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RESEARCH PRODUCT

Adipokines and Endotoxemia Correlate with Hepatic Steatosis in Non-Alcoholic Fatty Liver Disease (NAFLD)

Jörn M. SchattenbergMaurice MichelAnika NierIna BergheimYvonne HuberChristian Labenz

subject

Dietary FiberMale0301 basic medicinemedicine.medical_specialtyAdipokinelcsh:TX341-641GastroenterologyArticle03 medical and health sciences0302 clinical medicineAdipokinesRisk FactorsInternal medicineHumansMedicinehepatic fibrosisNutrition and DieteticsIntestinal permeabilityAdiponectinbusiness.industryLeptinFatty livernon-alcoholic fatty liver diseasedietary fiber consumptionnutritional and metabolic diseasesMiddle Agedmedicine.diseaseEndotoxemiadigestive system diseasesDietFatty Liver030104 developmental biologyCase-Control StudiesFemale030211 gastroenterology & hepatologyDisease Susceptibilitybacterial endotoxinInflammation MediatorsSteatosisbusinessHepatic fibrosislcsh:Nutrition. Foods and food supplyBody mass indexBiomarkersFood Science

description

(1) Background: The etiology of non-alcoholic fatty liver disease (NAFLD) is multifactorial. Dietary composition has been implicated as a factor modulating intestinal barrier and could affect disease severity. The aim of this study was to evaluate dietary intake and markers of intestinal permeability in patients with NAFLD. (2) Methods: We enrolled 63 patients with NAFLD and compared them to age-matched controls. (3) Results: body mass index (BMI) and leptin to adiponectin ratio&mdash

https://doi.org/10.3390/nu12030699