Search results for "toxin"

showing 10 items of 1434 documents

T-2 toxin and its metabolites: Characterization, cytotoxic mechanisms and adaptive cellular response in human hepatocarcinoma (HepG2) cells

2020

Abstract The T-2 toxin (T-2) is a type A trichothecene produced by Fusarium species, and the most cytotoxic mycotoxin of the group. A study was made to determine T-2 cytotoxicity in human hepatocarcinoma (HepG2) cells; evaluate whether there is an adaptive response of HepG2 cells exposed to low concentrations of T-2; identify the T-2 metabolites by LC-Q-TOF MS; and determine whether T-2 disrupts cell proliferation in HepG2 cells. The IC50 values obtained ranged from 61.9 ± 2.4 nM to 70.7 ± 7.4 nM. No adaptive response was observed. There was no evidence of extra- or intracellular accumulation of T-2 after 24 h of exposure as determined by LC-Q-TOF MS. However, some T-2 metabolites such as H…

NecrosisCell SurvivalApoptosisToxicologymedicine.disease_causeNecrosis03 medical and health sciences0404 agricultural biotechnologymedicineHumansCytotoxic T cellViability assayCytotoxicity030304 developmental biology0303 health sciencesCell growthChemistryToxinCell CycleHep G2 Cells04 agricultural and veterinary sciencesGeneral MedicineAdaptation Physiological040401 food scienceMolecular biologyT-2 ToxinApoptosismedicine.symptomIntracellularFood ScienceFood and Chemical Toxicology
researchProduct

Tinnitus Treatment: Botulinum Toxin

2011

1. Somatosensory tinnitus (objective or subjective) is tinnitus that can be modulated by stimulation of the somatosensory system. 2. Abnormal interactions between the auditory and the somatosensory nervous system that may occur at several levels of the central nervous system cause somatosensory tinnitus. 3. This chapter discusses how administration of a ­botulinum toxin can alleviate tinnitus and the mechanism of its action, and how that relates to its effects on chronic pain. 4. A proven benefit of botulinum toxin in patients with objective tinnitus is also discussed.

Nervous systemmedicine.medical_specialtybusiness.industryCentral nervous systemChronic painStimulationAudiologymedicine.diseaseSomatosensory systemBotulinum toxinmedicine.anatomical_structureotorhinolaryngologic diseasesmedicineIn patientmedicine.symptombusinessTinnitusmedicine.drug
researchProduct

Novel approaches in diagnosis and therapy of Creutzfeldt-Jakob disease.

2000

The scrapie prion protein, PrP(Sc), as well as its peptide fragment, PrP106-126, are toxic on neuronal cells, resulting in cell death by an apoptotic, rather than necrotic mechanism. The apoptotic process of neuronal cells induced by prion protein supports diagnosis and offers potential targets for therapeutic intervention of the prion diseases. Among the cerebrospinal fluid (CSF) proteins, which may serve as markers of neuronal cell death associated with prion diseases, the 14-3-3 protein(s) turned out to be the most promising one. A new sensitive assay allows the detection of even small changes in the normally low levels of these proteins. In vitro, the toxic effects displayed by PrP(Sc) …

NeuronsAgingCell DeathPrPSc ProteinsNeurotoxicityMemantinePrPSc ProteinsScrapieBiologyPharmacologymedicine.diseaseVirologyCreutzfeldt-Jakob Syndromenervous system diseasesPrion DiseasesmedicineNeurotoxinAnimalsHumansFlupirtineReceptor14-3-3 proteinDevelopmental Biologymedicine.drugMechanisms of ageing and development
researchProduct

Differences in the temperature dependencies of uptake of botulinum and tetanus toxins in Aplysia neurons

1992

The respective neuroselective actions of botulinum type A (BoNT) and tetanus (TeTx) neurotoxins on cholinergic and non-cholinergic synapses of Aplysia are mainly due to differences in their extracellular neuronal targetting. Further information was gained on this neuroselectivity by examining the temperature dependencies of binding, internalization and intracellular action of both toxins. After reduction of temperature from 22 degrees C to 10 degrees C, the binding of neither BoNT nor TeTx was significantly altered whereas the neuronal uptake of BoNT, but not of TeTx, was prevented. Although TeTx internalization could be detected at the low temperature, its intracellular activity was greatl…

NeuronsBotulinum ToxinsGeneral Neurosciencemedia_common.quotation_subjectTemperatureBiologybiology.organism_classificationAcetylcholineSynapseTetanus ToxinAplysiaAplysiamedicineExtracellularBiophysicsAnimalsNeurotoxinCholinergicInternalizationNeuroscienceAcetylcholineIntracellularmedia_commonmedicine.drugNeuroscience Letters
researchProduct

Neurodegeneration in excitotoxicity, global cerebral ischemia, and target deprivation: A perspective on the contributions of apoptosis and necrosis.

1998

In the human brain and spinal cord, neurons degenerate after acute insults (e.g., stroke, cardiac arrest, trauma) and during progressive, adult-onset diseases [e.g., amyotrophic lateral sclerosis, Alzheimer's disease]. Glutamate receptor-mediated excitotoxicity has been implicated in all of these neurological conditions. Nevertheless, effective approaches to prevent or limit neuronal damage in these disorders remain elusive, primarily because of an incomplete understanding of the mechanisms of neuronal death in in vivo settings. Therefore, animal models of neurodegeneration are crucial for improving our understanding of the mechanisms of neuronal death. In this review, we evaluate experimen…

NeuronsProgrammed cell deathNecrosisCell DeathGeneral Neurosciencemedicine.medical_treatmentNeurodegenerationNeurotoxinsGlutamate receptorExcitotoxicityApoptosisAxotomyBiologymedicine.diseasemedicine.disease_causeNeuroprotectionBrain IschemiaBrain ischemiaNecrosisNerve DegenerationmedicineAnimalsmedicine.symptomAxotomyNeuroscienceBrain research bulletin
researchProduct

Comparative analysis of eight cytotoxicity assays evaluated within the ACuteTox Project.

2013

A comparative analysis of eight cytotoxicity assays [the 3T3 and normal human keratinocytes Neutral Red Uptake (NRU) assay, the primary rat hepatocytes, human HepG2 and 3T3 MU assay, and the human A.704, SH-SY5Y and HepG2 cells propidium iodide (PI) assay] included in several work packages of the EU Integrated Project ACuteTox, has been carried out. The aim was to evaluate whether cells originating from liver, kidney and brain provided different in vitro acute toxicity results, and the influence of primary liver cells versus cell lines originated from the same tissue. Spearman rank correlation analysis and Hierarchical Cluster Analysis were performed based on the IC50 (50% inhibitory concen…

Neutral redCell SurvivalCytotoxicityBiologyToxicologyConcentration-response analysischemistry.chemical_compoundMiceCell Line TumorToxicity Tests AcuteAnimalsHumansMTT assayPropidium iodideCytotoxicityCells CulturedAcute toxicityCytotoxinsIn vitro toxicologyCorrelation analysisGeneral Medicine3T3 CellsMolecular biologyAcute toxicityRatsHierarchical Cluster AnalysischemistryCell cultureToxicityImmunologyIn vitro assaysHepatocytesToxicology in vitro : an international journal published in association with BIBRA
researchProduct

Reactive oxygen species induced by beauvericin, patulin and zearalenone in CHO-K1 cells

2008

The cytotoxic effects of mycotoxins, induction of reactive oxygen species (ROS) and generation of lipid peroxidation products in CHO-K1 cells were determined as function of increasing time of exposure and concentrations of beauvericin (BEA), patulin (PAT) and zearalenone (ZEA). The end points were evaluated after 24h of exposure, by the tetrazolium salt (MTT) and neutral red (NR) assays. The IC(50) values obtained on the MTT and NR assays ranged from 0.69 to 79.40 microM and 4.40 to 108.76 microM, respectively. To determine the intracellular production of ROS, the intensity of fluorescence emitted from the probe H(2)-DCFDA was measured. The relative intensity of fluorescence from cells incu…

Neutral redThiobarbituric acidCHO CellsToxicologyPatulinLipid peroxidationchemistry.chemical_compoundCricetulusCricetinaeDepsipeptidesMalondialdehydeTBARSAnimalschemistry.chemical_classificationReactive oxygen speciesfood and beveragesGeneral MedicineMycotoxinsMalondialdehydeBeauvericinPatulinchemistryBiochemistryZearalenoneLipid PeroxidationReactive Oxygen SpeciesToxicology in Vitro
researchProduct

Toxicological interactions between the mycotoxins beauvericin, deoxynivalenol and T-2 toxin in CHO-K1 cells in vitro.

2011

Abstract Beauvericin (BEA), deoxynivalenol (DON) and T-2 toxin (T-2) are important food-borne mycotoxins that have been implicated in human health. In this study, the acute toxicity of individual and combined mycotoxins (BEA, DON and T-2) were tested in immortalized hamster ovarian cells (CHO-K1) at 24, 48 and 72 h of exposure, by the tetrazolium salt (MTT) and neutral red (NR) assays. The IC50 values obtained for all mycotoxins by the MTT and NR assays ranged from 0.017 to 12.08 μM and from 0.042 to 17.22 μM, respectively. Both, individual and combined mycotoxins demonstrated a significant cytotoxic effect in CHO-K1 cells in a dose-dependent manner. When mycotoxins were assayed individuall…

Neutral redToxinHamsterCHO CellsPharmacologyToxicologymedicine.disease_causeAcute toxicityBeauvericinToxicologychemistry.chemical_compoundInhibitory Concentration 50T-2 ToxinchemistryCricetinaeDepsipeptidesmedicineToxicity Tests AcuteAnimalsAntagonismCytotoxicityMycotoxinTrichothecenesToxicon : official journal of the International Society on Toxinology
researchProduct

Cycloamphilectenes, a new type of potent marine diterpenes: inhibition of nitric oxide production in murine macrophages.

2003

The inhibitory effect of a series of 6 cycloamphilectenes, novel marine diterpenes based on amphilectene skeletons and isolated from the Vanuatu sponge Axinella sp., on NO, PGE(2) and TNFalpha production in murine peritoneal macrophages was studied. These compounds reduced potently nitric oxide production in a concentration-dependent manner with IC(50) values in the submicromolar range (0.1-4.3 microM). Studies on intact cells and Western blot analysis showed that the more potent cycloamphilectenes reduced the expression of inducible nitric oxide synthase without affecting cyclo-oxygenase-2 expression. Among them cycloamphilectene 2, the unique compound bearing an exocyclic methylene group,…

NeutrophilsBlotting WesternNitric Oxide Synthase Type IIElectrophoretic Mobility Shift AssayIn Vitro TechniquesNitric OxideGeneral Biochemistry Genetics and Molecular BiologyDinoprostoneNitric oxidechemistry.chemical_compoundMiceStructure-Activity RelationshipWestern blotmedicineAnimalsEdemaHumansGeneral Pharmacology Toxicology and PharmaceuticsMethylenebiologymedicine.diagnostic_testPancreatic ElastaseTumor Necrosis Factor-alphaMacrophagesZymosanAnti-Inflammatory Agents Non-SteroidalAxinellaNF-kappa BMembrane ProteinsGeneral Medicinebiology.organism_classificationPoriferaNitric oxide synthaseIsoenzymesSpongechemistryBiochemistryCyclooxygenase 2Prostaglandin-Endoperoxide Synthasesbiology.proteinMacrophages PeritonealTumor necrosis factor alphaFemaleMarine ToxinsDiterpenesNitric Oxide SynthaseLife sciences
researchProduct

Desensitization is a property of the cholinergic binding region of the nicotinic acetylcholine receptor, not of the receptor-integral ion channel

1991

AbstractThe reversible acetylcholine esterase inhibitor (−)-physostigmine (eserine) is the prototype of a new class of nicotinic acetylcholine receptor (nAChR) activating ligands: it induces cation fluxes into nAChR-rich membrane vesicles from Torpedo marmorala electric tissue even under conditions of antagonist blocked acetylcholine binding sites (Okonjo, Kuhlmann, Maclicke, Neuron, in press). This suggests that eserine exerts its channel-activating property via binding sites at the nAChR separate from those of the natural transmitter. We now report that eserine can activate the channel even when the receptor has been preincubated (desensitized) with elevated concentrations of acetylcholin…

Nicotinic acetylcholine receptorStereochemistryAcetylcholine-gated cation channelPhysostigmineBiophysicsCesiumIon fluxDesensitizationIn Vitro TechniquesReceptors NicotinicTorpedoBiochemistryIon ChannelsAnticholinesteraseAcetylcholine bindingGanglion type nicotinic receptorStructural BiologyMuscarinic acetylcholine receptor M5GeneticsmedicineAnimalsMolecular BiologyAcetylcholine receptorBinding SitesChemistryCell BiologyBungarotoxinsAcetylcholineNicotinic acetylcholine receptorNicotinic agonistCarbamateBiophysicsCholinergicAcetylcholineEserinemedicine.drugFEBS Letters
researchProduct