Search results for "trace"

showing 10 items of 3218 documents

Frequency of NOD2/CARD15 variants in both sporadic and familial cases of Crohn's disease across Italy. An Italian Group for Inflammatory Bowel Diseas…

2004

Abstract Background. Three variants of the NOD 2 /CARD 15 gene are strongly associated with susceptibility to Crohn’s disease; however, striking racial and geographic differences of their frequency have been described. Aims. We have compared the allele frequencies of familial cases of Crohn’s disease recruited in a multicentre study across Italy, in order to disclose possible geographic heterogeneity. Moreover, we also compared the allele frequencies in sporadic cases of Crohn’s disease and healthy controls from Southern Italy with those reported in other two populations from Central and Northern Italy. Subjects and Methods. A total of 731 subjects were genotyped for the polymorphism of thr…

AdultMalemedicine.medical_specialtyPathologyGenotypeNod2 Signaling Adaptor ProteinDiseaseInflammatory bowel diseaseGastroenterologyInflammatory bowel diseaseNOD2Frameshift mutationGeneticCrohn DiseaseGene FrequencyPolymorphism (computer science)NOD2Internal medicineMedicineHumansGenetic Predisposition to DiseaseCARD15Frameshift MutationAllele frequencyAgedCrohn's diseasePolymorphism GeneticHepatologybusiness.industrySignificant differenceGastroenterologyIntracellular Signaling Peptides and ProteinsMiddle Agedmedicine.diseaseCrohn's diseaseItalyFemalebusinessCarrier Proteins
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Immunolocalization of Tenascin-C in Human Type II Fiber Atrophy

2000

Tenascin-C is a multifunctional extracellular matrix glycoprotein with stimulatory and anti-adhesive or inhibitory properties for axon growth. Its location and discontinuous expression are restricted in innervated muscle tissues. Tenascin-C accumulated interstitially among human denervated muscle fibers and close to normal-sized fibers. To expand our knowledge of the expression of tenascin-C in human neuromuscular disorders, we investigated immunohistologically 20 human muscle specimens with type II myofiber atrophy of children and adults. Tenascin-C immunoreactivity in adult type II atrophy was frequent, and accumulation in children was sparse and weak. In both groups, tenascin-C immunorea…

AdultMalemedicine.medical_specialtyPathologyTenascinPlatelet membrane glycoproteinExtracellular matrixCellular and Molecular NeuroscienceAtrophyInternal medicinemedicineHumansMyocyteAgedDenervationbiologyChemistryTenascin CInfantTenascinGeneral MedicineMiddle Agedmedicine.diseaseImmunohistochemistryMuscular AtrophyEndocrinologyChild PreschoolMuscle Fibers Fast-Twitchembryonic structuresbiology.proteinImmunohistochemistryFemaleJournal of Molecular Neuroscience
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Familial Sotos syndrome caused by a novel missense mutation, C2175S, in NSD1 and associated with normal intelligence, insulin dependent diabetes, bro…

2009

We report a familial Sotos syndrome in two children, boy and girl, aged 17 and 8 years, and in their 44 year old mother, who displayed normal intelligence at adult age, but suffered from insulin dependent diabetes mellitus, bronchial asthma, and severe lipedema. The underlying missense mutation, C2175S, occurred in a conserved segment of the NSD1 gene. Our findings confirm that familial cases of SS are more likely to carry missense mutations. This case report may prove useful to avoid underestimation of the recurrence rate of SS, and to demonstrate that the developmental delay may normalize, enabling an independent life and having an own family.

AdultMalemedicine.medical_specialtyPediatricsAdolescentLipid Metabolism DisordersMutation MissenseGermanyInternal medicineImmunopathologyGeneticsHumansMedicineMissense mutationGrowth DisordersGenetics (clinical)AsthmaAutoimmune diseaseType 1 diabetesbusiness.industrySotos syndromeRespiratory diseaseIntracellular Signaling Peptides and ProteinsLipoedemaNuclear ProteinsHistone-Lysine N-MethyltransferaseSyndromeGeneral Medicinemedicine.diseaseAsthmaDiabetes Mellitus Type 1EndocrinologyHistone MethyltransferasesFemalebusinessEuropean Journal of Medical Genetics
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Acute Cerebrovascular Disease in the Young

2013

Background and Purpose— Strokes have especially devastating implications if they occur early in life; however, only limited information exists on the characteristics of acute cerebrovascular disease in young adults. Although risk factors and manifestation of atherosclerosis are commonly associated with stroke in the elderly, recent data suggests different causes for stroke in the young. We initiated the prospective, multinational European study Stroke in Young Fabry Patients (sifap) to characterize a cohort of young stroke patients. Methods— Overall, 5023 patients aged 18 to 55 years with the diagnosis of ischemic stroke (3396), hemorrhagic stroke (271), transient ischemic attack (1071) we…

AdultMalemedicine.medical_specialtyPediatricsAdolescentischemiccauseCohort StudiesYoung AdultRisk FactorsmedicineHumansrisk factorsProspective Studiescardiovascular diseasesYoung adultProspective cohort studyStrokeAdvanced and Specialized NursingIntracerebral hemorrhageFabry diseaseCerebral infarctionbusiness.industryAge Factorsimagingyoung strokeMiddle Agedmedicine.diseaseintracerebral hemorrhagestrokeFabry diseaseEuropeStrokeCerebrovascular DisordersNeurologytransient ischemic attackAcute DiseaseCohortPhysical therapyFabry DiseaseFemaleNeurology (clinical)Cardiology and Cardiovascular MedicinebusinessCohort studyStroke
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Increased prevalence of obesity in narcoleptic patients and relatives

2001

Increased Body Mass Indices (BMIs), increased prevalences of non insulin-dependent diabetes and sleep apnoe syndrome have been reported to be associated with narcolepsy. Our objective was to explore and possibly confirm the association of narcolepsy and increased BMI. In addition, we addressed the question whether increased BMIs also occur in relatives of narcoleptic patients. Together with narcolepsy-related clinical parameters we measured body weight and height of 132 narcoleptic patients who agreed to participate in our narcolepsy research program. In addition, 52 first degree relatives of 22 index patients, mostly from multiplex families, were included in the study. Data were compared t…

AdultMalemedicine.medical_specialtyPediatricseducationPopulationBody weightBody Mass IndexDiabetes mellitusmedicineHumansPharmacology (medical)ObesityFirst-degree relativesPsychiatryeducationBiological PsychiatryNarcolepsyPsychiatric Status Rating ScalesOrexinseducation.field_of_studybusiness.industryNeuropeptidesIntracellular Signaling Peptides and Proteinsnutritional and metabolic diseasesGeneral Medicinemedicine.diseaseObesityOrexinPsychiatry and Mental healthFemaleCarrier ProteinsbusinessBody mass indexNarcolepsyEuropean Archives of Psychiatry and Clinical Neuroscience
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Trace elements in scalp hair samples from patients with relapsing-remitting multiple sclerosis

2015

Background Epidemiological studies have suggested a possible role of trace elements (TE) in the etiology of several neurological diseases including Multiple Sclerosis (MS). Hair analysis provides an easy tool to quantify TE in human subjects, including patients with neurodegenerative diseases. Objective To compare TE levels in scalp hair from patients with MS and healthy controls from the same geographic area (Sicily). Methods ICP-MS was used to determine the concentrations of 21 elements (Ag, Al, As, Ba, Cd, Co, Cr, Cu, Fe, Li, Mn, Mo, Ni, Pb, Rb, Sb, Se, Sr, U, V and Zn) in scalp hair of 48 patients with relapsing–remitting Multiple Sclerosis compared with 51 healthy controls. Results MS …

AdultMalemedicine.medical_specialtyPercentilePathologylcsh:MedicineGastroenterologyIndirect evidenceMultiple sclerosisMultiple Sclerosis Relapsing-RemittingInternal medicinemedicineHuman scalp hairHumanslcsh:ScienceAgedTrace elementsMultidisciplinaryExpanded Disability Status ScaleScalpChemistryMultiple sclerosisSignificant differenceHair analysislcsh:RMiddle Agedmedicine.diseaseRubidiumTrace ElementsSettore GEO/08 - Geochimica E Vulcanologiamedicine.anatomical_structureRelapsing remittingScalpUraniumFemalelcsh:QAluminumHairResearch Article
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Low-density lipoproteins generated during an oral fat load in mild hypertriglyceridemic and healthy subjects are smaller, denser, and have an increas…

2005

Triglyceride-rich lipoproteins generated during the postprandial phase are atherogenic. Large very low-density lipoproteins (LDLs) or chylomicrons (CMs) are not as atherogenic as their remnants (Rem). Small and dense LDLs are associated with cardiovascular disease. Low-density lipoprotein size is partly under genetic control and is considered as a relatively stable LDL feature. In this article, we present data on retinyl palmitate kinetics correlated with the modification of LDL features in terms of size, density, and in vitro receptor binding affinity after an oral fat load. Six nondiabetic, hypertriglyceridemic (HTG) patients and 6 healthy controls were examined. Low-density lipoprotein s…

AdultMalemedicine.medical_specialtyRetinyl EstersSettore MED/09 - Medicina InternaEndocrinology Diabetes and MetabolismBinding CompetitiveModels Biologicalchemistry.chemical_compoundEndocrinologyInternal medicineRetinyl palmitateCell Line TumorChylomicronsmedicineHumansReceptorVitamin AHypertriglyceridemiaLow-density lipoproteins hypertriglyceridemia Fasting and postprandial LDLsTriglycerideCatabolismChemistryFastingFibroblastsPostprandial PeriodDietary FatsLipidsLipoproteins LDLKineticsEndocrinologyPostprandialReceptors LDLlipids (amino acids peptides and proteins)Density gradient ultracentrifugationElectrophoresis Polyacrylamide GelFemaleDiterpenesUltracentrifugationLipoproteinChylomicronMetabolism: clinical and experimental
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Increasing Burden of Stroke: The Dijon Stroke Registry (1987–2012)

2017

<b><i>Background:</i></b> We aimed to provide a representation of the global burden of stroke. <b><i>Methods:</i></b> All cases of stroke were prospectively identified through the population-based registry of Dijon, France (1987–2012). Attack rates and mortality rates (defined as stroke leading to death within 30 days) were standardized to the European standard. Sex differences and temporal trends were evaluated by calculating rate ratios (RRs). <b><i>Results:</i></b> In all, 5,285 stroke cases (52.7% women) were recorded. The standardized attack rate was 98.2/100,000/year and the mortality rate was 12/100,000/year, and…

AdultMalemedicine.medical_specialtyStroke registrySubarachnoid hemorrhageEpidemiologyAttack ratePopulation030204 cardiovascular system & hematologyBrain Ischemia03 medical and health sciencesSex Factors0302 clinical medicineRisk FactorsInternal medicineEpidemiologyPrevalenceHumansMedicineRegistrieseducationStrokeAgedAged 80 and overIntracerebral hemorrhageeducation.field_of_studybusiness.industryIncidenceMortality rateAge FactorsMiddle Agedmedicine.diseaseStrokeSurvival RateFemaleFranceNeurology (clinical)businessIntracranial Hemorrhages030217 neurology & neurosurgeryNeuroepidemiology
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Lattice Corneal Dystrophy Type 1

2014

PURPOSE To evaluate the question whether lattice corneal dystrophy type 1 (LCD1) is of epithelial or stromal origin. METHODS The landmark of advanced LCD1 shows central superficial haze and paracentral stromal lattice lines. In 16 eyes of 8 affected individuals of 2 families, a penetrating keratoplasty was performed. The follow-up was 8 to 16 years after penetrating keratoplasty. Slit-lamp documentation of the patients was evaluated in direct and indirect illumination with dilated pupil to assess the horizontal and vertical pattern of new deposits on the corneal graft. Three affected patients of 2 families are demonstrated in detail. A DNA analysis was performed. RESULTS Gene identification…

AdultMalemedicine.medical_specialtyStromal cellCorneal StromaDNA Mutational AnalysisCorneal graftCorneal KeratocytesCorneal erosionDirect illuminationTransforming Growth Factor betaOphthalmologyTGFBI genemedicineHumansCorneal Dystrophies HereditaryExtracellular Matrix Proteinsbusiness.industryEpithelium Cornealmedicine.diseaseeye diseasesOphthalmologyMutationLattice corneal dystrophyFemalebusinessKeratoplasty PenetratingTGFBICornea
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Acute symptomatic seizures in cerebral venous thrombosis

2020

© 2020 American Academy of Neurology

AdultMalemedicine.medical_specialtySubarachnoid hemorrhageInfarction030204 cardiovascular system & hematology03 medical and health sciences0302 clinical medicineModified Rankin ScaleRisk FactorsSeizuresInternal medicinemedicineHumans030212 general & internal medicineStrokeIntracerebral hemorrhageVenous Thrombosisbusiness.industrycerebral venous thrombosisSymptomatic seizuresOdds ratioMiddle Agedmedicine.diseaseThrombosisCerebral Veins3. Good healthVenous thrombosisAnesthesiaSettore MED/26 - NeurologiaFemaleNeurology (clinical)Intracranial Thrombosisbusiness030217 neurology & neurosurgery
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