Search results for " Hypertrophy"

showing 10 items of 281 documents

Chronic Prostatitis and Prostatodynia: Ultrasonographic Alterations of the Prostate, Bladder Neck, Seminal Vesicles and Periprostatic Venous Plexus

1988

Transrectal ultrasonography was performed in 121 patients with the chronic prostatitis syndrome (CPS), and in 20 patients with urological pathology not involving the prostate gland. The ultrasonographic aspects of the seminal vesicles (SV) in normal subjects and in patients with the CPS are described. In the latter group, characteristic ultrasonographic patterns can often be obtained, even in patients affected by prostatodynia, in whom all the other clinical and laboratory findings were absent. They consisted of: (a) dyshomogeneous echo-structure of the prostate; (b) constant dilatation of the periprostatic venous plexus, greater than 150 mm2; (c) dilated, elongated SV, with thickening of t…

MaleProstatic Diseasesmedicine.medical_specialtyPeriprostatic venous plexusmedicine.diagnostic_testbusiness.industryUrologyVesicleUrinary BladderProstateUrologySeminal VesiclesProstatitismedicine.diseaseProstatitisMuscle hypertrophyNeck of urinary bladdermedicine.anatomical_structureProstatemedicineHumansTransrectal ultrasonographyIn patientbusinessUltrasonographyEuropean Urology
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Immune Cell Toll-like Receptor 4 Mediates the Development of Obesity- and Endotoxemia-Associated Adipose Tissue Fibrosis

2014

International audience; Adipose tissue fibrosis development blocks adipocyte hypertrophy and favors ectopic lipid accumulation. Here, we show that adipose tissue fibrosis is associated with obesity and insulin resistance in humans and mice. Kinetic studies in C3H mice fed a high-fat diet show activation of macrophages and progression of fibrosis along with adipocyte metabolic dysfunction and death. Adipose tissue fibrosis is attenuated by macrophage depletion. Impairment of Toll-like receptor 4 signaling protects mice from obesity-induced fibrosis. The presence of a functional Toll-like receptor 4 on adipose tissue hematopoietic cells is necessary for the initiation of adipose tissue fibros…

LipopolysaccharidesMESH: Signal TransductionMESH: InflammationMESH : Toll-Like Receptor 4Adipose tissueMESH : AdipocytesMESH : LipopolysaccharidesMicechemistry.chemical_compoundFibrosisAdipocyteAdipocytes[ SDV.IMM ] Life Sciences [q-bio]/ImmunologyMESH: ObesityMESH: Animalslcsh:QH301-705.5Mice Inbred C3HToll-like receptorMESH : Diet High-FatMESH: Toll-Like Receptor 43. Good healthMESH: Insulin ResistanceAdipose TissueMESH: FibrosisMESH : Fibrosis[SDV.IMM]Life Sciences [q-bio]/ImmunologyMESH : ObesityMESH : Insulin ResistanceMESH: Adipose TissueSignal Transductionmedicine.medical_specialty[SDV.IMM] Life Sciences [q-bio]/ImmunologyAdipose tissue macrophagesBiologyDiet High-FatMESH : Adipose TissueGeneral Biochemistry Genetics and Molecular BiologyImmune systemMESH : Mice Inbred C3HInternal medicineMESH : MicemedicineAnimalsHumansObesityMESH: Mice Inbred C3HMESH: MiceMESH: AdipocytesInflammationMESH : Signal TransductionMESH : InflammationMESH: HumansMESH : EndotoxemiaMESH : Humans3T3-L1medicine.diseaseFibrosisMESH : Disease Models AnimalEndotoxemiaToll-Like Receptor 4Disease Models AnimalMESH: Diet High-FatEndocrinologylcsh:Biology (General)chemistryMESH: EndotoxemiaMESH : AnimalsInsulin ResistanceMESH: Disease Models AnimalMESH: LipopolysaccharidesAdipocyte hypertrophyCell Reports
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Absence of an independent association between serum uric acid and left ventricular mass in Caucasian hypertensive women and men

2013

Background and aim: Experimentally uric acid may induce cardiomyocyte growth and interstitial fibrosis of the heart. However, clinical studies exploring the relationship between serum uric acid (SUA) and left ventricular (LV) mass yielded conflicting results. The aim of our study was to evaluate the relationships between SUA and LV mass in a large group of Caucasian essential hypertensive subjects. Methods and results: We enrolled 534 hypertensive patients free of cardiovascular complications and without severe renal insufficiency. In all subjects routine blood chemistry, including SUA determination, echocardiographic examination and 24 h ambulatory blood pressure (BP) monitoring were obtai…

AdultMalemedicine.medical_specialtyAmbulatory blood pressureEndocrinology Diabetes and MetabolismHeart VentriclesPopulationLeft ventricular maMedicine (miscellaneous)Blood PressureLeft ventricular hypertrophyEssential hypertensionWhite PeopleEssential hypertensionBody Mass Indexchemistry.chemical_compoundSerum uric acidInternal medicinemedicineHumansEssential hypertension; Serum uric acid; Cardiovascular risk; Left ventricular mass; Left ventricular hypertrophyeducationeducation.field_of_studyCreatinineNutrition and Dieteticsbusiness.industryLeft ventricular hypertrophyBlood Pressure Monitoring AmbulatoryMiddle Agedmedicine.diseaseCardiovascular riskUric AcidEndocrinologyBlood pressureCross-Sectional StudieschemistryBlood chemistryEchocardiographyCreatinineHypertensionCardiologyFemaleCardiology and Cardiovascular MedicinebusinessBody mass index
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Increased Cx43 and Angiogenesis in Exercised Mouse Hearts

2007

Several studies focused on the macroscopic architecture of increased cardiac wall induced by exercise training. Our goal was to evaluate myocardiocyte, interstitial and vascular component, and connexin-43 expression in endurance-trained mouse hearts. Sixty-three 10-week-old male Swiss mice were divided into four sedentary groups (C0, C15, C30 and C45) and three groups exercised respectively for 15 (T15-D; running intensity [RI]: 3.18 m/min; running duration [RD]: 75 min/first week and 150 min/second week), 30 (T30-D; RI: 3.96 m/min; RD: 150 min/third week and 225 min/fourth week) and 45 days (T45-D; RI: 3.96 m/min and 4.8 m/min, respectively for the fifth and sixth week; RD: 300 min) on a t…

Malemedicine.medical_specialtyconnexin-43Neovascularization PhysiologicConnective tissueCardiomegalyPhysical Therapy Sports Therapy and RehabilitationheartMuscle hypertrophyNeovascularizationMiceangiogenesisendurance trainingEndurance trainingInternal medicinemedicineAnimalsOrthopedics and Sports MedicineInterventricular septumTreadmillSettore M-EDF/02 - Metodi E Didattiche Delle Attivita' Sportivebusiness.industryGap Junctionsmedicine.anatomical_structureEndocrinologyConnective TissueVentricleConnexin 43Circulatory systemPhysical Endurancemedicine.symptomhypertrophybusinessInternational Journal of Sports Medicine
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Neuromuscular adaptations to constant vs. variable resistance training in older men.

2013

This study examined the effects of constant or variable external resistance training on neuromuscular adaptations in the lower limbs of older men. 37 subjects (age 65±4 year) were quasi-randomly assigned to the constant or variable training group, or a non-training control group. Training consisted of a 20-week medium-intensity, high volume resistance training program. Maximum bilateral concentric and isometric force production of the leg extensors as well as repetitions-to-failure test were performed pre-, mid- and post-training. Vastus lateralis muscle cross-sectional area was assessed by ultrasound and lean leg mass was assessed by dual-energy x-ray absorptiometry. Both training groups s…

MaleMuscle Strength Dynamometermedicine.medical_specialtyVastus lateralis musclePhysical Therapy Sports Therapy and RehabilitationIsometric exerciseElectromyographyMuscle Strength DynamometerConcentricMuscle hypertrophylaw.inventionPhysical medicine and rehabilitationAbsorptiometry PhotonRandomized controlled triallawmedicineHumansOrthopedics and Sports MedicineMuscle StrengthMuscle SkeletalAgedUltrasonographymedicine.diagnostic_testbusiness.industryElectromyographyNeural adaptationResistance TrainingAdaptation Physiologicalmedicine.anatomical_structureLower ExtremityPhysical therapybusinessInternational journal of sports medicine
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Therapeutic Modulation of Urinary Bladder Function: Multiple Targets at Multiple Levels

2015

Storage dysfunction of the urinary bladder, specifically overactive bladder syndrome, is a condition that occurs frequently in the general population. Historically, pathophysiological and treatment concepts related to overactive bladder have focused on smooth muscle cells. Although these are the central effector, numerous anatomic structures are involved in their regulation, including the urothelium, afferent and efferent nerves, and the central nervous system. Each of these structures involves receptors for—and the urothelium itself also releases—many mediators. Moreover, hypoperfusion, hypertrophy, and fibrosis can affect bladder function. Established treatments such as muscarinic antago…

Central Nervous Systemmedicine.medical_specialtyUrinary BladderPopulationCentral nervous systemMuscarinic Antagonistsurologic and male genital diseasesToxicologyBioinformaticsMuscle hypertrophyNeurons EfferentFibrosisInternal medicinemedicineAnimalsHumansNeurons AfferentUrotheliumeducationPharmacologyeducation.field_of_studyUrinary bladderbusiness.industryUrinary Bladder DiseasesMuscle SmoothAdrenergic beta-AgonistsHyperplasiamedicine.diseasefemale genital diseases and pregnancy complicationsUrodynamicsTreatment OutcomeEndocrinologymedicine.anatomical_structureOveractive bladderAdrenergic alpha-1 Receptor AntagonistsUrological AgentsUrotheliumbusinessSignal TransductionAnnual Review of Pharmacology and Toxicology
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Genotype–phenotype correlation in a new Fabry-disease-causing mutation

2019

Background: Fabry disease (FD) is a rare X-linked inherited lysosomal storage disorder caused by α-galactosidase A deficiency leading to intracellular glycosphingolipid accumulation. FD manifestation is multisystem, and can differ depending on disease-related genetic variants. Currently, more than 700 different FD-causing mutations have been identified in the human GLA gene. We identified a novel mutation in a Lithuanian family with classical manifestations of Fabry disease, revealing severe effects to the cardiovascular systems of heterozygous women. Case presentation: A 49-year-old woman underwent echocardiography due to progressive dyspnea that lasted seven years, reduced physical a…

Probandmedicine.medical_specialtyAbdominal painMedicine (General)α-galactosidase ACase ReportLeft ventricular hypertrophyGastroenterologyclassical manifestationR5-920Internal medicinemedicineGLA geneFabry diseasemedicine.diagnostic_testbusiness.industryCardiac arrhythmiaGeneral MedicineFabry disease ; α-galactosidase A ; GLA gene ; novel mutation ; classical manifestationmedicine.diseaseFabry diseaseHyperintensityMutation (genetic algorithm)<i>GLA</i> geneRenal biopsymedicine.symptomnovel mutationbusiness
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Disturbed Glucose Metabolism and Left Ventricular Geometry in the General Population

2021

Background: This study sought to investigate the prevalence and clinical outcome of left ventricular (LV) geometry in prediabetes and type 2 diabetes mellitus (T2DM) and the impact of glucose metabolism on the incidence of left ventricular hypertrophy (LVH). Methods: 15,010 subjects (35–74 years) of the population-based Gutenberg Health Study were categorized into euglycemia, prediabetes, and T2DM according to clinical and metabolic (HbA1c) information. Clinical outcome was assessed via structured follow-up. Results: The study comprised 12,121 individuals with euglycemia (81.6%), 1415 with prediabetes (9.5%), and 1316 with T2DM (8.9%). Prevalence of LVH increased from euglycemia (10.2%) ove…

medicine.medical_specialtyendocrine system diseasestype 2 diabetes mellitusPopulationprediabetesCarbohydrate metabolismleft ventricular concentric remodelingLeft ventricular hypertrophyArticleInternal medicineMedicineMass indexPrediabetescardiovascular diseaseseducationeducation.field_of_studybusiness.industryIncidence (epidemiology)Hazard ratioRType 2 Diabetes Mellitusnutritional and metabolic diseasesGeneral Medicinemedicine.diseaseleft ventricular hypertrophyleft ventricular geometryCardiologyMedicinebusinessJournal of Clinical Medicine
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Prevalence and predictors of left ventricular hypertrophy in patients with hypertension and normal electrocardiogram.

2013

Background: Electrocardiography (ECG) has low sensitivity for detecting left ventricular hypertrophy (LVH), while echocardiography cannot be routinely performed. Design/methods: In this study we evaluate the prevalence of LVH and diastolic dysfunction in hypertensive patients with normal ECG. We excluded patients with cardiovascular (CV) diseases, diabetes, chronic kidney disease, or presenting ECG-LVH or other ECG anomalies. The enrolled 440 hypertensive patients underwent echocardiographic examination (Acuson Sequoia 512); LV mass was indexed by body surface area (LVMI) and LVH was defined as LVMI >125 g/m2 in men and >110 g/m2 in women. Diastolic function was evaluated by mitral inflow a…

AdultMalemedicine.medical_specialtySettore MED/09 - Medicina InternaTime FactorsEpidemiologyLeft ventricular hypertrophy hypertension electrocardiography echocardiographyDiastoleLeft ventricular hypertrophyDoppler imagingVentricular Function LeftElectrocardiographyVentricular Dysfunction LeftDiastolePredictive Value of TestsRisk FactorsInternal medicinemedicineOdds RatioPrevalenceHumanscardiovascular diseasesAgedBody surface areaChi-Square Distributionmedicine.diagnostic_testReceiver operating characteristicbusiness.industryPatient SelectionMiddle Agedmedicine.diseaseSettore MED/11 - Malattie Dell'Apparato CardiovascolareConfidence intervalEchocardiography DopplerLogistic ModelsItalyROC CurveArea Under CurveHypertensionMultivariate AnalysisCardiologyFemaleHypertrophy Left VentricularCardiology and Cardiovascular MedicinebusinessElectrocardiographyKidney diseaseEuropean journal of preventive cardiology
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Left ventricular hypertrophy or storage disease? the incremental value of speckle tracking strain bull's-eye

2017

Left ventricular hypertrophy (LVH) develops in response to a variety of physical, genetic, and biochemical stimuli and represents the early stage of ventricular remodeling. In patients with LVH, subclinical left ventricular (LV) dysfunction despite normal ejection fraction (EF) may be present before the onset of symptoms, which portends a dismal prognosis. Strain measurement with two-dimensional speckle tracking echocardiography (STE) represents a highly reproducible and accurate alternative to LVEF determination. The present review focuses on current available evidence that supports the incremental value of STE in the diagnostic and prognostic workup of LVH. When assessing the components o…

Radiology Nuclear Medicine and ImagingSpeckle tracking echocardiographyDisease030204 cardiovascular system & hematologyLeft ventricular hypertrophytwo-dimensional strain0302 clinical medicineCardiomegaly Exercise-Induced030212 general & internal medicineanabolic steroidSubclinical infectionamyloidosiEvidence-Based MedicineEjection fractionHypertrophic cardiomyopathyleft ventricular hypertrophyEchocardiographyCardiologyElasticity Imaging TechniquesHypertrophy Left VentricularRadiologyCardiomyopathiesCardiology and Cardiovascular MedicineHumanendocrine systemmedicine.medical_specialtyarterial hypertensionReproducibility of ResultSensitivity and SpecificityDiagnosis Differential03 medical and health sciencesElasticity Imaging TechniqueInternal medicinemedicineathlete's heartHumanscardiovascular diseasesVentricular remodelingspeckle tracking echocardiographyCardiomyopathiebusiness.industryReproducibility of ResultsStroke Volumeaortic stenosiImage Enhancementmedicine.diseasehypertrophic cardiomyopathyDifferential diagnosisMetabolism Inborn ErrorbusinessMetabolism Inborn Errors
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