Search results for "t-lymphocytes"

showing 10 items of 1380 documents

Effects of polyphenols and lipids from Pennisetum glaucum grains on T-cell activation: modulation of Ca2+ and ERK1/ERK2 signaling

2015

International audience; BACKGROUND: Pearl millet (PM), i.e., Pennisetum glaucum, is widely grown in Africa and known for its anti-oxidant and anti-hyperlipidemic properties.METHODS: The P. glaucum grains were obtained from the region of Ouled Aïssa (South of Algeria). We assessed the effects of phenolic compounds and lipids, extracted from seeds of P. glaucum, on rat lymphocyte proliferation, activated by phorbol 12-myristate 13-acetate and ionomycin. In order to explore signaling pathway, triggered by these compounds, we assessed interleukin-2 (IL-2) mRNA expression and extracellular signal-regulated kinase-1/2 (ERK1/ERK2) phosphorylation. Finally, we determined increases in free intracell…

PennisetumT-LymphocytesLymphocyte proliferationLymphocyte ActivationAntioxidantsPearl milletchemistry.chemical_compoundExtracellularAnimalsGallic acidExtracellular Signal-Regulated MAP KinasesCell ProliferationHypolipidemic AgentsMitogen-Activated Protein Kinase 1Mitogen-Activated Protein Kinase 3[SDV.MHEP] Life Sciences [q-bio]/Human health and pathologybiologyPlant ExtractsT-cellsPolyphenolsGeneral Medicinebiology.organism_classificationLipidsRatschemistryBiochemistryComplementary and alternative medicinePolyphenolIonomycinSeedsPhorbolPhosphorylationCalciumEdible GrainPennisetum[SDV.MHEP]Life Sciences [q-bio]/Human health and pathologySignal TransductionResearch Article
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CD8+CD45RA+CD27-CD28-T-cell subset in PBL of cervical cancer patients representing CD8+T-cells being able to recognize cervical cancer associated ant…

2003

Objective In response to antigenic stimulation, naive MHC-class I restricted and antigen-specific CD8+CD45RA+CD28+T-cells undergo clonal expansion and differentiate into CD8+CD45RO+ memory T-cells. Upon re- encounter with the nominal antigen, CD45RO+ T-cells are able to convert to CD8+CD45RA+CD28-T-cells displaying potent immune effector functions, including TNF-alpha production. This T-cell subpopulation constitutes a minor population in healthy individuals. In the present study we are currently evaluating whether this particular T-cell subset in PBL represents CD8+T-cells which may be able to recognize cervical cancer associated antigens provided by HPV 16 E7. Material and methods Flow-cy…

PopulationUterine Cervical Neoplasmschemical and pharmacologic phenomenaCD8-Positive T-LymphocytesBiologyEpitopeImmune systemCD28 AntigensAntigenAntigens CDT-Lymphocyte SubsetsmedicineHumansCytotoxic T cellAmino Acid SequenceeducationAntigens ViralPapillomaviridaeNeoplasm Stagingeducation.field_of_studyHistocompatibility TestingObstetrics and GynecologyCD28Cancerhemic and immune systemsmedicine.diseasePeptide FragmentsTumor Necrosis Factor Receptor Superfamily Member 7Lymphatic MetastasisImmunologyCytokinesLeukocyte Common AntigensFemaleCD8Zentralblatt für Gynäkologie
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Novel path to apoptosis: small transmembrane pores created by staphylococcal alpha-toxin in T lymphocytes evoke internucleosomal DNA degradation.

1994

Peripheral-blood human T lymphocytes were treated with Staphylococcus aureus alpha-toxin. Membrane permeabilization was assessed by measuring efflux of K+ and Rb+ and influx of Na+, Ca2+, and propidium iodide. Cellular ATP and [3H]thymidine incorporation following lectin stimulation were measured as parameters for cell viability. Internucleosomal cleavage characteristic of programmed cell death was assessed by agarose gel electrophoresis and by quantifying low-molecular-weight, [3H]thymidine-labeled DNA fragments. Nanomolar concentrations of alpha-toxin evoked protracted, irreversible ATP depletion in both activated and resting T lymphocytes. Toxin-damaged cells also lost their ability to i…

Programmed cell deathCell Membrane PermeabilityStaphylococcusT-LymphocytesImmunologyBacterial ToxinsApoptosisBiologyMicrobiologychemistry.chemical_compoundHemolysin ProteinsAdenosine TriphosphateHumansPropidium iodideViability assaySodiumT lymphocyteDNANucleosomesInfectious DiseaseschemistryBiochemistryApoptosisAgarose gel electrophoresisBiophysicsPotassiumParasitologyCalciumThymidineAdenosine triphosphateResearch Article
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Cytocidal effects of Escherichia coli hemolysin on human T lymphocytes.

1993

Escherichia coli hemolysin is the prototype of a large family of pore-forming toxins produced by gram-negative organisms. Besides its known cytotoxic activities against granulocytes, monocytes, endothelial cells, and renal epithelial cells, we now demonstrate that the toxin potently kills human T lymphocytes. Evidence based on different and independent approaches indicates that lymphocidal activity is due to formation of transmembrane pores. Additionally, cells prestimulated with phytohemagglutinin respond to low doses of E. coli hemolysin with DNA fragmentation similar to that observed in cells undergoing programmed cell death. Kinetic considerations lead us to conclude that DNA degradatio…

Programmed cell deathCell Membrane PermeabilityTime FactorsDNA damageT-LymphocytesImmunologyBiologyIn Vitro Techniquesmedicine.disease_causeHemolysin ProteinsLymphocyte ActivationMicrobiologyMicrobiologyHemolysin ProteinsAdenosine TriphosphatemedicineEscherichia coliCytotoxic T cellHumansEscherichia coliCell DeathDose-Response Relationship DrugHemolysinT lymphocyteDNAInfectious DiseasesDNA fragmentationParasitologyResearch ArticleDNA Damage
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Natural and induced apoptosis during lymphocyte development in the axolotl

1999

Lymphocytes apoptosis was characterized in a urodele amphibian, the axolotl, by morphology using electron microscopy and by flow cytometry after propidium iodide staining, as well as by biochemical criteria with the detection of DNA ladders after glucocorticoid treatment. The morphological and biochemical features observed in treated axolotls are in accordance with the criteria of apoptosis found in different models of mammalian lymphocyte programmed cell death. The onset of natural apoptosis was then detected by DNA fragmentation in thymus and in spleen during lymphocyte development and ontogenesis. A typical DNA ladder characteristic of apoptosis is detectable in the thymus as early as 5 …

Programmed cell deathHydrocortisoneT-LymphocytesLymphocyteImmunologyApoptosisBiologyAmbystomaFlow cytometryEnterotoxinschemistry.chemical_compoundAxolotlmedicineSuperantigenAnimalsLymphocytesPropidium iodideSuperantigensmedicine.diagnostic_testCell Differentiationbiology.organism_classificationMolecular biologymedicine.anatomical_structurechemistryApoptosisLarvaDNA fragmentationDevelopmental BiologyDevelopmental & Comparative Immunology
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Involvement of NO in contact hypersensitivity.

1998

The NO synthases (NOS) generate NO from L-arginine. High concentrations of NO have been shown to be responsible for tissue injury and cell death, while low concentrations of NO induce vasodilatation and other signaling effects. We have investigated the involvement of NO in contact hypersensitivity (CHS) reactions. CHS induced by treatment of BALB/c mice with the contact allergen 2,4-dinitrofluorobenzene (DNFB) was significantly reduced by the NOS inhibitor N-methyl-L-arginine (L-NMA), but not by the stereoisomer D-NMA, as shown by reduced ear swelling responses and evaluation of ear tissue sections. The CHS response was also reduced by aminoguanidine, which is known to preferentially inhibi…

Programmed cell deathLangerhans cellArginineInjections IntradermalT-LymphocytesImmunologyNitric Oxide Synthase Type IIBiologyArginineDermatitis ContactNitric OxideGuanidineschemistry.chemical_compoundMicemedicineImmunology and AllergyAnimalsRNA MessengerEnzyme InhibitorsSkinMice Inbred BALB Cintegumentary systemEpidermis (botany)Histocompatibility Antigens Class IIGeneral MedicineAllergensMolecular biologyPimagedineNitric oxide synthasemedicine.anatomical_structurechemistryLangerhans Cellsbiology.proteinDinitrofluorobenzeneSignal transductionNitric Oxide SynthaseKeratinocyteHaptensInternational immunology
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Apoptosis of T cells and the control of inflammatory bowel disease: therapeutic implications.

2007

Inflammatory bowel diseases (IBDs) such as Crohn’s disease and ulcerative colitis are the result of an imbalanced mucosal T cell response. Despite the identification of a genetic susceptibility region in the NOD2/CARD15 (nucleotide-binding oligomerisation domain 2/caspase recruitment domain 15) gene, the aetiology is still unclear. Thus, the hunt for disease-initiating factors such as defects in the mucosal barrier or pathogenic microorganisms is ongoing. By contrast, the immunopathogenesis in IBDs is better understood. The identification of cytokines that are involved in T cell and monocyte signalling led to specific therapeutic concepts. Recent data have clearly shown that the most powerf…

Programmed cell deathNecrosisCell Survivalmedicine.medical_treatmentT cellT-LymphocytesApoptosisImmune systemCrohn DiseaseNOD2AzathioprinemedicineHumansIntestinal MucosaMesalamineImmunity Mucosalbusiness.industryInterleukin-6Tumor Necrosis Factor-alphaAnti-Inflammatory Agents Non-SteroidalGastroenterologyRecent Advances in Basic ScienceInflammatory Bowel DiseasesInterleukin-12Immunosuppressive drugmedicine.anatomical_structureApoptosisImmunologyTumor necrosis factor alphamedicine.symptombusinessImmunosuppressive AgentsSignal TransductionGut
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Selective targeting of activated T cells in chronic intestinal inflammation

2009

Programmed cell death (apoptosis) has been implicated in normal biological processes as well as in the pathology of human diseases.1 The characterisation of genes involved in apoptosis has been pursued intensively and led to the identification of two major classes of genes: the bcl-2 family and the caspase family. Caspases are proteases that cleave their target substrates at specific peptide sequences and during apoptosis the activation of caspases takes place in a cascade fashion, leading to nuclear engulfment and cell death. Thus, caspases represent key functional components of the apoptosis pathway in human cells. Resistance against apoptosis is a key phenomenon in various chronic inflam…

Programmed cell deathRecombinant Fusion ProteinsT-LymphocytesT cellApoptosisLymphocyte ActivationProinflammatory cytokineImmune systemmedicineAnimalsHumansIntestinal MucosaCaspasebiologyCaspase 3Intrinsic apoptosisGastroenterologyColitisCell biologymedicine.anatomical_structureApoptosisChronic DiseaseModels Animalbiology.proteinInterleukin-2Tumor necrosis factor alphaGut
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Processing without proteolytic cleavage is required for recognition of insulin by T cells.

1990

Beef insulin as well as a chymotryptic A-chain fragment [BI-A1-14(SSO3-)3] need uptake by antigen-presenting cells (APC) for efficient presentation in combination with major histocompatibility complex class II molecules to insulin-specific T cells. This could be shown by the inability of aldehyde-fixed APC to present these antigens to T cells. Furthermore, presentation of the insulin fragment as well as presentation of ovalbumin (OVA) was inhibited by treatment of APC with chloroquine, cerulenin or tunicamycin. This was not the case for a processing-independent OVA peptide. Treatment of APC during antigen pulsing with various protease inhibitors, active on all classes of proteases, did not …

ProteasesOvalbuminmedicine.medical_treatmentT-LymphocytesImmunologyAntigen presentationAntigen-Presenting CellsBiologyIn Vitro TechniquesEpitopeCell Linechemistry.chemical_compoundMiceAntigenEndopeptidasesmedicineImmunology and AllergyAnimalsInsulinProtease InhibitorsAntigen-presenting cellProteaseInsulinTunicamycinChloroquineTunicamycinEndocytosischemistryBiochemistryEuropean journal of immunology
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Priming of Leishmania-Reactive CD8+ T cells In Vivo Does Not Require LMP7-Containing Immunoproteasomes

2012

Proteasome Endopeptidase ComplexLeishmaniasis CutaneousPriming (immunology)DermatologyCD8-Positive T-LymphocytesBiochemistryInterferon-gammaMiceIn vivomedicineAnimalsCytotoxic T cellInterferon gammaProteasome endopeptidase complexLeishmania majorMolecular BiologyLeishmania majorSkinMice KnockoutbiologyChemistryCell Biologybiology.organism_classificationCoculture TechniquesCell biologyMice Inbred C57BLDisease Models AnimalLangerhans CellsCoculture Techniquemedicine.drugJournal of Investigative Dermatology
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