0000000000549117

AUTHOR

Chen W

showing 8 related works from this author

Production of Charmed Mesons In Z Decays

1994

The production of charmed mesons $$\mathop {D^0 }\limits^{( - )} $$ ,D ± , andD *± is studied in a sample of 478,000 hadronicZ decays. The production rates are measured to be $$\begin{gathered} \frac{{\Gamma ({\rm Z} \to D^{* \pm } X)}}{{\Gamma _{had} }} = 0.187 \pm 0.015(\exp .) \pm 0.013(BR), \hfill \\ \frac{{\Gamma ({\rm Z} \to D^ \pm X)}}{{\Gamma _{had} }} = 0.251 \pm 0.026(\exp .) \pm 0.025(BR), \hfill \\ \frac{{\Gamma ({\rm Z} \to \mathop {D^0 }\limits^{( - )} X)}}{{\Gamma _{had} }} = 0.518 \pm 0.052(\exp .) \pm 0.035(BR), \hfill \\ \end{gathered} $$ where the errors from this analysis are separated from those coming from theD branching ratios (BR). TheD *± momentum distribution is ex…

PhysicsParticle physicsE+E ANNIHILATIONPhysics and Astronomy (miscellaneous)Meson010308 nuclear & particles physicsElectron–positron annihilationRESONANCEALEPH01 natural sciencesCharm quarkPseudoscalarNuclear physicsALEPH Experiment0103 physical sciencesFragmentation function[PHYS.HEXP]Physics [physics]/High Energy Physics - Experiment [hep-ex]HEAVY QUARKSFRAGMENTATION010306 general physicsEngineering (miscellaneous)Beam energyALEPH experimentParticle Physics - Experiment
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Measurements of mean lifetime and branching fractions of b hadrons decaying to J/ψ

1992

From a data sample of 450 000 hadronic events recorded with the ALEPH detector at LEP, 92 +/- 10 events are observed containing a J/psi meson decaying to mu+mu- or e+e-. From these data the measured inclusive branching fraction for a b flavoured hadron to decay to a J/psi is BR(b --> J/psiX) = (1.21 +/- 0.13 (stat.) +/- 0.08 (syst.))%, and the average b hadron lifetime in the events tagged with a J/psi is tau(b) = 1.35(-0.17)+0.19 +/- 0.05 ps. Five events are observed consistent with the exclusive decay B+/- --> J/psiK+/- and from these events the exclusive branching fraction is measured to be BR(B+/- --> J/psiK+/-) = (0.22 +/- 0.10 +/- 0.02)%. Upper limits for other exclusive branching rat…

PhysicsNuclear and High Energy PhysicsParticle physicsMesonBranching fractionElectron–positron annihilationHadronBOSONLEPBranching (polymer chemistry)Nuclear physicsMass spectrumHigh Energy Physics::ExperimentAstrophysics::Earth and Planetary AstrophysicsANNIHILATIONParticle Physics - Experiment
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Genome-wide association and longitudinal analyses reveal genetic loci linking pubertal height growth, pubertal timing and childhood adiposity

2013

The pubertal height growth spurt is a distinctive feature of childhood growth reflecting both the central onset of puberty and local growth factors. Although little is known about the underlying genetics, growth variability during puberty correlates with adult risks for hormone-dependent cancer and adverse cardiometabolic health. The only gene so far associated with pubertal height growth, LIN28B, pleiotropically influences childhood growth, puberty and cancer progression, pointing to shared underlying mechanisms. To discover genetic loci influencing pubertal height and growth and to place them in context of overall growth and maturation, we performed genome-wide association meta-analyses i…

MaleNetherlands Twin Register (NTR)Genetic LinkageMedizinGene ExpressionGenome-wide association studyVARIANTSBody Mass Index0302 clinical medicinegenetic linkageTransforming Growth Factor betaNeoplasmsmolecular biologygeneticsChildGenetics (clinical)Adiposity2. Zero hunger0303 health sciencesadiposityMitogen-Activated Protein Kinase 3Association Studies ArticlesAge FactorsACHONDROPLASIAGeneral MedicineGenome-Wide Association Study; pubertal height growth; pubertal timingPhenotypeOBESITYMenarche/dk/atira/pure/sustainabledevelopmentgoals/good_health_and_well_beingbody heightFemaleSignal Transductionmedicine.medical_specialtyage factorsCHROMOSOME 16P11.2AdolescentBIRTHQuantitative Trait Loci030209 endocrinology & metabolismContext (language use)BiologyChildhood obesitypubertal height growthMENARCHEYoung Adult03 medical and health sciencesAGESDG 3 - Good Health and Well-beingPrepubertyInternal medicineGeneticsmedicine/dk/atira/pure/keywords/cohort_studies/netherlands_twin_register_ntr_HumansMolecular Biology030304 developmental biologySignMenarcheFACTOR RECEPTOR-3MUTATIONSpubertal timingPubertyta3121medicine.diseaseObesityBody HeightGenetic architectureEndocrinologyPOPULATION COHORTgene expressionBody mass indexFollow-Up StudiesGenome-Wide Association Study
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A measurement of the b baryon lifetime

1992

In 451 000 hadronic Z0 decays, recorded with the ALEPH detector at LEP, the yields of lambdal- and lambdal+ combinations are measured. Semileptonic decays of b baryons result in a signal of 122 +/- 18(stat.)+22/-23(syst.) lambdal- combinations. From a fit to the impact parameter distribution of the leptons in the lambdal- sample, the lifetime of b baryons is measured to be 1.12+0.32/-0.29(stat.) +/- 0.16(syst.) ps.

PhysicsNuclear and High Energy PhysicsAlephParticle physicsHEAVY FLAVOR PRODUCTIONElectron–positron annihilationSEMILEPTONIC DECAYSHigh Energy Physics::PhenomenologyHadronAstrophysics::Cosmology and Extragalactic AstrophysicsALEPHMESONSNuclear physicsBaryonHigh Energy Physics::ExperimentAstrophysics::Earth and Planetary AstrophysicsImpact parameterNuclear ExperimentQUARK-MODELParticle Physics - ExperimentLepton
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Heavy Flavor Production and Decay With Prompt Leptons In the Aleph Detector

1994

In 431 000 hadronicZ decays recorded with the ALEPH detector at LEP, the yields of electrons and muons in events with one or more prompt leptons have been analysed to give information on the production and decay of heavy quarks. The fractions of $$b\bar b$$ and $$c\bar c$$ events are measured to be 0.219±0.006±0.005 and 0.165±0.005±0.020, and the corresponding forward-backward asymmetries at theZ mass are measured to be 0.090±0.013±0.003 and 0.111±0.021±0.018, after QED and QCD corrections. Measurements for the semileptonic branching ratios BR $$(b \to \ell ^ - \bar vX)$$ and BR (b→cl+ vX) yield 0.114±0.003±0.004 and 0.082±0.003±0.012, respectively. The dilepton events enable measurement of…

QuarkParticle physicsPhysics and Astronomy (miscellaneous)Elementary particleheavy flavour01 natural sciencesALEPH ExperimentNuclear physics0103 physical sciences[PHYS.HEXP]Physics [physics]/High Energy Physics - Experiment [hep-ex]Information retrieval010306 general physicsEngineering (miscellaneous)ALEPH experimentQuantum chromodynamicsPhysicskwnoledge organisationMuon010308 nuclear & particles physicsBranching fractionALEPH Experiment; LEP; heavy flavourserendipityWeinberg angleLEPALEPH detectorParticle Physics - ExperimentLepton
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Search for CP violation in ZmF ττ

1992

Abstract Using the 18.8 pb−1 of data accumulated at LEP in 1990 and 1991 with the ALEPH detector, a direct test of neutral current CP-invariance is performed by a search for CP-odd correlations in Z decays to τ pairs where both τ decay modes are identified. No evidence for CP-violation is observed. The weak dipole moment of the τ has been measured to be dτ(mZ) = (1.3 ± 1.4 ± 0.1) × 10−17 e·cm which results in an upper limit on the weak dipole moment of |dτ(mZ)| ⩽ 3.7 × 10−17 e·cm with 95% confidence level.

PhysicsNuclear and High Energy PhysicsParticle physicsNeutral currentElectron–positron annihilationAPLANARITIEST-ODDNuclear physicsMoment (mathematics)DipoleTAU-LEPTONSDirect testTESTSCP violationE+E COLLIDERSZ-DECAYSPhysics Letters B
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No association between two polymorphisms of the serotonin transporter gene and combined type attention deficit hyperactivity disorder.

2008

Contains fulltext : 71091.pdf (Publisher’s version ) (Closed access) Several independent studies have reported association between serotonin transporter gene (SLC6A4) polymorphisms and attention deficit hyperactivity disorder (ADHD). Five studies found evidence for association between the long-allele of a 44-bp insertion/deletion polymorphism (5-HTTLPR) and ADHD. Another two studies corroborated this finding while a further six studies did not find such an association. For a second polymorphism within the gene, a variable number tandem repeat (VNTR) within intron 2, one study demonstrated that the 12/12 genotype was significantly less frequent in ADHD cases compared to controls, while a sec…

Genetics and epigenetic pathways of disease [NCMLS 6]MedizinNeuroinformatics [DCN 3]0302 clinical medicinePolymorphism (computer science)GenotypePerception and Action [DCN 1]Childhealth care economics and organizationsSerotonin transporterGenetics (clinical)GeneticsSerotonin Plasma Membrane Transport Proteins0303 health sciencesDepression030305 genetics & heredityTransmission disequilibrium testhumanitiesVariable number tandem repeatPsychiatry and Mental healthChild Preschool/dk/atira/pure/sustainabledevelopmentgoals/good_health_and_well_beingFunctional Neurogenomics [DCN 2]medicine.medical_specialtyAdolescenteducationBiologybehavioral disciplines and activitiesMental health [NCEBP 9]Genomic disorders and inherited multi-system disorders [IGMD 3]03 medical and health sciencesCellular and Molecular NeuroscienceSDG 3 - Good Health and Well-beingCognitive neurosciences [UMCN 3.2]Internal medicinemental disordersmedicineAttention deficit hyperactivity disorderHumansGenetic Predisposition to Diseaseddc:610Medizinische Fakultät » Universitätsklinikum Essen » LVR-Klinikum Essen » Klinik für Psychiatrie Psychosomatik und Psychotherapie des Kindes- und JugendaltersAssociation (psychology)Gene030304 developmental biologyFamily HealthPolymorphism Geneticbusiness.industryMood Disordersmedicine.diseaseMoodEndocrinologyMood disordersGenetic defects of metabolism [UMCN 5.1]Attention Deficit Disorder with Hyperactivitybiology.proteinbusiness030217 neurology & neurosurgery
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The analysis of 51 genes in DSM-IV combined type attention deficit hyperactivity disorder: association signals in DRD4, DAT1 and 16 other genes.

2006

Contains fulltext : 35205.pdf (Publisher’s version ) (Closed access) Attention deficit hyperactivity disorder (ADHD) is a common neurodevelopmental disorder, starting in early childhood and persisting into adulthood in the majority of cases. Family and twin studies have demonstrated the importance of genetic factors and candidate gene association studies have identified several loci that exert small but significant effects on ADHD. To provide further clarification of reported associations and identify novel associated genes, we examined 1,038 single-nucleotide polymorphisms (SNPs) spanning 51 candidate genes involved in the regulation of neurotransmitter pathways, particularly dopamine, nor…

Candidate geneGenetics and epigenetic pathways of disease [NCMLS 6]MedizinReceptors NicotinicTryptophan HydroxylaseNeuroinformatics [DCN 3]0302 clinical medicinePerception and Action [DCN 1]Determinants in Health and Disease [EBP 1]ChildOncogene ProteinsGenetics0303 health sciencesbiologyDNA POOLING ANALYSISPedigree3. Good healthserotoninPsychiatry and Mental healthConduct disorderChild Preschool/dk/atira/pure/sustainabledevelopmentgoals/good_health_and_well_beingMonoamine oxidase AdopaminePsychologyFunctional Neurogenomics [DCN 2]Genetic MarkersAdolescentSynaptosomal-Associated Protein 25Single-nucleotide polymorphismassociation studyPolymorphism Single NucleotideMental health [NCEBP 9]Genetic determinismGenomic disorders and inherited multi-system disorders [IGMD 3]03 medical and health sciencesCellular and Molecular NeuroscienceMONOAMINE-OXIDASE-ACognitive neurosciences [UMCN 3.2]SDG 3 - Good Health and Well-beingmental disordersmedicineHumansAttention deficit hyperactivity disorderADHDGenetic Predisposition to Disease5-HT1B RECEPTOR GENEddc:610Medizinische Fakultät » Universitätsklinikum Essen » LVR-Klinikum Essen » Klinik für Psychiatrie Psychosomatik und Psychotherapie des Kindes- und JugendaltersMonoamine OxidaseMolecular Biology030304 developmental biologyGenetic associationDopamine Plasma Membrane Transport ProteinsSEROTONIN TRANSPORTER GENEDOPAMINE-BETA-HYDROXYLASESiblingsReceptors Dopamine D4candidate genemedicine.diseaseTwin studyPREFERENTIAL TRANSMISSIONHaplotypesCATECHOL-O-METHYLTRANSFERASEAttention Deficit Disorder with HyperactivityCONDUCT DISORDERbiology.proteinnoradrenalineDEFICIT/HYPERACTIVITY DISORDERNO EVIDENCE030217 neurology & neurosurgerylinkage disequilibriumMolecular Psychiatry
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