Search results for "vesicle"

showing 10 items of 787 documents

Opportunities and Challenges of Liquid Biopsy in Thyroid Cancer

2021

Thyroid cancer is the most common malignancy of the endocrine system, encompassing different entities with distinct histological features and clinical behavior. The diagnostic definition, therapeutic approach, and follow-up of thyroid cancers display some controversial aspects that represent unmet medical needs. Liquid biopsy is a non-invasive approach that detects and analyzes biological samples released from the tumor into the bloodstream. With the use of different technologies, tumor cells, free nucleic acids, and extracellular vesicles can be retrieved in the serum of cancer patients and valuable molecular information can be obtained. Recently, a growing body of evidence is accumulating…

0301 basic medicinediagnosisQH301-705.5differentiated thyroid cancerReviewNeoplastic CellsMalignancymedullary thyroid cancerCatalysisInorganic ChemistryExtracellular Vesicles03 medical and health sciences0302 clinical medicineCirculating tumor cellBiomarkers TumorCirculatingmedicineHumansThyroid NeoplasmsBiology (General)Physical and Theoretical ChemistryAnaplastic thyroid cancerLiquid biopsyQD1-999Molecular BiologyThyroid cancerSpectroscopyTumorliquid biopsybusiness.industryOrganic ChemistryThyroidMedullary thyroid cancerCancerGeneral MedicineNeoplastic Cells Circulatingmedicine.diseaseComputer Science ApplicationsChemistry030104 developmental biologymedicine.anatomical_structure030220 oncology & carcinogenesisCancer researchTherapyprognosisbusinessCell-Free Nucleic AcidsBiomarkersanaplastic thyroid cancerInternational Journal of Molecular Sciences
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Tissue factor prothrombotic activity is regulated by integrin-arf6 trafficking

2017

Objective— Coagulation initiation by tissue factor (TF) is regulated by cellular inhibitors, cell surface availability of procoagulant phosphatidylserine, and thiol-disulfide exchange. How these mechanisms contribute to keeping TF in a noncoagulant state and to generating prothrombotic TF remain incompletely understood. Approach and Results— Here, we study the activation of TF in primary macrophages by a combination of pharmacological, genetic, and biochemical approaches. We demonstrate that primed macrophages effectively control TF cell surface activity by receptor internalization. After cell injury, ATP signals through the purinergic receptor P2rx7 induce release of TF + microvesicles. T…

0301 basic medicinedynaminsIntegrin alpha4CellCardiorespiratory Medicine and Haematology030204 cardiovascular system & hematologyIntegrin alpha4beta1Inbred C57BLTransgenicMicechemistry.chemical_compound0302 clinical medicineAdenosine TriphosphateCell-Derived MicroparticlesReceptors2.1 Biological and endogenous factorsfibrinGene Knock-In TechniquesAetiologyPhospholipidsTumorbiologyChemistryADP-Ribosylation FactorsHematologyPhosphatidylserineCell biologyProtein Transportmedicine.anatomical_structurePhenotypeProteomeextracellular vesiclesCardiology and Cardiovascular MedicinePurinergic P2X7BiotechnologySignal TransductionGenotypeproteomeClinical SciencesIntegrinMice TransgenicFactor VIIaTransfectionExtracellular vesiclesFibrinArticleCell LineThromboplastin03 medical and health sciencesTissue factorCell Line TumormedicineAnimalsHumansBlood CoagulationMacrophagesThrombosisMice Inbred C57BL030104 developmental biologyCardiovascular System & HematologyADP-Ribosylation Factor 6biology.proteinReceptors Purinergic P2X7
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Phytoagent Deoxyelephantopin and Its Derivative Inhibit Triple Negative Breast Cancer Cell Activity through ROS-Mediated Exosomal Activity and Protei…

2017

A novel plant sesquiterpene lactone derivative, DET derivative (DETD)-35, originating from parental deoxyelephantopin (DET) was previously observed to effectively suppress human triple negative breast cancer (TNBC) MDA-MB-231 cell activity and tumor growth in mice. In this study, the mechanisms underlying the activity of DETD-35 were elucidated. DET and DETD-35 induced reactive oxygen species (ROS) which caused structural damage and dysfunction of mitochondria and increased cytosolic calcium level, subsequently evoking exosome release from the cancer cells. Intriguingly, exosomes induced by both compounds had an atypical function. Cancer cell-derived exosomes commonly show metastatic potent…

0301 basic medicineexosomal proteomeAngiogenesisMitochondrionBiologymedicine.disease_causeExosome03 medical and health sciencesbreast cancermedicinesesquiterpene lactoneoxidative stressPharmacology (medical)Cell adhesionOriginal ResearchPharmacologychemistry.chemical_classificationReactive oxygen specieslcsh:RM1-950MicrovesiclesCell biology030104 developmental biologylcsh:Therapeutics. PharmacologyBiochemistrychemistryCancer cellcancer therapyOxidative stressFrontiers in Pharmacology
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Platelets, endothelial cells and leukocytes contribute to the exercise-triggered release of extracellular vesicles into the circulation.

2019

ABSTRACT Physical activity initiates a wide range of multi-systemic adaptations that promote mental and physical health. Recent work demonstrated that exercise triggers the release of extracellular vesicles (EVs) into the circulation, possibly contributing to exercise-associated adaptive systemic signalling. Circulating EVs comprise a heterogeneous collection of different EV-subclasses released from various cell types. So far, a comprehensive picture of the parental and target cell types, EV-subpopulation diversity and functional properties of EVs released during exercise (ExerVs) is lacking. Here, we performed a detailed EV-phenotyping analysis to explore the cellular origin and potential …

0301 basic medicineimmunobead isolationCell typeHistologyCD14exosomes03 medical and health sciences0302 clinical medicinePlateletlcsh:QH573-671Antigen-presenting cellplasmaCluster of differentiationCD63exerciselcsh:CytologyChemistrysize exclusion chromatographyCell BiologyExtracellular vesiclesmultiplex phenotypingMicrovesiclesCell biology030104 developmental biology030220 oncology & carcinogenesisCD146extracellular vesiclesResearch Article
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Use of Stem Cell Extracellular Vesicles as a “Holistic” Approach to CNS Repair

2020

Neurodegeneration is a hallmark of many diseases and disorders of the central nervous system (CNS). High levels of neuroinflammation are often associated with irreparable damage to CNS cells due to the dysregulation of signaling cascades that are unable to restore a homeostatic balance. Due to the inherent complexity of the CNS, development of CNS-related therapeutics has met limited success. While stem cell therapy has been evaluated in the context of CNS repair, the mechanisms responsible for their functional properties have not been clearly defined. In recent years, there has been growing interest in the use of stem cell extracellular vesicles (EVs) for the treatment of various CNS patho…

0301 basic medicineinduced pluripotent stem cellsmedicine.medical_treatmentContext (language use)ReviewexosomesBiologyNeuroprotectionCell and Developmental Biology03 medical and health sciences0302 clinical medicinemedicineInduced pluripotent stem celllcsh:QH301-705.5Neuroinflammationmesenchymal stem cellsMesenchymal stem cellCell BiologyStem-cell therapycentral nervous systemMicrovesicles030104 developmental biologylcsh:Biology (General)030220 oncology & carcinogenesisStem cellextracellular vesiclesNeuroscienceDevelopmental BiologyFrontiers in Cell and Developmental Biology
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The Amino Acid Transporter JhI-21 Coevolves with Glutamate Receptors, Impacts NMJ Physiology, and Influences Locomotor Activity in Drosophila Larvae

2015

AbstractChanges in synaptic physiology underlie neuronal network plasticity and behavioral phenomena, which are adjusted during development. The Drosophila larval glutamatergic neuromuscular junction (NMJ) represents a powerful synaptic model to investigate factors impacting these processes. Amino acids such as glutamate have been shown to regulate Drosophila NMJ physiology by modulating the clustering of postsynaptic glutamate receptors and thereby regulating the strength of signal transmission from the motor neuron to the muscle cell. To identify amino acid transporters impacting glutmatergic signal transmission, we used Evolutionary Rate Covariation (ERC), a recently developed bioinforma…

0301 basic medicinejuvenile-hormonemelanogasterAmino Acid Transport Systemsextracellular glutamateprotein-protein interactionsPhysiology[ SDV.BA ] Life Sciences [q-bio]/Animal biologySynaptic Transmissionin-vivo0302 clinical medicinePostsynaptic potentialDrosophila Proteinsgenesglial xctMotor NeuronsAnimal biologyMultidisciplinary[SDV.BA]Life Sciences [q-bio]/Animal biologyGlutamate receptorBiological Evolutiondrosophilemedicine.anatomical_structureReceptors GlutamateLarvaExcitatory postsynaptic potentialDrosophila[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Drosophila ProteinSignal Transductionevolutionary rate covariationNeuromuscular JunctionPresynaptic TerminalsNeurotransmissionBiologyMotor ActivityArticlesynaptic vesicle03 medical and health sciencesGlutamatergicneuromuscular-junctionBiologie animalemedicineAnimalsAmino acid transporterevolutionary rate covariation;protein-protein interactions;juvenile-hormone;neuromuscular-junction;synaptic vesicle;in-vivo;extracellular glutamate;glial xct;melanogaster;genesfungiNeurosciencesExcitatory Postsynaptic PotentialsMotor neuron030104 developmental biology[ SDV.NEU ] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Neurons and CognitionMutation030217 neurology & neurosurgeryScientific Reports
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Proteolytic Enzymes Clustered in Specialized Plasma-Membrane Domains Drive Endothelial Cells’ Migration

2016

In vitro cultured endothelial cells forming a continuous monolayer establish stable cell-cell contacts and acquire a "resting" phenotype; on the other hand, when growing in sparse conditions these cells acquire a migratory phenotype and invade the empty area of the culture. Culturing cells in different conditions, we compared expression and clustering of proteolytic enzymes in cells having migratory versus stationary behavior. In order to observe resting and migrating cells in the same microscopic field, a continuous cell monolayer was wounded. Increased expression of proteolytic enzymes was evident in cell membranes of migrating cells especially at sprouting sites and in shed membrane vesi…

0301 basic medicinekalininsepraseCell Membranesbeta1 integrinCelllcsh:MedicineurokinaseBiochemistryEpitheliumCell membrane0302 clinical medicineAnimal CellsMedicine and Health Sciencesdipeptidyl peptidase IVlcsh:ScienceMultidisciplinarybiologyVesicleProteolytic enzymesCell migrationProteasesEnzymesCell biologyLaboratory EquipmentCell Motilitymedicine.anatomical_structureBiochemistry030220 oncology & carcinogenesisEngineering and TechnologyBiological Culturesmatrix metalloproteinase 14Cellular Structures and OrganellesCellular TypesAnatomyResearch ArticleEquipmentCell MigrationResearch and Analysis MethodsGelatin MediaCell Linegelatinase B03 medical and health sciencescollagen type 4fibronectinmedicineHumansVesiclescollagen type 1gelatinase Alcsh:RCell MembraneBiology and Life SciencesEndothelial CellsProteinsMembrane ProteinsEpithelial CellsCell BiologyCulture MediaFibronectinBiological Tissue030104 developmental biologyMembrane proteinCell cultureProteolysisMicroscopy Electron ScanningEnzymologybiology.proteinlcsh:QCollagensDevelopmental BiologyPLOS ONE
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Exosome-Mediated Signaling in Epithelial to Mesenchymal Transition and Tumor Progression

2018

Growing evidence points to exosomes as key mediators of cell–cell communication, by transferring their specific cargo (e.g., proteins, lipids, DNA and RNA molecules) from producing to receiving cells. In cancer, the regulation of the exosome-mediated intercellular communication may be reshaped, inducing relevant changes in gene expression of recipient cells in addition to microenvironment alterations. Notably, exosomes may deliver signals able to induce the transdifferentiation process known as Epithelial-to-Mesenchymal Transition (EMT). In this review, we summarize recent findings on the role of exosomes in tumor progression and EMT, highlighting current knowledge on exosome-mediated inter…

0301 basic medicinelcsh:MedicineReviewExosomeMetastasis03 medical and health sciencesepithelial-mesenchymal plasticity; cancer-derived exosomes; extracellular vesicles; metastasis; tumor nicheGene expressionmedicinemetastasisEpithelial–Mesenchymal plasticityEpithelial–mesenchymal transitionbusiness.industrytumor nicheTransdifferentiationlcsh:RRNAGeneral Medicinemedicine.diseasecancer-derived exosomeMicrovesiclesCell biology030104 developmental biologyepithelial-mesenchymal plasticityTumor progressionmetastasiextracellular vesiclebusinesscancer-derived exosomesextracellular vesicles
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H1.0 Linker Histone as an Epigenetic Regulator of Cell Proliferation and Differentiation

2018

H1 linker histones are a class of DNA-binding proteins involved in the formation of supra-nucleosomal chromatin higher order structures. Eleven non-allelic subtypes of H1 are known in mammals, seven of which are expressed in somatic cells, while four are germ cell-specific. Besides having a general structural role, H1 histones also have additional epigenetic functions related to DNA replication and repair, genome stability, and gene-specific expression regulation. Synthesis of the H1 subtypes is differentially regulated both in development and adult cells, thus suggesting that each protein has a more or less specific function. The somatic variant H1.0 is a linker histone that was recognized…

0301 basic medicinelcsh:QH426-470Somatic cellRNA-binding proteinhistone H1.0RNA-binding proteinsReviewBiologymedicine.disease_cause03 medical and health sciencesSettore BIO/10 - BiochimicaGeneticsmedicineEpigeneticsSettore BIO/06 - Anatomia Comparata E CitologiaGenetics (clinical)linker histonesCell growthChromatinCell biologylcsh:Geneticslinker histone030104 developmental biologyHistoneCancer cellbiology.proteinStem cellextracellular vesiclesCarcinogenesisGenes
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Extracellular Vesicle‐Associated RNA as a Carrier of Epigenetic Information

2017

Post-transcriptional regulation of messenger RNA (mRNA) metabolism and subcellular localization is of the utmost importance both during development and in cell differentiation. Besides carrying genetic information, mRNAs contain cis-acting signals (zip codes), usually present in their 5'- and 3'-untranslated regions (UTRs). By binding to these signals, trans-acting factors, such as RNA-binding proteins (RBPs), and/or non-coding RNAs (ncRNAs), control mRNA localization, translation and stability. RBPs can also form complexes with non-coding RNAs of different sizes. The release of extracellular vesicles (EVs) is a conserved process that allows both normal and cancer cells to horizontally tran…

0301 basic medicinelcsh:QH426-470mRNAnon‐coding RNA (ncRNA)RNA-binding proteinReviewBiology03 medical and health sciencesRNA‐binding proteins (RBPs)Settore BIO/10 - Biochimicanon-coding RNA (ncRNA)Gene expressionGeneticsSettore BIO/06 - Anatomia Comparata E CitologiaTranscription factorGenetics (clinical)GeneticsmRNA; non-coding RNA(ncRNA); RNA-binding proteins (RBPs); extracellular vesicles (EVs)Messenger RNARNATranslation (biology)Extracellular vesicleCell biologyChromatinlcsh:Genetics030104 developmental biologyRNA-binding proteins (RBPs)extracellular vesicles (EVs)non-coding RNA(ncRNA)Genes
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