Search results for "tumor necrosis factor alpha"

showing 10 items of 479 documents

Ovarian stimulation does not influence the uterine immune environment in healthy infertile women.

2019

Abstract Research question There is some controversy regarding the impact of ovarian stimulation on immune cells in women undergoing IVF. The study's aim was to determine whether ovarian stimulation affected immune uterine cells in healthy women undergoing IVF. Design This prospective cohort study included 28 patients undergoing IVF and 47 healthy oocyte donors. Endometrial biopsies were taken in a natural cycle and after ovarian stimulation. All participants had a normal karyotype, pelvic ultrasound and cervical cytology results and thyroid-stimulating hormone concentration, as well as normal glucose and insulin concentrations and inherited and acquired thrombophilia test results. Screenin…

AdultNecrosisendocrine system diseasesPopulationStimulationEndometrial biopsies Immune cell populations Natural cycles Ovarian stimulation Peripheral blood natural killer cells Uterine natural killer cellsHuman leukocyte antigenAndrologyImmune systemOvulation InductionmedicineHumansEmbryo ImplantationProspective Studieseducationreproductive and urinary physiologyMenstrual Cycleeducation.field_of_studybusiness.industryUterusObstetrics and GynecologyFOXP3biochemical phenomena metabolism and nutritionfemale genital diseases and pregnancy complicationsKiller Cells NaturalReproductive MedicinebacteriaTumor necrosis factor alphaFemalemedicine.symptombusinessInfertility FemaleDevelopmental BiologyHormoneReproductive biomedicine online
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NO Reduces PMN Adhesion to Human Vascular Endothelial Cells Due to Downregulation of ICAM-1 mRNA and Surface Expression

2000

Reperfusion damage is largely due to the adherence of polymorphonuclear leukocytes to the endothelium initiated by adhesion molecule upregulation. The reduced endothelial nitric oxide release during ischemia may be involved in the upregulation of intercellular adhesion molecule 1. In this study, we tested if nitric oxide donors suppress polymorphonuclear leukocyte adherence to activated endothelial cells by inhibition of the intercellular adhesion molecule 1 surface expression. Confluent human umbilical vein endothelial cells were stimulated with tumor necrosis factor alpha (300 U/mL) after preincubation with increasing concentrations of the nitric oxide donors CAS 1609 (0.005-5 mM/L) and 3…

AdultUmbilical VeinsEndotheliumNeutrophilsIntercellular Adhesion Molecule-1Cell Culture TechniquesDown-RegulationNitric Oxide Synthase Type IINitric OxideTransfectionUmbilical veinNitric oxidechemistry.chemical_compoundmedicineCell AdhesionHumansSaphenous VeinRNA MessengerICAM-1biologyTumor Necrosis Factor-alphaMembrane ProteinsHematologyIntercellular Adhesion Molecule-1Molecular biologyEndothelial stem cellNitric oxide synthasemedicine.anatomical_structurechemistryBiochemistryGene Expression Regulationbiology.proteinTumor necrosis factor alphaEndothelium VascularNitric Oxide Synthase
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C-reactive protein and intercellular adhesion molecule-1 are stronger predictors of oxidant stress than blood pressure in established hypertension

2007

Oxidant stress is implicated in the pathogenesis of atherosclerosis in cardiovascular diseases. Our aim was to test oxidative stress, as 8-iso-prostaglandin F2alpha (8-iso-PGF2alpha), and its relationship with inflammation markers C-reactive protein (CRP) and tumour necrosis factor-alpha (TNFalpha), and endothelial activation assayed as soluble intercellular adhesion molecule (ICAM)-1 and vascular cell adhesion molecule (VCAM)-1 in essential hypertension.In 216 essential hypertensive patients and 55 healthy control individuals, plasma levels of high-sensitivity CRP and TNFalpha, 8-iso-PGF2alpha, ICAM-1 and VCAM-1 were measured in basal conditions. Moreover, basal and 24-h ambulatory blood p…

Adultmedicine.medical_specialtyPhysiologyIntercellular Adhesion Molecule-1Vascular Cell Adhesion Molecule-1InflammationBlood Pressuremedicine.disease_causeDinoprostC-reactive protein oxidant stress hypertensionPathogenesisPredictive Value of TestsInternal medicineInternal MedicinemedicineHumansEndothelial dysfunctionInflammationbiologybusiness.industryTumor Necrosis Factor-alphaC-reactive proteinBlood Pressure Monitoring AmbulatoryMiddle Agedmedicine.diseaseIntercellular Adhesion Molecule-1Oxidative StressBlood pressureEndocrinologyC-Reactive ProteinCase-Control StudiesImmunologyHypertensionbiology.proteinTumor necrosis factor alphamedicine.symptomCardiology and Cardiovascular MedicinebusinessOxidative stressBiomarkers
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The use of computer-assisted video image analysis for the quantification of CD8+ T lymphocytes producing tumor necrosis factor alpha spots in respons…

1997

Enzyme-linked immunospot (ELISPOT) analysis is a sensitive technique for the detection and quantification of single T lymphocytes forming cytokine spots after antigen contact in vitro. Herein computer-assisted video image analysis (CVIA) was applied to automatically determine the number and size of tumor necrosis factor alpha (TNF-alpha) spots formed by single blood-derived CD8+ T cells after contact with peptide-loaded target cells. With CVIA and TNF-alpha ELISPOT analysis we quantified CD8+ T cells responsive to HLA-A2.1-binding tyrosinase and influenza matrix peptides in healthy donors. We followed the course of the virus-specific T cell response in two HLA-A2-positive patients with reac…

Adultmedicine.medical_treatmentT cellImmunologyCytomegalovirusEnzyme-Linked Immunosorbent AssayBiologyCD8-Positive T-LymphocytesCell LineAntigenViral Envelope ProteinsHLA-A2 AntigenmedicineImage Processing Computer-AssistedImmunology and AllergyHumansLymphocyte CountMicroscopy VideoTumor Necrosis Factor-alphaELISPOTMiddle AgedMolecular biologyIn vitroCytokinemedicine.anatomical_structureCell cultureImmunologyCytomegalovirus InfectionsTumor necrosis factor alphaPeptidesCD8Journal of immunological methods
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Inflammation, genes and zinc in ageing and age-related diseases.

2006

Lifelong antigenic burden determines a condition of chronic inflammation, with increased lymphocyte activation and pro-inflammatory cytokine production. A large number of studies have documented changes in Zn metabolism in experimental animal models of acute and chronic inflammation and in human chronic inflammatory diseases. In particular, modification of zinc plasma concentration as well as intracellular disturbance of antioxidant intracellular pathways have been found associated to age-related inflammatory diseases, like atherosclerosis. Zinc deficiency is extremely diffused in aged people that are educated to avoid meat and other high Zn-content foods due to fear of cholesterol. Rather,…

Agingmedicine.medical_treatmentLongevityGene ExpressionInflammationBiologychemistry.chemical_compoundmedicinecytokine interleukin 6 metallothionein tumor necrosis factor alpha zincAnimalsHumansGeneTranscription factorCellular SenescenceInflammationPolymorphism GeneticCholesterolInterleukin-6Tumor Necrosis Factor-alphamedicine.diseaseAtherosclerosisImmunity InnateZincCytokinechemistryAgeingImmunologyZinc deficiencyCytokinesMetallothioneinGeriatrics and Gerontologymedicine.symptomGerontologyIntracellular
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Cancer relapse under chemotherapy: why TLR2/4 receptor agonists can help.

2007

Liver or lung metastases usually relapse under chemotherapy. Such life-threatening condition urgently needs new, systemic anticancer compounds, with original and efficient mechanisms of action. In B16 melanoma mice treated with cyclophosphamide, D'Agostini et al. [D'Agostini, C., Pica, F., Febbraro, G., Grelli, S., Chiavaroli, C., Garaci, E., 2005. Antitumour effect of OM-174 and Cyclophosphamide on murine B16 melanoma in different experimental conditions. Int. Immunopharmacol. 5, 1205-1212.] recently found that OM-174, a chemically defined Toll-like receptor(TLR)2/4 agonist, reduces tumor progression and prolongs survival. Here we review 149 articles concerning molecular mechanisms of TLR2…

AgonistLipopolysaccharidesCyclophosphamidemedicine.drug_classmedicine.medical_treatmentNitric Oxide Synthase Type IIAntineoplastic AgentsApoptosisNitric oxidechemistry.chemical_compoundRecurrenceNeoplasmsMedicineAnimalsHumansPharmacologyChemotherapybusiness.industryTumor Necrosis Factor-alphaCancerDendritic Cellsmedicine.diseaseNeoadjuvant TherapyToll-Like Receptor 2Interleukin-10Toll-Like Receptor 4TLR2Lipid ATreatment OutcomechemistryTumor progressionChemotherapy AdjuvantDrug Resistance NeoplasmEnzyme InductionImmunologyCancer researchBCG VaccineTumor necrosis factor alphaImmunotherapybusinessmedicine.drugSignal TransductionT-Lymphocytes CytotoxicEuropean journal of pharmacology
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Selection and characterization of a novel agonistic human recombinant anti-Trail-R2 minibody with anti-leukemic activity

2009

Tumor necrosis factor-related apoptosis-inducing ligand (TRAIL) is a promising natural anticancer therapeutic agent because through its “death receptors”, TRAIL-R1 and TRAIL-R2, it induces apoptosis in many transformed tumor cells, but not in the majority of normal cells. Hence, agonistic compounds directed against TRAIL death receptors have the potential of being excellent cancer therapeutic agents, with minimal cytotoxicity in normal tissues. Here, we report the selection and characterization of a new single-chain fragment variable (scFv) to TRAIL-R2 receptor isolated from a human phage-display library, produced as minibody (MB), and characterized for the in vitro anti-leukemic tumoricid…

Agonistmedicine.drug_classTRAIL; TRAIL-R2; minibody; anticancer therapyImmunologylymphoma; therapy; recombinant antibodyTRAILApoptosislymphomaCHO CellsCricetulusPeptide LibraryTRAIL-R2CricetinaeImmunoglobulin FragmentmedicineAnimalsHumansImmunology and Allergyrecombinant antibodyanticancer therapyReceptorCytotoxicityImmunoglobulin FragmentsPharmacologytherapyLeukemiaChemistryAnimalChinese hamster ovary cellAntibody-Dependent Cell CytotoxicityminibodyApoptosiIn vitroRecombinant ProteinsReceptors TNF-Related Apoptosis-Inducing LigandCHO CellCell cultureApoptosisImmunologyCancer researchTumor necrosis factor alphaCricetuluHuman
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Mast cells in allergic asthma and beyond.

2010

Mast cells have been regarded for a long time as effector cells in IgE mediated type I reactions and in host defence against parasites. However, they are resident in all environmental exposed tissues and express a wide variety of receptors, suggesting that these cells can also function as sentinels in innate immune responses. Indeed, studies have demonstrated an important role of mast cells during the induction of life-saving antibacterial responses. Furthermore, recent findings have shown that mast cells promote and modulate the development of adaptive immune responses, making them an important hinge of innate and acquired immunity. In addition, mast cells and several mast cell-produced me…

AllergyLeukotrienesmast cellsReview ArticleImmunoglobulin EModels BiologicalClassical complement pathwaychemistry.chemical_compoundMiceImmune systemAnti-Infective AgentsThymic Stromal LymphopoietinmedicineHypersensitivityAnimalsHumansmediatorsInnate immune systembiologyTumor Necrosis Factor-alphaGeneral MedicineImmunoglobulin Emedicine.diseaseAcquired immune systemallergyAsthmachemistryImmune SystemImmunologybiology.proteinProstaglandinsCytokinesTumor necrosis factor alphaHistamineHistamineYonsei medical journal
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T cell killing by tolerogenic dendritic cells protects mice from allergy.

2011

It is well established that allergy development can be prevented by repeated low-dose exposure to contact allergens. Exactly which immune mechanisms are responsible for this so-called low zone tolerance (LZT) is not clear, although CD8⁺ suppressor T cells are known to have a role. Here, we show that TNF released by tolerogenic CD11⁺CD8⁺ DCs located in skin-draining lymph nodes is required and sufficient for development of tolerance to contact allergens in mice. DC-derived TNF protected mice from contact allergy by inducing apoptosis in allergen-specific effector CD8⁺ T cells via TNF receptor 2 but did not contribute to the generation and function of the regulatory T cells associated with LZ…

AllergyT cellApoptosisBiologyCD8-Positive T-LymphocytesDermatitis Contactlaw.inventionImmune toleranceMicelawmedicineHypersensitivityImmune ToleranceAnimalsReceptors Tumor Necrosis Factor Type IIReceptorMice KnockoutEffectorTumor Necrosis Factor-alphaGeneral MedicineDendritic CellsAllergensmedicine.diseaseMice Inbred C57BLmedicine.anatomical_structureApoptosisReceptors Tumor Necrosis Factor Type IImmunologySuppressorTumor necrosis factor alphaThe Journal of clinical investigation
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4-dimethylamino-3′,4′-dimethoxychalcone downregulates iNOS expression and exerts anti-inflammatory effects

2001

Abstract Reactive oxygen and nitrogen species contribute to the pathophysiology of inflammatory conditions. We have studied the effects of a novel superoxide scavenger, 4-dimethylamino-3′,4′-dimethoxychalcone (CH11) in macrophages and in vivo. CH11 has been shown to inhibit the chemiluminescence induced by zymosan in mouse peritoneal macrophages and the cytotoxic effects of superoxide. In the same cells, the modulation by superoxide of nitric oxide (NO) production in response to zymosan was investigated. CH11 was more effective than the membrane-permeable scavenger Tiron for inhibition of inducible nitric oxide synthase (iNOS) protein expression and nitrite production. We have shown that CH…

Anti-Inflammatory AgentsNitric Oxide Synthase Type IIPharmacologyCarrageenanNitric OxideBiochemistryGene Expression Regulation EnzymologicNitric oxideMicechemistry.chemical_compoundChalconeChalconesSuperoxidesIn vivoPhysiology (medical)AnimalsEdemaEnzyme InhibitorsRespiratory BurstInflammationTironbiologySuperoxideZymosanZymosanFree Radical ScavengersNitric oxide synthaseOxidative StresschemistryBiochemistryEicosanoidLuminescent Measurements12-Dihydroxybenzene-35-Disulfonic Acid Disodium SaltMacrophages Peritonealbiology.proteinFemaleTumor necrosis factor alphaNitric Oxide SynthaseFree Radical Biology and Medicine
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