Search results for "toxin"

showing 10 items of 1434 documents

In vivo formation of aflatoxin B1-DNA adducts in parenchymal and non-parenchymal cells of rat liver

1992

The induction of hepatocellular carcinoma from liver parenchymal cells in laboratory animals by aflatoxin B1 (AFB1) is well documented. In contrast no tumours arising from the sinusoidal cell population have been reported after exposure to AFB1. The apparent resistance of the latter cell type was investigated at the level of DNA adduct formation in vivo in male Sprague-Dawley rats. Liver parenchymal and non-parenchymal cell populations were isolated from rats at 20 min and 1, 24 and 72 h after administration of 240 microCi (0.6 mg) [G-3H]AFB1/kg. AFB1-DNA binding was observed in both liver cell subpopulations and was 3- to 5-fold higher in parenchymal cells than in non-parenchymal cells. Th…

MaleCancer ResearchCell typePathologymedicine.medical_specialtyAflatoxin B1Time FactorsPopulationCellBiologyDNA AdductsIn vivoDNA adductmedicineAnimalseducationeducation.field_of_studyLiver cellRats Inbred StrainsDNAGeneral Medicinemedicine.diseaseRats Inbred F344Ratsmedicine.anatomical_structureLiverHepatocyteHepatocellular carcinomaCarcinogenesis
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Clobetasol promotes neuromuscular plasticity in mice after motoneuronal loss via sonic hedgehog signaling, immunomodulation and metabolic rebalancing

2021

AbstractMotoneuronal loss is the main feature of amyotrophic lateral sclerosis, although pathogenesis is extremely complex involving both neural and muscle cells. In order to translationally engage the sonic hedgehog pathway, which is a promising target for neural regeneration, recent studies have reported on the neuroprotective effects of clobetasol, an FDA-approved glucocorticoid, able to activate this pathway via smoothened. Herein we sought to examine functional, cellular, and metabolic effects of clobetasol in a neurotoxic mouse model of spinal motoneuronal loss. We found that clobetasol reduces muscle denervation and motor impairments in part by restoring sonic hedgehog signaling and …

MaleCancer ResearchPhysiology129 StrainBiochemistryMiceDatabases GeneticMedicineMyocyteMotor NeuronsNeuronal PlasticitySkeletalSmoothened ReceptorHedgehog signaling pathwayMuscle atrophyMitochondriaAstrogliosisNeuroprotective AgentsMusclemedicine.symptomInflammation MediatorsSignal TransductionCholera ToxinMice 129 StrainhedgehogImmunologyMotor ActivityNeuroprotectionArticleDatabasesCellular and Molecular NeurosciencesmoothenedGeneticAnimalsHumansHedgehog ProteinsMuscle SkeletalHedgehogGlucocorticoidsMuscle DenervationQH573-671Animalbusiness.industryAmyotrophic Lateral SclerosisGlial biologyCell Biologymedicine.diseaseSaporinsSpineMitochondria MuscleDisease Models AnimalclobetasolinflammationCase-Control StudiesDisease ModelsDiseases of the nervous systemCytologySmoothenedbusinessEnergy MetabolismNeuroscienceOpen Field Test
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A restricted population of CB1 cannabinoid receptors with neuroprotective activity.

2014

The CB1 cannabinoid receptor, the main molecular target of endocannabinoids and cannabis active components, is the most abundant G protein-coupled receptor in the mammalian brain. Of note, CB1 receptors are expressed at the synapses of two opposing (i.e., GABAergic/inhibitory and glutamatergic/excitatory) neuronal populations, so the activation of one and/or another receptor population may conceivably evoke different effects. Despite the widely reported neuroprotective activity of the CB1 receptor in animal models, the precise pathophysiological relevance of those two CB1 receptor pools in neurodegenerative processes is unknown. Here, we first induced excitotoxic damage in the mouse brain b…

MaleCannabinoid receptorPopulationNeurotoxinsExcitotoxicityGlutamic AcidBiologymedicine.disease_causeNeuroprotectionGlutamatergicMiceOrgan Culture TechniquesReceptor Cannabinoid CB1medicineAnimalsHumansGABAergic NeuronsReceptoreducationCaenorhabditis elegans ProteinsAgedCerebral CortexMice KnockoutNeuronseducation.field_of_studyMultidisciplinaryIntegrasesmusculoskeletal neural and ocular physiologyNeurodegenerative DiseasesBiological SciencesMiddle AgedReceptors GABA-AEndocannabinoid systemCorpus Striatumnervous systemGABAergiclipids (amino acids peptides and proteins)FemaleNeurosciencepsychological phenomena and processesEndocannabinoidsSynaptosomesProceedings of the National Academy of Sciences of the United States of America
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Noradrenaline release from permeabilized synaptosomes is inhibited by the light chain of tetanus toxin

1992

AbstractNoradrenaline release from rat brain cortical synaptosomes permeabilized with streptolysin O can be triggered by μM concentrations of free Ca2+. This process was inhibited within minutes by tetanus toxin and its isolated light chain, but not by its heavy chain. The data demonstrate that the effect of tetanus toxin on NA release from purified synaptosomes is caused by the intraterminal action of its light chain.

MaleCell Membrane PermeabilityClostridium tetaniBiophysicsBiologymedicine.disease_causeImmunoglobulin light chainBiochemistryExocytosisExocytosisGeneeskundeNorepinephrineStructural BiologyPermeabilizationGeneticsmedicineSynaptosomeAnimalsNeurotoxinRats WistarStreptolysin OMolecular BiologySynaptosomeToxinCell BiologyRatsTetanus toxinMechanism of actionBiochemistryStreptolysinmedicine.symptomSynaptosomesFEBS Letters
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Variant toxin B and a functional toxin A produced by Clostridium difficile C34.

2001

A particular property of Clostridium difficile strain C34 is an insertion of approximately 2 kb in the tcdA-C34 gene that does not hinder expression of a fully active TcdA-C34 molecule. Intoxication with TcdA-C34 induced an arborized appearance in eukaryotic cells (D-type cytopathic effect); intoxication with TcdB-C34 induced a spindle-like appearance of cells (S-type cytopathic effect). Inactivation of GTPases with purified toxins revealed that Rho, Rac, Cdc42, and Rap are substrates of TcdA-C34. The variant cytotoxin TcdB-C34 inactivated Rho, Rac, Cdc42, Rap, Ral, and R-Ras. Hence, this is the first ‘S-type’ cytotoxin which inactivates both Rho and R-Ras, and is coexpressed with a ‘D-type…

MaleCell SurvivalBacterial ToxinsClostridium difficile toxin AClostridium difficile toxin BGTPaseEnterotoxinCHO CellsBiologymedicine.disease_causeMicrobiologyMicrobiologyEnterotoxinsBacterial ProteinsCricetinaeGeneticsmedicineAnimalsHumansMolecular BiologyCells CulturedCytopathic effectSkinToxinClostridioides difficileCytotoxinsGenetic VariationClostridium difficileMolecular biologyCdc42 GTP-Binding ProteinDNA Transposable ElementsMicroscopy Electron ScanningFEMS microbiology letters
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An anti-interleukin-2 receptor drug attenuates T- helper 1 lymphocytes-mediated inflammation in an acute model of endotoxin-induced uveitis.

2014

The aim of the present study was to evaluate the anti-inflammatory efficacy of Daclizumab, an anti-interleukin-2 receptor drug, in an experimental uveitis model upon a subcutaneous injection of lipopolysaccharide into Lewis rats, a valuable model for ocular acute inflammatory processes. The integrity of the blood-aqueous barrier was assessed 24 h after endotoxin-induced uveitis by evaluating two parameters: cell count and protein concentration in aqueous humors. The histopathology of all the ocular structures (cornea, lens, sclera, choroid, retina, uvea, and anterior and posterior chambers) was also considered. Enzyme-linked immunosorbent assays of the aqueous humor samples were performed t…

MaleChemokineAnatomy and PhysiologyDaclizumabmedicine.medical_treatmentlcsh:MedicineBiochemistryRedox SignalingOxidative DamageDaclizumabImmune PhysiologyMolecular Cell BiologyBasic Cancer ResearchSignaling in Cellular ProcessesInterferon gammalcsh:ScienceMultidisciplinarybiologyT CellsOxygen MetabolismCytokinemedicine.anatomical_structureOncologyAcute DiseaseCytokinesMedicineImmunotherapymedicine.symptomUveitismedicine.drugResearch ArticleSignal TransductionInterleukin 2Cell PhysiologyImmune CellsImmunologyInflammationAntibodies Monoclonal HumanizedSignaling PathwaysUveitisOcular SystemmedicineAnimalsBiologyInflammationbusiness.industrylcsh:RImmunityReceptors Interleukin-2UveaTh1 Cellsmedicine.diseaseeye diseasesEndotoxinsDisease Models AnimalMetabolismRats Inbred LewImmune SystemImmunoglobulin GImmunologybiology.proteinlcsh:Qsense organsMolecular NeurosciencebusinessNeurosciencePLoS ONE
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Anterograde tracing of retinal afferents to the tree shrew hypothalamus and raphe

2000

The anterograde neuronal transport of Cholera toxin B subunit (CTB) was used in this study to label the termination of retinal afferents in the hypothalamus of the tree shrew Tupaia belangeri. Upon pressure-injection of the substance into the vitreous body of one eye, a major projection of the retinohypothalamic tract (RHT) was found to the hypothalamic suprachiasmatic nuclei (SCN). Although the innervation pattern was bilateral, the ipsilateral SCN received a somewhat stronger projection. Labeling was also found in the supraoptic nucleus and its perinuclear zone, respectively, mainly ipsilaterally as well as in the bilateral para- and periventricular hypothalamic regions without lateral pr…

MaleCholera ToxinHypothalamusBiologySynaptic TransmissionRetinaSupraoptic nucleusAnimalsNeurons AfferentMolecular BiologyNeuronal transportRapheSuprachiasmatic nucleusGeneral NeuroscienceTupaiidaeGeniculate BodiesAnatomyAnterograde tracingHypothalamusRaphe NucleiFemaleSuprachiasmatic NucleusNeurology (clinical)Raphe nucleiSupraoptic NucleusNeuroscienceRetinohypothalamic tractDevelopmental BiologyBrain Research
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Prevalence of diphtheria toxin antibodies in human sera from a cross-section of the Italian population.

1999

Abstract A polycentric study was carried out between 1993 and 1995 in order to evaluate diphtheria immunity on a representative sample of population from different areas of Italy. To determine diphtheria antitoxin, sera from 5187 apparently healthy subjects, divided according to sex and age groups, were titrated using an ELISA indirect method. A basic protective titre of diphtheria antitoxin (>0.01 IU ml −1 ) was found in 4080 (78.6%) subjects. No statistically significant differences between males and females were observed. Our findings show that the proportion of susceptibles increases with age and a high proportion of adults no longer has diphtheria antitoxin at protective levels since t…

MaleCross-sectional studySerologySeroepidemiologic StudiesPrevalenceCluster AnalysisDiphtheria ToxinChildAged 80 and overeducation.field_of_studybiologyImmunoenzyme techniquesDiphtheriaMiddle AgedAntibodies BacterialInfectious DiseasesItalyChild PreschoolCross-sectional studiesMolecular MedicineFemaleAdults; Cross-sectional studies; Diphtheria; Diphtheria antitoxin; Immunoenzyme techniques; ItalyAntibodyAdultDiphtheria antitoxinAdolescentPopulationcomplex mixturesNOAge DistributionImmunitymedicineHumansAdultsSex DistributioneducationAgedDiphtheria toxinGeneral VeterinaryGeneral Immunology and Microbiologybusiness.industryDiphtheriaPublic Health Environmental and Occupational HealthInfantmedicine.diseaseDiphtheria; Diphtheria antitoxin; Cross-sectional studies; Immunoenzyme techniques; Adults; ItalyImmunologyHumoral immunitybiology.proteinbusiness
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Release of [3H]acetylcholine from a modified rat phrenic nerve-hemidiaphragm preparation

1986

Two different preparations of the rat phrenic nerve-hemidiaphragm (whole nerve-muscle preparation, end-plate preparation) were used for studying synthesis and release of radioactive acetylcholine in the absence and presence of cholinesterase inhibitors. When the whole nerve-muscle preparation (110-180 mg) was incubated with [3H]choline, only small amounts of radioactive acetylcholine were synthesized within the tissue. Electrical nerve stimulation of the whole nerve-muscle preparation produced no increase in tritium outflow. Incubation of the end-plate preparation (16-29 mg) which was obtained after removal of most of the muscle mass led to the formation of large amounts of [3H]acetylcholin…

MaleDiaphragmNeuromuscular JunctionStimulationIn Vitro TechniquesNeuromuscular junctionCholinechemistry.chemical_compoundmedicineAnimalsCholinePhrenic nerveCholinesterasePharmacologyNeurotransmitter AgentsbiologyRats Inbred StrainsHemicholinium 3General MedicineAcetylcholineMuscle DenervationRatsPhrenic Nervemedicine.anatomical_structurechemistryAnesthesiaTetrodotoxinbiology.proteinBiophysicsLiberationAcetylcholinemedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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alpha-Bungarotoxin, kappa-bungarotoxin, alpha-cobratoxin and erabutoxin-b do not affect [3H]acetylcholine release from the rat isolated left hemidiap…

1995

Endplate preparations of the rat left hemidiaphragm were incubated with [3H]choline to label neuronal transmitter stores. Nerve evoked release of newly-synthesized [3H]acetylcholine was measured in the absence of cholinesterase inhibitors to investigate whether snake venom neurotoxins by blocking presynaptic nicotinic autoreceptors affect evoked transmitter release. Contractions of the indirectly stimulated hemidiaphragm were recorded to characterize the blocking effect of alpha-neurotoxins at the post-synaptic nicotinic receptors. Neither the long chain neurotoxins alpha-cobratoxin (1 microgram ml-1) and alpha-bungarotoxin (5 microgram ml-1) nor the short chain neurotoxin erabutoxin-b (0.1…

MaleDiaphragmNeurotoxinsPharmacologyReceptors NicotinicTritiumSynaptic TransmissionPostsynaptic potentialmedicineAnimalsCobra Neurotoxin ProteinsChromatography High Pressure LiquidCholinesterasePharmacologyErabutoxinsbiologyChemistryMuscle SmoothGeneral MedicineBungarotoxinmusculoskeletal systemBungarotoxinsAcetylcholineRatsPhrenic NerveNicotinic agonistSnake venomIsotope Labelingbiology.proteinAutoreceptorFemaleCobratoxinNeuroscienceAcetylcholinemedicine.drugMuscle ContractionSnake VenomsNaunyn-Schmiedeberg's archives of pharmacology
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