Search results for "taxi"

showing 10 items of 515 documents

THE BEHAVIORAL RESPONSE OF AMPHIPODS HARBORING CORYNOSOMA CONSTRICTUM (ACANTHOCEPHALA) TO VARIOUS COMPONENTS OF LIGHT

2006

Many studies have shown that photic behavior of amphipods is subject to parasitic manipulation. However, all these investigations have focused on but one property of light (i.e., intensity). This study investigated the possibility that variable wavelength sensitivity, as a potentially important component of amphipod ecology, is subject to parasitic manipulation. The photic behavior of freshwater amphipods Hyalella azteca, infected with the duck acanthocephalan Corynosoma constrictum, was tested. The phototactic responses of infected and uninfected amphipods to various wavelengths in the visible spectrum were compared, and to delineate the effects of intensity and wavelength on behavior, the…

AmphipodaBehavior AnimalLightgenetic structuresbiologyEcologyMovementHyalella aztecabiology.organism_classificationCrustaceanAcanthocephalaHost-Parasite InteractionsLight intensityDucksBehavioral responsePhototaxisAnimalsAmphipodaFemaleParasitologyPhotic zonesense organsAcanthocephalaEcology Evolution Behavior and SystematicsJournal of Parasitology
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Emerging contributions of formyl peptide receptors to neurodegenerative diseases.

2021

Abstract Inflammation is a central element of many neurodegenerative diseases. Formyl peptide receptors (FPRs) can trigger several receptor-dependent signal transduction pathways that play a key role in neuroinflammation and neurodegeneration. They are chemotactic receptors that help to regulate pro- and anti-inflammatory responses in most mammals. FPRs are primarily expressed in the immune and nervous systems where they interact with a complex pattern of pathogen-derived and host-endogenous molecules. Mounting evidence points towards a contribution of FPRs – via neuropathological ligands such as Amyloid beta, and neuroprotective ligands such as Humanin, Lipoxin A4, and Annexin A1 – to mult…

Amyloid beta-PeptidesClinical BiochemistryNeurodegenerationChemotaxisNeurodegenerative DiseasesBiologymedicine.diseaseLigandsBiochemistryNeuroprotectionReceptors Formyl PeptideNeuroinflammatory DiseasesmedicineFunctional selectivityAnimalsHumansSignal transductionMolecular BiologyCentral elementNeuroscienceNeuroinflammationHumaninBiological chemistryReferences
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Anaphylatoxin-like molecules generated during complement activation induce a dramatic enhancement of particle uptake in rainbow trout phagocytes.

2004

Here we have identified a serum fraction containing approximately 8-kDa molecules with an unexpected capacity to greatly enhance particle uptake in trout head kidney leukocytes (HKLs). This approximately 8-kDa particle-uptake enhancing fraction (PUEF-8) was purified from complement-activated serum by gel filtration chromatography. Mass spectrometric analysis and reactivity of anti-trout C3-1 and C4 antibodies, indicated the presence of C3a, C4a and C5a molecules in PUEF-8. Using a newly developed flow cytometric assay that measures the capacity of cells to ingest fluorescent beads, we showed that PUEF-8 induced a striking enhancement (344+/-50% higher than the PBS control value) in the numb…

AnaphylatoxinsTime FactorsEvolutionPhagocytosisImmunologySize-exclusion chromatographyComplementAnaphylatoxinPhagocytosisCell MovementLeukocytesAnimalsAnaphylatoxinPhagocytesPhagocytosibiologyChemotaxisC4AChemotaxisComplement System Proteinsbiology.organism_classificationFlow CytometryMolecular biologyComplement systemTroutRainbow troutOncorhynchus mykissImmunologybiology.proteinAntibodyDevelopmental Biology
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The Clinical Enzymology of the Complement System

1989

The complement (C) system is one of the most important humoral systems mediating many activities that contribute to inflammation and host defense, e.g. various anaphylatoxin activities, Chemotaxis and opsonization for phagocytosis. The C system is similar to other humoral systems, such as coagulation, fibrinolysis and the kinin system, a multifactoral system whose activation represents sequentially occurring multi-step activation cascades of the “classical” as well as the “alternative” pathway.

Antibody opsonizationClassical complement pathwayChemistrymedicineAnaphylatoxinChemotaxisInflammationKininComplement deficiencymedicine.symptommedicine.diseaseCell biologyComplement system
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Migration of Leukocytes into Filters Coated Homogeneously with Immune Complexes, Antigens, Lectins or Tripeptides

1980

Cellulose nitrate filters were incubated in solutions of albumin, a chemotactically active tripeptide (f-Met-Leu-Phe), immune complexes or lectins and afterwards washed with buffer. They showed a dose-dependent increased leukocyte migration, when tested in typical Boyden chambers in comparison to filters treated only with buffer. The tripeptide, the immune complexes and the lectins were stimulatory at very low concentrations and acted inhibitory at high concentrations. Treating filters with formaldehyde or glutardialdehyde had no clear stimulatory effect. These findings extend earlier observations obtained with casein. They show that cells move very effectively on solid substrata in the abs…

Antigen-Antibody ComplexLeukocyte migrationGuinea PigsImmunologySerum albuminAntigen-Antibody ComplexTripeptideAntigenCell MovementLectinsCaseinConcanavalin AAnimalsImmunology and AllergyPhytohemagglutininsSerum AlbuminOligopeptideChemotactic FactorsbiologyChemistryMicropore FiltersHematologyChemotaxis LeukocyteBiochemistryConcanavalin AImmunoglobulin Gbiology.proteinOligopeptidesGranulocytesImmunobiology
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Sobre conectores, expletivos y muletillas

1992

-Salvador.Pons@uv.es

Análisis oracional; Conectores; Expletivos; Muletillas; Español habladoConectores:LINGÜÍSTICA::Lingüística sincrónica::Sintaxis análisis sintáctico [UNESCO]ExpletivosEspañol habladoMuletillasUNESCO::LINGÜÍSTICA::Lingüística sincrónica::Sintaxis análisis sintácticoAnálisis oracional
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La expansión del posesivo pospuesto a la luz de documentación navarra (y aragonesa) del siglo XIII*

2016

Resumen: En los primitivos documentos romances el posesivo solo aparece antepuesto al sustantivo (“(el) nuestro padre”). La posposicion (“padre nuestro”) se empieza a registrar en el siglo XIII en aragones y en dialecto navarro. Considerando principalmente la documentacion de esta segunda variedad, el presente trabajo tiene como objetivo determinar el modo en que el posesivo adquirio la capacidad de aparecer pospuesto. Este cambio se vio impulsado por factores de naturaleza estructural. Uno de ellos es la existencia de la construccion posesiva con de y pronombre personal (“por racon de mi ”); otro, la concurrencia del posesivo en coordinacion, bien con el sintagma preposicional (“nin subdit…

AragonésHistoryUNESCO::CIENCIAS DE LAS ARTES Y LAS LETRASNavarroLiterature and Literary TheoryFilologíasPhilosophyaragonéslcsh:Literature (General)lcsh:D111-203lcsh:Medieval historyFilologías. GeneralidadesSintaxis históricalcsh:PN1-6790sintaxis históricaLanguage and LinguisticsPosesivoslcsh:D204-475:CIENCIAS DE LAS ARTES Y LAS LETRAS [UNESCO]navarroHumanitieslcsh:Modern history 1453-
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Granulocyte functions are independent of arginine availability.

2014

Abstract Arginine depletion via myeloid cell arginase is critically involved in suppression of the adaptive immune system during cancer or chronic inflammation. On the other hand, arginine depletion is being developed as a novel anti-tumor metabolic strategy to deprive arginine-auxotrophic cancer cells of this amino acid. In human immune cells, arginase is mainly expressed constitutively in PMNs. We therefore purified human primary PMNs from healthy donors and analyzed PMN function as the main innate effector cell and arginase producer in the context of arginine deficiency. We demonstrate that human PMN viability, activation-induced IL-8 synthesis, chemotaxis, phagocytosis, generation of RO…

ArginineHydrolasesNeutrophilsPhagocytosisImmunologyPrimary Cell CultureInflammationAntineoplastic AgentsApoptosisBiologyPharmacologyArgininePolyethylene GlycolsMiceImmune systemPhagocytosismedicineImmunology and AllergyAnimalsHumansLungCells CulturedRespiratory BurstInnate immune systemArginaseAspergillus fumigatusInterleukin-8ChemotaxisCell BiologyAcquired immune systemImmunity InnateArginaseMice Inbred C57BLChemotaxis LeukocyteImmunologyCitrullinePulmonary Aspergillosismedicine.symptomReactive Oxygen SpeciesBronchoalveolar Lavage FluidJournal of leukocyte biology
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Phenotype Correlation and Intergenerational Dynamics of the Friedreich Ataxia GAA Trinucleotide Repeat

1997

Summary The Friedreich ataxia (FA) mutation has recently been identified as an unstable trinucleotide GAA repeat present 7–22 times in the normal population but amplified as many as > 1, 000 times in FA. Since it is an autosomal recessive disease, FA does not show typical features observed in other dynamic mutation disorders, such as genetic anticipation. We have analyzed the GAA repeat in 104 FA patients and 163 carrier relatives previously defined by linkage analysis. The GAA expansion was detected in all patients, most (94%) of them being ho-mozygous for the mutation. We have demonstrated that clinical variability in FA is related to the size of the expanded alleles: milder forms of the …

AtaxiaAdolescentGenetic LinkagePopulationBiologyTrinucleotide RepeatsMeiosisGenetic linkageGene duplicationGeneticsmedicineHumansGenetics(clinical)AlleleChildeducationGenetics (clinical)Geneticseducation.field_of_studyGene AmplificationPhenotypeFriedreich AtaxiaMutationMutation (genetic algorithm)Dynamic mutationmedicine.symptomResearch ArticleThe American Journal of Human Genetics
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Causative role of oxidative stress in a Drosophila model of Friedreich ataxia

2006

Friedreich ataxia (FA), the most common form of hereditary ataxia, is caused by a deficit in the mitochondrial protein frataxin. While several hypotheses have been suggested, frataxin function is not well understood. Oxidative stress has been suggested to play a role in the pathophysiology of FA, but this view has been recently questioned, and its link to frataxin is unclear. Here, we report the use of RNA interference (RNAi) to suppress the Drosophila frataxin gene (fh) expression. This model system parallels the situation in FA patients, namely a moderate systemic reduction of frataxin levels compatible with normal embryonic development. Under these conditions, fh-RNAi flies showed a shor…

AtaxiaBlotting WesternLongevityGene ExpressionCHO Cellsmedicine.disease_causeBiochemistryAconitaseMitochondrial ProteinsCricetulusRNA interferenceCricetinaeIron-Binding ProteinsGeneticsmedicineAnimalsDrosophila ProteinsRNA MessengerMolecular BiologyGeneAconitate HydrataseHyperoxiaGeneticsElectron Transport Complex IbiologyReverse Transcriptase Polymerase Chain ReactionSuccinate dehydrogenasefungiImmunohistochemistryCell biologySuccinate DehydrogenaseOxidative StressDrosophila melanogasterFriedreich AtaxiaFrataxinbiology.proteinRNA Interferencemedicine.symptomOxidative stressBiotechnologyThe FASEB Journal
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