Search results for "script"

showing 10 items of 5143 documents

Cytokine Gene Transcription By NF-kappaB Family Members in Patients with Inflammatory Bowel Disease

1998

We examined the expression of the transcription factor NF-kappa B, a nuclear trans-acting factor known to play a key role in cytokine gene regulation, in patients with inflammatory bowel disease (IBD). It was found that LP macrophages in Crohn's disease (CD) and ulcerative colitis (UC) display high levels of NF-kappa B DNA-binding activity accompanied by an increased production of interleukin (IL)-1, IL-6, and tumor necrosis factor (TNF) alpha. Western blot studies showed an increased expression of the p50 and c-rel subunits of NF-kappa B; however, the most striking finding was an increased expression level of NF-kappa B p65 in patients with CD and UC. Selective downregulation of p65 in IBD…

AdultMaleShort Bowel SyndromeAdolescentTranscription GeneticColonBiologyInflammatory bowel diseaseGeneral Biochemistry Genetics and Molecular BiologyProinflammatory cytokinechemistry.chemical_compoundCrohn DiseaseHistory and Philosophy of ScienceDownregulation and upregulationmedicineHumansIntestinal MucosaTranscription factorCells CulturedRegulation of gene expressionMacrophagesGeneral NeuroscienceNF-kappa BInterleukinNF-κBMiddle Agedmedicine.diseaseGene Expression RegulationchemistryImmunologyCancer researchCytokinesColitis UlcerativeFemaleTumor necrosis factor alphaAnnals of the New York Academy of Sciences
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Cordycepin is an immunoregulatory active ingredient of Cordyceps sinensis.

2008

We have reported that cordycepin, an adenosine derivative from the fungus Cordyceps, increased interleukin (IL)-10 expression, decreased IL-2 expression and suppressed T lymphocyte activity. In the present study, we further characterized the regulatory effects of cordycepin on human immune cells. Moreover, a traditional Chinese drug, Cordyceps sinensis (CS) that contains cordycepin, was also investigated. Cytometric Bead Array (CBA) was used to determine the concentrations of IL-1beta, IL-2, IL-4, IL-5, IL-6, IL-8, IL-10, IL-12, TNF-alpha and IFN-gamma in culture of peripheral blood mononuclear cells (PBMCs). The results showed that both cordycepin and CS up-regulated IL-10, IL-1beta, IL-6,…

AdultMaleT-LymphocytesGene ExpressionBiologyPeripheral blood mononuclear cellchemistry.chemical_compoundImmune systemmedicineHumansTranscription factorCells CulturedCell ProliferationCordycepsCordycepinDeoxyadenosinesPlant ExtractsInterleukinGeneral Medicinebiology.organism_classificationAdenosineComplementary and alternative medicinechemistryBiochemistryCell cultureCordycepsLeukocytes MononuclearCytokinesFemalemedicine.drugProtein BindingTranscription FactorsThe American journal of Chinese medicine
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Naturally Occurring Telomerase-Specific CD4 T-Cell Immunity in Melanoma.

2020

CD4 T cells play a key role in anticancer immunity. In this study, we investigate the clinical relevance of circulating CD4 T helper type 1 (Th1) response against telomerase (anti-TERT Th1 response) in patients with melanoma. The spontaneous anti-TERT Th1 response was detected in 54.5% (85/156) of patients with melanoma before treatment. The prevalence of this systemic response was inversely related to Breslow thickness >1 mm and American Joint Committee on Cancer stage ≥II (P = 0.001 and 0.032, respectively). In contrast to patients treated with targeted therapies, the anti-TERT Th1 immunity was associated with an objective response after immune checkpoint inhibitors treatment. Hence, 86% …

AdultMaleTelomeraseSkin Neoplasmsmedicine.medical_treatmentDermatologyBiochemistryBreslow ThicknessImmunitymedicineHumansTelomerase reverse transcriptaseProgression-free survivalProspective StudiesMolecular BiologyImmune Checkpoint InhibitorsMelanomaTelomeraseAgedNeoplasm Stagingbusiness.industryMelanomaCell BiologyImmunotherapyMiddle AgedTh1 Cellsmedicine.diseaseProgression-Free SurvivalDrug Resistance NeoplasmCancer researchBiomarker (medicine)FemalebusinessFollow-Up StudiesThe Journal of investigative dermatology
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Deficiency of the autoimmune regulator AIRE in thymomas is insufficient to elicit autoimmune polyendocrinopathy syndrome type 1 (APS‐1)

2007

Thymomas are thymic epithelial neoplasms, associated with a variety of autoimmune disorders (especially myasthenia gravis), that apparently result from aberrant intra-tumourous thymopoiesis and export of inefficiently tolerized T-cells to the periphery. The autoimmune regulator (AIRE) drives the expression of self-antigens in the thymic medulla and plays an essential role in ‘central’ tolerance in both humans and mice. However, while inactivating AIRE mutations result in the ‘autoimmune polyendocrinopathy syndrome type 1’ (APS-1), its major features are not well reproduced in AIRE-knock-out mice. Therefore, alternative human disease scenarios with concomitant AIRE deficiency may be valuable…

AdultMaleThymomaAdolescentThymomaAntibodies NeoplasmThymus Glandmedicine.disease_causeAutoantigensAutoimmune DiseasesPathology and Forensic MedicineAutoimmunity03 medical and health sciences0302 clinical medicineAntigens NeoplasmInterferonMyasthenia GravismedicineHumansPolyendocrinopathies AutoimmuneAgedAutoantibodies030304 developmental biologyAged 80 and over0303 health sciencesbiologybusiness.industryAutoantibodyThymus NeoplasmsMiddle AgedAutoimmune regulatormedicine.diseaseImmunohistochemistryMyasthenia gravisNeoplasm Proteins3. Good healthThymic Tissue030220 oncology & carcinogenesisInterferon Type IImmunologybiology.proteinCytokinesFemaleAntibodybusinessTranscription Factorsmedicine.drugThe Journal of Pathology
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Studies on the subcellular pathophysiology of sublethal chronic cell injury.

1974

Summary This paper summarizes some of the important subcellular events occurring after chronic sublethal cell injury. Chronic cell injury is defined as the result of injurious stimuli which permit cell survival though in altered steady states for protracted periods of time. The importance of ultrastructural and biochemical studies of these phenomena is emphasized. Among the phenomena discussed are alterations in lysosomes, cellular hypertrophy, fatty metamorphosis, alterations in microfilaments and microtubules, alterations in mechanisms of transcription and replication, disturbances in the cell surface and transport across the cell membrane, and alterations in intracellular transport.

AdultMaleTime FactorsTranscription GeneticSurface PropertiesCellsCellGuinea PigsBronchiBiologyMicrofilamentMicrotubulesPathology and Forensic MedicineMuscle hypertrophyCell Physiological PhenomenaCell membraneMiceMicrotubuleTranscription (biology)medicineAnimalsHumansCerebral CortexMacrophagesMusclesCell MembraneBiological TransportGeneral MedicineHypertrophyMiddle AgedPathophysiologyCell biologyMicroscopy Electronmedicine.anatomical_structureLiverUltrastructureRabbitsLysosomesCell DivisionBeitrage zur Pathologie
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Influence of sex and genetic variability on expression of X-linked genes in human monocytes

2011

Abstract In humans, the fraction of X-linked genes with higher expression in females has been estimated to be 5% from microarray studies, a proportion lower than the 25% of genes thought to escape X inactivation. We analyzed 715 X-linked transcripts in circulating monocytes from 1,467 subjects and found an excess of female-biased transcripts on the X compared to autosomes (9.4% vs 5.5%, p  −5 ). Among the genes not previously known to escape inactivation, the most significant one was EFHC2 whose 20% of variability was explained by sex. We also investigated cis expression quantitative trait loci (eQTLs) by analyzing 15,703 X-linked SNPs. The frequency and magnitude of X-linked cis eQTLs were…

AdultMaleTranscription GeneticMicroarrayQuantitative Trait LociSingle-nucleotide polymorphismBiologyPolymorphism Single NucleotideMonocytesX-inactivationSex FactorsGenes X-LinkedX Chromosome InactivationGene expressionGeneticsHumansGenetic variabilityGeneAgedGeneticsChromosomes Human XAutosomeCalcium-Binding ProteinsGenderGenetic VariationMiddle AgedExpression Quantitative Trait LocusExpression quantitative trait lociFemaleGene expressionGenome-Wide Association StudyGenomics
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Different Transcriptional Activity and In Vitro TNF-α Production in Psoriasis Patients Carrying the TNF-α 238A Promoter Polymorphism

2000

Genes encoded on chromosome 6 within the major histocompatibility complex region are thought to play an important role in the pathogenesis of psoriasis. A potential candidate gene is tumor necrosis factor alpha. The tumor necrosis factor alpha promoter contains several polymorphisms including two G--A transitions at position -308 and -238, which are the most common in Caucasian populations. The TNF238.2 (-238A) allele has been strongly associated with psoriasis. We have investigated the effect of the -238 and -308 variants on transcription of the tumor necrosis factor alpha gene in luciferase reporter gene assays. In addition, peripheral blood mononuclear cells of 47 patients with psoriasis…

AdultMaleTranscription Geneticmedicine.medical_treatmentT cellDermatologyBiologyBiochemistryPeripheral blood mononuclear cellAntigenPsoriasisTNFαmedicineSuperantigenHumansPsoriasisPromoter Regions GeneticMolecular Biologytranscriptional activityAgedAged 80 and overPBMGPolymorphism GeneticTumor Necrosis Factor-alphaPromoterCell BiologyMiddle Agedmedicine.diseaseMolecular biologypromoter polymorphismCytokinemedicine.anatomical_structureImmunologyLeukocytes MononuclearFemaleTumor necrosis factor alphaJournal of Investigative Dermatology
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Neural Correlates of Visual versus Abstract Letter Processing in Roman and Arabic Scripts

2013

In alphabetic orthographies, letter identification is a critical process during the recognition of visually presented words. In the present experiment, we examined whether and when visual form influences letter processing in two very distinct alphabets (Roman and Arabic). Disentangling visual versus abstract letter representations was possible because letters in the Roman alphabet may look visually similar/dissimilar in lowercase and uppercase forms (e.g., c-C vs. r-R) and letters in the Arabic alphabet may look visually similar/dissimilar, depending on their position within a word (e.g., [Formula: see text] - [Formula: see text] vs. [Formula: see text] - [Formula: see text]). We employed a…

AdultMaleVisual perceptionLetter processingArabicCognitive Neurosciencemedia_common.quotation_subjectDecision Makingcomputer.software_genreArticle050105 experimental psychologyYoung Adult03 medical and health sciences0302 clinical medicineReading (process)Reaction TimeHumans0501 psychology and cognitive sciencesEvoked PotentialsLanguagemedia_commonAnalysis of VarianceNeural correlates of consciousnessbusiness.industry05 social sciencesElectroencephalographyRecognition PsychologyEvent-Related Potentials P300language.human_languageLinguisticsPattern Recognition VisualReadingScripting languageData Interpretation StatisticalVisual PerceptionlanguageFemaleArtificial intelligencePsychologybusinesscomputerPriming (psychology)Psychomotor Performance030217 neurology & neurosurgeryWord (group theory)Natural language processingJournal of Cognitive Neuroscience
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COMT genotype predicts BOLD signal and noise characteristics in prefrontal circuits.

2006

Abstract Objective: Prefrontal dopamine (DA) is catabolized by the COMT (catechol- O -methyltransferase) enzyme. Literature suggests that the Val/Met single nucleotide polymorphism (SNP) in the COMT gene predicts executive cognition in humans with Val carriers showing poorer performance due to less available synaptic DA. Recent fMRI studies are thought to agree with these studies having demonstrated prefrontal hyperactivation during n -back and attention-requiring tasks. This was interpreted as “less efficient” processing due to impaired signal-to-noise ratio (SNR) of neuronal activity. However, electrophysiological studies of neuronal SNR in primates and humans imply that prefrontal cortex…

AdultMaleVisual perceptiongenetic structuresGenotypeCognitive NeurosciencePrefrontal CortexCatechol O-Methyltransferasebehavioral disciplines and activitiesDopaminemedicineImage Processing Computer-AssistedPremovement neuronal activityHumansPrefrontal cortexOddball paradigmReverse Transcriptase Polymerase Chain ReactionSMA*Magnetic Resonance ImagingDorsolateral prefrontal cortexOxygenElectrophysiologymedicine.anatomical_structurenervous systemNeurologyRegression AnalysisFemalePsychologyNeurosciencePhotic StimulationPsychomotor Performancemedicine.drugNeuroImage
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Genotypic and phenotypic spectrum in tricho-rhino-phalangeal syndrome types I and III

2000

Tricho-rhino-phalangeal syndrome (TRPS) is characterized by craniofacial and skeletal abnormalities. Three subtypes have been described: TRPS I, caused by mutations in the TRPS1 gene on chromosome 8; TRPS II, a microdeletion syndrome affecting the TRPS1 and EXT1 genes; and TRPS III, a form with severe brachydactyly, due to short metacarpals, and severe short stature, but without exostoses. To investigate whether TRPS III is caused by TRPS1 mutations and to establish a genotype-phenotype correlation in TRPS, we performed extensive mutation analysis and evaluated the height and degree of brachydactyly in patients with TRPS I or TRPS III. We found 35 different mutations in 44 of 51 unrelated p…

AdultMaleanimal structuresAdolescentGenotypeDNA Mutational AnalysisMolecular Sequence DataLimb Deformities CongenitalBiologyOsteochondrodysplasiasPolymorphism Single NucleotideShort statureLanger–Giedion syndromeGeneticsmedicineHumansMissense mutationTricho–rhino–phalangeal syndromeGenetics(clinical)Amino Acid SequenceChildGenetics (clinical)GeneticsAnthropometryBase SequenceBrachydactylyInfantZinc FingersExonsSyndromeArticlesMiddle AgedMicrodeletion syndromemedicine.diseasePenetranceBody HeightPedigreeDNA-Binding ProteinsRadiographyPhenotypeChild PreschoolMutationTrichorhinophalangeal Syndrome Type IErythroid-Specific DNA-Binding FactorsFemalemedicine.symptomChromosomes Human Pair 8Transcription Factors
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