Search results for " Growth factors"

showing 10 items of 100 documents

Intravitreal dexamethasone implant versus anti-vascular endothelial growth factor therapy combined with cataract surgery in patients with diabetic ma…

2021

Objective: to compare outcomes of cataract surgery combined with either anti-Vascular Endothelial Growth Factor (anti-VEGF) therapy or dexamethasone implant (DEX) in patients with diabetic macular oedema (DMO). Methods: Pubmed and Embase databases were searched for studies reporting outcomes of diabetic cataract surgery combined with either anti-VEGF or DEX, with a follow-up ≥3 months. The primary outcome was the mean change in central macular thickness (CMT). Mean change in best corrected visual acuity (BCVA) was considered as a secondary outcome. The mean difference between baseline and post-treatment values (MD) with 95%-Confidence Interval (95%CI) was calculated and meta-analyses were p…

Vascular Endothelial Growth Factor Amedicine.medical_specialtygenetic structuresmedicine.medical_treatmentVisual AcuityEndothelial Growth FactorsMacular EdemaDexamethasoneCataractOphthalmologyDiabetes MellitusHumansMedicineIn patientDexamethasoneDiabetic Retinopathybusiness.industryDEXcataract surgeryCataract surgeryDiabetic cataractBevacizumabOphthalmologyAnti–vascular endothelial growth factor therapyDiabetic macular oedemaanti-VEGFdiabetic macular oedemaMeta-analysissense organsImplantbusinessmedicine.drugEye
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Early OCTA Changes of Type 3 Macular Neovascularization Following Brolucizumab Intravitreal Injections

2022

Background and Objectives: Brolucizumab is a novel anti-vascular endothelial growth factor (VEGF), whose efficacy has been shown in the Hawk and Harrier phase 3 clinical studies. The goal of the present case series is to report initial results of brolucizumab intravitreal injections (IVI) on type 3 neovascularization in neovascular age-related macular degeneration (nAMD), evaluated by optical coherence tomography angiography (OCTA). Materials and Methods: This is a bicentric retrospective case series. Patients with newly diagnosed type 3 MNV treated with brolucizumab IVI and at least 6 months follow-up were enrolled. OCTA en face images and B-scans were analyzed for lesions at baseline, 1 m…

Vascular Endothelial Growth Factor Atype 3 neovascularization.brolucizumabNeovascularization PathologicVisual Acuityneovascular age-related macular degeneration; type 3 neovascularization; retinal angiomatous proliferation; brolucizumab; optical coherence tomography angiography; intravitreal injectionintravitreal injectionretinal angiomatous proliferationEndothelial Growth Factorsneovascular age-related macular degenerationGeneral MedicineAntibodies Monoclonal Humanizedoptical coherence tomography angiographyIntravitreal InjectionsHumansFluorescein AngiographyTomography Optical CoherenceRetrospective StudiesMedicina
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Longitudinal Study of Cytokine Expression, Lipid Profile and Neuronal Growth Factors in Human Breast Milk from Term and Preterm Deliveries

2015

Breast milk (BM) is considered as a reference for infant nutrition. The role of bioactive components, such as cytokines, hormones, growth factors (GFs) and fatty acids (FAs) is poorly known, but they might be implicated in immune response development. The aim of this study was to identify the lipid profile and the spectrum of cytokines and neuronal GF in BM samples and analyse the influence of gestational age and lactation time on these components. This study used a longitudinal prospective method for the characterization of cytokines, FAs and GFs global profiles in 120 BM samples from 40 healthy mothers (20 preterm and 20 term) collected as colostrum, transitional and mature milk. The cyto…

breastfeedingmedicine.medical_treatmentNeurotrophic factorsPregnancyLactationneurotrophic factorNutrition and Dieteticsmedicine.diagnostic_testHuman milkhuman milkdocosahexaenoic acidDocosahexaenoic acidmedicine.anatomical_structureCytokineBreast FeedingDocosahexaenoic acidCytokinesbreast milkFemaleGrowth factorslcsh:Nutrition. Foods and food supplyInfant Prematurepolyunsaturated fatty acidsAdultmedicine.medical_specialtyBreast milkBreastfeedingNeurotrophic factorlcsh:TX341-641Gestational AgeBiologyBreast milkfatty acidsArticleInternal medicinegrowth factorsFatty Acids Omega-3medicineHumansLactationNerve Growth FactorsFatty acidsMilk HumanInfant Newbornhuman milk; breast milk; breastfeeding; fatty acids; cytokines; growth factors; neurotrophic factor; polyunsaturated fatty acids; docosahexaenoic acidcytokinesEndocrinologyColostrumPolyunsaturated fatty acidsLipid profileBreast feedingFood ScienceNutrients
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Estrogens and Stem Cells in Thyroid Cancer

2014

Recent discoveries highlight the emerging role of estrogens in the initiation and progression of different malignancies through their interaction with stem cell compartment. Estrogens play a relevant role especially for those tumors bearing a gender disparity in incidence and aggressiveness, as occurs for most thyroid diseases. Although several experimental lines suggest that estrogens promote thyroid cell proliferation and invasion, their precise contribution in stem cell compartment still remains unclear. This review underlines the interplay between hormones and thyroid function, which could help to complete the puzzle of gender discrepancy in thyroid malignancies. Defining the associatio…

cancer stem cellsendocrine systemendocrine system diseasesMini ReviewEndocrinology Diabetes and MetabolismEstrogen receptorlcsh:Diseases of the endocrine glands. Clinical endocrinologyEndocrinologyCancer stem cellstem cellsgrowth factorsmedicinethyroid cancerThyroid cancerSettore MED/04 - Patologia Generalecancer stem cells (CSC)thyroid hormoneslcsh:RC648-665business.industryThyroidmedicine.diseasethyroid cancer stem cells cancer stem cells estrogens thyroid hormones growth factorsmedicine.anatomical_structureImmunologyCancer researchSignal transductionStem cellThyroid functionbusinessHormoneestrogensFrontiers in Endocrinology
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Targeting the vascular endothelial growth factor system to prevent ovarian hyperstimulation syndrome

2008

BACKGROUND: Ovarian hyperstimulation syndrome (OHSS) typically occurs when ovaries are primed with FSH/ LH and subsequently exposed to hCG. The ultimate pathophysiological step underlying this clinical picture is increased vascular permeability (VP). METHODS: A search of the literature was carried out using PubMed and the authors’ files. RESULTS: In rodents and humans, the expression of vascular endothelial growth factor (VEGF) and VEGF receptor 2 (VEGFR-2) mRNA increases during ovarian stimulation. With the administration of hCG, the expression of each rises to a maximum. Expression of VEGF/VEGFR-2 mRNAs correlates with enhanced VP, with both peaking 48 h following an injection of hCG. Imm…

endocrine systemmedicine.medical_specialtyCabergolineIndolesOvarian hyperstimulation syndromeVascular permeabilityBiologyChorionic GonadotropinDopamine agonistCapillary PermeabilityOvarian Hyperstimulation Syndromechemistry.chemical_compoundInternal medicineCabergolinemedicineAnimalsHumansPyrrolesErgolinesPhosphorylationCells CulturedClinical Trials as TopicEstradiolVascular Endothelial Growth FactorsObstetrics and GynecologyEstrogensKinase insert domain receptormedicine.diseaseRatsVascular endothelial growth factorActin CytoskeletonVascular endothelial growth factor Amedicine.anatomical_structureEndocrinologyReproductive MedicinechemistryDopamine AgonistsReproductive Control AgentsFemaleCorpus luteumSignal Transductionmedicine.drugHuman Reproduction Update
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Expression of neurotrophins, GDNF, and their receptors in rat thyroid tissue

1999

Levels of mRNA for neurotrophins (brain-derived neurotrophic factor, BDNF; neurotrophin 3, NT-3; neurotrophin 4, NT-4) and their receptors (trkA, trkB, trkC) and for glial cell line-derived neurotrophic factor (GDNF) and its receptors (ret, GDNFR-alpha) were measured in rat thyroid tissue by ribonuclease protection assays. In thyroid tissue the NT-3 mRNA level was threefold lower and the NT-4 mRNA level sixfold higher than those detected in adult rat hippocampus, while BDNF mRNA was undetectable. Very low levels of mRNA for truncated trkB and trkC receptors and no catalytic trkA, trkB or trkC were found. In conclusion NT-3 and NT-4, but not the corresponding functional receptors, are expres…

endocrine systemmedicine.medical_specialtyGlial Cell Line-Derived Neurotrophic Factor ReceptorsHistologyendocrine system diseasesThyroid GlandGene ExpressionNerve Tissue ProteinsReceptors Nerve Growth FactorNeurotrophin-3Tropomyosin receptor kinase AFollicular cellPathology and Forensic MedicineNeurotrophin 3Proto-Oncogene ProteinsInternal medicinemedicineGlial cell line-derived neurotrophic factorAnimalsDrosophila ProteinsHumansLow-affinity nerve growth factor receptorReceptor trkCGlial Cell Line-Derived Neurotrophic FactorNerve Growth FactorsRNA MessengerReceptor trkAReceptor Ciliary Neurotrophic FactorbiologyBrain-Derived Neurotrophic FactorProto-Oncogene Proteins c-retReceptor Protein-Tyrosine KinasesCell BiologyRatsCell biologyEndocrinologynervous systemProto-Oncogene Proteins c-retbiology.proteinGDNF family of ligandsNeurotrophinCell and Tissue Research
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BDNF is essentially required for the early postnatal survival of nociceptors

2010

AbstractNeurotrophins promote the survival of specific types of neurons during development and ensure proper maintenance and function of mature responsive neurons. Significant effects of BDNF (Brain-Derived Neurotrophic Factor) on pain physiology have been reported but the contribution of this neurotrophin to the development of nociceptors has not been investigated. We present evidence that BDNF is required for the survival of a significant fraction of peptidergic and non-peptidergic nociceptors in dorsal root ganglia (DRG) postnatally. Bdnf homozygous mutant mice lose approximately half of all nociceptive neurons during the first 2 weeks of life and adult heterozygotes exhibit hypoalgesia …

medicine.medical_specialtySkin innervationCell SurvivalNeurotrophic factorMice Inbred StrainsNeuronal survivalMiceNeurotrophic factorsGanglia SpinalInternal medicineGlial cell line-derived neurotrophic factormedicineAnimalsGlial Cell Line-Derived Neurotrophic FactorNerve Growth FactorsDorsal root gangliaAutocrine signallingMolecular BiologyCells CulturedSensory neuronHypoalgesiabiologyBrain-Derived Neurotrophic FactorNociceptorsAnatomyCell BiologyBdnf knockout miceEmbryo MammalianSensory neuronmedicine.anatomical_structureEndocrinologynervous systemPeripheral nervous systembiology.proteinNociceptorNeurotrophinPeripheral nervous systemSignal TransductionNeurotrophinDevelopmental BiologyDevelopmental Biology
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The nicotinic acetylcholine receptor agonist (±)-epibatidine increases FGF-2 mRNA and protein levels in the rat brain

2000

Abstract In a previous work, we showed that acute intermittent nicotine treatment up-regulates the level of fibroblast growth factor-2 (FGF-2) mRNA in brain regions of tel- and mesencephalon of rats suggesting that neuroprotective effect of (−)nicotine may, at least in part, involve an activation of the neuronal FGF-2 signalling. The present experiments were designed to extend the study on the nicotinic receptor mediated up-regulation of FGF-2 mRNA levels to the use of the potent nicotinic acetylcholine receptor (nAChR) agonist (±)-epibatidine. The (±)-epibatidine treatment led to a strong and long lasting up-regulation of FGF-2 mRNA expression in the cerebral cortex, in the hippocampal for…

medicine.medical_specialtyTime FactorsNicotinic acetylcoline receptor agonistPyridinesBlotting WesternNerve Tissue ProteinsNicotinic AntagonistsFibroblast growth factor-2MecamylamineBiologyHippocampusRats Sprague-DawleyNicotineCellular and Molecular NeuroscienceInternal medicineMecamylaminemedicineGlial cell line-derived neurotrophic factorAnimalsGlial Cell Line-Derived Neurotrophic FactorNerve Growth FactorsNicotinic AgonistsRNA MessengerMolecular BiologyIn Situ HybridizationEpibatidineCerebral CortexBrain-derived neurotrophic factorDose-Response Relationship DrugBrain-Derived Neurotrophic FactorBrainBridged Bicyclo Compounds HeterocyclicCorpus StriatumRatsSpecific Pathogen-Free OrganismsNicotinic acetylcholine receptorEndocrinologyNicotinic agonistGene Expression RegulationEpibatidinebiology.proteinFibroblast Growth Factor 2Alpha-4 beta-2 nicotinic receptorFibroblast growth factor receptor-1medicine.drugMolecular Brain Research
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Stem Cell Therapy

2019

The necessity of improving the health and quality of life of subjects affected by diverse injuries and chronic diseases, has led to develop a new branch of translational medicine, called Regenerative Medicine (RegMed). RegMed has the principal objects of restoring, maintaining or enhancing structures and functions of tissues and organs. In order to achieve these goals, the RegMed researchers have proposed and tested diverse approaches. The Stem Cell Therapy represents the central focus of RegMed, even if its applications may imply the onset of adverse conditions. Here, an overview of this topic will be reported, by pointing advantages and disadvantages.

mesenchymal stem cellsbusiness.industrymedicine.medical_treatmentfibrosisregenerative medicineStem-cell therapybenefits and limitationsstem cell therapyangiogenesistranslational medicinegrowth factors/cytokinesCancer researchSettore MED/05 - Patologia ClinicaMedicinebusinessangiogenesis; benefits and limitations; fibrosis; growth factors/cytokines; mesenchymal stem cells; regenerative medicine; stem cell therapy; translational medicine
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Nicotinic receptor agonists as neuroprotective/neurotrophic drugs. Progress in molecular mechanisms

2007

In the present work we reviewed recent advances concerning neuroprotective/neurotrophic effects of acute or chronic nicotine exposure, and the signalling pathways mediating these effects, including mechanisms implicated in nicotine addiction and nAChR desensitization. Experimental and clinical data largely indicate long-lasting effects of nicotine and nicotinic agonists that imply a neuroprotective/neurotrophic role of nAChR activation, involving mainly alpha 7 and alpha 4 beta 2 nAChR subtypes, as evidenced using selective nAChR agonists. Compounds interacting with neuronal nAChRs have the potential to be neuroprotective and treatment with nAChR agonists elicits long-lasting neurotrophic e…

neuroplasticitydesensitizationFGF-2nAChRReceptors NicotinicBiologyPharmacologySettore BIO/09 - FisiologiaNeuroprotectionNicotineNeurotrophic factorsmedicineAnimalsHumansnicotinic agonistNerve Growth FactorsNicotinic Agonistsneurotrophic factorBiological PsychiatryNeuronal PlasticitynAChR; nicotinic agonists; neurotrophic factors; FGF-2; neuroprotection; neurotrophism; addiction; desensitization; neuroplasticityBrainNeurodegenerative DiseasesTobacco Use DisorderPsychiatry and Mental healthNeuroprotective AgentsNerve growth factorNicotinic agonistNeurologySynaptic plasticityneurotrophismbiology.proteinneuroprotectionaddictionNeurology (clinical)Signal transductionNeuroscienceSignal Transductionmedicine.drugNeurotrophinJournal of Neural Transmission
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