Search results for "trigeminal ganglion"

showing 6 items of 16 documents

Neuropeptides in Primary Afferent Neurons

1990

Traditionally, the primary sensory neurons, having their perikarya in the spinal or cranial sensory ganglia with processes directed towards the periphery and the central nervous system, have been regarded to function as receptive and afferent systems which reflexly activate central effector systems.1 This, however, does not apply to the small diameter primary afferents as suggested by the observation made about a century ago that antidromic stimulation of transected dorsal roots or sensory nerves caused vasodilatation and inflammatory signs in the skin.2,3 The novel concept which has been confirmed by many investigators ascribes to small diameter (particularly unmyelinated C) primary sensor…

Trigeminal ganglionmedicine.anatomical_structurenervous systemDorsal root ganglionCentral nervous systemmedicineNeuropeptideSensory systemStimulationBiologyNeuroscienceSensory neuronAntidromic
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Lesions of single nerves

2017

The trigeminal nerve exits the brain in its anatomical course in the area of the pons and proceeds to the anterior edge of the petrous bone. There, the nerve forms the trigeminal ganglion – also known as the Gasserian ganglion – which, sheathed by a dural sleeve, gives off three branches (V1–3).

Trigeminal nervebusiness.industryAnatomyFacial nerveMedian nervePonsGanglionTrigeminal ganglionmedicine.anatomical_structureMedicinesense organsSciatic nervebusinessBrachial plexus
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Trigeminal innervation of the mammalian pineal gland.

1999

There is evidence that the trigeminal (Gasserian) ganglia innervate the mamma- lian pineal gland and serve in its regulation in addition to the sympathetic and cholinergic as well as further influences. By means of immunohistochemical methods, previous studies demonstrated fibers containing calcitonin gene-related peptide (CGRP-LI) or substance P (SP) in the superficial pineal of various mammalian species. In addition, SP and the related tachykinin, neurokinin A, were detected by radioimmunoassay and HPLC, respectively, in the rat and human pineal gland. In the present study, retrograde neuronal tracing upon injection of a tracer substance into the superfical pineal gland of rats was used i…

endocrine systemmedicine.medical_specialtyHistologyCalcitonin Gene-Related PeptideNeuropeptideSubstance PBiologyCalcitonin gene-related peptideSubstance PPineal Glandchemistry.chemical_compoundPineal glandTrigeminal ganglionInternal medicinemedicineAnimalsHumansInstrumentationRatsNeuronal tracingMedical Laboratory TechnologyEndocrinologymedicine.anatomical_structurenervous systemchemistryTrigeminal GanglionCholinergicNeurokinin AAnatomyhormones hormone substitutes and hormone antagonistsMicroscopy research and technique
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Catestatin-like immunoreactivity in the rat eye

2014

The aim of the study was to investigate the presence and distribution of the chromogranin A-derived peptide catestatin in the rat eye and trigeminal ganglion by immunofluorescence using an antibody which recognizes not only free catestatin but also larger fragments containing the sequence of catestatin. Western blots were performed in an attempt to characterize the immunoreactivities detected by the catestatin antiserum. Sparse immunoreactive nerve fibers were visualized in the corneal stroma, in the chamber angle, in the sphincter muscle but also in association with the dilator muscle, in the stroma of the ciliary body and processes, but dense in the irideal stroma, around blood vessels at…

medicine.medical_specialtyPathologygenetic structuresFluorescent Antibody TechniqueNeuropeptideSubstance PBiologyEyeCellular and Molecular NeuroscienceTrigeminal ganglionEndocrinologyCiliary bodyInternal medicinemedicineAnimalsRetinaEndocrine and Autonomic SystemsColocalizationChromogranin AGeneral MedicinePeptide Fragmentseye diseasesRatsGanglionEndocrinologymedicine.anatomical_structureTrigeminal GanglionNeurologyRats Inbred Lewbiology.proteinChromogranin Asense organsChoroidNeuropeptides
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A Novel Pathophysiological Mechanism Contributing to Trigeminal Neuralgia

2016

Trigeminal neuralgia (TN) is a form of neuropathic pain that affects the fifth cranial nerve, the most widely distributed nerve in the head. Although TN has been associated with a variety of pathological conditions, neurovascular compression on the trigeminal nerve, as it exits the brain stem, is the most frequent reported cause. This compression provides a progressive demyelination of the nerve and a subsequent aberrant neural transmission. Although several studies have clarified some physiopathological mechanisms underlying TN, the molecular basis remains vague. Very recently the substitution of methionine 136 by valine (MET126Val) in sodium channel Nav1.6 in a case study of typical TN ha…

trigeminal ganglionlcsh:Biochemistry03 medical and health sciencesTrigeminal ganglion0302 clinical medicineaction potentialTrigeminal neuralgianeurovascualr compressionGeneticsmedicinelcsh:QD415-436Molecular BiologyPathologicalTrigeminal neuralgia; action potential; molecular mechanism; neurovascualr compression; trigeminal ganglionGenetics (clinical)Trigeminal nervebusiness.industrySodium channellcsh:RM1-950medicine.diseasePathophysiologylcsh:Therapeutics. Pharmacology030220 oncology & carcinogenesisAnesthesiaNeuropathic painMolecular MedicineBrainstemmolecular mechanismbusinessNeuroscience030217 neurology & neurosurgeryTrigeminal neuralgiaResearch Article
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Enhancement by TNF-alpha of reactivation and replication of latent herpes simplex virus from trigeminal ganglia of mice.

1995

The influence of tumor-necrosis-factor-alpha (TNF-alpha), granulocyte-macrophage colony-stimulating factor (GM-CSF), interleukine-1 (IL-1) and IL-3 on the in vitro reactivation frequency and replication rate of trigeminal ganglia of mice latently infected with herpes simplex virus (HSV) strain KOS was studied. It could be demonstrated that TNF-alpha and possibility GM-CSF, but not IL-1 and IL-3, enhanced the reactivation frequency and replication of HSV. Interferon alpha/beta (IFN alpha/beta) prevented reactivation and replication.

virusesmedicine.medical_treatmentHerpesvirus 1 HumanBiologymedicine.disease_causeVirus ReplicationVirusHerpesviridaeMiceInterferonVirologyAlphaherpesvirinaeChlorocebus aethiopsmedicineAnimalsHumansVero CellsMice Inbred BALB CTumor Necrosis Factor-alphaGranulocyte-Macrophage Colony-Stimulating FactorInterferon-alphaGeneral MedicineInterferon-betabiology.organism_classificationVirologyIn vitroVirus LatencyCytokineHerpes simplex virusViral replicationTrigeminal GanglionInterleukin-3Virus Activationmedicine.drugInterleukin-1Archives of virology
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