Search results for "taxi"

showing 10 items of 515 documents

Prognostic significance of the chemokine CXCL13 in node-negative breast cancer

2013

615 Background: The chemokine CXCL13 is chemotactic for B cells. We examined the prognostic significance of CXCL13 mRNA expression in node-negative breast cancer. Methods: Microarray based gene-expression data for CXCL13 (205242_at) were analysed in four previously published cohorts (Mainz, Rotterdam, Transbig, Yu) of node-negative breast cancer patients not treated with adjuvant therapy (n=824). A meta-analysis of previously published cohorts was performed using a random effects model. Prognostic significance of CXCL13 on metastasis-free survival (MFS) was examined in the whole cohort and in different molecular subtypes (ER+/HER2-, ER-/HER2-, HER2+). Independent prognostic relevance was a…

OncologyCancer Researchmedicine.medical_specialtyChemokineMicroarraybiologybusiness.industryMrna expressionObstetrics and GynecologyChemotaxismedicine.diseaseNode negativeBreast cancerOncologyInternal medicineMaternity and MidwiferyImmunologyCancer researchbiology.proteinMedicineCXCL13businessGeburtshilfe und Frauenheilkunde
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17. Masseter reflex abnormalities with meso-diencephalic lesions

2012

different papers and some eponyms used to characterize a certain neurological state differ from the original description. Except for Wallenberg‘s syndrome, classical brainstem syndromes are rarely seen in clinical practice. Lacunar brainstem syndromes are the consequence of infarcts, which involve long tracts and spare intra-axial cranial nerve segments. This group includes pure motor or pure sensory stroke, dysarthriaclumsy hand syndrome, or ataxic hemiparesis. Such infarcts may also cause body lateropulsion with or without limb ataxia, internuclear ophthalmoplgia, skew-torsion sign, or ocular tilt reaction. Small deep infarcts, which solely affect certain nuclei, may be followed by horizo…

Palsybusiness.industryLimb ataxiaSensory systemAnatomymedicine.diseaseSensory SystemsNeurologyPhysiology (medical)VomitingMedicinecardiovascular diseasesNeurology (clinical)BrainstemUpbeat nystagmusmedicine.symptombusinessStrokeJaw jerk reflexClinical Neurophysiology
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Late-Onset Globoid Cell Leukodystrophy: Unusual Ultrastructural Pathology and Subtotal β-Galactocerebrosidase Deficiency

1990

An 11-year-old girl was found to have severely reduced β-galactocerebrosidase activity as evidence of late-onset globoid cell leukodystrophy, while her mother had almost normal enzyme activity in circulating white blood cells. Clinically, the patient showed a remitting course marked by seizures, ataxia, white-matter disease on computed tomographic scan, and reduced conduction velocities of peripheral nerves. Symptoms improved somewhat around the age of 10 years. Two sural nerve biopsies, performed 6 years apart, disclosed a demyelinating neuropathy. By electron microscopy, membrane-bound vacuolar lysosomes in Schwann cells of myelinated axons, unlike the typical needlelike inclusions seen …

Pathologymedicine.medical_specialtyAtaxiaAdolescentBiopsyCellSural nerveBiologyUltrastructural Pathology03 medical and health sciences0302 clinical medicineSural Nerve030225 pediatricsBiopsymedicineHumansChildMyelin SheathInclusion Bodiesmedicine.diagnostic_testLeukodystrophymedicine.diseaseAxonsLeukodystrophy Globoid CellMicroscopy Electronmedicine.anatomical_structureLate-Onset Globoid Cell LeukodystrophyChild PreschoolPediatrics Perinatology and Child HealthFemaleSchwann CellsNeurology (clinical)medicine.symptom030217 neurology & neurosurgeryβ galactocerebrosidaseFollow-Up StudiesGalactosylceramidaseJournal of Child Neurology
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Early-onset ataxia with cardiomyopathy and retained tendon reflexes maps to the friedreich's ataxia locus on chromosome 9q

1995

Absence of lower limb tendon reflexes has been considered an essential diagnostic criterion for Friedreich's ataxia (FA). However, preservation of knee and ankle jerks has been reported in a few patients. Linkage analysis to FA locus (FRDA) on chromosome 9q13-21.1 was performed in 11 patients from 6 families with FA phenotype, including cardiomyopathy, but retained reflexes (FARR). A maximal lod score of 3.38 at recombination fraction theta equal to 0.00 was obtained demonstrating that FARR maps to the FRDA locus. These results suggest that FARR is a variant phenotype of FA.

Pathologymedicine.medical_specialtyAtaxiaCardiomyopathyLocus (genetics)Biologymedicine.diseaseTendon reflexCentral nervous system diseaseDegenerative diseaseAtrophymedicine.anatomical_structureNeurologyGenetic linkagemedicineNeurology (clinical)medicine.symptomAnnals of Neurology
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Paraneoplastic Focal Outer Retinitis and Optic Neuropathy in a Patient with Small Cell Lung Carcinoma and Anti-CRMP5, Anti-HU and Anti-Amphiphysin An…

2020

Our aim is to describe clinical and optical coherence tomographic features of acute paraneoplastic focal outer retinitis associated with optic neuropathy in a patient diagnosed with small cell lung carcinoma. Bilateral focal outer retinitis, bilateral optic disc oedema and vitritis were identified in a patient with progressive bilateral visual loss and ataxia. Spectral domain optical coherence tomography (SD-OCT) revealed various extents of granular hyperreflectivity and atrophy of the macular outer retinal layers. Serum and cerebrospinal fluid positivity for anti-CRMP5, anti-HU and anti-amphiphysin antibodies intensified the search for an underlying malignancy, and a small cell lung carcin…

Pathologymedicine.medical_specialtyAtaxiagenetic structuresmedicine.medical_treatmentRetinitisCase ReportMalignancy01 natural sciencesOptic neuropathy03 medical and health scienceschemistry.chemical_compound0302 clinical medicineAtrophylcsh:Ophthalmologymedicine0101 mathematicsOptic disc oedemabusiness.industry010102 general mathematicsRetinitisRetinalmedicine.diseaseCollapsin response mediator protein 5eye diseasesRadiation therapyOphthalmologychemistrylcsh:RE1-994030221 ophthalmology & optometryParaneoplastic syndromeAtaxiaSmall Cell Lung Carcinomasense organsmedicine.symptombusinessCase Reports in Ophthalmology
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Superficial Siderosis of the Central Nervous System associated with Hemophilia A: A case report

2021

Abstract Superficial Siderosis of the Central Nervous System (SSCNS) is a condition secondary to the deposition of hemosiderin within the subpial layers of central nervous system leading to its progressive degeneration, clinically responsible for hearing impairment, cerebellar ataxia and pyramidal syndrome. Here we report the case of a 61-year-old man with medical history of congenital hemophilia A presenting with typical clinical features of SSCNS associated with extensive hypo-intensity on fast 2D gradient-echo-weighted sequences, along the spinal cord, posterior fossa's structures and cerebral cortex. Interestingly, although his disorder was revealed by a lumbar spinal stenosis, presurgi…

Pathologymedicine.medical_specialtyCentral nervous systemlcsh:SurgerySuperficial Siderosis of the Central Nervous System[INFO] Computer Science [cs]lcsh:RC346-429030218 nuclear medicine & medical imaging03 medical and health sciences0302 clinical medicinemedicineMedical history[INFO]Computer Science [cs]lcsh:Neurology. Diseases of the nervous systemSubclinical infectionetiological diagnosisCerebellar ataxiabusiness.industryLumbar spinal stenosislcsh:RD1-811medicine.diseaseSpinal cordSuperficial siderosis3. Good healthmedicine.anatomical_structureCongenital Hemophilia AHemosiderinchronical bleedingSurgeryNeurology (clinical)medicine.symptombusiness030217 neurology & neurosurgery
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Endothelial capillaries chemotactically attract tumour cells.

2001

Directional migration of capillaries towards tumour implants is generally assumed to be regulated by chemotaxis. Preliminary evidence has also been presented for the existence of a reverse chemotactic signalling pathway, with capillaries attracting tumour cells via paracrine factors. By using a variety of endothelial cell types and tumour cell lines, this study has systematically investigated chemotaxis between endothelial cells and tumour cells in two- and three-dimensional systems. Checkerboard analysis revealed faint attraction of human umbilical vein endothelial cells (HUVECs), but not porcine aortic endothelial cells (PAECs), by tumour cells. In reverse, both PAECs and HUVECs potently …

Pathologymedicine.medical_specialtyEndotheliumAngiogenesisSwineCell Culture TechniquesCell CommunicationBiologyPathology and Forensic MedicineParacrine signallingVasculogenesisNeoplasmsmedicineTumor Cells CulturedAnimalsHumansMicroscopy Phase-ContrastMelanomaFibrinNeovascularization PathologicChemotaxisMicrocarrierChemotaxisCell biologyCapillariesEndothelial stem cellmedicine.anatomical_structureCell cultureCulture Media Conditionedcardiovascular systemEndothelium VascularGlioblastomaThe Journal of pathology
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Microvascular in vivo assessment of reperfusion injury: significance of prostaglandin E1 and I2 in postischemic “no-reflow” and “reflow-paradox”

2004

Microvascular ischemia-reperfusion (I/R) injury is characterized by failure of capillary perfusion ("no-reflow") and reoxygenation-associated phenomena ("reflow-paradox"), including activation of leukocyte-endothelium interaction with cytotoxic mediator-induced loss of endothelial integrity. The objectives of this study were to elucidate the impact of both prostaglandins E(1) (PGE(1)) and I(2) (PGI(2)) in microvascular reperfusion injury, with special focus on the distinct pathophysiology of no-reflow- and reflow-paradox phenomena.By use of the hamster dorsal skinfold preparation and in vivo fluorescence microscopy, the microcirculation of a striated skin muscle was assessed before 4 h of p…

Pathologymedicine.medical_specialtyEndotheliummedicine.medical_treatmentIschemiaPharmacologyMicrocirculationCapillary Permeabilitychemistry.chemical_compoundIn vivoCricetinaemedicineAnimalsVascular Diseasescardiovascular diseasesAlprostadilMuscle SkeletalProstaglandin E1SkinMicroscopyMesocricetusbusiness.industryMicrocirculationmedicine.diseaseEpoprostenolPathophysiologyCapillariesChemotaxis Leukocytemedicine.anatomical_structurechemistryReperfusion InjuryModels Animalcardiovascular systemSurgeryEndothelium VascularbusinessReperfusion injuryPlatelet Aggregation InhibitorsProstaglandin EJournal of Surgical Research
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A potential role of interferon-gamma in the pathogenesis of venous leg ulcers.

2005

Venous leg ulcer is the most severe expression of chronic venous insufficiency. Venous ulcerations are always associated with venous ambulatory hypertension, but the exact mechanism leading from pathological hemodynamics in venous circulation to the necrotic lesions in the skin still remains undiscovered. It has been shown that tissue injury in venous ulcer patients was induced by leukocytes. However, though infiltrating leukocytes have at their disposal a powerfully cytotoxic arsenal, it has not been discovered which molecular mechanisms may contribute to the skin damage. The search for this hypothetical factor responsible for the development of ulceration should be focused on mechanisms l…

Pathologymedicine.medical_specialtyNecrosisChronic venous insufficiencybusiness.industrymedicine.medical_treatmentLeg UlcerModels ImmunologicalGeneral Medicinemedicine.diseaseVenous leg ulcerPathophysiologyProinflammatory cytokineVaricose UlcerPathogenesisInterferon-gammaCytokinemedicineHumansmedicine.symptombusinessLeukocyte chemotaxisMedical hypotheses
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Determination of neuronal antibodies in suspected and definite Creutzfeldt-Jakob disease

2014

IMPORTANCE: Creutzfeldt-Jakob disease (CJD) and autoimmune encephalitis with antibodies against neuronal surface antigens (NSA-abs) may present with similar clinical features. Establishing the correct diagnosis has practical implications in the management of care for these patients. OBJECTIVE: To determine the frequency of NSA-abs in the cerebrospinal fluid of patients with suspected CJD and in patients with pathologically confirmed (ie, definite) CJD. DESIGN, SETTING, AND PARTICIPANTS: A mixed prospective (suspected) and retrospective (definite) CJD cohort study was conducted in a reference center for detection of NSA-abs. The population included 346 patients with suspected CJD and 49 pati…

Pathologymedicine.medical_specialtyPopulationAntígensArticleNeurologiamental disordersmedicineMalaltia de Creutzfeldt-JakobCognitive declineAntigenseducationAutoimmune encephalitiseducation.field_of_studyCerebellar ataxiabusiness.industryEncefalitisDiagnòstic diferencialCreutzfeldt-Jakob Syndromemedicine.diseaseCreutzfeldt-Jakob diseasenervous system diseasesEstudi de casosNeurologyEncephalitisDifferential diagnosisNeurology (clinical)Case studiesDifferential diagnosismedicine.symptombusinessMyoclonusEncephalitis
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