106 history of social sciencesinvestment regulation systemMarket economy0502 economics and businessEconomicsta615market failure0601 history and archaeology050207 economicsFinlandTrade associationMarket failure05 social sciencesCartel06 humanities and the arts15. Life on landInvestment (macroeconomics)Natural resourceIntervention (law)Economic interventionism8. Economic growthBusiness Management and Accounting (miscellaneous)cartelsEnterprise & Society
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UME6, a Novel Filament-specific Regulator ofCandida albicansHyphal Extension and Virulence

2008

The specific ability of the major human fungal pathogen Candida albicans, as well as many other pathogenic fungi, to extend initial short filaments (germ tubes) into elongated hyphal filaments is important for a variety of virulence-related processes. However, the molecular mechanisms that control hyphal extension have remained poorly understood for many years. We report the identification of a novel C. albicans transcriptional regulator, UME6, which is induced in response to multiple host environmental cues and is specifically important for hyphal extension. Although capable of forming germ tubes, the ume6Δ/ume6Δ mutant exhibits a clear defect in hyphal extension both in vitro and during i…

HyphaGenes FungalRegulatorGerm tubeVirulenceBiologymedicine.disease_causeModels BiologicalMicrobiologyFungal ProteinsProtein filamentMiceGene Expression Regulation FungalCandida albicansmedicineAnimalsHumansDNA FungalCandida albicansMolecular BiologyOligonucleotide Array Sequence AnalysisMice Inbred BALB CFungal proteinMutationVirulenceCandidiasisGene Expression Regulation DevelopmentalArticlesCell Biologybiology.organism_classificationRepressor ProteinsDisease Models AnimalMutationFemaleTranscription FactorsMolecular Biology of the Cell
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Pseudohyphal Regulation by the Transcription Factor Rfg1p in Candida albicans

2010

ABSTRACT The opportunistic human fungal pathogen Candida albicans is a major cause of nosocomial infections. One of the fundamental features of C. albicans pathogenesis is the yeast-to-hypha transition. Hypha formation is controlled positively by transcription factors such as Efg1p and Cph1p, which are required for hyphal growth, and negatively by Tup1p, Rfg1p, and Nrg1p. Previous work by our group has shown that modulating NRG1 gene expression, hence altering morphology, is intimately linked to the capacity of C. albicans to cause disease. To further dissect these virulence mechanisms, we employed the same strategy to analyze the role of Rfg1p in filamentation and virulence. Studies using …

Hyphal growthHyphaHyphaeVirulenceBiologyMicrobiologyMicrobiologyFungal ProteinsMiceGene Expression Regulation FungalCandida albicansAnimalsHumansCandida albicansMolecular BiologyRegulator geneMice Inbred BALB CFungal proteinVirulenceCandidiasisGene Expression Regulation DevelopmentalArticlesGeneral Medicinebiology.organism_classificationCorpus albicansRepressor ProteinsComplementationFemaleEukaryotic Cell
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Nouvelles perspectives concernant la structure et la fonction du domaine carboxyl terminal de Hfq

2015

Accumulating evidence indicates that RNA metabolism components assemble into supramolecular cellular structures to mediate functional compartmentalization within the cytoplasmic membrane of the bacterial cell. This cellular compartmentalization could play important roles in the processes of RNA degradation and maturation. These components include Hfq, the RNA chaperone protein, which is involved in the post-transcriptional control of protein synthesis mainly by the virtue of its interactions with several small regulatory ncRNAs (sRNA). The Escherichia coli Hfq is structurally organized into two domains. An N-terminal domain that folds as strongly bent β-sheets within individual protomers to…

IDP intrinsically-disordered proteinslcsh:Lifelcsh:QR1-502sub-membrane macromolecular assemblyPlasma protein bindingsRNA small non-coding RNABiochemistrylcsh:Microbiologyamyloid fibrilsProtein biosynthesis0303 health sciences[SDV.BBM.BS]Life Sciences [q-bio]/Biochemistry Molecular Biology/Structural Biology [q-bio.BM]Escherichia coli Proteins030302 biochemistry & molecular biologyHfqCTRp Hfq C-terminal peptideFTIR Fourier transform infrared spectroscopyNTR N-terminal regionCompartmentalization (psychology)Cell biology[SDV.BBM.BP]Life Sciences [q-bio]/Biochemistry Molecular Biology/BiophysicsRNA Bacterialsmall non-coding ribonucleic acid (RNA)BiochemistryFSD Fourier self-deconvolutionTransfer RNAAmyloid fibrilProtein BindingBiophysicsBiologyHost Factor 1 Protein03 medical and health sciencesEscherichia coliThT thioflavin T[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologyProtein Structure QuaternaryncRNA regulatory non-coding RNAPost-transcriptional regulationMolecular Biology030304 developmental biologyOriginal PaperC-terminusRNA[SDV.BBM.BM]Life Sciences [q-bio]/Biochemistry Molecular Biology/Molecular biologyCell Biologycellular compartmentalizationWT wild-typeProtein Structure Tertiarylcsh:QH501-531Host Factor 1 ProteinCTR Hfq C-terminal regionribonucleic acid (RNA) processing and degradationBiophysicpost-transcriptional regulationBioscience Reports
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Dominating Clasp of the Financial Sector Revealed by Partial Correlation Analysis of the Stock Market

2010

What are the dominant stocks which drive the correlations present among stocks traded in a stock market? Can a correlation analysis provide an answer to this question? In the past, correlation based networks have been proposed as a tool to uncover the underlying backbone of the market. Correlation based networks represent the stocks and their relationships, which are then investigated using different network theory methodologies. Here we introduce a new concept to tackle the above question--the partial correlation network. Partial correlation is a measure of how the correlation between two variables, e.g., stock returns, is affected by a third variable. By using it we define a proxy of stoc…

INFORMATIONEconomicsPORTFOLIO OPTIMIZATIONEconomic Modelslcsh:MedicineNetwork theorySocial and Behavioral SciencesFinancial correlationStock exchangeMicroeconomicsEconometricsEconomicslcsh:ScienceMathematical ComputingMarketingMultidisciplinarySystems BiologyApplied MathematicsPhysicsStatisticsComplex SystemsMathematical EconomicsModels EconomicInterdisciplinary PhysicsAlgorithmsResearch ArticleCORRELATION-BASED NETWORKS; PORTFOLIO OPTIMIZATION; CORRELATION-MATRICES; TIME-SERIES; INFORMATIONNew YorkTIME-SERIESHumansInvestmentsStatistical MethodsCorrelation swapBiologyStructure of MarketsStock (geology)Partial correlationCORRELATION-BASED NETWORKSRegulatory NetworksModels Statisticallcsh:RFinancial marketComputational BiologyIndustrial OrganizationModels TheoreticalCORRELATION-MATRICESlcsh:QStock marketMathematicsForecasting
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Lymphokine profile and activation pattern of two unrelated antigen- or idiotype-specific T suppressor cell clones.

1992

Two T suppressor (Ts) clones of different specificity have been analyzed for their lymphokine spectrum. BVI/5 is an I-Ek-restricted bovine serum albumin (BSA)-specific Ts cell clone from a CBA/J mouse tolerized by low doses of BSA. It affects directly or indirectly the function of BSA-specific T helper (Th) cells. The Ts cell clone 178-4 from a BALB/c mouse is I-Ed restricted and recognizes the public J558 Id on B cells. It prevents alpha(1----3)dextran B 1355S (Dex)-specific IgG antibody production and drives Dex-specific J558 idiotype-bearing B cells into an anergic B IgG memory cell state. Both Ts cell clones thus cause specific suppression, yet in different experimental systems using di…

IdiotypeMalemedicine.medical_treatmentImmunologyLymphocyte ActivationT-Lymphocytes RegulatoryInterferon-gammaMiceAntigenInterferonAntibody SpecificityCell ClonemedicineImmunology and AllergyAnimalsAntigensLymphokinesCD40biologyLymphokineHistocompatibility Antigens Class IISerum Albumin BovineT-Lymphocytes Helper-InducerMolecular biologyClone CellsCytokineImmunologybiology.proteinMice Inbred CBAClone (B-cell biology)medicine.drugInterleukin-1European journal of immunology
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Interferon-α Abrogates Tolerance Induction by Human Tolerogenic Dendritic Cells

2011

Background Administration of interferon-α (IFN-α) represents an approved adjuvant therapy as reported for malignancies like melanoma and several viral infections. In malignant diseases, tolerance processes are critically involved in tumor progression. In this study, the effect of IFN-α on tolerance induction by human tolerogenic dendritic cells (DC) was analyzed. We focussed on tolerogenic IL-10-modulated DC (IL-10 DC) that are known to induce anergic regulatory T cells (iTregs). Methodology/Principal Findings IFN-α promoted an enhanced maturation of IL-10 DC as demonstrated by upregulation of the differentiation marker CD83 as well as costimulatory molecules. IFN-α treatment resulted in an…

Immune CellsT cellImmunologylcsh:MedicineAntigen-Presenting CellsPriming (immunology)Adaptive ImmunityBiologyLymphocyte ActivationImmune SuppressionT-Lymphocytes RegulatoryImmunophenotypingImmune toleranceImmune ActivationImmunomodulationImmune TolerancemedicineHumansCytotoxic T celllcsh:ScienceAntigen-presenting cellBiologyImmune ResponseClonal AnergyMultidisciplinaryClonal anergyT Cellslcsh:RImmunityImmunoregulationInterferon-alphaCell DifferentiationDendritic CellsInterleukin-10Tolerance inductionmedicine.anatomical_structureImmune SystemImmunologyCancer researchCytokineslcsh:QImmunizationCD8Research ArticlePLoS ONE
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Expression profiling of autoimmune regulator AIRE mRNA in a comprehensive set of human normal and neoplastic tissues.

2006

Defects in the autoimmune regulator (AIRE) gene cause the monogenic autoimmune disease autoimmune polyendocrinopathy syndrome type 1 (APS-1), which is characterized by a loss of self-tolerance to multiple organs. In concordance with its role in immune tolerance, AIRE is strongly expressed in medullary thymic epithelial cells (mTECs). Data on mechanisms controlling AIRE activation and the expression of this gene in other tissues are fragmentary and controversial. We report here AIRE mRNA expression profiling of a large set of normal human tissues and cells, tumor specimen and methylation deficient cell lines. On this broad data basis we found that AIRE mRNA expression is confined to mTECs in…

Immune Tolerance/geneticsThymus Gland/immunologyTranscription GeneticImmunologyTranscription Genetic/immunologyThymus GlandBiologyLymph Nodes/immunologymedicine.disease_causeAutoimmunityImmune toleranceCell Line TumorNeoplasmsmedicineTranscriptional regulationImmune ToleranceImmunology and AllergyHumansRNA MessengerPolyendocrinopathies AutoimmuneGeneTranscription Factors/biosynthesisAutoimmune diseaseReverse Transcriptase Polymerase Chain ReactionGene Expression ProfilingRNA Messenger/biosynthesisDNA Methylationmedicine.diseaseAutoimmune regulatorNeoplasms/geneticsPolyendocrinopathies Autoimmune/geneticsGene expression profilingGene Expression Regulation NeoplasticDNA methylationImmunologyCancer researchLymph NodesGene Expression Profiling/methodsTranscription FactorsImmunology letters
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