Search results for " High-Fat"

showing 10 items of 95 documents

NUPR1 protects liver from lipotoxic injury by improving the endoplasmic reticulum stress response

2021

AbstractBackground and AimsNon-alcoholic fatty liver disease and related hepatic syndromes affect up to one third of the adult population. The molecular mechanisms underlying NAFL etiology remain elusive. Nuclear Protein 1 (NUPR1) expression increases upon cell injury in all organs and recently we report its active participation in the activation of the Unfolded Protein Response (UPR). The UPR typically maintains protein homeostasis, but downstream mediators of the pathway regulate metabolic functions, including lipid metabolism. NUPR1 and UPR increase have been reported in obesity and liver pathologies and the goal of this study was to investigate the roles of NUPR1 in this context.Methods…

0301 basic medicinemedicine.medical_specialtySettore MED/09 - Medicina InternaPPAR-a signalling UPRPeroxisome proliferator-activated receptorContext (language use)UPRDiet High-FatBiochemistry03 medical and health sciencesLiver diseaseMice0302 clinical medicineInternal medicineCell Line TumorGeneticsmedicineBasic Helix-Loop-Helix Transcription FactorsAnimalsHomeostasisHumansMolecular Biologychemistry.chemical_classificationbusiness.industryEndoplasmic reticulumFatty liverNASHLipid metabolismlipotoxicitymedicine.diseaseEndoplasmic Reticulum StressLipid MetabolismNeoplasm Proteins030104 developmental biologyEndocrinologychemistryLipotoxicityLiverNAFLKnockout mouseUnfolded protein responseUnfolded Protein ResponsePPAR-a signallingSteatosisSteatohepatitisbusiness030217 neurology & neurosurgeryNUPR1Biotechnology
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3,5-DIIODO-L-THYRONINE-INDUCED MODIFICATION IN PITUITARY-THYROID AXIS IN RATS FED HIGH-FAT DIET. A PRELIMINARY REPORT

2014

Experimental observations highlight that the administration of 3,5-diiodo-L-thyronine (T2) may decrease the body weight and the plasma levels of cholesterol and triglycerides and may prevent the onset of hepatic steatosis in rats fed diets rich in lipids (HFD). On the basis of these findings we have carried out some in vivo studies to evaluate the effects of increased levels of T2 on pituitary thyroid axis function in HFD rats. Fifteeen Wistar male rats were divided in 3 groups. The first group (N) was fed with a standard diet. The second group (G) was fed with a diet high in fat (HDF), while the third group (GT2) was additionally administered intraperitoneally with T2 (70 ug/100g body weig…

35-DIIODO-L-THYRONINE HIGH-FAT DIETSettore BIO/09 - Fisiologia
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Neddylation inhibition ameliorates steatosis in NAFLD by boosting hepatic fatty acid oxidation via the DEPTOR-mTOR axis

2021

Objective Neddylation is a druggable and reversible ubiquitin-like post-translational modification upregulated in many diseases, including liver fibrosis, hepatocellular carcinoma, and more recently, non-alcoholic fatty liver disease (NAFLD). Herein, we propose to address the effects of neddylation inhibition and the underlying mechanisms in pre-clinical models of NAFLD. Methods Hepatic neddylation measured by immunohistochemical analysis and NEDD8 serum levels measured by ELISA assay were evaluated in NAFLD clinical and pre-clinical samples. The effects of neddylation inhibition by using a pharmacological small inhibitor, MLN4924, or molecular approaches were assessed in isolated mouse hep…

AdultMaleCoronavirus disease 2019 (COVID-19)AdolescentMLN4924[SDV.BC]Life Sciences [q-bio]/Cellular BiologyDiet High-Fat03 medical and health sciencesMiceYoung Adult0302 clinical medicineNon-alcoholic Fatty Liver DiseasePolitical scienceNAFLDmedia_common.cataloged_instanceAnimalsHumansEuropean unionNeddylationMolecular BiologyInternal medicineComputingMilieux_MISCELLANEOUS030304 developmental biologymedia_commonAged0303 health sciencesTOR Serine-Threonine KinasesFatty AcidsIntracellular Signaling Peptides and ProteinsCell BiologyMiddle AgedRC31-12453. Good healthMice Inbred C57BLRare tumorDisease Models AnimalDeptor; Fatty acid oxidation; MLN4924; mTOR; NAFLD; NeddylationDeptorFatty acid oxidationHepatocytesmTOR030211 gastroenterology & hepatologyChristian ministryOriginal ArticleHumanitiesSignal Transduction
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Low-Carbohydrate High-Fat Diet and Exercise: Effect of a 10-Week Intervention on Body Composition and CVD Risk Factors in Overweight and Obese Women—…

2020

We assessed the effect of weight-loss induced with a low-carbohydrate-high-fat diet with and without exercise, on body-composition, cardiorespiratory fitness and cardiovascular risk factors. A total of 57 overweight and obese women (age 40 &plusmn

Adultcardiovascular riskNormal diet030209 endocrinology & metabolismlcsh:TX341-641lean body mass030204 cardiovascular system & hematologyOverweightDiet High-FatArticleFat masslaw.invention03 medical and health sciencesDiet Carbohydrate-Restricted0302 clinical medicineAnimal scienceRandomized controlled triallawRisk Factorsdietary interventionmedicineHumansVDP::Medisinske Fag: 700ObesityTriglyceridesNutrition and Dieteticscardiorespiratory fitnessmedicine.diagnostic_testexercisebusiness.industryCardiorespiratory fitnessfat massMiddle Agedketogeniclipid profileCholesterolCardiovascular DiseasesLean body massBody CompositionFemalemedicine.symptombusinessLipid profilelow-carbohydrate high-fat-dietBody mass indexlcsh:Nutrition. Foods and food supplyFood ScienceNutrients
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Inhibition of endocannabinoid-degrading enzyme fatty acid amide hydrolase increases atherosclerotic plaque vulnerability in mice

2013

The role of endocannabinoids such as anandamide during atherogenesis remains largely unknown. Fatty acid amide hydrolase (FAAH) represents the key enzyme in anandamide degradation, and its inhibition is associated with subsequent higher levels of anandamide. Here, we tested whether selective inhibition of FAAH influences the progression of atherosclerosis in mice. Selective inhibition of FAAH using URB597 resulted in significantly increased plasma levels of anandamide compared to control, as assessed by mass spectrometry experiments in mice. Apolipoprotein E-deficient (ApoE(-/-)) mice were fed a high-fat, cholesterol-rich diet to induce atherosclerotic conditions. Simultaneously, mice recei…

Apolipoprotein Emedicine.medical_specialtyApolipoprotein BNeutrophilsPolyunsaturated Alkamidesmedicine.medical_treatmentIntraperitoneal injectionGene ExpressionArachidonic AcidsDiet High-FatAmidohydrolasesMicechemistry.chemical_compoundApolipoproteins EWestern blotCell MovementSuperoxidesFatty acid amide hydrolaseInternal medicinemedicineAnimalsEnzyme InhibitorsMolecular BiologyMice Knockoutbiologymedicine.diagnostic_testChemistryMacrophagesAnandamideURB597Dietary FatsEndocannabinoid systemPlaque AtheroscleroticEndocrinologyBenzamidesbiology.proteinCarbamatesCardiology and Cardiovascular MedicineEndocannabinoidsJournal of Molecular and Cellular Cardiology
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GLP2: An underestimated signal for improving glycaemic control and insulin sensitivity

2016

Glucagon-like peptide 2 (GLP2) is a proglucagon-derived peptide produced by intestinal enteroendocrine L-cells and by a discrete population of neurons in the brainstem, which projects mainly to the hypothalamus. The main biological actions of GLP2 are related to the regulation of energy absorption and maintenance of mucosal morphology, function and integrity of the intestine; however, recent experimental data suggest that GLP2 exerts beneficial effects on glucose metabolism, especially in conditions related to increased uptake of energy, such as obesity, at least in the animal model. Indeed, mice lacking GLP2 receptor selectively in hypothalamic neurons that express proopiomelanocortin show…

Blood Glucose0301 basic medicinemedicine.medical_specialtyEndocrinology Diabetes and MetabolismPopulation030209 endocrinology & metabolismEnteroendocrine cellType 2 diabetesBiologyCarbohydrate metabolismDiet High-FatModels BiologicalType 2 diabeteMice03 medical and health sciences0302 clinical medicineInsulin resistanceEndocrinologyInternal medicineGlucagon-Like Peptide 2medicineAnimalsHomeostasisHumansGlucose homeostasisObesityeducationeducation.field_of_studyGLP2Insulin resistanceGlucagon-like peptide-2medicine.disease030104 developmental biologyEndocrinologyDiabetes Mellitus Type 2GLP2; Insulin resistance; Obesity; Type 2 diabetes; Endocrinology; Endocrinology Diabetes and MetabolismHomeostasisSignal Transduction
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Early Low-Fat Diet Enriched With Linolenic Acid Reduces Liver Endocannabinoid Tone and Improves Late Glycemic Control After a High-Fat Diet Challenge…

2016

International audience; Evidence suggests that alterations of glucose and lipid homeostasis induced by obesity are associated with the elevation of endocannabinoid tone. The biosynthesis of the two main endocannabinoids, N-arachidonoylethanolamine and 2-arachidonoyl-glycerol, which derive from arachidonic acid, is influenced by dietary fatty acids (FAs). We investigated whether exposure to n-3 FA at a young age may decrease tissue endocannabinoid levels and prevent metabolic disorders induced by a later high-fat diet (HFD) challenge. Three-week-old mice received a 5% lipid diet containing lard, lard plus safflower oil, or lard plus linseed oil for 10 weeks. Then, mice were challenged with a…

Blood Glucose0301 basic medicinemedicine.medical_specialty[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionEndocrinology Diabetes and MetabolismMice TransgenicCarbohydrate metabolismBiologyDiet High-FatMice03 medical and health scienceschemistry.chemical_compoundInternal medicineInternal MedicinemedicineAnimalsHomeostasisObesityDiet Fat-RestrictedGlycemic2. Zero hungerdiabetesalpha-Linolenic acidBody WeightFatty liveralpha-Linolenic AcidLipid metabolismLipid Metabolismmedicine.diseaseEndocannabinoid system3. Good healthFatty LiverMice Inbred C57BL[SDV.AEN] Life Sciences [q-bio]/Food and Nutrition030104 developmental biologyEndocrinologyLiverchemistryendocananbinoid systemCarbohydrate MetabolismArachidonic acidlipids (amino acids peptides and proteins)Metabolic syndrome[SDV.AEN]Life Sciences [q-bio]/Food and NutritionEndocannabinoids
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Chronic exposure of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) induces an obesogenic effect in C57BL/6J mice fed a high fat diet

2017

IF 3.582; International audience; Contaminant involvement in the pathophysiology of obesity is widely recognized. It has been shown that low dose and chronic exposure to endocrine disruptor compounds (EDCs) potentiated diet- induced obesity. High and acute exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD), a persistent organic pollutant (POP) and an EDC with anti-estrogenic property, causes wasting syndrome . However at lower doses, the TCDD metabolic effects remain poorly understood. We investigated the obesogenic effect during chronic exposure of TCDD at 1μg/kg body weight (bw)/week in adult C57BL/6J mice fed with a high fat diet (HFD) and exposed from 10 to 42 weeks old to TCDD or e…

Blood GlucoseLeptinMale0301 basic medicineTCDDPolychlorinated DibenzodioxinsTime FactorsAdipose tissue010501 environmental sciencesToxicology01 natural sciencesBasic Helix-Loop-Helix Transcription FactorsInsulinAdiposity2. Zero hunger[ SDV.MHEP.EM ] Life Sciences [q-bio]/Human health and pathology/Endocrinology and metabolism[SDV.MHEP.EM]Life Sciences [q-bio]/Human health and pathology/Endocrinology and metabolism3. Good healthLiverEndocrine disruptorReceptors AndrogenCytokinesEnvironmental PollutantsFemaleInflammation Mediatorsmedicine.symptomStearoyl-CoA Desaturasemedicine.medical_specialtyLipolysisInflammationchronic exposureIntra-Abdominal FatDiet High-FatRisk Assessment03 medical and health sciencesSex FactorsobesogenInternal medicinemedicineAnimalsEndocrine systemObesityRNA MessengerWasting SyndromeTriglycerides0105 earth and related environmental sciencesbusiness.industrymedicine.diseaseObesityMice Inbred C57BL030104 developmental biologyEndocrinologyReceptors Aryl HydrocarbonInsulin ResistancebusinessBiomarkersObesogenDrug metabolism
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Asperuloside Enhances Taste Perception and Prevents Weight Gain in High-Fat Fed Mice

2021

Asperuloside is an iridoid glycoside found in many medicinal plants that has produced promising anti-obesity results in animal models. In previous studies, three months of asperuloside administration reduced food intake, body weight, and adipose masses in rats consuming a high fat diet (HFD). However, the mechanisms by which asperuloside exerts its anti-obesity properties were not clarified. Here, we investigated homeostatic and nutrient-sensing mechanisms regulating food intake in mice consuming HFD. We confirmed the anti-obesity properties of asperuloside and, importantly, we identified some mechanisms that could be responsible for its therapeutic effect. Asperuloside reduced body weight …

Blood GlucoseLeptinMalecannabinoid (CB) receptor 10301 basic medicineTastePro-Opiomelanocortinfood intakeEndocrinology Diabetes and MetabolismAdipose tissueWeight Gainnutrient-sensing mechanismslcsh:Diseases of the endocrine glands. Clinical endocrinologyCyclopentane MonoterpenesEnergy homeostasisMiceEndocrinology0302 clinical medicineGlucosidesWeight lossInsulinasperuloside; cannabinoid (CB) receptor 1; CD36; FFAR1-4; food intake; nutrient-sensing mechanisms; TAS1R2-3; weight lossReceptorOriginal ResearchLeptindigestive oral and skin physiologyTaste PerceptionGhrelinTAS1R2-3Ghrelinmedicine.symptommedicine.medical_specialtyHypothalamusBiologyDiet High-Fatasperuloside03 medical and health sciencesInternal medicinemedicineAnimalsPyranslcsh:RC648-665Body WeightFFAR1-4030104 developmental biologyEndocrinologyAnti-Obesity Agentsweight lossEnergy IntakeCD36Weight gain030217 neurology & neurosurgery
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Bile acid receptor TGR5 is critically involved in preference for dietary lipids and obesity

2020

International audience; We investigated the implication of Takeda G protein-coupled receptor 5 (TGR5) in fat preference and fat sensing in taste bud cells (TBC) in C57BL/6 wild-type (WT) and TGR5 knock out (TGR5-/-) male mice maintained for 20 weeks on a high-fat diet (HFD). We also assessed the implication of TGR5 single nucleotide polymorphism (SNP) in young obese humans. The high-fat diet (HFD)-fed TGR5-/- mice were more obese, marked with higher liver weight, lipidemia and steatosis than WT obese mice. The TGR5-/- obese mice exhibited high daily food/energy intake, fat mass and inflammatory status. WT obese mice lost the preference for dietary fat, but the TGR5-/- obese mice exhibited n…

Blood GlucoseLipopolysaccharidesMale0301 basic medicine[SDV]Life Sciences [q-bio]Endocrinology Diabetes and MetabolismClinical BiochemistryBiochemistryReceptors G-Protein-CoupledMice0302 clinical medicineInsulinReceptorMice Knockout2. Zero hungerchemistry.chemical_classificationNutrition and DieteticsLipidsG protein-coupled bile acid receptor[SDV] Life Sciences [q-bio]medicine.anatomical_structuremedicine.medical_specialtyMice Transgenic030209 endocrinology & metabolismSingle-nucleotide polymorphismDiet High-FatPolymorphism Single NucleotideBile Acids and SaltsFood Preferences03 medical and health sciencesInternal medicineTaste budmedicineAnimalsObesityMolecular BiologyInflammationbusiness.industryTaste budFatty acidFatty acidmedicine.diseaseDietary FatsObesityIn vitroFatty LiverMice Inbred C57BLDisease Models Animal030104 developmental biologyEndocrinologychemistryFatCalciumSteatosisbusinessThe Journal of Nutritional Biochemistry
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