Search results for "type II"

showing 10 items of 607 documents

Determinants of intracellular RNA pharmacokinetics: Implications for RNA-based immunotherapeutics

2011

RNAs with optimized properties are increasingly investigated as a tool to deliver the genetic information of complete antigens into professional antigen-presenting dendritic cells for HLA haplotype-independent antigen-specific vaccination against cancer. As the dose of the antigen and duration of its presentation are critical factors for generating strong and sustained antigen-specific immune responses, improvement of the immunobioavailability of RNA-based vaccines has been a recurrent subject of research. Substantial increase of the amount of antigen produced from RNA can be achieved by optimizing RNA stability and translational efficiency. Both features are determined by cis-acting elemen…

RNA CapsRNA StabilityPolyadenylationTranslational efficiencyRNA Stabilitymedicine.medical_treatmentHuman leukocyte antigenComputational biologyBiologyPolyadenylationCancer VaccinesPoly(A)-Binding ProteinsAntigenNeoplasmsmedicineHumansDeoxyribonucleases Type II Site-Specific3' Untranslated RegionsMolecular BiologyAntigen PresentationThree prime untranslated regionRNADendritic CellsCell BiologyImmunotherapyVirologyRNAImmunotherapyPoly ARNA Biology
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The function of the soluble interleukin 6 (IL-6) receptor in vivo: sensitization of human soluble IL-6 receptor transgenic mice towards IL-6 and prol…

1996

Interleukin 6 (IL-6) is considered an important mediator of acute inflammatory responses. Moreover, IL-6 functions as a differentiation and growth factor of hematopoietic precursor cells, B cells, T cells, keratinocytes, neuronal cells, osteoclasts, and endothelial cells. IL-6 exhibits its action via a receptor complex consisting of a specific IL-6 receptor (IL-6R) and a signal transducing subunit (gp130). Soluble forms of both receptor components are generated by shedding and are found in patients with various diseases such as acquired immune deficiency syndrome, rheumatoid arthritis, and others. The function of the soluble (s)IL-6R in vivo is unknown. Since human (h)IL-6 acts on human and…

Receptor complexImmunologyMice TransgenicInterleukin 1 receptor type IIBiologyMiceSpecies SpecificityAntigens CDInterleukin-4 receptorImmunology and AllergyAnimalsHumansAcute-Phase ReactionInterleukin 12 receptor beta 1 subunitInterleukin 3HaptoglobinsInterleukin-6Receptors InterleukinArticlesMolecular biologyReceptors Interleukin-6Interleukin 10LiverSolubilityInterleukin-6 receptorPhosphoenolpyruvate Carboxykinase (GTP)Interleukin 1 receptor type ICarrier ProteinsHalf-LifeThe Journal of experimental medicine
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The polypyrimidine tract-binding protein (PTB) is involved in the post-transcriptional regulation of human inducible nitric oxide synthase expression.

2006

Human inducible nitric oxide synthase (iNOS) expression is regulated by transcriptional and post-transcriptional mechanisms. We have recently shown that the multifunctional RNA-binding proteins KH-type splicing regulatory protein and tristetraprolin are critically involved in the post-transcriptional regulation of human iNOS expression. Several reports have shown that KH-type splicing regulatory protein colocalizes with the polypyrimidine tract-binding protein (PTB), and both RNA-binding proteins seem to interact with the same mRNAs. Therefore we analyzed the involvement of PTB in human iNOS expression. In human DLD-1 cells, cytokine incubation necessary to induce iNOS expression did not ch…

Recombinant Fusion ProteinsTristetraprolinGreen Fluorescent ProteinsNitric Oxide Synthase Type IImacromolecular substancesBiologyIn Vitro TechniquesTransfectionenvironment and public healthBiochemistryGene Expression Regulation EnzymologicCell LineCell Line TumorHumansPolypyrimidine tract-binding proteinRNA MessengerEnzyme InhibitorsPromoter Regions GeneticMolecular BiologyPost-transcriptional regulationRegulation of gene expressionMessenger RNAintegumentary systemCarcinomaEpithelial CellsCell BiologyTransfectionMolecular biologyNitric oxide synthaseRNA splicingColonic Neoplasmsbiology.proteinCytokinesRNA InterferenceProtein Processing Post-TranslationalDichlororibofuranosylbenzimidazolePolypyrimidine Tract-Binding ProteinThe Journal of biological chemistry
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Preferential Type II Muscle Fiber Damage From Plyometric Exercise

2012

Context: Plyometric training has been successfully used in different sporting contexts. Studies that investigated the effect of plyometric training on muscle morphology are limited, and results are controversial with regard to which muscle fiber type is mainly affected. Objective: To analyze the skeletal muscle structural and ultrastructural change induced by an acute bout of plyometric exercise to determine which type of muscle fibers is predominantly damaged. Design: Descriptive laboratory study. Setting: Research laboratory. Patients or Other Participants: Eight healthy, untrained individuals (age = 22 ± 1 years, height = 179.2 ± 6.4 cm, weight = 78.9 ± 5.9 kg). Intervention(s): Partici…

Reflex Stretchmedicine.medical_specialtyPhysical Therapy Sports Therapy and RehabilitationContext (language use)Plyometric ExerciseSarcomereYoung AdultHumansPlyometricsMedicineOrthopedics and Sports MedicineExercise physiologyMuscle SkeletalCreatine KinaseExerciseOriginal ResearchbiologySettore BIO/16 - Anatomia Umanabusiness.industrySkeletal muscleGeneral Medicinemedicine.anatomical_structureMuscle Fibers Fast-Twitchbiology.proteinPhysical therapyCreatine kinasemedicine.symptombusinessType II Muscle Fibereccentric exercise sarcomere Z-disk streaming electron transmission microscopy creatine kinase dystrophinMuscle ContractionMuscle contractionJournal of Athletic Training
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A Unified Approach to Measuring Accuracy of Error Indicators

2014

In this paper, we present a unified approach to error indication for elliptic boundary value problems. We introduce two different definitions of the accuracy (weak and strong) and show that various indicators result from one principal relation. In particular, this relation generates all the main types of error indicators, which have already gained high popularity in numerical practice. Also, we discuss some new forms of indicators that follow from a posteriori error majorants of the functional type and compare them with other indicators. Finally, we discuss another question related to accuracy of error indicators for problems with incompletely known data.

Relation (database)Computer sciencePrincipal (computer security)Functional typeA priori and a posterioriApplied mathematicsBoundary value problemPopularityType I and type II errors
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Short-Time Ocular Ischemia Induces Vascular Endothelial Dysfunction and Ganglion Cell Loss in the Pig Retina

2019

Visual impairment and blindness are often caused by retinal ischemia-reperfusion (I/R) injury. We aimed to characterize a new model of I/R in pigs, in which the intraocular pathways were not manipulated by invasive methods on the ocular system. After 12 min of ischemia followed by 20 h of reperfusion, reactivity of retinal arterioles was measured in vitro by video microscopy. Dihydroethidium (DHE) staining, qPCR, immunohistochemistry, quantification of neurons in the retinal ganglion cell layer, and histological examination was performed. Retinal arterioles of I/R-treated pigs displayed marked attenuation in response to the endothelium-dependent vasodilator, bradykinin, compared to sham-tre…

Retinal Ganglion CellsVascular Endothelial Growth Factor A0301 basic medicinePathologySwineNitric Oxide Synthase Type IIVasodilationendothelial dysfunctionlcsh:Chemistrychemistry.chemical_compound0302 clinical medicineIschemiaEndothelial dysfunctionlcsh:QH301-705.5SpectroscopyGeneral MedicineComputer Science ApplicationsArteriolesmedicine.anatomical_structureRetinal ganglion cellReperfusion InjuryNADPH Oxidase 2medicine.medical_specialtyEndotheliumRetinal ArteryI/R injuryIschemiaretinal arteriolesBradykininRetinal ganglionRetinaArticleCatalysisganglion cell lossInorganic Chemistry03 medical and health sciencesmedicineAnimalsPhysical and Theoretical ChemistryMolecular BiologyRetinabusiness.industryOrganic ChemistryRetinalHypoxia-Inducible Factor 1 alpha Subunitmedicine.disease030104 developmental biologylcsh:Biology (General)lcsh:QD1-999chemistry030221 ophthalmology & optometryEndothelium VascularReactive Oxygen SpeciesbusinessInternational Journal of Molecular Sciences
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Role of nitric oxide synthase isoforms for ophthalmic artery reactivity in mice.

2014

Abstract Nitric oxide synthases (NOS) are involved in regulation of ocular vascular tone and blood flow. While endothelial NOS (eNOS) has recently been shown to mediate endothelium-dependent vasodilation in mouse retinal arterioles, the contribution of individual NOS isoforms to vascular responses is unknown in the retrobulbar vasculature. Moreover, it is unknown whether the lack of a single NOS isoform affects neuron survival in the retina. Thus, the goal of the present study was to examine the hypothesis that the lack of individual nitric oxide synthase (NOS) isoforms affects the reactivity of mouse ophthalmic arteries and neuron density in the retinal ganglion cell (RGC) layer. Mice defi…

Retinal Ganglion CellsVasodilator AgentsNitric Oxide Synthase Type IIVideo microscopyVasodilationCell CountNitric Oxide Synthase Type IMuscle Smooth Vascularchemistry.chemical_compoundMiceOphthalmic ArteryPhenylephrineEnosEnzyme InhibitorsMice KnockoutbiologyAnatomySensory SystemsNitric oxide synthaseIsoenzymesVasodilationmedicine.anatomical_structureNG-Nitroarginine Methyl EsterRetinal ganglion cellKnockout mouseRetinal NeuronsNitroprussidemedicine.medical_specialtyNitric Oxide Synthase Type IIIEndothelial NOSNitric oxideCellular and Molecular NeuroscienceTonometry OcularInternal medicinemedicineAnimalsNitric Oxide DonorsIntraocular Pressurebusiness.industrybiology.organism_classificationAcetylcholineMice Inbred C57BLOphthalmologyEndocrinologychemistryVasoconstrictionbiology.proteinAdrenergic alpha-1 Receptor AgonistsEndothelium VascularbusinessExperimental eye research
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Role of nitric oxide during rotavirus infection.

2006

The pathophysiological mechanisms behind rotavirus-induced diarrhoea still remain incomplete. Current views suggest that the non-structural protein 4 (NSP4) of rotavirus and the enteric nervous system (ENS) participate in water secretion and diarrhoea. In the present work the role of nitric oxide (NO) in rotavirus infection and disease has been studied in vitro, mice and humans. Incubation of human intestinal epithelial cells (HT-29) with purified NSP4 but not with infectious virus produced NO2/NO3 accumulation in the incubation media. The NSP4-induced release of NO metabolites occurred within the first minutes after the addition of the toxin. Mice infected with murine rotavirus (strain EDI…

RotavirusvirusesReoviridaeNitric Oxide Synthase Type IIIleumIn Vitro TechniquesViral Nonstructural Proteinsmedicine.disease_causeNitric OxideVirusRotavirus InfectionsMicrobiologyNitric oxideCell LineJejunumchemistry.chemical_compoundMiceVirologyRotavirusmedicineAnimalsHumansProspective StudiesRNA MessengerGlycoproteinsToxins BiologicalMice Inbred BALB CbiologyBase SequenceToxinInfantbiology.organism_classificationVirologyGastroenteritisDiarrheaInfectious Diseasesmedicine.anatomical_structurechemistryAnimals NewbornCase-Control StudiesImmunologymedicine.symptomJournal of medical virology
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SPARC is a new myeloid-derived suppressor cell marker licensing suppressive activities

2019

Myeloid-derived suppressor cells (MDSC) are well-known key negative regulators of the immune response during tumor growth, however scattered is the knowledge of their capacity to influence and adapt to the different tumor microenvironments and of the markers that identify those capacities. Here we show that the secreted protein acidic and rich in cysteine (SPARC) identifies in both human and mouse MDSC with immune suppressive capacity and pro-tumoral activities including the induction of epithelial-to-mesenchymal transition (EMT) and angiogenesis. In mice the genetic deletion of SPARC reduced MDSC immune suppression and reverted EMT. Sparc−/− MDSC were less suppressive overall and the granu…

STAT3 Transcription Factorlcsh:Immunologic diseases. Allergy0301 basic medicineEpithelial-Mesenchymal TransitionAngiogenesisImmunologyneutrophil extracellular trapsNitric Oxide Synthase Type IIInflammationExtracellular TrapsMice03 medical and health sciences0302 clinical medicineImmune systemBreast cancermedicineMyeloid-derived suppressor cellAnimalsHumansImmunology and AllergyOsteonectinOriginal ResearchMice KnockoutMice Inbred BALB CTumor microenvironmentArginaseChemistryNeutrophilNF-kappa B p50 SubunitSPARCNeutrophil extracellular trapsmyeloid-derived suppressor cells030104 developmental biologyCancer researchMyeloid-derived Suppressor CellTumor necrosis factor alphaSignal transductionmedicine.symptomlcsh:RC581-607Neutrophil extracellular trapBiomarkers030215 immunology
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The interleukin-22/STAT3 pathway potentiates expression of inducible nitric-oxide synthase in human colon carcinoma cells.

2007

Inducible nitric-oxide synthase (iNOS) has been identified as a marker and mediator of disease in human colonic inflammation and carcinogenesis. Accordingly, identification of mediators that trigger iNOS in colon carcinoma/epithelial cells is an important topic of current research. Here we demonstrate that interleukin (IL)-22, a newly described member of the IL-10 cytokine family, potently synergizes with interferon (IFN)-gamma for iNOS expression in human DLD-1 colon carcinoma cells. Detection of both IL-22 receptor chains and STAT3 phosphorylation proved robust IL-22 responsiveness of these cells. Short interfering RNA technology identified STAT3 as being crucial for up-regulation of iNOS…

STAT3 Transcription Factormedicine.medical_treatmentNitric Oxide Synthase Type IIBiologymedicine.disease_causeBiochemistryGene Expression Regulation EnzymologicInterleukin 22InterferonmedicineHumansRNA MessengerRNA NeoplasmSTAT3Promoter Regions GeneticMolecular BiologyInflammationInterleukinsNF-kappa BInterleukinCell BiologyTransfectionReceptors InterleukinMolecular biologyNeoplasm ProteinsGene Expression Regulation NeoplasticCytokineSTAT1 Transcription FactorColonic Neoplasmsbiology.proteinCancer researchCytokinesIntercellular Signaling Peptides and ProteinsTumor necrosis factor alphaImmunotherapyCaco-2 CellsCarcinogenesismedicine.drugSignal TransductionThe Journal of biological chemistry
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