Search results for "transgene"

showing 10 items of 259 documents

Human endogenous retrovirus rec interferes with germ cell development in mice and may cause carcinoma in situ, the predecessor lesion of germ cell tu…

2005

Germ cell tumors (GCTs) are among the most common malignancies in young men. We have previously documented that patients with GCT frequently produce serum antibodies directed against proteins encoded by human endogenous retrovirus (HERV) type K sequences. Transcripts originating from the env gene of HERV-K, including the rec-relative of human immunodeficiency virus rev, are highly expressed in GCTs. We report here that mice that inducibly express HERV-K rec show a disturbed germ cell development and may exhibit, by 19 months of age, changes reminiscent of carcinoma in situ, the predecessor lesion of classic seminoma in humans. This provides the first direct evidence that the expression of a…

MaleCancer ResearchTime FactorsvirusesTransgeneBlotting WesternEndogenous retrovirusApoptosisMice TransgenicEndogenyBiologymedicine.disease_causeMiceViral Envelope ProteinsCell Line TumorGeneticsmedicineAnimalsHumansHuman endogenous retrovirus KRNA MessengerMolecular BiologyModels GeneticReverse Transcriptase Polymerase Chain ReactionEndogenous RetrovirusesSeminomaNeoplasms Germ Cell and EmbryonalSeminiferous Tubulesmedicine.diseaseVirologyProtein Structure TertiaryAlternative SplicingGerm Cellsmedicine.anatomical_structureMicroscopy FluorescenceCancer researchGerminomaGerm cell tumorsCarcinogenesisCarcinoma in SituGerm cellOncogene
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Aspartoacylase-lacZ knockin mice: an engineered model of Canavan disease.

2011

Canavan Disease (CD) is a recessive leukodystrophy caused by loss of function mutations in the gene encoding aspartoacylase (ASPA), an oligodendrocyte-enriched enzyme that hydrolyses N-acetylaspartate (NAA) to acetate and aspartate. The neurological phenotypes of different rodent models of CD vary considerably. Here we report on a novel targeted aspa mouse mutant expressing the bacterial β-Galactosidase (lacZ) gene under the control of the aspa regulatory elements. X-Gal staining in known ASPA expression domains confirms the integrity of the modified locus in heterozygous aspa lacZ-knockin (aspa(lacZ/+)) mice. In addition, abundant ASPA expression was detected in Schwann cells. Homozygous (…

MaleCentral Nervous SystemCerebellumPathologyAnatomy and PhysiologyCanavan DiseaseMouseMutantlcsh:MedicineNeural HomeostasisBiochemistryMiceNeurobiology of Disease and Regenerationlcsh:ScienceSex CharacteristicsMultidisciplinaryNeuromodulationNeurochemistryGenomicsAnimal ModelsFunctional Genomicsmedicine.anatomical_structureLac OperonNeurologyHomeostatic MechanismsMedicineFemaleNeurochemicalsGenetic EngineeringResearch ArticleNervous System PhysiologyBiotechnologymedicine.medical_specialtyTransgeneCentral nervous systemNeurophysiologyMice TransgenicNeuroimagingBiologyNeurological SystemAmidohydrolasesWhite matterModel OrganismsGeneticsmedicineAnimalsBiologyNeuropeptidesLeukodystrophylcsh:RComputational Biologymedicine.diseaseMolecular biologyCanavan diseaseAspartoacylaseDisease Models AnimalMetabolismnervous systemSmall MoleculesCellular NeuroscienceMetabolic DisordersMutationGenetics of DiseaseNervous System Componentslcsh:QGene FunctionMolecular NeuroscienceAnimal GeneticsNeurosciencePLoS ONE
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A human leucocyte antigen-DR1 transgene confers susceptibility to experimental allergic encephalomyelitis elicited by an epitope of myelin basic prot…

2003

Much evidence now indicates that human leucocyte antigen (HLA) class I and class II transgenic (Tg) mice can be of value in analysing HLA-restricted presentation of T-cell epitopes relevant to experimental models of autoimmune diseases. One area where this has been applied is the characterization of myelin epitopes presented by HLA class II molecules in experimental model of multiple sclerosis (experimental allergic encephalomyelitis (EAE)). As a first step towards humanized disease models in HLA Tg mice, we have analysed immune response of lymph node cells of HLA-DR1 Tg mice immunized with the human myelin basic protein (MBP) peptides 13–33, 87–106 and 139–154 bound by HLA-DR1. We report h…

MaleEncephalomyelitis Autoimmune ExperimentalMultiple SclerosisEncephalomyelitisTransgeneImmunologyMolecular Sequence DataEpitopes T-LymphocyteMice TransgenicHuman leukocyte antigenEpitopeMyelinMiceImmune systemmedicineAnimalsHumansGenetic Predisposition to DiseaseAmino Acid SequencebiologyHLA-DR1 AntigenMyelin Basic ProteinGeneral Medicinemedicine.diseaseIn vitroPeptide FragmentsMyelin basic proteinDisease Models Animalmedicine.anatomical_structureImmunologybiology.proteinFemaleLymph NodesScandinavian journal of immunology
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Hepatocellular expression of a dominant-negative mutant TGF-β type II receptor accelerates chemically induced hepatocarcinogenesis

2001

The potent growth-inhibitory activity of cytokines of the transforming growth factor-beta (TGF-beta) superfamily and their widespread expression in epithelia suggest that they may play an important role in the maintenance of epithelial homeostasis. To analyse TGF-beta mediated tumor suppressor activity in the liver, we generated transgenic mice overexpressing a dominant negative type II TGF-beta receptor in hepatocytes under control of the regulatory elements of the human C-reactive protein gene promoter. Transgenic animals exhibited constitutive and liver-specific transgene expression. The functional inactivation of the TGF-beta signaling pathway in transgenic hepatocytes was shown by redu…

MaleGenetically modified mouseCancer Researchmedicine.medical_specialtyCarcinoma HepatocellularTransgeneMice TransgenicProtein Serine-Threonine KinasesBiologymedicine.disease_causeMiceLiver Neoplasms ExperimentalTransforming Growth Factor betaInternal medicineGeneticsmedicineAnimalsRNA MessengerMolecular BiologyCells CulturedTissue homeostasisDNA synthesisReceptor Transforming Growth Factor-beta Type IICell biologyC-Reactive ProteinEndocrinologymedicine.anatomical_structureHepatocyteMutationHepatocytesSignal transductionCarcinogenesisReceptors Transforming Growth Factor betaTransforming growth factorOncogene
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4-Epidoxycycline: an alternative to doxycycline to control gene expression in conditional mouse models

2004

Since the pioneering work by Gossen and Bujard in 1992 demonstrating the usefulness of the Escherichia coli derived tet resistance operon for regulating gene expression a large collection of doxycycline-controlled transgenic mice has been established. Gene switching in eukaryotic tissue culture cells or mice requires administration of tetracycline, anhydrotetracycline or doxycycline to efficiently inactivate the transactivator protein tTA (TET-OFF system) or alternatively to activate the reverse transactivator protein rtTA (TET-ON system). However, the antibiotic activity of doxycycline can create an imbalance of the intestinal flora, resulting in diarrhoea and in a smaller number of animal…

MaleGenetically modified mouseReceptor ErbB-2TransgeneBiophysicsAdministration OralMice NudeAntineoplastic AgentsBreast NeoplasmsMice TransgenicBiologyPharmacologyBiochemistryMiceTransactivationCell Line TumorGene expressionmedicineAnimalsMolecular BiologyDoxycyclineRegulation of gene expressionDose-Response Relationship DrugOncogeneStereoisomerismCell BiologyRatsGene Expression Regulation NeoplasticDisease Models AnimalTreatment OutcomeTetracyclinesCell cultureDoxycyclineImmunologyNIH 3T3 Cellsmedicine.drugBiochemical and Biophysical Research Communications
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Extensive characterization of the human DDAH1 transgenic mice

2009

Abstract Purpose of the research Overexpression of the human dimethylarginine dimethylaminohydrolase type 1 (hDDAH1) gene was reported to have beneficial cardiovascular effects in mice. To date, it is unclear whether these effects are related to enhanced metabolic clearance of asymmetric dimethylarginine (ADMA) and l - N G -mono-methyl- l -arginine ( l -NMMA) or increased DDAH1 expression and activity in cardiovascular tissues of hDDAH1 transgenic mice. Principal results DDAH activity (DDAH1 + DDAH2) was found to be markedly increased in aortic and heart tissues but unaltered in liver and kidney tissues of hDDAH1 transgenic as compared to wild-type (WT) mice. In WT mice, DDAH activity was m…

MaleGenetically modified mousemedicine.medical_specialtyEndotheliumArginineTransgeneMice TransgenicArginineNitric OxideGene Expression Regulation EnzymologicAmidohydrolasesMicechemistry.chemical_compoundEnosInternal medicinemedicineAnimalsHumansTissue DistributionRNA MessengerPharmacologyKidneybiologyChemistryArteriosclerosismedicine.diseasebiology.organism_classificationIsoenzymesMice Inbred C57BLmedicine.anatomical_structureEndocrinologyOrgan SpecificityFemaleAsymmetric dimethylarginineSignal TransductionPharmacological Research
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Genetic elimination of known pheromones reveals the fundamental chemical bases of mating and isolation in Drosophila

1999

Overexpression of the UAS-tra transgene in Drosophila melanogaster females led to the complete elimination of their cuticular pheromones. According to current models of Drosophila behavior, these flies should induce no courtship. In fact, they are still attractive to conspecific males. Three classes of stimuli are shown to induce courtship, with different effects on male behavior: ( i ) known pheromones produced by control females, ( ii ) stimuli produced by living control and transgenic flies, and ( iii ) as-yet-undetermined pheromones present on both control and transgenic flies. Only the latter class of pheromones are required for mating. They appear to represent a layer of ancestral at…

MaleHot TemperaturePheromones/genetics/*physiologyPheromonesAnimals Genetically ModifiedCourtshipSexual Behavior AnimalAnimal/*physiologyMelanogasterMatingreproductive and urinary physiologymedia_commonGeneticsMultidisciplinarybiologyBiological SciencesDNA-Binding ProteinsDrosophila melanogasterSocial IsolationSex pheromonebehavior and behavior mechanismsDrosophilaFemaleDrosophila melanogasteranimal structuresSaccharomyces cerevisiae ProteinsGenotypeRecombinant Fusion ProteinsRecombinant Fusion Proteins/biosynthesisSexual BehaviorTransgenemedia_common.quotation_subjectGenetically ModifiedCrossesHSP70 Heat-Shock Proteins/biosynthesis/genetics/physiologyFungal ProteinsGeneticSibling speciesAnimalsHSP70 Heat-Shock ProteinsDrosophilaCrosses Geneticfungibiology.organism_classificationHeatTranscription Factors/biosynthesis/geneticsFungal Proteins/biosynthesis/geneticsHydrocarbonsDrosophila melanogaster/genetics/*physiologyEvolutionary biologyDrosophila/genetics/*physiologyTranscription FactorsProceedings of the National Academy of Sciences
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Tenectin is a novel alphaPS2betaPS integrin ligand required for wing morphogenesis and male genital looping in Drosophila.

2010

International audience; Morphogenesis of the adult structures of holometabolous insects is regulated by ecdysteroids and juvenile hormones and involves cell-cell interactions mediated in part by the cell surface integrin receptors and their extracellular matrix (ECM) ligands. These adhesion molecules and their regulation by hormones are not well characterized. We describe the gene structure of a newly described ECM molecule, tenectin, and demonstrate that it is a hormonally regulated ECM protein required for proper morphogenesis of the adult wing and male genitalia. Tenectin's function as a new ligand of the PS2 integrins is demonstrated by both genetic interactions in the fly and by cell s…

MaleMESH: Extracellular Matrix ProteinsMESH: DrosophilaMESH : Immunohistochemistry[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionIntegrinLigandsLooping morphogenesisExtracellular matrixchemistry.chemical_compound0302 clinical medicineMESH: Genitalia MaleMorphogenesisMESH: LigandsDrosophila ProteinsWings AnimalMESH: AnimalsTransgenesIn Situ Hybridization0303 health sciencesExtracellular Matrix ProteinsMESH : Genitalia MaleMESH : LigandsIntegrin alpha ChainsCell adhesion moleculeMESH : In Situ HybridizationImmunohistochemistry3. Good healthCell biologyLarvaMESH : Integrin alpha ChainsAdhesionDrosophilaMESH : MutationMESH : TransgenesTenectinIntegrin alpha ChainsDrosophila ProteinEcdysoneEcdysoneMESH: MutationMESH: Drosophila ProteinsMESH : MaleIntegrinMorphogenesisMESH : WingMESH: TransgenesBiologyGenitalia MaleArticle03 medical and health sciencesMESH : Extracellular Matrix ProteinsMESH: In Situ HybridizationAnimalsMESH : DrosophilaCell adhesionMolecular Biology030304 developmental biologyMESH : LarvaMetamorphosisMESH: Integrin alpha ChainsLeft–right asymmetryMESH: ImmunohistochemistryCell BiologyMESH : Drosophila ProteinsMESH: WingMESH: MaleMESH: MorphogenesischemistryMESH : MorphogenesisMutationbiology.proteinMESH : AnimalsMESH: Larva[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition030217 neurology & neurosurgeryDevelopmental Biology
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Genetic identification of neurons controlling a sexually dimorphic behaviour

2000

0960-9822 (Print) Journal Article Research Support, Non-U.S. Gov't; In the fruit fly Drosophila melanogaster, locomotor activity is sexually dimorphic: female flies constantly modulate their activity pattern whereas males show a steadier, stereotyped walking pace [1]. Here, we mapped the area of the brain controlling this behavioural dimorphism. Adult male Drosophila expressing a dominant feminising transgene in a small cluster of neurons in the pars intercerebralis exhibited a female-like pattern of locomotor activity. Genetic ablation of these neurons prevented the feminisation of the locomotor activity of transgenic males. The results suggest that this cluster of neurons modulates sex-sp…

MaleMESH: NeuronsCourtshipAnimals Genetically ModifiedSexual Behavior Animal0302 clinical medicineMESH: Saccharomyces cerevisiae ProteinsDrosophila ProteinsNervous System Physiological PhenomenaMESH: AnimalsMESH: Sexual Behavior AnimalDrosophila melanogaster/*physiologymedia_commonNeurons0303 health sciencesFungal proteinSex CharacteristicsbiologyAgricultural and Biological Sciences(all)Nuclear ProteinsAnatomyMESH: Transcription FactorsMotor Activity/*physiologyMESH: Motor ActivityDNA-Binding ProteinsFungal Proteins/geneticsNuclear Proteins/*genetics/physiologyDrosophila melanogasterMESH: Fungal Proteins[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]FemaleDrosophila melanogasterGeneral Agricultural and Biological SciencesLocomotionSex characteristicsMESH: Sex CharacteristicsNervous System PhysiologySaccharomyces cerevisiae ProteinsTransgenemedia_common.quotation_subjectRecombinant Fusion ProteinsRecombinant Fusion Proteins/biosynthesisSexual BehaviorMESH: LocomotionTranscription Factors/geneticsGenetically ModifiedMotor ActivityGeneral Biochemistry Genetics and Molecular BiologyMESH: Drosophila melanogasterFungal ProteinsMESH: Animals Genetically Modified03 medical and health sciencesMESH: Recombinant Fusion ProteinsAnimalsDrosophila030304 developmental biologyBiochemistry Genetics and Molecular Biology(all)Animalfungibiology.organism_classificationMESH: MaleSexual dimorphismMale courtship behaviourMESH: Nervous System PhysiologyNeuroscienceMESH: FemaleMESH: Nuclear ProteinsNeurons/*physiology030217 neurology & neurosurgeryTranscription Factors
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Gene Expression Profiling of Facilitated L-LTP in VP16-CREB Mice Reveals that BDNF Is Critical for the Maintenance of LTP and Its Synaptic Capture

2011

Expression of VP16-CREB, a constitutively active form of CREB, in hippocampal neurons of the CA1 region lowers the threshold for eliciting the late, persistent phase of long-term potentiation (L-LTP) in the Schaffer collateral pathway. This VP16-CREB-mediated L-LTP differs from the conventional late phase of LTP in not being dependent on new transcription. This finding suggests that in the transgenic mice the mRNA transcript(s) encoding the protein(s) necessary for this form of L-LTP might already be present in CA1 neurons in the basal condition. We used high-density oligonucleotide arrays to identify the mRNAs differentially expressed in the hippocampus of transgenic and wild-type mice. We…

MalePatch-Clamp TechniquesTime FactorsTransgeneNeuroscience(all)Long-Term PotentiationNerve Tissue ProteinsDynorphinHippocampal formationCREBHippocampusSynaptic TransmissionMiceNeurotrophic factorsMHC class ImedicineAnimalsRNA MessengerIn Situ HybridizationMice KnockoutNeuronsNeuronal PlasticitybiologyReverse Transcriptase Polymerase Chain ReactionBrain-Derived Neurotrophic FactorGene Expression Profilingmusculoskeletal neural and ocular physiologyGeneral NeuroscienceExcitatory Postsynaptic PotentialsHerpes Simplex Virus Protein Vmw65Long-term potentiationExonsCREB-Binding ProteinMolecular biologyCell biologymedicine.anatomical_structurenervous systemSchaffer collateralSynapsesbiology.proteinFemaleNeuron
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