Search results for "wistar"

showing 10 items of 1094 documents

Brain-Derived Neurotrophic Factor But Not Forced Arm Use Improves Long-Term Outcome After Photothrombotic Stroke and Transiently Upregulates Binding …

2008

Background and Purpose— Both application of neurotrophic factors like brain-derived neurotrophic factor (BDNF) and constraint-induced movement therapy like forced arm use have been shown to potentially improve outcome after stroke. The aim of the present study was to check whether postischemic long-term outcome correlates to specific modifications in the abundance of various neurotransmitter receptors. Methods— Adult male Wistar rats were subjected to photothrombotic ischemia and assigned to various treatment groups (n=5 each) with end points at 3 and 6 weeks: (1) ischemic control (saline); (2) BDNF (ischemia, 20 μg BDNF); (3) forced arm use (ischemia, saline, and ipsilateral plaster cast …

MaleRestraint PhysicalMotor ActivityLigandsDownregulation and upregulationNeurotrophic factorsForelimbmedicineAnimalsRats WistarReceptorGABA Agonistsalpha-Amino-3-hydroxy-5-methyl-4-isoxazolepropionic AcidStrokePhysical Therapy ModalitiesAdvanced and Specialized NursingBrain-derived neurotrophic factorMuscimolbusiness.industryBrain-Derived Neurotrophic FactorGlutamate receptorBrainCerebral Infarctionmedicine.diseaseRatsUp-RegulationStrokemedicine.anatomical_structureReceptors Glutamatenervous systemAnesthesiaExcitatory postsynaptic potentialAutoradiographyNeurology (clinical)Dizocilpine MaleateIntracranial ThrombosisForelimbCardiology and Cardiovascular MedicinebusinessExcitatory Amino Acid AntagonistsNeuroscienceStroke
researchProduct

Impaired hippocampal neuroligin-2 function by chronic stress or synthetic peptide treatment is linked to social deficits and increased aggression.

2014

Neuroligins (NLGNs) are cell adhesion molecules that are important for proper synaptic formation and functioning, and are critical regulators of the balance between neural excitation/inhibition (E/I). Mutations in NLGNs have been linked to psychiatric disorders in humans involving social dysfunction and are related to similar abnormalities in animal models. Chronic stress increases the likelihood for affective disorders and has been shown to induce changes in neural structure and function in different brain regions, with the hippocampus being highly vulnerable to stress. Previous studies have shown evidence of chronic stress-induced changes in the neural E/I balance in the hippocampus. Ther…

MaleRestraint PhysicalhippocampusmoodCell Adhesion Molecules NeuronalNeurexinstress disordersHippocampusPoison controlNeuroliginNerve Tissue ProteinsReceptors Cell Surfacebehavioral scienceHippocampal formationneuropharmacologyHippocampussocial behaviorRats Sprague-DawleystressmedicineNeuritesAnimalsChronic stressRats WistarSocial BehaviorCells CulturedPharmacologyNeuronsAggressionaggressionneuropeptideschronic restraint stressOrgan SizeanxietyRatsAggressionsociabilityPsychiatry and Mental healthChronic DiseaseOriginal Articleneuroliginmedicine.symptomPsychologyCorticosteronePeptidesNeuroscienceStress PsychologicalSocial behaviorNeuropsychopharmacology : official publication of the American College of Neuropsychopharmacology
researchProduct

Hypertonic fluid resuscitation from subarachnoid hemorrhage in rats: A comparison between small volume resuscitation and mannitol

2005

Abstract Objective Death and severe morbidity after subarachnoid hemorrhage (SAH) are mainly caused by global cerebral ischemia through increased intracranial pressure (ICP) and decreased cerebral blood flow (CBF). We have recently demonstrated neuroprotective effects of small volume resuscitation (7.5% saline in combination with 6% dextran 70) in an animal model of SAH, leading to normalization of increased ICP, reduced morphological damage and improved neurological recovery. In the present study, we compared the concept of small volume resuscitation represented by two clinically licenced hypertonic–hyperoncotic saline solutions with the routinely used hyperosmotic agent–mannitol–and inves…

MaleResuscitationTime FactorsSubarachnoid hemorrhageIntracranial PressureResuscitationmedicine.medical_treatmentHypertonic SolutionsFunctional LateralityRandom AllocationmedicineAnimalsMannitolcardiovascular diseasesRats WistarSalineIntracranial pressureNeurologic Examinationbusiness.industryDextransSubarachnoid Hemorrhagemedicine.diseaseRatsnervous system diseasesDextran 70Hypertonic salineDisease Models AnimalNeurologyCerebral blood flowCerebrovascular CirculationAnesthesiaTonicityNeurology (clinical)businessJournal of the Neurological Sciences
researchProduct

Bioavailability and pharmacokinetic model for ritonavir in the rat.

2007

The aim of this study is to investigate in vivo the oral bioavailability of ritonavir and to evaluate the pharmacokinetic model that best describes the plasma concentration behavior after oral and intravenous administration. Male Wistar rats were intravenously administered at 3 mg dose of pure ritonavir and oral administered at 4.6 +/- 2.5 mg of diluted Norvir. Blood samples were taken by means of the jugular vein for a 24 h period of time. An analytical high-performance liquid chromatography (HPLC) technique was developed in order to quantify ritonavir plasma concentrations. A nonlinear modeling approach was used to estimate the pharmacokinetic parameters of interest. Results showed that a…

MaleRitonavirbiologyChemistryPharmaceutical ScienceBiological AvailabilityAbsorption (skin)PharmacologyHigh-performance liquid chromatographyModels BiologicalBioavailabilityAbsorptionRatsPharmacokineticsIn vivoEnzyme inhibitormedicinebiology.proteinAnimalsRitonavirProtease inhibitor (pharmacology)Rats Wistarmedicine.drugJournal of pharmaceutical sciences
researchProduct

In vivo GSH depletion induces c-myc expression by modulation of chromatin protein complexes.

2009

Abstract We hypothesize that glutathione (GSH) fluctuations could have a prominent role in the modulation of c-myc expression through a mechanism affecting chromatin remodeling complexes. This could lead to an open chromatin structure accessible to transcription factors. We studied the in vivo effect of GSH depletion on these complexes bound to the c-myc promoter in the liver of l-buthionine-(S,R)-sulfoximine (BSO)-treated rats. Using chromatin immunoprecipitation we found that 3 h after BSO treatment the repressing complexes Id2 and Sin3A (part of a histone–deacetylase complex) were released from the c-myc promoter. STAT3 was phosphorylated and associated with its coactivator p300 with int…

MaleSTAT3 Transcription FactorTranscriptional ActivationTime FactorsBiologyBiochemistryChromatin remodelingHistone DeacetylasesProto-Oncogene Proteins c-mycHistone H3Physiology (medical)Gene expressionCoactivatorTranscriptional regulationAnimalsp300-CBP Transcription FactorsPhosphorylationRats WistarTranscription factorButhionine SulfoximineInhibitor of Differentiation Protein 2AcetylationChromatin Assembly and DisassemblyMolecular biologyGlutathioneChromatinRatsRepressor ProteinsSin3 Histone Deacetylase and Corepressor ComplexGene Expression RegulationLiverChromatin immunoprecipitationProtein BindingFree radical biologymedicine
researchProduct

Antimutagenic activity of organosulfur compounds from Allium is associated with phase II enzyme induction

2001

In a previous study, we showed that naturally occurring organosulfur compounds (OSCs) from garlic and onion modulated the activation of carcinogen via the alteration of cytochromes P450. The present study was undertaken to determine the incidence of the in vivo induction of phase II enzymes by individual OSCs on the genotoxicity of several carcinogens. Diallyl sulfide (DAS), diallyl disulfide (DADS), dipropyl sulfide (DPS) and dipropyl disulfide (DPDS), were administered by gavage (1mmol/kg) to male SPF Wistar rats for 4 consecutive days. The effects of treatments on phase II enzymes and on the genotoxicity of carcinogens were evaluated with hepatic cytosols and microsomes from OSCs-treated…

MaleSalmonella typhimuriumHealth Toxicology and Mutagenesis[SDV]Life Sciences [q-bio]Allyl compoundAdministration OralSulfidesmedicine.disease_causeAmes testAllium03 medical and health scienceschemistry.chemical_compoundPropane0302 clinical medicineGeneticsmedicineNAD(P)H Dehydrogenase (Quinone)AnimalsDisulfidesRats WistarEpoxide hydrolaseCarcinogenComputingMilieux_MISCELLANEOUS030304 developmental biologyGlutathione TransferaseEpoxide Hydrolases0303 health sciencesDose-Response Relationship DrugChemistryDiallyl disulfideMutagenicity TestsAntimutagenic Agents3. Good healthRatsSpecific Pathogen-Free Organisms[SDV] Life Sciences [q-bio]Allyl CompoundsBiochemistryAntimutagenic AgentsLiver030220 oncology & carcinogenesisEnzyme InductionAntimutagenGenotoxicityMutagensSubcellular Fractions
researchProduct

Expression of α4-1 and α5 Nicotinic Cholinoceptor mRNA in the Aging Rat Cerebral Cortex

1997

Although important in neurodegeneration, systematic studies of nicotinic acetylcholine receptor expression in normal aging human brains are difficult to perform. We have studied the expression of nicotinic receptor alpha 4-1 and alpha 5 mRNA in the frontal and parietal isocortex of 3- (young adult), 24- (late middle aged), and 33-month-old (old) rats by nonisotopic in situ hybridization. In all groups transcripts were mainly present in layer II/III and V pyramidal neurons. The numerical densities of alpha 4-1 mRNA-containing neurons with respect to those of cresyl violet-stained neurons decreased with aging in the rat frontal and parietal cortex, while those of alpha 5 mRNA-containing neuro…

MaleSenescenceAgingmedicine.medical_specialtyPosterior parietal cortexReceptors NicotinicBiologyNicotineInternal medicinemedicineAnimalsRNA MessengerRats WistarIn Situ HybridizationAcetylcholine receptorCerebral CortexGeneral NeuroscienceNeurodegenerationmedicine.diseaseImmunohistochemistryRatsNicotinic acetylcholine receptorNicotinic agonistEndocrinologymedicine.anatomical_structureCerebral cortexNeurology (clinical)Geriatrics and GerontologyDevelopmental Biologymedicine.drugNeurobiology of Aging
researchProduct

Lateral habenula and hippocampal units: electrophysiological and iontophoretic study

1995

In previous works we studied, on cats, the effects of lateral habenula (LH) stimulation on hippocampal units. In particular, the results showed an excitation or an inhibition in relation to the stimulation frequency (0.5-3.0 Hz or 5.0-20 Hz, respectively). All the LH stimulation effects were antagonised by iontophoretic intrahippocampal application of methysergide (MS). In this series of experiments it was possible to demonstrate, on rats, that LH stimulation causes an excitatory effect in a major number of hippocampal units in relation to the frequency increase. The inhibitory effect by iontophoretic serotonine application and the reversible blockade of habenular modulation after iontophor…

MaleSerotoninN-MethylaspartateMethysergideStimulationHippocampal formationHippocampusMicromanipulationDorsal raphe nucleusThalamusmedicineAnimalsRats WistarNeuronsAnalysis of VarianceRapheMethysergideChemistryGeneral NeuroscienceIontophoresisElectric StimulationRatsHabenulaExcitatory postsynaptic potentialRaphe NucleiRaphe nucleiNeurosciencemedicine.drugBrain Research Bulletin
researchProduct

Electrophysiological and Iontophoretic Aspects of the Habenular Influence on Hippocampal Neurones

1995

In previous experimental studies, carried out on cats, we demonstrated that electrical stimulation of lateral habenula (LH) at 0.5-3.0 Hz or 5-20 Hz had a double effect (low frequency-excitation; high frequency-inhibition) on the spontaneous firing rate of single hippocampal neurones. Our results, in agreement with similar case studies, allowed us to hypothesise that in the habenular modulation of the hippocampus the raphe nucleus is probably involved. In fact, all the effects of LH stimulation were antagonised by the iontophoretic intrahippocampal application of methysergide. In the present series of experiments, performed on rats, it was possible to demonstrate that LH stimulation at 1-10…

MaleSerotoninN-MethylaspartatePhysiologyMethysergideHippocampusStimulationHippocampal formationHippocampusPhysiology (medical)medicineAnimalsRats WistarNeuronsIontophoresisMethysergideChemistryGeneral MedicineIontophoresisElectric StimulationCochleaRatsElectrophysiologyElectrophysiologyRaphe NucleiNMDA receptorRaphe nucleiNeurosciencemedicine.drugArchives of Physiology and Biochemistry
researchProduct

Effect of chronic citalopram on serotonin-related and stress-regulated genes in the dorsal raphe nucleus of the rat

2007

Using a model of depression in which chronic social stress induces depressive-like symptoms, we investigated effects of the selective serotonin-reuptake inhibitor (SSRI) citalopram on gene expression in the dorsal raphe nucleus of male rats. Expression of tryptophan hydroxylase (TPH) protein was found to be upregulated by the stress and normalized by citalopram, while mRNAs for genes TPH 1 and 2 were differentially affected. Citalopram had no effect on serotonin transporter mRNA but reduced serotonin-1A autoreceptor mRNA in stressed animals. The SSRI prevented the stress-induced upregulation of mRNA for CREB binding protein, synaptic vesicle glycoprotein 2b and the glial N-myc downstream-re…

MaleSerotoninendocrine systemmedicine.medical_specialtyCitalopramTryptophan HydroxylaseBiologyCitalopramPolymerase Chain Reactionbehavioral disciplines and activitiesGene Expression Regulation Enzymologic03 medical and health scienceschemistry.chemical_compound0302 clinical medicineDorsal raphe nucleusStress PhysiologicalInternal medicinemental disordersmedicineAnimalsPharmacology (medical)Rats WistarSocial BehaviorNeurotransmitterBiological PsychiatrySerotonin transporter030304 developmental biologyPharmacology0303 health sciencesTryptophan hydroxylaseRatsPsychiatry and Mental healthEndocrinologyNeurologychemistryModels Animalbiology.proteinSynaptic vesicle glycoprotein 2BRNARaphe Nuclei5-HT1A receptorNeurology (clinical)SerotoninSelective Serotonin Reuptake Inhibitors030217 neurology & neurosurgerymedicine.drugEuropean Neuropsychopharmacology
researchProduct