Search results for " GABA"

showing 10 items of 127 documents

What is the form of muscimol from fly agaric mushroom (Amanita muscaria) in water? An insight from NMR experiment supported by molecular modeling.

2020

ARTYKUŁ Z BADAŃ SPECJALNYCH

Models MolecularMagnetic Resonance SpectroscopyAmanita010402 general chemistry01 natural scienceschemistry.chemical_compoundReceptors GABAComputational chemistryMoleculeHumansGeneral Materials ScienceGABA-A Receptor Antagonistsindirect spin‐spin coupling constantsNMR in waterDensity Functional TheoryCarbon Isotopesbiology010405 organic chemistryAgaricmolecular modelingMuscimolChemical shiftWaterGeneral ChemistryCarbon-13 NMRbiology.organism_classification0104 chemical sciencesSolventnervous systemchemistryMuscimolGIAO NMRZwitterionProtonsAmanita muscariaMagnetic resonance in chemistry : MRCREFERENCES
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Synthesis of GABAA receptor agonists and evaluation of their alpha-subunit selectivity and orientation in the GABA binding site.

2008

Drugs used to treat various disorders target GABA A receptors. To develop alpha subunit selective compounds, we synthesized 5-(4-piperidyl)-3-isoxazolol (4-PIOL) derivatives. The 3-isoxazolol moiety was substituted by 1,3,5-oxadiazol-2-one, 1,3,5-oxadiazol-2-thione, and substituted 1,2,4-triazol-3-ol heterocycles with modifications to the basic piperidine substituent as well as substituents without basic nitrogen. Compounds were screened by [(3)H]muscimol binding and in patch-clamp experiments with heterologously expressed GABA A alpha ibeta 3gamma 2 receptors (i = 1-6). The effects of 5-aminomethyl-3 H-[1,3,4]oxadiazol-2-one 5d were comparable to GABA for all alpha subunit isoforms. 5-pipe…

Models MolecularPatch-Clamp TechniquesStereochemistryAlpha (ethology)gamma-Aminobutyric acidArticleGABAA-rho receptorCell Linechemistry.chemical_compoundStructure-Activity RelationshipXenopus laevisPiperidinesDrug DiscoverymedicineAnimalsHumansGABA-A Receptor AgonistsBinding siteReceptorgamma-Aminobutyric AcidG alpha subunitBinding SitesMolecular StructureChemistryGABAA receptorMuscimolBrainIsoxazolesReceptors GABA-ARatsElectrophysiologyProtein SubunitsBiochemistryMuscimolMutationOocytesMolecular MedicineFemalemedicine.drugJournal of medicinal chemistry
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Putative excitatory and putative inhibitory inputs are localised in different dendritic domains in aDrosophilaflight motoneuron

2012

Input-output computations of individual neurons may be affected by the three-dimensional structure of their dendrites and by the targeting of input synapses to specific parts of their dendrites. However, only few examples exist where dendritic architecture can be related to behaviorally relevant computations of a neuron. By combining genetic, immunohistochemical, and confocal laser scanning methods this study estimates the location of the spike initiating zone and the dendritic distribution patterns of putative synaptic inputs on an individually identified Drosophila flight motorneuron, MN5. MN5 is a monopolar neuron with more than 4000 dendritic branches. The site of spike initiation was e…

Motor NeuronsDendritic spikeGABAA receptorGeneral NeuroscienceAction PotentialsDendritesVoltage-Gated Sodium ChannelsReceptors NicotinicBiologyReceptors GABA-AInhibitory postsynaptic potentialArticleTonic (physiology)SynapseProtein TransportDrosophila melanogastermedicine.anatomical_structureSynapsesmedicineExcitatory postsynaptic potentialAnimalsDrosophila ProteinsGABAergicNeuronNeuroscienceEuropean Journal of Neuroscience
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NITRIC OXIDE-ACTIVE COMPOUNDS MODULATE IN VIVO GABA-EVOKED RESPONSES IN THE GLOBUS PALLIDUS OF RAT

2012

Nitric oxide (NO) acts as a messenger in the central nervous system; it affects the synaptic activity by modulating neurotransmitter release and/or receptor function. We previously observed that NO-active compounds modify the bioelectric activity of basal ganglia (BG) units. In this study, we applied microiontophoresis to extracellular in vivo recordings to investigate the effect of NO-active compounds on GABA-evoked responses in the globus pallidus (GP) of rats. The response to GABA release was tested on recorded GP neurons before and during the administration of S-nitroso-glutathione (SNOG, NO donor) and/or Nω-nitro-L-arginine methyl ester (L-NAME), inhibitor of nitric oxide synthase (NOS…

Nitric oxide Basal ganglia Globus pallidus Microiontophoresis GABA transmissionSettore BIO/09 - Fisiologia
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Kavapyrone enriched extract fromPiper methysticum as modulator of the GABA binding site in different regions of rat brain

1994

Regional differences in the modulation of [3H] muscimol binding to GABAA receptor complexes by kavapyrones, compounds of the rhizome of the plant Piper methysticum which possess sedative activity, were demonstrated using membrane fractions obtained from target brain centers of kavapyrone action: hippocampus (HIP), amygdala (AMY) and medulla oblongata (MED), and from brain centers outside the main kavapyrone effects as frontal cortex (FC) and cerebellum (CER). The kava extract enhanced the binding of [3H] muscimol in a concentration-dependent manner with maximal potentiation of 358% over control in HIP followed by AMY and MED (main target brain centers). Minimal stimulation was observed in C…

OvariectomyStimulationIn Vitro TechniquesPharmacologyBiologyBinding CompetitiveRats Sprague-Dawleychemistry.chemical_compoundReceptors GABAmedicineAnimalsBinding siteKavainReceptorPentobarbitalBrain ChemistryPharmacologyDiazepamPlants MedicinalMuscimolPlant ExtractsGABAA receptorLong-term potentiationRatsnervous systemMechanism of actionMuscimolchemistryPyronesFemaleSteroidsmedicine.symptomNeurosciencePsychopharmacology
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GABA-A Receptors Regulate Neocortical Neuronal Migration In Vitro and In Vivo

2006

The cortical migration process depends on a number of trophic factors and on the activation of different voltage- and ligand-gated channels. We investigated the role of gamma-aminobutyric acid (GABA) type A receptors in the neuronal migration process of the newborn rat parietal cortex in vivo and in vitro. Local in vivo application of the GABA-A antagonist bicuculline methiodide (BMI) or the agonist muscimol via cortical surface Elvax implants induced prominent alterations in the cortical architecture when compared with untreated or sham-operated controls. BMI- and muscimol-treated animals revealed heterotopic cell clusters in the upper layers and a complete loss of the cortical lamination …

Patch-Clamp TechniquesAntimetabolitesCognitive NeuroscienceNeocortexIn Vitro TechniquesBiologyBicucullineReceptors N-Methyl-D-Aspartategamma-Aminobutyric acidGABA AntagonistsCellular and Molecular Neurosciencechemistry.chemical_compoundCell MovementPostsynaptic potentialGlial Fibrillary Acidic ProteinmedicineAnimalsRats WistarReceptorGABA AgonistsDrug ImplantsNeuronsMuscimolGABAA receptorBicucullineReceptors GABA-AImmunohistochemistryRatsCell biologyElectrophysiologyKineticsmedicine.anatomical_structureAnimals NewbornBromodeoxyuridinenervous systemMuscimolchemistryCerebral cortexGABAergicCalciumNeurosciencemedicine.drugCerebral Cortex
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Effect of depolarizing GABAA-mediated membrane responses on excitability of Cajal-Retzius cells in the immature rat neocortex

2011

In immature neurons activation of ionotropic GABA receptors induces depolarizing membrane responses due to a high intracellular Cl− concentration ([Cl−]i). However, it is difficult to draw conclusions about the functional consequences of subthreshold GABAergic depolarizations, since GABAergic membrane shunting and additional effects on voltage-dependent ion channels or action potential threshold must be considered. To systematically investigate factors that determine the GABAergic effect on neuronal excitability we performed whole cell patch-clamp recordings from Cajal-Retzius cells in immature rat neocortex, using [Cl−]i between 10 and 50 mM. The effect of focal GABA application was quant…

Patch-Clamp TechniquesPhysiologyModels NeurologicalAction PotentialsDifferential ThresholdNeocortexMembrane PotentialsGABA AntagonistsChloridesInterneuronsmedicineAnimalsPatch clampGABAergic NeuronsRats WistarReceptorgamma-Aminobutyric AcidNeocortexGABAA receptorChemistryGeneral NeuroscienceReceptors GABA-ARatsPyridazinesRheobasemedicine.anatomical_structureAnimals NewbornIon Channel GatingNeuroscienceShunting inhibitionIntracellularIonotropic effectJournal of Neurophysiology
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GABA and GABA receptors in the gastrointestinal tract: from motility to inflammation

2015

Although an extensive body of literature confirmed γ-aminobutyric acid (GABA) as mediator within the enteric nervous system (ENS) controlling gastrointestinal (GI) function, the true significance of GABAergic signalling in the gut is still a matter of debate. GABAergic cells in the bowel include neuronal and endocrine-like cells, suggesting GABA as modulator of both motor and secretory GI activity. GABA effects in the GI tract depend on the activation of ionotropic GABAA and GABAC receptors and metabotropic GABAB receptors, resulting in a potential noteworthy regulation of both the excitatory and inhibitory signalling in the ENS. However, the preservation of GABAergic signalling in the gut …

PharmacologyChemistryGABAA receptorGABAB receptorPharmacologyInflammatory Bowel DiseasesInhibitory postsynaptic potentialSettore BIO/09 - FisiologiaGastrointestinal TractMetabotropic receptorReceptors GABAGABA receptorAnimalsHumansGABAergicEnteric nervous systemGastrointestinal MotilityGABA • GABAA receptors • GABAB receptors • Gastrointestinal motility • Inflammationgamma-Aminobutyric Acid5-HT receptorPharmacological Research
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GABAC receptors are functionally expressed in the intermediate zone and regulate radial migration in the embryonic mouse neocortex

2010

Radial neuronal migration in the cerebral cortex depends on trophic factors and the activation of different voltage- and ligand-gated channels. To examine the func- tional role of GABAC receptors in radial migration we ana- lyzed the effects of specific GABAA and GABAC receptor antagonists on the migration of BrdU-labeled neurons in vitro using organotypic neocortical slice cultures. These experi- ments revealed that the GABAA specific inhibitor bicuculline methiodide facilitated neuronal migration, while the GABAC specific inhibitor (1,2,5,6-tetrahydropyridine-4-yl) methylphos- phinic-acid (TPMPA) impeded migration. Co-application of TPMPA and bicuculline methiodide or the unspecific ionot…

PyridinesNeocortexIn Vitro TechniquesBiologyBicucullineGABAA-rho receptorGABA AntagonistsMicechemistry.chemical_compoundReceptors GABACell MovementmedicineAnimalsPicrotoxinGABA-A Receptor AntagonistsRNA MessengerReceptorGABA AgonistsNeuronsNeocortexGABAA receptorGeneral NeuroscienceGABA receptor antagonistReceptors GABA-APhosphinic AcidsCell biologyMice Inbred C57BLmedicine.anatomical_structurechemistryCrotonatesGABAergicNeurosciencePicrotoxinIonotropic effectNeuroscience
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Inhibition of GABA and benzodiazepine receptor binding by penicillins.

1980

Penicillins are thought to be GABA receptor antagonists. In order to determine the affinities of various penicillin derivatives for the GABA receptor, their potencies as inhibitors of specific [3H]GABA binding to rat brain membranes were investigated. All investigated penicillins inhibit specific [3H]GABA binding, with IC50 values ranging from 2 to 60 mM. The results are consistent with the assumption that penicillins are weak GABA receptor antagonists.

Receptors Cell SurfaceFlunitrazepamPenicillinsPharmacologygamma-Aminobutyric acidBenzodiazepinesStructure-Activity RelationshipGABA receptorpolycyclic compoundsmedicineStructure–activity relationshipAnimalsgamma-Aminobutyric AcidBenzodiazepine receptor bindingChemistryGeneral NeuroscienceBrainGABA receptor antagonistReceptors GABA-AAffinitiesRatsPenicillinnervous systemBiochemistryFlunitrazepammedicine.drugNeuroscience letters
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