Search results for "response"

showing 10 items of 4136 documents

Early Response to treatment in Eating Disorders: A Systematic Review and a Diagnostic Test Accuracy Meta-Analysis

2016

Objective: Early response to eating disorders treatment is thought to predict a later favourable outcome. A systematic review of the literature and meta-analyses examined the robustness of this concept. Method: The criteria used across studies to define early response were summarised following the Preferred Reporting Items for Systematic Reviews and Meta-Analyses guidelines. Diagnostic Test Accuracy methodology was used to estimate the size of the effect. Results: Findings from 24 studies were synthesized and data from 14 studies were included in the meta-analysis. In Anorexia Nervosa, the odds ratio of early response predicting remission was 4.85(95%CI: 2.94–8.01) and the summary Area Unde…

Time FactorsAnorexia NervosaDiagnostic Tests RoutineReproducibility of Resultscognitive behavioural therapyearly responseFeeding and Eating DisordersClinical PsychologyTreatment OutcomePsychiatry and Mental Healtheating disorderSettore M-PSI/08 - Psicologia Clinicabinge eating disorderHumansfamily therapyBulimia NervosaBinge-Eating DisorderRandomized Controlled Trials as Topic
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Oxidative DNA damage and disturbance of antioxidant capacity by alternariol in Caco-2 cells

2015

Oxidative stress occurs as a consequence of an imbalance between the prooxidant/antioxidant systems, causing an increase of intracellular generation of reactive oxygen species. Alternariol (AOH), a mycotoxin produced by Alternaria sp. can alter the action of glutathione (GSH) and the enzymes involved in the redox system, causing damage to cellular macromolecules such as DNA. The aims of this work were to determine the induction of oxidative stress by the antioxidant defenses imbalance in relation to glutathione (GSH), glutathione reductase (GR), glutathione transferase (GST), glutathione peroxidase (GPx) levels and DNA damage in Caco-2 cells derived from adenocarcinoma human colon. Oxidativ…

Time FactorsAntioxidantDNA damagemedicine.medical_treatmentGlutathione reductaseAlternariolBiologyToxicologymedicine.disease_causeAntioxidantsLactoneschemistry.chemical_compoundmedicineHumansGlutathione Transferasechemistry.chemical_classificationGlutathione PeroxidaseDose-Response Relationship DrugGlutathione peroxidaseGeneral MedicineGlutathioneMycotoxinsGlutathioneComet assayOxidative StressGlutathione ReductaseBiochemistrychemistryComet AssayCaco-2 CellsOxidative stressDNA DamageToxicology Letters
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Differential effects of cysteine and methionine residues in the antioxidant activity of human serum albumin

2005

Antioxidant properties of human serum albumin (HSA) may explain part of its beneficial role in various diseases related to free radical attack. In the present study, the antioxidant role of Cys and Met was studied by copper-mediated oxidation of human low density lipoproteins and by free radical-induced blood hemolysis which essentially assessed metal-chelating and free radical scavenging activities, respectively. Mild conditions were set up to specifically modify Cys and Met residues by N-ethylmaleimide (NEM) and chloramine T treatments, respectively. We found that Met and Cys accounted for 40-80% of total antioxidant activity of HSA. Copper binding to HSA was decreased by about 50% with c…

Time FactorsAntioxidantFree Radicalsmedicine.medical_treatmentDithionitrobenzoic AcidHemolysisBiochemistryAntioxidantsTosyl Compoundschemistry.chemical_compoundMethioninemedicineHumansChelationCysteineSerum AlbuminMethionineDose-Response Relationship DrugChloraminesFree Radical ScavengersGeneral MedicineFree radical scavengerHuman serum albuminmedicine.diseaseHemolysisLipoproteins LDLOxygenOxidative StresschemistryBiochemistryEthylmaleimideChloramine-TOxidation-ReductionCopperPhenanthrolinesProtein Bindingmedicine.drugCysteineFree Radical Research
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PHEA-graft-polybutylmethacrylate copolymer microparticles for delivery of hydrophobic drugs.

2012

Abstract Polymeric microparticles encapsulating two model hydrophobic drugs, beclomethasone dipropionate (BDP) and flutamide (FLU) were prepared by using the high pressure homogenization-solvent evaporation method starting from a oil-in-water emulsion. For the preparation of polymeric microparticles a α,β-poly(N-2-hydroxyethyl)- d , l -aspartamide (PHEA) graft copolymer with comb like structure was properly synthesized via grafting from atom transfer radical polymerization (ATRP) technique, by using two subsequent synthetic steps. In the first step a polymeric multifunctional macroinitiator was obtained by the conjugation of a proper number of 2-bromoisobutyryl bromide (BIB) residues to the…

Time FactorsBioadhesivePharmaceutical ScienceCell LineDrug Delivery SystemsPolymethacrylic AcidsPolymer chemistryMucoadhesionCopolymerSide chainHumansPhea polybutylmethacrylate microparticles drug deliveryParticle SizeGlucocorticoidsDrug CarriersDose-Response Relationship DrugChemistryAtom-transfer radical-polymerizationBeclomethasoneAdhesivenessAndrogen AntagonistsGraftingFlutamideMicrospheresPolymerizationDelayed-Action PreparationsEmulsionSolventsNanoparticlesEmulsionsCaco-2 CellsPeptidesHydrophobic and Hydrophilic InteractionsInternational journal of pharmaceutics
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Secretion of neutral and acid DNases in cultivated human lymphocytes after incubation with DNA; possible consequences for inhalation anesthesia.

1995

Abstract After incubation with DNA human lymphocytes release neutral and acid DNase activities into the culture medium; the release depends on DNA concentration and time of cultivation. The electrophoretic mobility of the released neutral DNase activity is in accordance with DNase I and the electrophoretic mobility of the released acid DNase activity with DNase II. The released DNase activities do not originate from dead cells and are not influenced by blast cell formation. The anesthetic halothane can inhibit the released neutral and acid DNase activities. Inhalation anesthesia can possibly disturb the correlation between DNA and DNases in human blood.

Time FactorsBiologyGeneral Biochemistry Genetics and Molecular Biologychemistry.chemical_compoundPrecursor cellmedicineDeoxyribonuclease IHumansSecretionLymphocytesIncubationCells CulturedDeoxyribonucleasesEndodeoxyribonucleasesInhalationDose-Response Relationship DrugDNAHydrogen-Ion ConcentrationDose–response relationshipKineticschemistryBiochemistryAnestheticHalothaneAnesthesia InhalationHalothaneDNAmedicine.drugZeitschrift fur Naturforschung. C, Journal of biosciences
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Does low concentration mycotoxin exposure induce toxicity in HepG2 cells through oxidative stress?

2020

The purpose of this study was to determine whether exposure to low concentrations of deoxynivalenol (DON), T-2 toxin (T-2) and patulin (PAT) in a human hepatocellular carcinoma cell line (HepG2) exerts toxic effects through mechanisms related to oxidative stress, and how cells deal with such exposure. Cell viability was determined by the MTT and protein content (PC) assays over 24, 48 and 72 h. The IC

Time FactorsCell SurvivalHealth Toxicology and MutagenesisMitochondria LiverHepatic carcinoma010501 environmental sciencesToxicologymedicine.disease_cause01 natural sciencesPatulinInhibitory Concentration 5003 medical and health scienceschemistry.chemical_compoundmedicineHumansMycotoxinVolume concentration0105 earth and related environmental sciencesMembrane Potential Mitochondrial0303 health sciencesDose-Response Relationship DrugToxinChemistry030302 biochemistry & molecular biologyfood and beveragesHep G2 CellsMycotoxinsMolecular biologydigestive system diseasesOxidative StressT-2 ToxinPatulinHepg2 cellsToxicityHepatocytesLipid PeroxidationReactive Oxygen SpeciesTrichothecenesOxidative stressToxicology Mechanisms and Methods
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pRb suppresses camptothecin-induced apoptosis in human osteosarcoma Saos-2 cells by inhibiting c-Jun N-terminal kinase

2001

AbstractThis paper studies the cytotoxic effect induced by the topoisomerase I inhibitor camptothecin in human osteosarcoma Saos-2 cells, which lack p53 and contain a non-functional form of the product of the retinoblastoma gene, pRb. Cytotoxicity induced by camptothecin was dose- and time-dependent; the treatment with 100 nM camptothecin reduced cell viability by 50% at 32 h and by 75% at 72 h of exposure. The cytotoxic effect was caused by apoptosis, as ascertained by morphological evidence, acridine orange-ethidium bromide staining and flow cytometric analysis. Apoptosis was accompanied by both the activation of caspase-3 and the fragmentation of poly(ADP-ribose) polymerase. Treatment wi…

Time FactorsCell SurvivalProto-Oncogene Proteins c-junBlotting WesternBiophysicsApoptosisBiologyTransfectionRetinoblastoma ProteinBiochemistryStructural BiologyTumor Cells CulturedpRb JNK topoisomerase I inhibitors osteosarcomaGeneticsmedicineHumansCytotoxic T cellViability assayPhosphorylationFragmentation (cell biology)neoplasmsMolecular BiologySaos-2 cellsc-Jun N-terminal kinaseCell SizeDose-Response Relationship DrugCaspase 3Cell growthCell Cyclec-junJNK Mitogen-Activated Protein KinasesHydrogen PeroxideCell BiologyFlow CytometryGlutathioneMolecular biologyEnzyme ActivationOxidative StresspRbDNA Topoisomerases Type IApoptosisCaspasesCamptothecinMitogen-Activated Protein KinasesPoly(ADP-ribose) PolymerasesTopoisomerase I InhibitorsCamptothecinmedicine.drugFEBS Letters
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Side-specific effects by cadmium exposure: Apical and basolateral treatment in a coculture model of the blood–air barrier

2010

Cadmium (Cd{sup 2+}) is a widespread environmental pollutant, which is associated with a wide variety of cytotoxic and metabolic effects. Recent studies showed that intoxication with the heavy metal most importantly targets the integrity of the epithelial barrier. In our study, the lung epithelial cell line, NCI H441, was cultured with the endothelial cell line, ISO-HAS-1, as a bilayer on a 24-well HTS-Transwell (registered) filter plate. This coculture model was exposed to various concentrations of CdCl{sub 2}. The transepithelial electrical resistance decreased on the apical side only after treatment with high Cd{sup 2+} concentrations after 48 h. By contrast, a breakdown of TER to less t…

Time FactorsCell SurvivalToxicologyTight JunctionsProinflammatory cytokineAlveolar cellsCadmium ChlorideCell Line TumorElectric ImpedancemedicineHumansViability assayRespiratory systemFragmentation (cell biology)Cell ShapePharmacologyBlood-Air BarrierDose-Response Relationship DrugChemistryCell PolarityEndothelial CellsEpithelial CellsBlood–air barrierAdherens JunctionsMolecular biologyCoculture TechniquesEndothelial stem cellmedicine.anatomical_structureCytoprotectionImmunologyCytokinesCalciumInflammation MediatorsIntracellularToxicology and Applied Pharmacology
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Cadmium regulation of apoptotic and stress response genes in tumoral and immortalized epithelial cells of the human breast

2008

Cadmium (Cd) is a widely-disseminated metal which can be imported and accumulated in living cells thereby drastically interfering with their biological mechanisms. Increasing interest has been recently focused on the elucidation of the cellular and molecular aspects of Cd-dependent regulation of gene expression and signal transduction pathways in different model system. Concerning breast cancer, very limited studies have been produced so far on the role played by Cd on estrogen receptor-negative human breast cancer cells, that are expected to be insensitive to the already-proven metallo-estrogenic effect exerted by Cd on the estrogen receptor-positive cell counterparts. Here, we have examin…

Time FactorsCellApoptosisBiologyBiochemistryHsp27Cell Line TumorHeat shock proteincadmium apoptosis stress response tumor cells human breastmedicineAnimalsHumansBreastSettore BIO/06 - Anatomia Comparata E CitologiaRegulation of gene expressionDose-Response Relationship DrugEpithelial CellsGeneral MedicineMolecular biologyCell biologyGene Expression Regulation NeoplasticOxidative StressSettore BIO/18 - Geneticamedicine.anatomical_structureReceptors EstrogenApoptosisCell cultureCancer cellbiology.proteinCattleEnvironmental PollutantsSignal transductionCadmium
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SYNTHESIS, CHARACTERIZATION AND IN VITRO CYTOTOXICITY STUDIES OF A MACROMOLECULAR CONJUGATE OF PACLITAXEL BEARING OXYTOCIN AS TARGETING MOIETY.

2007

The present study describes the experimental synthetic procedure and the characterization of a new polyaspartamide macromolecular prodrug of paclitaxel, bearing oxytocin residues as targeting moieties. In vitro stability studies of bioconjugate, performed in media mimicking biological fluids (buffer solutions at pH 7.4 and 5.5) and in human plasma, evidenced the high stability of the targeting portion (oxytocin)-polymer linkage and the ability of this conjugate to release linked paclitaxel in a prolonged way in plasma. Moreover, preliminary in vitro antiproliferative studies, carried out on MCF-7 cells, that are oxytocin receptor positive cells, showed that the polymeric conjugate has the s…

Time FactorsChemistry PharmaceuticalDrug CompoundingpolyaspartamidePharmaceutical ScienceBreast NeoplasmsPolyethylene Glycolschemistry.chemical_compoundpaclitaxelDrug StabilityCell Line TumoroxytocinHumansMoietyProdrugsbioconjugateCytotoxicityCell ProliferationDrug CarriersDose-Response Relationship DrugMolecular StructureHydrolysisdrug targetingGeneral MedicineHydrogen-Ion ConcentrationAntineoplastic Agents PhytogenicOxytocin receptorIn vitroSolubilityPaclitaxelchemistryBiochemistryTargeted drug deliveryReceptors OxytocinDelayed-Action PreparationsFemalePeptidesDrug carrierBiotechnologyConjugate
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