Search results for " ALPHA"

showing 10 items of 1610 documents

PAR1 signaling regulates the retention and recruitment of EPCR-expressing bone marrow hematopoietic stem cells

2015

Retention of long-term repopulating hematopoietic stem cells (LT-HSCs) in the bone marrow is essential for hematopoiesis and for protection from myelotoxic injury. We report that signaling cascades that are traditionally viewed as coagulation related also control retention of endothelial protein C receptor-positive (EPCR(+)) LT-HSCs in the bone marrow and their recruitment to the blood via two pathways mediated by protease activated receptor 1 (PAR1). Thrombin-PAR1 signaling induces nitric oxide (NO) production, leading to EPCR shedding mediated by tumor necrosis factor-α-converting enzyme (TACE), enhanced CXCL12-CXCR4-induced motility and rapid stem and progenitor cell mobilization. Conver…

Receptors CXCR4Receptors Cell SurfaceADAM17 ProteinIntegrin alpha4beta1BiologyNitric OxideArticleGeneral Biochemistry Genetics and Molecular BiologyMiceBone MarrowCell MovementCell AdhesionmedicineAnimalsReceptor PAR-1Progenitor cellcdc42 GTP-Binding ProteinCell adhesionEndothelial protein C receptorThrombinEndothelial Protein C ReceptorGeneral MedicineHematopoietic Stem CellsChemokine CXCL12Cell biologyMice Inbred C57BLTransplantationADAM ProteinsHaematopoiesismedicine.anatomical_structureCdc42 GTP-Binding ProteinImmunologyBone marrowStem cellProtein CSignal TransductionNature Medicine
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Adhesion of 8701-BC breast cancer cells to type V collagen and 67 kDa receptor

1992

Ductal infiltration carcinomas (d.i.c.) of the breast are potentially highly metastatic tumours, associated with drastic alterations of the architecture and molecular composition of the extracellular matrix at the tumour-host interface. 8701-BC, a recently characterized cell line, isolated from primary d.i.c., was used to study different aspects of tumor cell-substratum interactions. Since type V collagen deposition is augmented in d.i.c. we have examined the ability of 8701-BC cells to interact with this collagen species. We have found that cell binding to type V collagen was mediated by protein homologous to the 67 kDa laminin receptor (67-R). This conclusion is substantiated by the follo…

Receptors CollagenbiologyIntegrinMammary Neoplasms ExperimentalLactoseReceptors Cell SurfaceCell BiologyMolecular biologyChromatography AffinityCollagen receptorExtracellular matrixCollagen type I alpha 167 kDa Laminin ReceptorMembrane proteinCell AdhesionTumor Cells Culturedbiology.proteinAnimalsCollagenCell adhesionReceptorJournal of Cell Science
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Xanthine dehydrogenase processes retinol to retinoic acid in human mammary epithelial cells

2008

Retinoic acid is considered to be the active metabolite of retinol, able to control differentiation and proliferation of epithelia. Retinoic acid biosynthesis has been widely described with the implication of multiple enzymatic activities. However, our understanding of the cell biological function and regulation of this process is limited. In a recent study we evidenced that milk xanthine oxidase (E.C. 1.17.3.2.) is capable to oxidize all-trans-retinol bound to CRBP (holo-CRBP) to all-trans-retinaldehyde and then to all-trans-retinoic acid. To get further knowledge regarding this process we have evaluated the biosynthetic pathway of retinoic acid in a human mammary epithelial cell line (HME…

Receptors Retinoic AcidXanthine dehydrogenaseCellRetinoic acidOxypurinolTretinoinRetinoic acid receptor betaBiologychemistry.chemical_compoundSettore BIO/10 - BiochimicaDrug DiscoverymedicineHumansMammary Glands HumanVitamin AXanthine oxidaseHMECPharmacologyRetinolEpithelial CellsRetinol-Binding Proteins CellularGeneral MedicineMilk ProteinsNADRetinoic acid receptormedicine.anatomical_structurechemistryBiochemistryXanthine dehydrogenaseRetinol oxidationRetinoic acid receptor alphaRetinoid AcidMetabolic Networks and PathwaysJournal of Enzyme Inhibition and Medicinal Chemistry
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Intercellular communication and human hepatocellular carcinoma.

2005

We have previously reported that gap junction-mediated intercellular communication (GJIC) can be restored in junctionally deficient human prostate epithelial cells, also suggesting that GJIC activity is regulated by estrogen. In the present work, we report studies on sex steroid regulation of GJIC and proliferative activity in both nontumoral (Chang liver, CL) and malignant (HepG2, Huh7) human liver cells. Junctional activity and liver cell growth were measured using the scrape-loading/dye-transfer (SL/DT) and the MTS assay, respectively. Using the SL/DT, only Huh7 cells exhibited a moderate degree of Junctional activity in basic conditions, while neither CL nor HepG2 cells showed functiona…

Receptors SteroidTime FactorsProliferationCell Communicationchemistry.chemical_compoundNeoplasmsReceptorTumorGeneral NeuroscienceLiver cellLiver NeoplasmsGap JunctionsGap junction-mediated intercellular communication (GJIC)ImmunohistochemistryLiverLiver NeoplasmReceptors AndrogenGap JunctionReceptors ProgesteroneHumanmedicine.medical_specialtyCell signalingCarcinoma HepatocellularTime Factormedicine.drug_classEstroneBiologyGeneral Biochemistry Genetics and Molecular BiologyCell LineHistory and Philosophy of ScienceInternal medicineCell Line TumormedicineCarcinomaEstrogen Receptor betaHumansHepatocellular carcinoma (HCC)SteroidCell ProliferationBiochemistry Genetics and Molecular Biology (all)Cell growthEstrogen Receptor alphamedicine.diseasedigestive system diseasesEndocrinologychemistryEstrogenCell cultureCancer researchNeoplasmAnnals of the New York Academy of Sciences
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Species-specific mechanisms for cholesterol 7alpha-hydroxylase (CYP7A1) regulation by drugs and bile acids.

2005

The gene encoding cholesterol 7alpha-hydroxylase (CYP7A1) is tightly regulated in order to control intrahepatic cholesterol and bile acid levels. Ligands of the xenobiotic-sensing pregnane X receptor inhibit CYP7A1 expression. To retrace the evolution of the molecular mechanisms underlying CYP7A1 inhibition, we used a chicken hepatoma cell system that retains the ability to be induced by phenobarbital and other drugs. Whereas bile acids regulate CYP7A1 via small heterodimer partner and liver receptor homolog-1, mRNA expression of these nuclear receptors is unchanged by xenobiotics. Instead, drugs repress chicken hepatic nuclear factor 4alpha (HNF4alpha) transcript levels concomitant with a …

Receptors Steroidmedicine.drug_classMolecular Sequence DataBiophysicsReceptors Cytoplasmic and NuclearBiologyIn Vitro TechniquesCholesterol 7 alpha-hydroxylaseBiochemistryGene Expression Regulation EnzymologicBile Acids and SaltsMiceSpecies SpecificitymedicineAnimalsHumansRNA MessengerCholesterol 7-alpha-HydroxylaseMolecular BiologyCells CulturedMice KnockoutPregnane X receptorBile acidLiver receptor homolog-1Pregnane X ReceptorPhosphoproteinsRecombinant ProteinsDNA-Binding ProteinsBiochemistryNuclear receptorHepatocyte Nuclear Factor 4PhenobarbitalSmall heterodimer partnerHepatocytesFarnesoid X receptorSignal transductionChickensSignal TransductionTranscription FactorsArchives of biochemistry and biophysics
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Soluble interleukin-6 receptor (sIL-6R) makes IL-6R negative T cell line respond to IL-6: it inhibits TNF production

2000

The receptor for interleukin-6 (IL-6) consists of two subunits: a ligand specific IL-6Ralpha and gp130 that is responsible for signal-transduction. A soluble form of the ligand specific chain was described that when complexed to IL-6 is capable of binding to the membrane-bound gp130 subunit and thus can elicit signal-transduction. This soluble receptor can act on cells that express only the gp130 but not the ligand-specific subunit of the IL-6R. This phenomenon, called trans-signaling, introduced a novel aspect of cytokine action. In this study we examined the response of Jurkat cells, that are known not to express IL-6Ralpha, to IL-6, the soluble IL-6 receptor (sIL-6R) and a covalent compl…

Recombinant Fusion ProteinsT-Lymphocytesmedicine.medical_treatmentT cellImmunologyBiologyJurkat cellsCell LineInterferon-gammaJurkat CellsAntigens CDCytokine Receptor gp130medicineHumansImmunology and AllergyInterferon gammaElméleti orvostudományokReceptorLymphotoxin-alphaMembrane GlycoproteinsInterleukin-6Tumor Necrosis Factor-alphaReceptors InterleukinOrvostudományokLigand (biochemistry)Glycoprotein 130Receptors Interleukin-6Molecular biologyCytokinemedicine.anatomical_structureSolubilityTumor necrosis factor alphamedicine.drug
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Functional regulation of HIF-1α under normoxia--is there more than post-translational regulation?

2011

The hypoxia-inducible factor-1 (HIF-1) is an oxygen-regulated transcriptional activator playing a pivotal role in mammalian physiology and disease pathogenesis, e.g., HIF-1 is indispensable in a broad range of developmental stages in different tumors. Its post-translational regulation via PHDs under the influence of hypoxia is widely investigated and accepted. Different non-hypoxic stimuli such as lipopolysaccharides (LPS), thrombin, and angiotensin II (Ang II), have been proven to enhance HIF-1 levels through activation of regulative mechanisms distinct from protein stabilization. Some of these stimuli specifically regulate HIF-1α at the transcriptional, post-transcriptional, or translatio…

Regulation of gene expressionMammalsHypoxia-Inducible Factor 1PhysiologyClinical BiochemistryCell BiologyHypoxia (medical)BiologyDisease pathogenesisHypoxia-Inducible Factor 1 alpha SubunitAngiotensin IICell biologyOxygenThrombinBiochemistryGene Expression RegulationmedicineAnimalsPost-translational regulationmedicine.symptomProtein stabilizationmedicine.drugJournal of cellular physiology
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Large scale preparation of human MHC class II+ integrin beta(1)+ Tregs.

2010

Abstract The human CD4 + CD25 + FoxP3 + regulatory T cell population (Tregs) contains both MHC class II + and MHC class II − cells. MHC class II + Tregs belong to the integrin α 4 β 1 + subpopulation and exclusively execute contact-dependent suppressive activity. Here we present a method optimized for isolation of these MHC class II expressing Tregs from large leukaphereses products using magnetic microbeads that achieves a reproducible purity of more than 90% and enables the use of this small-sized Treg population in pre-clinical application and basic research.

Regulatory T cellImmunologyPopulationIntegrinchemical and pharmacologic phenomenaIntegrin alpha4beta1T-Lymphocytes RegulatoryT-Lymphocyte SubsetsmedicineImmune ToleranceImmunology and AllergyHumansIL-2 receptorLeukapheresiseducationCells CulturedMHC class IIeducation.field_of_studybiologyImmunomagnetic SeparationHistocompatibility Antigens Class IIInterleukin-2 Receptor alpha SubunitFOXP3hemic and immune systemsForkhead Transcription FactorsT lymphocyteMHC restrictionFlow CytometryCell biologyHigh-Throughput Screening Assaysmedicine.anatomical_structureImmunologyCD4 Antigensbiology.proteinJournal of immunological methods
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Revista electrónica de investigación y evaluación educativa

2016

Este estudio evalúa un procedimiento nuevo para el análisis “micro-sociométrico” de los patrones relacionales de género en el alumnado adolescente, lo que se analiza aplicando Análisis de Correspondencias Múltiples (ACM). El propósito del estudio es desarrollar y validar la Escala de Preferencias Relacionales de Género (EPRG), con el fin de obtener datos válidos y fiables que permitan reconocer la predisposición de las chicas y los chicos adolescentes a relacionarse con sus iguales; y, si las relaciones que establecen están condicionadas por la socialización diferencial de género que propone el patriarcado (Bosch, Ferrer & Alzamora, 2006; Lagarde, 1996; Lomas, 2007; Lorente, 2007; Rodríguez…

Reliability & ValidityEducaciónrelaciones intergrupalesPatriarchyMultiple Correspondence AnalysisScalesAnálisis de Correspondencias MúltiplesEducationDevelopmental psychologyFiabilidad y Validezrol sexualCronbach's alphaMultiple correspondence analysissocializaciónSocial DistanceUNESCO::PEDAGOGÍAReliability (statistics)Gender DifferencesAnálisis SociométricoSocial distanceSocializationCoeducationConstruct validityDistancia SocialEducational AssessmentCoeducaciónDiferencias de GéneroPeer RelationsAdolescenceScale (social sciences)AdolescenciaSociometric Analysisdiferencia de sexoEscalasPsicología y educaciónDiagnóstico en Educación:PEDAGOGÍA [UNESCO]PsychologySocial psychologyRelaciones entre IgualesRELIEVE - Revista Electrónica de Investigación y Evaluación Educativa
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Activation of Mast Cells by Streptolysin O and Lipopolysaccharide

2005

This chapter provides protocols to measure the reversible permeabilization of mast cells by streptolysin O (SLO) and to follow SLO-induced activation of mast cells by monitoring degranulation, activation of mitogen-activated protein kinases, and production of tumor necrosis factor-alpha. A method that uses SLO to deliver molecules into the cytosol of living cells also is described. Furthermore, we outline a procedure to measure the activation of nuclear factor-kappaB by lipopolysaccharide and ionomycin using transfection of mast cells with reporter genes by electroporation. These protocols should be widely applicable in mast cell research.

Reporter genegenetic structuresElectroporationDegranulationTransfectionMast cellCell biologychemistry.chemical_compoundmedicine.anatomical_structurechemistryIonomycinmedicineTumor necrosis factor alphaStreptolysin
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