Search results for "script"

showing 10 items of 5143 documents

Tissue bioaccumulation patterns, xenobiotic biotransformation and steroid hormone levels in Atlantic salmon (Salmo salar) fed a diet containing perfl…

2011

In the present study, groups of juvenile Atlantic salmon (Salmo salar) were fed gelatine capsules containing fish-food spiked with PFOA or PFOS (0.2 mg kg(-1) fish) and solvent (methanol). The capsules were given at days 0, 3 and 6. Blood, liver and whole kidney samples were collected prior to exposure (no solvent control), and at days 2, 5, 8 and 14 after exposure (Note: that day 14 after exposure is equal to 7d recovery period). We report on the differences in the tissue bioaccumulation patterns of PFOS and PFOA, in addition to tissue and compound differences in modulation pattern of biotransformation enzyme genes. We observed that the level of PFOS and PFOA increased in the blood, liver …

medicine.medical_specialtyReceptors SteroidEnvironmental EngineeringHydrocortisoneTranscription GeneticCYP3AEstroneHealth Toxicology and MutagenesisSalmo salarEstroneBiologyKidneyXenobioticschemistry.chemical_compoundInternal medicineMethyltestosteronemedicineCytochrome P-450 CYP1A1Environmental ChemistryAnimalsCytochrome P-450 CYP3ATestosteroneMethyltestosteroneBiotransformationGlutathione TransferaseFluorocarbonsPublic Health Environmental and Occupational HealthPregnane X ReceptorKidney metabolismGeneral MedicineGeneral ChemistryPollutionPerfluorooctaneEndocrinologychemistryAlkanesulfonic AcidsLiverBioaccumulationToxicityCaprylatesXenobioticmedicine.drugChemosphere
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Repression of Cyclic Adenosine Monophosphate Upregulation Disarms and Expands Human Regulatory T Cells

2011

Abstract The main molecular mechanism of human regulatory T cell (Treg)-mediated suppression has not been elucidated. We show in this study that cAMP represents a key regulator of human Treg function. Repression of cAMP production by inhibition of adenylate cyclase activity or augmentation of cAMP degradation through ectopic expression of a cAMP-degrading phosphodiesterase greatly reduces the suppressive activity of human Treg in vitro and in a humanized mouse model in vivo. Notably, cAMP repression additionally abrogates the anergic state of human Treg, accompanied by nuclear translocation of NFATc1 and induction of its short isoform NFATc1/αA. Treg expanded under cAMP repression, however,…

medicine.medical_specialtyRegulatory T cellImmunologychemical and pharmacologic phenomenaBiologyT-Lymphocytes RegulatoryMicechemistry.chemical_compoundInternal medicineCyclic AMPmedicineAnimalsHumansImmunology and AllergyCyclic adenosine monophosphatePsychological repressionCell ProliferationClonal AnergyNFATC Transcription FactorsClonal anergyPhosphodiesterasehemic and immune systemsUp-RegulationCell biologyEndocrinologymedicine.anatomical_structurechemistryHumanized mousecAMP-dependent pathwayCyclase activityThe Journal of Immunology
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Exercise and ankle sprain injuries: a comprehensive review.

2014

Ankle sprains are common in team sports and sports played on courts, and often result in structural and functional alterations that lead to a greater reinjury risk. Specific exercises are often used to promote neuromuscular improvements in the prevention and rehabilitation of ankle injuries. This literature review summarizes the neuromuscular characteristics of common ankle sprains and the effectiveness of exercise as an intervention for improving neuromuscular function and preventing reinjury. Our review found that appropriate exercise prescription can increase static and dynamic balance and decrease injury recurrence. In particular, the addition of dynamic activities in the exercise progr…

medicine.medical_specialtyRehabilitationbusiness.industrymedicine.medical_treatmentPhysical Therapy Sports Therapy and RehabilitationNeuromuscular trainingExercise programmedicine.anatomical_structurePhysical medicine and rehabilitationIntervention (counseling)Injury preventionPhysical therapySprains and StrainsMedicineHumansOrthopedics and Sports MedicineAnkle InjuriesAnkleExercise prescriptionbusinessAnkle sprainhuman activitiesExerciseAnkle JointThe Physician and sportsmedicine
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Is Structured Exercise Performed with Supplemental Oxygen a Promising Method of Personalized Medicine in the Therapy of Chronic Diseases?

2020

Aim: This systematic review aimed to explore the literature to identify in which types of chronic diseases exercise with supplemental oxygen has previously been utilized and whether this type of personalized therapy leads to superior effects in physical fitness and well-being. Methods: Databases (PubMed/MEDLINE, CINHAL, EMBASE, Web of knowledge and Cochrane Library) were searched in accordance with PRISMA. Eligibility criteria included adult patients diagnosed with any type of chronic diseases engaging in supervised exercise training with supplemental oxygen compared to normoxia. A random-effects model was used to pool effect sizes by standardized mean differences (SMD). Results: Out of the…

medicine.medical_specialtySupplemental oxygenmedicine.medical_treatmentPhysical fitnessMEDLINEMedicine (miscellaneous)lcsh:Medicineoxygen therapyCochrane LibraryArticleindividualized exercise prescriptionCoronary artery disease03 medical and health sciences0302 clinical medicineOxygen therapyMedicine030212 general & internal medicineFiO2krooniset tauditexercise medicinesystemaattiset kirjallisuuskatsauksetindividualized exercise prescription FiO2individualized exercise prescription FiO<sub>2</sub>happihoitoCOPDkuntoliikuntabusiness.industryclinical exercise sciencelcsh:Rclinical exercise science; exercise medicine; hyperoxia; individualized exercise prescription FiO2; oxygen therapymedicine.disease030228 respiratory systemPhysical therapyhyperoxiaPersonalized medicinebusinessliikuntahoitoJournal of Personalized Medicine
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Metabolic adaptation and neuroprotection differ in the retina and choroid in a piglet model of acute postnatal hypoxia.

2013

Hypoxic-ischemic insults to the neonatal brain may cause neurodevelopmental disorders. Vulnerability of different areas of the neural tissue to hypoxic-ischemic stress might be explained by either heterogeneous sensitivity to oxygen or neuroprotective capability. Our understanding of regional heterogeneity is still incomplete in terms of metabolic reconfiguration and/or activation of neuroprotective mechanisms.We studied, by western blotting, reverse-transcriptase PCR, and tandem mass spectrometry, the response of retina and choroid at protein, gene, and metabolic levels during hypoxia in a piglet model of acute postnatal hypoxia.We evidenced a metabolic shift towards glycolysis in choroid …

medicine.medical_specialtySwineanimal diseasesBlotting WesternMetabolic adaptationNeuroprotectionRetinafluids and secretionsStress PhysiologicalTandem Mass SpectrometryInternal medicinemedicineAnimalsHypoxiaRetinaintegumentary systembusiness.industryChoroidReverse Transcriptase Polymerase Chain ReactionHypoxia (medical)Hypoxia-Inducible Factor 1 alpha Subuniteye diseasesBlotmedicine.anatomical_structureEndocrinologyAnimals NewbornAnesthesiaPediatrics Perinatology and Child Healthsense organsChoroidmedicine.symptombusinessEnergy MetabolismGlycolysisSignal TransductionPediatric research
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Retarded thymic involution and massive germinal center formation in NF-ATp-deficient mice.

1998

NF-ATp and NF-ATc are the most prominent nuclear NF-AT transcription factors in peripheral T lymphocytes. After T cell activation both factors bind to and control the promoters and enhancers of numerous lymphokine and receptor ligand genes. In order to define a specific role for NF-ATp in vivo we have inactivated the NF-ATp gene by gene targeting in mice. We show that NF-ATp deficiency leads to the accumulation of peripheral T cells with a “preactivated” phenotype, enhanced immune responses of T cells after secondary stimulation in vitro and severe defects in the proper termination of antigen responses, as shown by a reduced deletion of superantigen-reactive CD4+ T cells. These alterations …

medicine.medical_specialtyT cellT-LymphocytesImmunologyApoptosisThymus GlandBiologyPolymerase Chain ReactionMiceImmune systemAntigenInternal medicinemedicineImmunology and AllergyCytotoxic T cellAnimalsfas ReceptorDNA PrimersMice KnockoutThymic involutionSuperantigensBase SequenceNFATC Transcription FactorsLymphokineGerminal centerNuclear ProteinsGerminal CenterMolecular biologyDNA-Binding ProteinsEndocrinologymedicine.anatomical_structureLymphatic systemPhenotypeTranscription FactorsEuropean journal of immunology
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Aryl hydrocarbon receptor-dependent cell cycle arrest in isolated mouse oval cells

2013

The aryl hydrocarbon receptor (AhR) is a ligand-activated transcription factor, which mediates toxic responses to environmental pollutants, such as 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) and related compounds. Besides its well known role in induction of xenobiotic metabolizing enzymes, for instance CYP1A1, the AhR is also involved in tumor promotion in rodents although the underlying mechanisms are still poorly understood. Additionally, the AhR is known to regulate cellular proliferation, which might result in either inhibition or stimulation of proliferation depending on the cell-type studied. Potential targets in hepatocarcinogenesis are liver oval (stem/progenitor) cells. In the pres…

medicine.medical_specialtyTCDDPolychlorinated DibenzodioxinsCell cycle checkpointBlotting WesternCyclin AMice TransgenicCyclin ATransfectionToxicologyRetinoblastoma ProteinCell LineMiceCyclin D1Proliferating Cell Nuclear AntigenInternal medicinemedicineAnimalsCyclin D1RNA Small InterferingTranscription factorCell Proliferationbiologyaryl hydrocarbon receptorRetinoblastoma proteinmouse oval cellsCell Cycle CheckpointsGeneral MedicineCell cycleAryl hydrocarbon receptorCell biologyEndocrinologyLiverReceptors Aryl Hydrocarbonbiology.proteinEnvironmental PollutantsTumor promotioncell cycle
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Corticotropin-Releasing Hormone-Mediated Induction of Intracellular Signaling Pathways and Brain-Derived Neurotrophic Factor Expression Is Inhibited …

2005

CRH receptor (CRHR) 1 and the cannabinoid receptor 1 (CB1) are both G protein-coupled receptors. Activation of CRHR1 leadstoincreasesincAMPproductionandphosphorylationof the transcription factor cAMP response element-binding protein (CREB). In contrast, CB1 is negatively coupled to the cAMP signaling cascade. In this study, we analyzed a putative interaction between these two systems focusing on the regulation of the expression of brain-derived neurotrophic factor (BDNF), a CREB-regulated gene. In situ hybridization revealed coexpression of CRHR1 and CB1 receptors in the granular layer of the cerebellum. Therefore, we analyzed the effects of CRH and the CB1 agonist WIN-55,212-2 on BDNF expr…

medicine.medical_specialtyTime FactorsCorticotropin-Releasing HormoneMorpholinesmedicine.medical_treatmentImmunoblottingEnzyme-Linked Immunosorbent AssayTropomyosin receptor kinase BNaphthalenesCREBModels BiologicalRats Sprague-DawleyMiceEndocrinologyNeurotrophic factorsCerebellumInternal medicineCannabinoid Receptor ModulatorsCyclic AMPmedicineAnimalsRNA MessengerCyclic AMP Response Element-Binding ProteinReceptorEgtazic AcidCells CulturedIn Situ HybridizationNeuronsBrain-derived neurotrophic factorSulfonamidesbiologyReverse Transcriptase Polymerase Chain ReactionBrain-Derived Neurotrophic FactorCalcium Channel BlockersIsoquinolinesEndocannabinoid systemBenzoxazinesRatsMice Inbred C57BLPyrimidinesEndocrinologynervous systembiology.proteinCalciumCannabinoidSignal transductionEndocannabinoidsProtein BindingSignal TransductionEndocrinology
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US Food and Drug Administration's Risk Evaluation and Mitigation Strategy for Extended-Release and Long-Acting Opioids Pros and Cons, and a European …

2012

Prescriptions for opioid analgesics to manage moderate-to-severe chronic non-cancer pain have increased markedly over the last decade. An unintentional consequence of greater prescription opioid utilization has been the parallel increase in misuse, abuse and overdose, which are serious risks associated with all opioid analgesics. In response to disturbing rises in prescription opioid abuse, the US Food and Drug Administration (FDA) has proposed the implementation of aggressive Risk Evaluation and Mitigation Strategies (REMS). While REMS could dramatically change the development, release, marketing and prescription of extended-release opioids, questions remain on how these programmes may inf…

medicine.medical_specialtyTime FactorsSettore MED/41 - AnestesiologiaLong-Acting Opioids Food and drug administration AnalgesiaRisk AssessmentFood and drug administrationPatient safetyPharmacotherapymedicineHumansPharmacology (medical)Medical prescriptionIntensive care medicineUnited States Food and Drug Administrationbusiness.industryPerspective (graphical)Chronic painLong-Acting Opioids Food and drug administration AnalgesiaOpioid-Related Disordersmedicine.diseaseUnited StatesRisk evaluationAnalgesics OpioidEuropeDelayed-Action PreparationsMedical emergencyChronic PainRisk assessmentbusiness
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Acute exercise induced changes in rat skeletal muscle mRNAs and proteins regulating type IV collagen content

2001

This experiment tested the hypothesis that running-induced damage to rat skeletal muscle causes changes in synthesis and degradation of basement membrane type IV collagen and to proteins regulating its degradation. Samples from soleus muscle and red and white parts of quadriceps femoris muscle (MQF) were collected 6 h or 1, 2, 4, or 7 days after downhill running. Increased muscle β-glucuronidase activity indicated greater muscle damage in the red part of MQF than in the white part of MQF or soleus. In the red part of MQF, type IV collagen expression was upregulated at the pretranslational level and the protein concentration decreased, whereas matrix metalloproteinase-2 (MMP-2), a protein th…

medicine.medical_specialtyTime FactorsTranscription GeneticPhysiologyPhysical ExertionMatrix metalloproteinaseBiologyRunningType IV collagenPhysiology (medical)Internal medicineGene expressionmedicineAnimalsRNA MessengerRats WistarMuscle SkeletalGlucuronidaseSoleus muscleBasement membranechemistry.chemical_classificationTissue Inhibitor of Metalloproteinase-2Tissue Inhibitor of Metalloproteinase-1Skeletal muscleTissue inhibitor of metalloproteinaseRatsmedicine.anatomical_structureEndocrinologyGene Expression RegulationMatrix Metalloproteinase 9chemistryProtein BiosynthesisMuscle Fibers Fast-TwitchMatrix Metalloproteinase 2FemaleCollagenGlycoproteinAmerican Journal of Physiology-Regulatory, Integrative and Comparative Physiology
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