Search results for " Helper-Inducer"

showing 10 items of 81 documents

IL-21 Regulates the Differentiation of a Human γδ T Cell Subset Equipped with B Cell Helper Activity

2012

Vγ9Vδ2 T lymphocytes recognize nonpeptidic antigens without presentation by MHC molecules and display pleiotropic features. Here we report that coculture of Vγ9Vδ2 cells with phosphoantigen and IL-21 leads to selective expression of the transcription repressor Bcl-6 and polarization toward a lymphocyte subset displaying features of follicular B-helper T (T(FH)) cells. T(FH) like Vγ9Vδ2 cells have a predominant central memory (CD27(+)CD45RA(-)) phenotype and express ICOS, CD40L and CXCR5. Upon antigen activation, they secrete IL-4, IL-10 and CXCL13, and provide B-cell help for antibody production in vitro. Our findings delineate a subset of human Vγ9Vδ2 lymphocytes, which, upon interaction w…

Anatomy and PhysiologyImmunoglobulin delta-ChainsB CellsCellular differentiationAntibody Affinitylcsh:MedicineAdaptive Immunitychemistry.chemical_compoundWhite Blood CellsSpectrum Analysis TechniquesCell MovementAnimal CellsImmune PhysiologyMedicine and Health SciencesCytotoxic T celllcsh:ScienceImage CytometryB-LymphocytesMultidisciplinarybiologyT CellsCell DifferentiationT-Lymphocytes Helper-InducerFlow CytometryInnate ImmunityDNA-Binding Proteinsmedicine.anatomical_structureIL-21 differentiation Vγ9Vδ2 T lymphocytesSpectrophotometryCD4 AntigensProto-Oncogene Proteins c-bcl-6CytokinesMedicineCytophotometryChemokinesCellular TypesResearch ArticleCD4 antigenImmunoglobulin gamma-ChainsImmune CellsImmunologyMajor histocompatibility complexResearch and Analysis MethodsAntigenmedicineHumansCXCL13Antibody-Producing CellsBiologyB cellCell ProliferationSettore MED/04 - Patologia GeneraleCD40Blood CellsInterleukinsFluorimetrylcsh:RImmunityBiology and Life SciencesCell BiologyMolecular biologyRetractionchemistryGene Expression RegulationHumoral Immunitybiology.proteinClinical Immunologylcsh:QDevelopmental BiologyPloS one
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Requirements for the growth of TH1 lymphocyte clones.

1990

Besides the signal generated in a T lymphocyte after triggering the T cell receptor (TcR), most lymphocytes need a "second signal" to become fully activated. The necessity and nature of the "second signal" differs between different types of T cells. At the level of CD4-positive T helper lymphocytes interleukin 1 (IL 1) serves as "second signal" for those of the TH2 subtype (IL4, 5, 6 producer) but not for those of the TH1 subtype (IL 2, IFN-gamma producer). This correlates with the absence of the IL 1 receptor at the surface of TH1 clones. We report herein the further purification of T cell stimulating factor (TSF), a soluble mediator involved in the proliferation of TH1 lymphocytes. A prep…

Antigens Differentiation T-LymphocyteCD3 Complexmedicine.medical_treatmentT cellLymphocyteImmunologyReceptors Antigen T-CellAntigen-Presenting CellsBiologyLymphocyte ActivationMicemedicineImmunology and AllergyAnimalsAntigen-presenting cellInterleukin 4Mice Inbred BALB CCell growthMacrophage Colony-Stimulating FactorMacrophagesT-cell receptorAntibodies MonoclonalReceptors Interleukin-2T lymphocyteT-Lymphocytes Helper-InducerMolecular biologyCytokinemedicine.anatomical_structureImmunologyInterleukin-1European journal of immunology
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Different response of TH1 cells for stimulation with anti-CD3 antibodies.

1990

In this report, evidence is provided for a further subdivision of CD4+ T helper cell lines. The earlier definition of the TH1 and TH2 subtypes was confirmed by their differential response to interleukin (IL) 1. An additional subdivision of the TH1 subset was revealed when TH1 cell lines were costimulated with anti-CD3 antibodies and IL2. The IL2-induced proliferation of three of the resulting TH1 lines was blocked by anti-CD3 antibodies. By contrast, no such block was observed in a fourth TH1 cell line. In all four lines anti-CD3 triggering caused production of IL2. The block of proliferation was reversed neither by antigen-presenting cells nor by phorbol 12-myristate 13-acetate, a protein …

Antigens Differentiation T-LymphocyteCD4-Positive T-LymphocytesCD3 ComplexCell Survivalmedicine.medical_treatmentImmunologyDose-Response Relationship ImmunologicReceptors Antigen T-CellMice Inbred StrainsBiologyLymphocyte Activationchemistry.chemical_compoundMiceAntigenmedicineImmunology and AllergyAnimalsInterleukin 4Cell growthInterleukinAntibodies MonoclonalT helper cellT-Lymphocytes Helper-InducerMolecular biologyCytokinemedicine.anatomical_structurechemistryCell cultureImmunologyPhorbolInterleukin-2Tetradecanoylphorbol AcetateInterleukin-4SpleenEuropean journal of immunology
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An antigen-independent physiological activation pathway for L3T4+ T lymphocytes.

1987

The data presented in this report describe an antigen-independent activation pathway leading to reinduction of proliferation of class II major histocompatibility complex (MHC)-restricted murine T cell lines that after previous antigen-specific stimulation reverted to a resting state. Antigen-independent proliferation and interleukin 2 (IL2)-receptor expression occur in the presence of splenic accessory cells, exogenous IL2 and a soluble factor(s) provisionally termed T cell-stimulating factor(s) (TSF). Each of these components is essential for inducing growth. TSF is found in the supernatant of an autoreactive T cell line upon stimulation with syngeneic accessory cells. Neither TSF nor acce…

Antigens Differentiation T-LymphocyteT cellImmunologyReceptors Antigen T-CellMice Inbred StrainsGrowthBiologyMajor histocompatibility complexLymphocyte ActivationCell LineTosyl CompoundsMiceAntigenmedicineImmunology and AllergyCytotoxic T cellAnimalsIL-2 receptorAntigensReceptors ImmunologicAntigen-presenting cellMice Inbred BALB CHistocompatibility Antigens Class IICD28Receptors Interleukin-2T-Lymphocytes Helper-InducerCell biologymedicine.anatomical_structurePhenotypeImmunologyAntigens Surfacebiology.proteinInterleukin-2CD8SpleenEuropean journal of immunology
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Th9 Cells: A Novel CD4 T-cell Subset in the Immune War against Cancer

2015

Abstract CD4 T cells are key components of the immune system that shape the anticancer immune response in animal models and in humans. The biology of CD4 T cells is complex because naïve T cells can differentiate into various subpopulations with various functions. Recently, a new population called Th9 cells was described. These cells are characterized by their ability to produce IL9 and IL21. They were first described in the context of parasite infections and allergic processes. However, some reports described their presence in the tumor bed in mice and humans. Their high secretion of IL9 and IL21 in the tumor bed contributes to their anticancer functions. Indeed, these cytokines trigger th…

CD4-Positive T-LymphocytesCancer ResearchTranscription GeneticT-LymphocytesAntigen presentationCD8-Positive T-LymphocytesBiologyMiceImmune systemNeoplasmsAnimalsHumansCytotoxic T cellAntigen-presenting cellGene Expression ProfilingInterleukinsInterleukin-9LymphokineCell DifferentiationT-Lymphocytes Helper-InducerNatural killer T cellAcquired immune systemOncologyImmunologyInterleukin 12Cancer researchCancer Research
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Uptake and presentation of exogenous antigen and presentation of endogenously produced antigen by skin dendritic cells represent equivalent pathways …

2008

Gene gun-mediated biolistic DNA vaccination with beta-galactosidase (betaGal)-encoding plasmid vectors efficiently modulated antigen-induced immune responses in an animal model of type I allergy, including the inhibition of immunoglobulin E (IgE) production. Here we show that CD4(+) as well as CD8(+) T cells from mice biolistically transfected with a plasmid encoding betaGal under the control of the fascin promoter (pFascin-betaGal) are capable of inhibiting betaGal-specific IgE production after adoptive transfer into naïve recipients. Moreover, suppression of IgE production was dependent on interferon (IFN)-gamma. To analyse the modalities of activation of CD4(+) and CD8(+) T cells regardi…

CD4-Positive T-LymphocytesCytotoxicity ImmunologicKeratinocytesAdoptive cell transferGenetic VectorsImmunologyAntigen presentationPriming (immunology)CD8-Positive T-LymphocytesBiologyImmunoglobulin GDNA vaccinationInterferon-gammaMiceCross-PrimingImmune systemAntigenHypersensitivityVaccines DNAAnimalsImmunology and AllergyCytotoxic T cellPromoter Regions GeneticMice KnockoutAntigen PresentationInterleukin-12 Subunit p40Keratin-15VaccinationT-Lymphocytes Helper-InducerOriginal ArticlesBiolisticsImmunoglobulin Ebeta-GalactosidaseAdoptive TransferMolecular biologyImmunoglobulin GLangerhans CellsImmunologybiology.proteinKeratin-5FemaleImmunology
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Comparison of allergen-stimulated dendritic cells from atopic and nonatopic donors dissecting their effect on autologous naive and memory T helper ce…

2000

Abstract Background: Because of their production of IL-12, mature dendritic cells (DC) are potent inducers of T H 1 responses. However, recent reports have demonstrated that DCs can also induce T H 2 differentiation. Objective: In the current study we investigated which immune response is induced by DCs in naive CD45RA + or memory CD45R0 + CD4 + T cells from atopic individuals (patients with grass pollen, birch pollen, or house dust mite allergy) compared with nonatopic control subjects. Methods: Immature DCs, generated from peripheral blood monocytes from atopic and nonatopic donors, were pulsed with the respective allergen and fully matured. Then the mature DCs were cocultured in vitro wi…

CD4-Positive T-LymphocytesHypersensitivity ImmediateAllergymedicine.medical_treatmentImmunologyAntigen presentationImmunoglobulin ETh2 CellsImmune systemmedicineHumansImmunology and AllergyB-LymphocytesbiologyAntibodies MonoclonalDendritic CellsT-Lymphocytes Helper-InducerT lymphocyteDendritic cellAllergensImmunoglobulin Emedicine.diseaseInterleukin-12PhenotypeCytokineImmunologybiology.proteinInterleukin 12CytokinesLeukocyte Common AntigensImmunologic MemoryCell DivisionJournal of Allergy and Clinical Immunology
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Hematopoietic Growth Factors Are Differentially Regulated in Monocytes and CD4+T Lymphocytes: Influence of IFN-α and Interleukin-4

1998

We investigated the influence of interferon-alpha (IFN-alpha) on the synthesis of granulocyte-macrophage colony-stimulating factor (GM-CSF) and interleukin-3 (IL-3) by monocytes and activated T helper cells. IFN-alpha inhibited the production of GM-CSF in unstimulated and lipopolysaccharide (LPS)-activated monocytes to the same extent as was observed in the presence of IL-4. In highly purified CD4+ T cells, which were activated by incubation with immobilized anti-CD3 antibody and anti-CD28, IFN-alpha reduced production of GM-CSF to 47%. In contrast, GM-CSF production in activated T cells was unaffected by exogenously added IL-4. The production of IL-3 by T helper cells was significantly inh…

CD4-Positive T-LymphocytesImmunologyGranulocyte-Macrophage Colony-Stimulating FactorInterferon-alphaT-Lymphocytes Helper-InducerCell BiologyBiologyMonocytesInterferon-gammaInterleukin 21HaematopoiesisTransforming Growth Factor betaVirologyImmunologyHumansCytotoxic T cellInterleukin-3Interleukin-4Cells CulturedInterleukin 4Journal of Interferon & Cytokine Research
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Defective T helper response of hepatocyte-stimulated CD4 T cells impairs antiviral CD8 response and viral clearance.

2007

Background & Aims: In hepatitis, hepatocytes gain the ability to express major histocompatibility complex (MHC) class II molecules and to present antigen to CD4 T cells. Here, we investigated whether MHC class II-expressing hepatocytes influence in vitro the differentiation of CD4 T cells and in vivo the T-cell response to and control of viral infection. Methods: Class II transactivator-transgenic hepatocytes that constitutively express MHC class II molecules were used to stimulate CD4 T cells in vitro, and the effector response type of the stimulated CD4 T cells was determined. The in vivo relevance of the obtained findings was confirmed by infecting nontransgenic or class II transactivato…

CD4-Positive T-LymphocytesMHC class IIHepatologybiologyCD8 AntigensGastroenterologyCD1Mice TransgenicT helper cellT-Lymphocytes Helper-InducerMHC restrictionCD8-Positive T-LymphocytesMicemedicine.anatomical_structureMHC class IImmunologyCD4 Antigensbiology.proteinmedicineHepatocytesCytotoxic T cellAnimalsAntigen-presenting cellCD8Gastroenterology
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The Transcription Factor T-bet Regulates Mucosal T Cell Activation in Experimental Colitis and Crohn's Disease

2002

The balance between pro and antiinflammatory cytokines secreted by T cells regulates both the initiation and perpetuation of inflammatory bowel diseases (IBD). In particular, the balance between interferon (IFN)-gamma/interleukin (IL)-4 and transforming growth factor (TGF)-beta activity controls chronic intestinal inflammation. However, the molecular pathways that evoke these responses are not well understood. Here, we describe a critical role for the transcription factor T-bet in controlling the mucosal cytokine balance and clinical disease. We studied the expression and function of T-bet in patients with IBD and in mucosal T cells in various T helper (Th)1- and Th2-mediated animal models …

CD4-Positive T-LymphocytesMalecolitisGenes RAG-1T-Lymphocytesmedicine.medical_treatmentMice SCIDGATA-3Polymerase Chain ReactionMiceInterleukin 210302 clinical medicineCrohn DiseaseT-Lymphocyte SubsetsImmunology and AllergyCytotoxic T cellIL-2 receptorIFN-γMice Inbred BALB C0303 health sciencesGene Transfer Techniqueshemic and immune systemsT-Lymphocytes Helper-InducerMiddle Aged3. Good healthCytokinemedicine.anatomical_structureFemaleAdultT cellImmunologychemical and pharmacologic phenomenaBiologyT-betArticleTCIRG103 medical and health sciencesmedicineAnimalsHumansColitisImmunity MucosalInterleukin 4DNA Primers030304 developmental biologyHomeodomain ProteinsBase Sequencemedicine.diseasecytokinesDisease Models AnimalGene Expression RegulationImmunologyT-Box Domain ProteinsSpleenTranscription Factors030215 immunologyJournal of Experimental Medicine
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