Search results for "type II"

showing 10 items of 607 documents

Twelve Variants Polygenic Score for Low-Density Lipoprotein Cholesterol Distribution in a Large Cohort of Patients With Clinically Diagnosed Familial…

2022

: Background A significant proportion of individuals clinically diagnosed with familial hypercholesterolemia (FH), but without any disease-causing mutation, are likely to have polygenic hypercholesterolemia. We evaluated the distribution of a polygenic risk score, consisting of 12 low-density lipoprotein cholesterol (LDL-C)-raising variants (polygenic LDL-C risk score), in subjects with a clinical diagnosis of FH. Methods and Results Within the Lipid Transport Disorders Italian Genetic Network (LIPIGEN) study, 875 patients who were FH-mutation positive (women, 54.75%; mean age, 42.47±15.00 years) and 644 patients who were FH-mutation negative (women, 54.21%; mean age, 49.73±13.54 years) wer…

MaleAdultMultifactorial InheritanceSettore MED/09 - Medicina Internafamilial hypercholesterolemia; molecular diagnosis; polygenic risk score; Adult; Cholesterol LDL; Female; Humans; Middle Aged; Multifactorial Inheritance; Mutation; Gene Regulatory Networks; Hyperlipoproteinemia Type IIfamilial hypercholesterolemiaCholesterol LDLMiddle AgedLDLHyperlipoproteinemia Type IICholesterolmolecular diagnosispolygenic risk scoreMutationHumansFemaleGene Regulatory Networksmolecular diagnosifamilial hypercholesterolemia; molecular diagnosis; polygenic risk score
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Glutathione Peroxidase-1 Deficiency Potentiates Dysregulatory Modifications of Endothelial Nitric Oxide Synthase and Vascular Dysfunction in Aging

2014

Recently, we demonstrated that gene ablation of mitochondrial manganese superoxide dismutase and aldehyde dehydrogenase-2 markedly contributed to age-related vascular dysfunction and mitochondrial oxidative stress. The present study has sought to investigate the extent of vascular dysfunction and oxidant formation in glutathione peroxidase-1–deficient ( GPx-1 −/− ) mice during the aging process with special emphasis on dysregulation (uncoupling) of the endothelial NO synthase. GPx-1 −/− mice on a C57 black 6 (C57BL/6) background at 2, 6, and 12 months of age were used. Vascular function was significantly impaired in 12-month-old GPx-1 −/− -mice as compared with age-matched controls. Oxidan…

MaleAgingmedicine.medical_specialtyGPX1Nitric Oxide Synthase Type IIIBiologymedicine.disease_causeMicechemistry.chemical_compoundGlutathione Peroxidase GPX1Internal medicineLeukocytesInternal MedicinemedicineAnimalsHumansPhosphorylationEndothelial dysfunctionProtein kinase ACells CulturedAgedMice Knockoutchemistry.chemical_classificationGlutathione PeroxidaseGlutathione peroxidaseEndothelial CellsNitric Oxide Synthase Type IIIGlutathioneOxidantsmedicine.diseaseMice Inbred C57BLOxidative StressEndocrinologychemistryImmunologyPhosphorylationEndothelium VascularOxidative stressHypertension
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Dietary soy isoflavone induced increases in antioxidant and eNOS gene expression lead to improved endothelial function and reduced blood pressure in …

2005

Epidemiological evidence suggests that populations consuming large amounts of soy protein have a reduced incidence of coronary heart disease (1-5). The cardiovascular risks associated with conventional hormone replacement therapy in postmenopausal women (5-7) have precipitated a search for alternative estrogen receptor modulators. Here we report that long-term feeding of rats with a soy protein-rich (SP) diet during gestation and adult life results in decreased oxidative stress, improved endothelial function, and reduced blood pressure in vivo measured by radiotelemetry in aged male offspring. Improved vascular reactivity in animals fed an SP diet was paralleled by increased mitochondrial g…

MaleAntioxidantTime Factorsmedicine.medical_treatmentBlood PressureCoronary Disease030204 cardiovascular system & hematologymedicine.disease_causeBiochemistryAntioxidantschemistry.chemical_compound0302 clinical medicineEnosMalondialdehydeSoy proteinAorta2. Zero hungerRegulation of gene expression0303 health sciencesReverse Transcriptase Polymerase Chain ReactionGenistein3. Good healthmedicine.anatomical_structureLiverFemaleBiotechnologymedicine.medical_specialtyEndotheliumNitric Oxide Synthase Type IIIPhytoestrogensBiologyModels BiologicalGene Expression Regulation Enzymologic03 medical and health sciencesInternal medicineGeneticsmedicineAnimalsRNA MessengerRats WistarMolecular Biology030304 developmental biologybiology.organism_classificationAnimal FeedIsoflavonesRatsOxidative StressBlood pressureEndocrinologychemistryModels ChemicalPhytoestrogensEndothelium VascularSoybeansOxidative stressFASEB journal : official publication of the Federation of American Societies for Experimental Biology
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Conditioned Media from Adipose-Tissue-Derived Mesenchymal Stem Cells Downregulate Degradative Mediators Induced by Interleukin-1β in Osteoarthritic C…

2013

Osteoarthritis (OA) is the most frequent joint disorder and an important cause of disability. Recent studies have shown the potential of adipose-tissue-derived mesenchymal stem cells (AD-MSC) for cartilage repair. We have investigated whether conditioned medium from AD-MSC (CM) may regulate in OA chondrocytes a number of key mediators involved in cartilage degeneration. CM enhanced type II collagen expression in OA chondrocytes while decreasing matrix metalloproteinase (MMP) activity in cell supernatants as well as the levels of MMP-3 and MMP-13 proteins and mRNA in OA chondrocytes stimulated with interleukin- (IL-) 1β. In addition, CM increased IL-10 levels and counteracted the stimulating…

MaleArticle Subjectmedicine.medical_treatmentImmunologyInterleukin-1betaType II collagenAdipose tissueDown-RegulationNitric OxideChondrocytesMatrix Metalloproteinase 13Osteoarthritislcsh:PathologymedicineHumansProstaglandin E2Interleukin 6Collagen Type IICells CulturedAgedbiologyChemistryInterleukin-6Tumor Necrosis Factor-alphaMesenchymal stem cellNF-kappa BInterleukinMesenchymal Stem CellsCell BiologyMiddle AgedCell biologyAdipose TissueCulture Media ConditionedImmunologybiology.proteinTumor necrosis factor alphaFemaleMatrix Metalloproteinase 3Inflammation Mediatorslcsh:RB1-214Prostaglandin Emedicine.drugResearch Article
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Right ventricular collagen type III and IV gene expression increases during early phases of endurance training in hypobaric hypoxic condition

1997

The objective of this study was to examine the effects of prolonged exposure to hypobaric hypoxic condition, physical training and their combination on collagen type I, III and IV gene expression in the ventricles and atria of rat heart. Male rats were assigned to four groups: normobaric sedentary (NS) and trained (NT), and hypobaric sedentary (HS) and trained (HT). Exposure to and treadmill running training in hypobaric condition were carried out in a hypobaric chamber (770–740 mbar, 2250–2550 m). Experimental periods were 10, 21 and 56 days; the groups of 91 days served as recovery groups from experimental settings of 56 days. Exposure to hypobaric condition as such and in combination wit…

MaleAtmosphere Exposure Chambersmedicine.medical_specialtyPhysiologyHeart VentriclesProcollagen-Proline DioxygenaseGene ExpressionCardiomegalyBiologyLeft ventricular hypertrophyCollagen Type IIIEndurance trainingRight ventricular hypertrophyPhysical Conditioning AnimalPhysiology (medical)Internal medicineGene expressionmedicineAnimalsHeart AtriaRNA MessengerRats WistarHypoxiaCardiac muscleBlotting Northernmedicine.diseaseRatsHydroxyprolineAtmospheric PressureEndocrinologymedicine.anatomical_structureMrna levelHypobaric chamberPhysical EnduranceCollagenCardiology and Cardiovascular MedicineBasic Research in Cardiology
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iNOS-derived nitric oxide mediates the increase in TFF2 expression associated with gastric damage: role of HIF-1.

2009

Trefoil (TFF) peptides are involved in gastrointestinal mucosal restitution. An hypoxia inducible factor 1 (HIF-1)-dependent induction of TFF genes has been reported in gastric epithelial cells. Nitric oxide (NO) is associated with mucosal damage and modulates HIF-1 activity. The aim of the present study was to analyze the role of iNOS-derived NO in HIF-1alpha stabilization and TFF gene expression in damaged gastric mucosa. Aspirin caused gastric injury that peaked 6 h after dosing and returned to normality at 24 h. iNOS mRNA expression occurs in the corpus in parallel with damage. Blockade of iNOS activity did not modify gastric lesions induced by aspirin but delayed mucosal healing. Aspir…

MaleBenzylaminesAmidinesNitric Oxide Synthase Type IINitric OxideBiochemistryNitric oxideCell LineRats Sprague-Dawleychemistry.chemical_compoundMiceDownregulation and upregulationGene expressionGeneticsGastric mucosamedicineGene silencingAnimalsHumansRNA MessengerEnzyme InhibitorseducationMolecular BiologyDNA Primerseducation.field_of_studyWound HealingAspirinBase SequenceAnti-Inflammatory Agents Non-SteroidalTrefoil factor 2Macrophage ActivationHypoxia-Inducible Factor 1 alpha SubunitCoculture TechniquesRatsUp-RegulationMicroRNAsmedicine.anatomical_structurechemistryCell cultureGastric MucosaCancer researchTrefoil Factor-2Wound healingPeptidesBiotechnologyFASEB journal : official publication of the Federation of American Societies for Experimental Biology
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Inhibition of iNOS activity by 1400W decreases glutamate release and ameliorates stroke outcome after experimental ischemia

2005

Background and purpose. It has been shown that the reversed operation of glutamate transporters when ATP levels fall accounts for most glutamate release induced by severe cerebral ischemia. Nitric oxide (NO) is formed after ischemia and causes ATP depletion. Our purpose is to test if NO release from inducible NO synthase (iNOS) after stroke may cause a delayed glutamate release due to ATP depletion that might underlie progression of the ischemic infarct. We have studied the effect of the highly selective inhibitor of iNOS activity 1400W on brain ATP levels, extracellular glutamate, and stroke outcome after transient focal cerebral ischemia in rats. Methods. To induce focal ischemia, the mid…

MaleBenzylaminesAmino Acid Transport System X-AGIschemiaAmidinesInfarctionDown-RegulationGlutamic AcidNitric Oxide Synthase Type IIL-argininePharmacologyNeuroprotectionNitric oxidelcsh:RC321-571chemistry.chemical_compoundAdenosine TriphosphateWestern blotmedicine.arteryStroke outcomeMedicineAnimalscardiovascular diseasesEnzyme InhibitorsRats Wistarlcsh:Neurosciences. Biological psychiatry. Neuropsychiatrymedicine.diagnostic_testbusiness.industryGlutamate receptorBrainInfarction Middle Cerebral ArteryNitric oxideCerebral Infarctionmedicine.diseaseNeuroprotectionRatsATPStrokeDisease Models AnimalNeuroprotective AgentsTreatment OutcomeNeurologychemistryCytoprotectionIschemic Attack TransientAnesthesiaMiddle cerebral arteryNitric Oxide SynthaseGlutamatebusinessNeurobiology of Disease
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Effect of autologous blood transfusion on cerebral cytokine expression.

2011

BACKGROUND: Autologous blood transfusion (ABT), for example, by means of cell saver equipment, is used to reduce the need for allogenic blood transfusion in patients with high perioperative blood loss. This study investigated the effect of blood/extracorporal surface interaction during withdrawal and retransfusion of shed autologous blood on cerebral inflammation in rats. Rats subjected to hypotension with cerebral ischemia served as positive controls. METHODS: Eighty-eight male Sprague-Dawley rats were anesthetized with sevoflurane, instrumented, and randomly assigned to the following groups: sham-operation (SHAM), autologous blood withdrawal/transfusion only (ABT), or bilateral carotid ar…

MaleBlood transfusionmedicine.medical_treatmentInterleukin-1betaIschemiaNitric Oxide Synthase Type IIInflammationPharmacologySevofluraneRats Sprague-DawleyBlood Transfusion AutologousmedicineHippocampus (mythology)AnimalsInterleukin 6biologybusiness.industryInterleukin-6Reverse Transcriptase Polymerase Chain ReactionTumor Necrosis Factor-alphaBrainmedicine.diseaseRatsNitric oxide synthaseAnesthesiology and Pain MedicineCyclooxygenase 2biology.proteinCytokinesSurgeryTumor necrosis factor alphaNeurology (clinical)medicine.symptombusinessmedicine.drugFollow-Up StudiesJournal of neurosurgical anesthesiology
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Phytochemical and pharmacological studies on the acetonic extract of Marrubium globosum ssp. libanoticum

2006

A new natural labdane diterpene, marrulibanoside, was isolated from the acetonic extract of aerial parts of M. globosum Montbr. et Auch. ex Benth. ssp. libanoticum Boiss. (Lamiaceae). Its structure was determined by spectroscopic methods such as 1D and 2D NMR and mass spectrometry. Pharmacological studies have shown that the extract of M. globosum exerts anti-inflammatory effects in the rat paw oedema induced by carrageenin resulting in reduced paw swelling. This activity, which seems due to marrulibanoside, is a consequence of iNOS and COX-2 activities inhibition.

MaleBlotting WesternPharmaceutical ScienceNitric Oxide Synthase Type IIPharmacognosyLABDANE DITERPENOIDSCarrageenanDinoprostoneAnalytical Chemistrylaw.inventionLabdanechemistry.chemical_compoundMicelawDrug DiscoveryBotanyVULGAREAnimalsEdemaRats WistarPharmacologybiologyTraditional medicinePlant ExtractsOrganic ChemistryAnti-Inflammatory Agents Non-SteroidalCYLLENEUMPlant Components Aerialbiology.organism_classificationCarrageenanRatsComplementary and alternative medicinechemistryPhytochemicalMacrophages PeritonealMolecular MedicineLamiaceaeVELUTINUMDiterpenePhytotherapyMarrubiumMarrubiumPhytotherapy
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Role of endothelial nitric oxide in pulmonary and systemic arteries during hypoxia

2014

Abstract Our aim was to investigate the role played by endothelial nitric oxide (NO) during acute vascular response to hypoxia, as a modulator of both vascular tone (through guanylate cyclase (sGC) activation) and mitochondrial O2 consumption (through competitive inhibition of cytochrome-c-oxydase (CcO)). Organ bath experiments were performed and O2 consumption (Clark electrode) was determined in isolated aorta, mesenteric and pulmonary arteries of rats and eNOS-knockout mice. All pre-contracted vessels exhibited a triphasic hypoxic response consisting of an initial transient contraction (not observed in vessels from eNOS-knockout mice) followed by relaxation and subsequent sustained contra…

MaleCancer ResearchContraction (grammar)Nitric Oxide Synthase Type IIIEndotheliumPhysiologyClinical BiochemistryVasodilationPulmonary ArteryMitochondrionPharmacologyNitric OxideBiochemistryNitric oxideRats Sprague-DawleyMicechemistry.chemical_compoundNon-competitive inhibitionEnosmedicineAnimalsHypoxiaAortaMice KnockoutbiologyMyxothiazolEndothelial Cellsbiology.organism_classificationMesenteric ArteriesRatsMice Inbred C57BLmedicine.anatomical_structurechemistryAnesthesiacardiovascular systemNitric Oxide
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