0000000000465991

AUTHOR

Hans Hilmar Goebel

showing 4 related works from this author

Pigment variant of neuronal ceroid-lipofuscinosis

1995

A 6-year-old girl had progressive ataxia, and visual disturbances resulting in blindness. She died in her sleep at age 22 years. She shared with her sister and paternal relatives bilateral pes cavus deformities and impaired deep-tendon reflexes which suggested Charcot-Marie-Tooth disease. Her sister, who also had both polyneuropathy and a progressive central nervous system (CNS) disease, did not have pigmentary retinopathy. At autopsy, the patient was found to have neuronal ceroid-lipofuscinosis (NCL) marked by intraneuronal accumulation of autofluorescent granular lipopigments in ballooned perikarya and conspicuous extraneuronal pigmentation of subcortical grey matter, but without axonal s…

Malemedicine.medical_specialtyPathologyPostmortem studiesNeurologyCentral nervous systemAutopsyBiologyGrey matterEpitheliumNuclear FamilyDiagnosis DifferentialCharcot-Marie-Tooth DiseaseNeuronal Ceroid-LipofuscinosesmedicineNeuropilHumansChildGenetics (clinical)Cerebral CortexNeuronsPigmentationPigments BiologicalAnatomymedicine.diseaseMicroscopy ElectronKidney Tubulesmedicine.anatomical_structureSpinal CordFemaleNeuronal ceroid lipofuscinosisPolyneuropathyAmerican Journal of Medical Genetics
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Retina in various animal models of neuronal ceroid-lipofuscinosis

1992

The childhood forms of human neuronal ceroid-lipofuscinosis (NCL) are invariably associated with a severe progressive retinopathy which commences at the photoreceptor level morphologically and proceeds to a final loss of neuronal cells accompanied by severe gliosis. In respective spontaneous animal conditions of NCL, in English setters, Dalmatian dogs, and New Zealand sheep, retinal involvement is not commensurate although the retina does not seem to be completely unaffected. In canine NCL, there might be functional and electro-physiological impairment of retinal cells, but retinal atrophy is not obvious. In ovine NCL, the retina, apart from accumulating NCL-specific lipopigments within neu…

Retinal degenerationPathologymedicine.medical_specialtyBiologyRetinachemistry.chemical_compoundDogsNeuronal Ceroid-LipofuscinosesmedicineCarnivoraAnimalsPigment Epithelium of EyeGenetics (clinical)RetinaSheepRetinal DegenerationRetinalPigments BiologicalAnatomymedicine.diseaseLipidseye diseasesRetinal atrophyDisease Models Animalmedicine.anatomical_structurechemistryGliosisNeuronal ceroid lipofuscinosissense organsmedicine.symptomRetinopathyAmerican Journal of Medical Genetics
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205th ENMC International Workshop: Pathology diagnosis of idiopathic inflammatory myopathies Part II 28-30 March 2014, Naarden, The Netherlands.

2015

The idiopathic inflammatory myopathies (IM) are a heterogeneous group of diseases and diagnosis often necessitates a muscle biopsy. Five main entities are recognized: (1) dermatomyositis (DM); (2) polymyositis (PM); (3) necrotizing autoimmune myopathy (NAM); (4) sporadic inclusion body myositis (IBM); and (5) non-specific myositis. Other entities include granulomatous myopathy, macrophagic myofasciitis, and eosinophilic fasciitis (Shulman's syndrome). The pathological classification and subsequent identification of disease subgroups are extremely important for assessing treatment options and prognosis in the individual patient, yet classification criteria have not been standardized and vali…

2716 Genetics (clinical)medicine.medical_specialtyConsensusBiopsy10208 Institute of Neuropathology610 Medicine & healthPolymyositismedicineHumans2735 Pediatrics Perinatology and Child HealthColoring AgentsMyopathyGenetics (clinical)MyositisNetherlandsMuscle biopsyMyositismedicine.diagnostic_testbusiness.industryMusclesMacrophagic myofasciitisDermatomyositismedicine.diseaseDermatologyEosinophilic fasciitis2728 Neurology (clinical)Neurology2808 NeurologyPediatrics Perinatology and Child HealthPhysical therapy570 Life sciences; biologyNeurology (clinical)medicine.symptomInclusion body myositisbusinessNeuromuscular Disorders
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Significance of lipopigments with fingerprint profiles in eccrine sweat gland epithelial cells.

1995

Lipopigments with fingerprint profiles in eccrine sweat gland epithelial cells are regular findings in childhood NCL. They have also been described in adult NCL (ANCL) a few times, but not consistently. However, they have been considered nonspecific when not matched by similar abnormal profiles in noneccrine sweat gland epithelial cells. These conflicting reports may pose a diagnostic dilemma as outlined in the following 2 examples. Patient 1 is a 20-year-old man who developed severe tetraparesis and dementia over 2 years. Electroencephalogram was abnormal with epileptiform discharges. The patient died at age 21 years without autopsy ; no other relatives are known to have a similar disease.…

AdultMalemedicine.medical_specialtyPathologyAtaxiaAutopsyBiologyEccrine GlandsEpitheliumLipofuscinNeuronal Ceroid-LipofuscinosesSweat glandInternal medicinemedicineHumansEccrine sweat glandChildGenetics (clinical)Skinmedicine.diagnostic_testPigments BiologicalMiddle Agedmedicine.diseaseLipidsMicroscopy ElectronEndocrinologymedicine.anatomical_structureNeuronal ceroid lipofuscinosisFemalemedicine.symptomElectroretinographyRetinopathyAmerican journal of medical genetics
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