Search results for "reverse transcriptase"

showing 10 items of 715 documents

Interplay between RNA structure and protein evolution in HIV-1.

2010

The genomes of many RNA viruses contain abundant secondary structures that have been shown to be important for understanding the evolution of noncoding regions and synonymous sites. However, the consequences for protein evolution are less well understood. Recently, the secondary structure of the HIV-1 RNA genome has been experimentally determined. Using this information, here we show that RNA structure and proteins do not evolve independently. A negative correlation exists between the extent of base pairing in the genomic RNA and amino acid variability. Relaxed RNA structures may favor the accumulation of genetic variation in proteins and, conversely, sequence changes driven by positive sel…

GeneticsBase SequenceBase pairMolecular Sequence DataRNAGenome ViralBiologyGenomeBiological EvolutionReverse transcriptaseViral ProteinsGenetic variationGeneticsHIV-1HumansNucleic Acid ConformationRNANucleic acid structureMolecular BiologyGeneProtein secondary structureEcology Evolution Behavior and SystematicsMolecular biology and evolution
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Genomic response programs of Saccharomyces cerevisiae following protoplasting and regeneration.

2007

Abstract Global transcription profiling during regeneration of Saccharomyces cerevisiae protoplasts was explored. DNA microarrays measured the expression of 6388 genes and wall removal resulted initially in over-expression of 861 genes that decayed later on, a behaviour expected from a transient stress response. Kinetics of expression divided the genes into 25 clusters. Transcription of the genes from clusters 14–25 was initially up-regulated, suggesting that the grouped genes permitted cell adaptation to the removal of the wall. Clustering of genes involved in “wall structure and biosynthesis” showed that most of them had initially low levels of expression that increased along the process.…

GeneticsSaccharomyces cerevisiae ProteinsbiologyReverse Transcriptase Polymerase Chain ReactionGene Expression ProfilingProtoplastsSaccharomyces cerevisiaeGenomicsSaccharomyces cerevisiaeProtoplastbiology.organism_classificationMicrobiologyCell biologyGene expression profilingTranscription (biology)Cell WallGene Expression Regulation FungalGene expressionGeneticsDNA microarrayCandida albicansGeneOligonucleotide Array Sequence AnalysisFungal genetics and biology : FGB
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The role of telomeres in predicting individual radiosensitivity of patients with cancer in the era of personalized radiotherapy.

2014

Radiotherapy plays a key role in cancer treatments, but tumor cell death differs from one tumor to another. The response of patients to radiotherapy varies considerably and adverse side effects are difficult to prevent. The mechanisms involved in the heterogeneity of this response are not well understood. In order to enhance the efficacy and safety of radiotherapy, it is important to identify subpopulations most at risk of developing a late adverse response to radiotherapy. Telomeres are composed of multiple repeats of a unique sequence of nucleotides forming a TTAGGG pattern. They protect chromosomes from end-to-end fusion and maintain genomic stability. Telomeres have been shown to be ext…

GeneticsTelomerasebusiness.industrymedicine.medical_treatmentCancerGeneral MedicineTelomeremedicine.diseaseRadiation ToleranceTelomereRadiation therapyTelomerase RNA componentOncologyMRN complexPredictive Value of TestsChromosome instabilityNeoplasmsmedicineCancer researchAnimalsHumansRadiology Nuclear Medicine and imagingTelomerase reverse transcriptasePrecision MedicinebusinessCancer treatment reviews
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Simultaneous Aurora-A/STK15 overexpression and centrosome amplification induce chromosomal instability in tumour cells with a MIN phenotype

2007

Abstract Background Genetic instability is a hallmark of tumours and preneoplastic lesions. The predominant form of genome instability in human cancer is chromosome instability (CIN). CIN is characterized by chromosomal aberrations, gains or losses of whole chromosomes (aneuploidy), and it is often associated with centrosome amplification. Centrosomes control cell division by forming a bipolar mitotic spindle and play an essential role in the maintenance of chromosomal stability. However, whether centrosome amplification could directly cause aneuploidy is not fully established. Also, alterations in genes required for mitotic progression could be involved in CIN. A major candidate is represe…

Genome instabilityCancer ResearchCellular differentiationAneuploidyApoptosisCell CommunicationSpindle ApparatusBiologyProtein Serine-Threonine Kinaseslcsh:RC254-282Aurora KinasesChromosome instabilityChromosomal InstabilitymedicineTumor Cells CulturedGeneticsHumansRNA Small InterferingMitosisIn Situ Hybridization FluorescenceAurora Kinase ACentrosomePloidiesReverse Transcriptase Polymerase Chain ReactionAurora-A centrosomes amplification aneuploidyCell Differentiationlcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensmedicine.diseaseAneuploidyCell biologySpindle apparatusUp-RegulationSettore BIO/18 - GeneticaCell Transformation NeoplasticPhenotypeMicroscopy FluorescenceOncologyCentrosomeColonic NeoplasmsEctopic expressionMicrosatellite InstabilityResearch ArticleBMC Cancer
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CENPA overexpression promotes genome instability in pRb-depleted human cells

2009

Abstract Background Aneuploidy is a hallmark of most human cancers that arises as a consequence of chromosomal instability and it is frequently associated with centrosome amplification. Functional inactivation of the Retinoblastoma protein (pRb) has been indicated as a cause promoting chromosomal instability as well centrosome amplification. However, the underlying molecular mechanism still remains to be clarified. Results Here we show that pRb depletion both in wild type and p53 knockout HCT116 cells was associated with the presence of multipolar spindles, anaphase bridges, lagging chromosomes and micronuclei harbouring whole chromosomes. In addition aneuploidy caused by pRb acute loss was…

Genome instabilityCancer ResearchChromosomal Proteins Non-HistoneBlotting WesternBiologyAutoantigensRetinoblastoma Proteinlcsh:RC254-282Genomic InstabilityRNA interferenceChromosome instabilityCentromere Protein ACell Line TumorHumansRNA Processing Post-TranscriptionalDNA PrimersCENPABase SequenceReverse Transcriptase Polymerase Chain ReactionResearchRetinoblastoma proteincentromere protein aneuploidy pRBlcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensMolecular biologyCell biologySettore BIO/18 - GeneticaSpindle checkpointOncologyMicroscopy FluorescenceCentrosomebiology.proteinMolecular MedicineRNA Interferencebiological phenomena cell phenomena and immunityCentromere Protein AMolecular Cancer
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Identification of sapovirus infection among Japanese infants in a day care center.

2005

A total of 921 fecal specimens collected from 44 infants in a day care center in Tokyo, Japan during June 1999 to July 2000 were tested for the presence of sapovirus by reverse transcription-polymerase chain reaction (RT-PCR). Of 88 fecal specimens from infants with acute gastroenteritis, 2.3% (2) were found to be positive for sapovirus. Twenty-two of 833 (2.6%) fecal specimens collected from asymptomatic infants were also infected with this virus. Another interesting feature was the demonstration of high incidence of sapovirus infection (95.5%, 21 of 22) identified in a single day care center, which was not due to viral shedding after the latest acute gastroenteritis. Sapovirus was subject…

GenotypeAsymptomaticVirusSapovirusFecesJapanVirologyGenotypeMedicineHumansViral sheddingPathogenFecesPhylogenyCaliciviridae Infectionsbiologybusiness.industryReverse Transcriptase Polymerase Chain ReactionAge FactorsInfant NewbornOutbreakGenetic VariationInfantSapovirusChild Day Care Centersbiology.organism_classificationVirologyGastroenteritisInfectious DiseasesAcute DiseaseDiarrhea InfantileSeasonsmedicine.symptombusinessJournal of medical virology
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Functional and genetic characterization of the non-lysosomal glucosylceramidase 2 as a modifier for Gaucher disease.

2013

Background: Gaucher disease (GD) is the most common inherited lysosomal storage disorder in humans, caused by mutations in the gene encoding the lysosomal enzyme glucocerebrosidase (GBA1). GD is clinically heterogeneous and although the type of GBA1 mutation plays a role in determining the type of GD, it does not explain the clinical variability seen among patients. Cumulative evidence from recent studies suggests that GBA2 could play a role in the pathogenesis of GD and potentially interacts with GBA1. Methods: We used a framework of functional and genetic approaches in order to further characterize a potential role of GBA2 in GD. Glucosylceramide (GlcCer) levels in spleen, liver and brain…

GenotypeDiseaseBiologymedicine.disease_causePolymorphism Single NucleotidePathogenesis03 medical and health sciencesMice0302 clinical medicineGenotypemedicineAnimalsGenetics(clinical)Pharmacology (medical)GeneGenetics (clinical)Cells Cultured030304 developmental biologyMedicine(all)Mice Knockout0303 health sciencesMutationGaucher DiseaseReverse Transcriptase Polymerase Chain ReactionResearchGeneral MedicineHematologyFibroblastsHuman genetics3. Good healthGlucosylceramidaseImmunologyGlucosylceramidaseGlucocerebrosidase030217 neurology & neurosurgery
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Establishment of a quantitative RT-pCR for detection of vascular cell adhesion molecule-1 transcripts in endothelial cells after stimulation with adv…

1998

Advanced glycation endproducts (AGE) are supposed to increase endothelial expression of adhesion molecules like vascular cell adhesion molecule-1 (VCAM-1) by inducing an intracellular stress with subsequent activation of nuclear transcription factor NF-kappa-B. Quantitative analysis of VCAM-1-transcription has not been demonstrated concerning this topic. Thus, the aim of this study was to establish quantitative reverse transcription polymerase chain reaction (RT-PCR) assays using a spacer gene in order to measure the amounts of specific mRNA for VCAM-1 in human umbilical vein endothelial cells (HUVEC) which were stimulated with AGE-albumin (AGE-BSA). A recombinant RNA-standard was synthesiz…

Glycation End Products AdvancedCell adhesion moleculeReverse Transcriptase Polymerase Chain ReactionCellEndothelial CellsReproducibility of ResultsVascular Cell Adhesion Molecule-1Serum Albumin BovineGeneral MedicineCell cycleBiologyUmbilical veinCell biologyReverse transcription polymerase chain reactionReal-time polymerase chain reactionmedicine.anatomical_structureGeneticsmedicineHumansEndothelium VascularRNA MessengerCell adhesionIntracellularCells CulturedInternational journal of molecular medicine
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8-Oxoguanine DNA glycosylase (Ogg1) causes a transcriptional inactivation of damaged DNA in the absence of functional Cockayne syndrome B (Csb) prote…

2008

We have analysed the effect of oxidative guanine lesions on the expression of a transfected reporter gene in mouse embryonic fibroblasts deficient in Cockayne syndrome B protein (Csb) and/or the 8-oxoguanine DNA glycosylase (Ogg1). We used a highly sensitive flow cytometry-based approach and quantitative real-time PCR to measure the changes in gene expression caused by the presence of oxidised guanine residues generated by photosensitisation in the vector DNA. In wild-type cells, small numbers (one or three) of oxidised guanines did not affect gene expression at short times after transfections, whereas progressive reduction of the transgene expression was observed at later time points. Alth…

GuanineGuanineGreen Fluorescent ProteinsGene ExpressionBiologyHost-Cell ReactivationBiochemistryCell LineDNA GlycosylasesMicechemistry.chemical_compoundGenes ReporterGene expressionAnimalsHumansGene SilencingPoly-ADP-Ribose Binding ProteinsMolecular BiologyGeneReporter genePhotosensitizing AgentsReverse Transcriptase Polymerase Chain ReactionDNA HelicasesCell BiologyBase excision repairFlow CytometryMolecular biologyDNA Repair EnzymeschemistryDNA glycosylaseDNADNA DamageDNA Repair
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Quantification of CD8+ T lymphocytes responsive to human immunodeficiency virus (HIV) peptide antigens in HIV-infected patients and seronegative pers…

1998

/ T cells responding to HIV-1 peptides were observed in none of 11 HIV- seronegative donors without a history of HIV exposure. ELISPOT assays are relatively fast and easy to perform and appear to reliably detect T cell reactivity due to previous exposure to HIV. These findings support the use of the ELISPOT assay for monitoring T cell responsiveness to HIV peptides. In acute infection with the human immunodeficiency virus We described recently an enzyme-linked immunospot (ELISPOT) assay to detect and quantitate single blood-de- type 1 (HIV-1), initial reduction in virus load is associated with the appearance of a high frequency of antiviral cytotoxic T rived CD8 / T lymphocytes forming tumo…

HIV AntigensT cellHIV Core Protein p24HIV InfectionsBiologyCD8-Positive T-LymphocytesHLA-A3 AntigenVirusAntigenRisk FactorsHLA-A2 AntigenmedicineImmunology and AllergyCytotoxic T cellHumansAntigen PresentationELISPOTT lymphocytebiology.organism_classificationVirologyHIV Reverse TranscriptaseInfectious Diseasesmedicine.anatomical_structureImmunologyLentivirusPeptidesCD8The Journal of infectious diseases
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