Search results for " Immune system"

showing 10 items of 893 documents

Specific and Redundant Roles for NFAT Transcription Factors in the Expression of Mast Cell-Derived Cytokines

2006

Abstract By virtue of their ability to express a plethora of biologically highly active mediators, mast cells (MC) are involved in both adaptive and innate immune responses. MC-derived Th2-type cytokines are thought to act as local amplifiers of Th2 reactions, including chronic inflammatory disorders such as allergic asthma, whereas MC-derived TNF-α is a critical initiator of antimicrobial defense. In this study, we demonstrate that the transcription factors NFATc1 and NFATc2 are part of a MC-specific signaling network that regulates the expression of TNF-α and IL-13, whereas NFATc3 is dispensable. Primary murine bone marrow-derived MC from NFATc2−/− mice, activated by either ionomycin or I…

ImmunologyDown-RegulationImmunoglobulin EMicechemistry.chemical_compoundTh2 CellsCell Line TumormedicineAnimalsImmunology and AllergyMast CellsTranscription factorCells CulturedMice KnockoutMice Inbred BALB CGene knockdownInterleukin-13Innate immune systemNFATC Transcription FactorsbiologyTumor Necrosis Factor-alphaDegranulationNFATMast cellUp-RegulationCell biologymedicine.anatomical_structurechemistryIonomycinImmunologybiology.proteinCytokinesThe Journal of Immunology
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Altered metabolism of gut microbiota contributes to chronic immune activation in HIV-infected individuals.

2015

Altered interplay between gut mucosa and microbiota during treated HIV infection may possibly contribute to increased bacterial translocation and chronic immune activation, both of which are predictors of morbidity and mortality. Although a dysbiotic gut microbiota has recently been reported in HIV + individuals, the metagenome gene pool associated with HIV infection remains unknown. The aim of this study is to characterize the functional gene content of gut microbiota in HIV + patients and to define the metabolic pathways of this bacterial community, which is potentially associated with immune dysfunction. We determined systemic markers of innate and adaptive immunity in a cohort of HIV-in…

ImmunologyHIV InfectionsBiologyGut floraAdaptive ImmunityMicrobiologyMetabolomicsImmunityAntiretroviral Therapy Highly ActiveRNA Ribosomal 16SMetabolomeImmunology and AllergyCluster AnalysisHumansMetabolomicsGeneCase-control studyBayes TheoremBiodiversityAcquired immune systembiology.organism_classificationImmunity InnateMarkov ChainsGastrointestinal MicrobiomeMetabolic pathwayCase-Control StudiesImmunologyDisease ProgressionHIV-1MetabolomeMetagenomeMucosal immunology
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Mast cells are crucial for early inflammation, migration of Langerhans cells, and CTL responses following topical application of TLR7 ligand in mice.

2007

Abstract Until recently, IgE-activated mast cells have been regarded merely as effector cells of adaptive immune responses, involved in allergic reactions and mucosal immunity to parasites. Herein, we report that murine dermal mast cells, activated by local administration of a cream containing the synthetic TLR7 ligand imiquimod, are essential to initiate an early inflammatory reaction. The mast-cell–derived cytokines TNF-α and IL-1β play an important role in this process. Furthermore, TLR7-activated mast cells are also able to promote the emigration of Langerhans cells, which partly depends on the expression of mast-cell–derived IL-1β. We have previously shown that TLR7 ligation enhances t…

ImmunologyInterleukin-1betaInflammationImmunoglobulin ELigandsBiochemistryMiceImmune systemAdjuvants ImmunologicCell MovementmedicineCytotoxic T cellAnimalsMast CellsAntigensSkinInflammationImmunity CellularMice Inbred BALB CVaccinesImiquimodMembrane GlycoproteinsbiologyTumor Necrosis Factor-alphaDegranulationCell BiologyHematologyTLR7Immunoglobulin EAcquired immune systemImmunity InnateInterleukin 33Toll-Like Receptor 7Langerhans CellsImmunologybiology.proteinAminoquinolinesImmunizationmedicine.symptomAgranulocytosisT-Lymphocytes CytotoxicBlood
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CD4-mediated functional activation of human CD4+CD25+ regulatory T cells

2007

Naturally occurring CD4(+)CD25(+)FoxP3(+) regulatory T cells (CD25(+) Tregs) constitute a specialized population of T cells that is essential for the maintenance of peripheral self-tolerance. The immune regulatory function of CD25(+) Tregs depends upon their activation. We found that anti-CD4 antibodies activate the suppressive function of human CD25(+) Tregs in a dose-dependent manner. We demonstrate that CD4-activated CD25(+) Tregs suppress the proliferation of CD4(+) and CD8(+) T cells, their IL-2 and IFN-gamma production as well as the capacity of CD8(+) T cells to re-express CD25. By contrast, anti-CD4 stimulation did not induce suppressive activity in conventional CD4(+) T cells. Thes…

ImmunologyInterleukin-2 Receptor alpha SubunitAntibodies MonoclonalFOXP3hemic and immune systemschemical and pharmacologic phenomenaCD8-Positive T-LymphocytesBiologyFlow CytometryLymphocyte ActivationT-Lymphocytes RegulatoryCoculture TechniquesImmune toleranceCell biologyInterleukin 21Immune systemCD4 AntigensImmunologyImmune ToleranceHumansImmunology and AllergyCytotoxic T cellIL-2 receptorCell activationCD8European Journal of Immunology
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Mechanisms underlying lineage commitment and plasticity of human γδ T cells.

2012

Phenotypic and functional heterogeneity are the hallmarks of effector and memory T cells. Upon antigen stimulation, γδ T cells differentiate into two major types of memory T cells: central memory cells, which patrol the blood and secondary lymphoid organs, and effector memory cells, which migrate to peripheral tissues. γδ T cells display in vitro a certain degree of plasticity in their function that is reminiscent of that which is observed in conventional CD4 T cells. Similar to CD4 T cells, in which a plethora of specialized subsets affect the host response, γδ T cells may readily and rapidly assume distinct Th1-, Th2-, Th17-, T(FH) and T regulatory-like effector functions, suggesting that…

ImmunologyReviewT-Lymphocytes RegulatoryInterleukin 21Cell MovementImmunology and AllergyCytotoxic T cellHumansIL-2 receptorAntigen-presenting cellhuman gamma delta T cells lineage subsets.Interleukin 3Settore MED/04 - Patologia GeneraleImmunity CellularCD40biologycytokines; effector and memory cells; γδ T cells; lineage-specifying factors; T-cell subsetsCell DifferentiationReceptors Antigen T-Cell gamma-deltaT-Lymphocytes Helper-InducerNatural killer T cellAcquired immune systemImmunity HumoralInfectious DiseasesImmunologybiology.proteinImmunologic Memory
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Protection from graft-versus-host disease by HIV-1 envelope protein gp120-mediated activation of human CD4+CD25+ regulatory T cells.

2009

AbstractNaturally occurring CD4+CD25+ regulatory T cells (Tregs) represent a unique T-cell lineage that is endowed with the ability to actively suppress immune responses. Therefore, approaches to modulate Treg function in vivo could provide ways to enhance or reduce immune responses and lead to novel therapies. Here we show that the CD4 binding human immunodeficiency virus-1 envelope glycoprotein gp120 is a useful and potent tool for functional activation of human Tregs in vitro and in vivo. Gp120 activates human Tregs by binding and signaling through CD4. Upon stimulation with gp120, human Tregs accumulate cyclic adenosine monophosphate (cAMP) in their cytosol. Inhibition of endogeneous cA…

ImmunologyTransplantation HeterologousGraft vs Host Diseasechemical and pharmacologic phenomenaCHO CellsMice SCIDBiologyHIV Envelope Protein gp120Lymphocyte ActivationBiochemistryT-Lymphocytes RegulatoryImmune tolerancechemistry.chemical_compoundMiceImmune systemCricetulusIn vivoMice Inbred NODCricetinaeCyclic AMPImmune ToleranceAnimalsHumansCyclic adenosine monophosphateIL-2 receptorhemic and immune systemsCell BiologyHematologyEnvelope glycoprotein GP120Cell biologyTransplantationchemistryImmunologyCD4 Antigensbiology.proteinHIV-1Signal transductionSignal TransductionBlood
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Differentiation of Effector/Memory Vδ2 T Cells and Migratory Routes in Lymph Nodes or Inflammatory Sites

2003

Vδ2 T lymphocytes recognize nonpeptidic antigens without presentation by MHC molecules and mount both immediate effector functions and memory responses after microbial infection. However, how Vδ2 T cells mediate different facets of a memory response remains unknown. Here, we show that the expression of CD45RA and CD27 antigens defines four subsets of human Vδ2 T cells with distinctive compartmentalization routes. Naive CD45RA+CD27+ and memory CD45RA−CD27+ cells express lymph node homing receptors, abound in lymph nodes, and lack immediate effector functions. Conversely, memory CD45RA−CD27− and terminally differentiated CD45RA+CD27− cells, which express receptors for homing to inflamed tissu…

Immunologychemical and pharmacologic phenomenachemokine receptorsBiologyMajor histocompatibility complexArticleeffector functions03 medical and health sciences0302 clinical medicineAntigenimmune system diseasesCell MovementT-Lymphocyte SubsetsLymph node stromal cellImmunology and AllergyAnimalsHumansCell LineageIL-2 receptorAntigen-presenting cell030304 developmental biologyγδ cellsInflammation0303 health sciencesEffectorvirus diseasesphosphoantigenshemic and immune systemsfunctional subsetsCell DifferentiationTumor Necrosis Factor Receptor Superfamily Member 7PhenotypeImmunologybiology.proteinLeukocyte Common AntigensLymphLymph NodesImmunologic Memory030215 immunologyHoming (hematopoietic)The Journal of Experimental Medicine
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Immunosenescence, inflammation and Alzheimer’s disease

2012

Abstract Ageing impacts negatively on the development of the immune system and its ability to fight pathogens. Progressive changes in the T-cell and B-cell systems over the lifespan of individuals have a major impact on the capacity to respond to immune challenges. The cumulative age-associated changes in immune competence are termed immunosenescence that is characterized by changes where adaptive immunity deteriorates, while innate immunity is largely conserved or even upregulated with age. On the other hand, ageing is also characterized by “inflamm-ageing”, a term coined to explain the inflammation commonly present in many age-associated diseases. It is believed that immune inflammatory p…

ImmunosenescenceImmunosenescence; Alzheimer’s disease; Inflammation; Cytokine; Chemokine; Lymphocyte; AgeingInflammationReviewDiseaseImmune systemmedicineDementiaCytokineInflammationSettore MED/04 - Patologia GeneraleInnate immune systembusiness.industryImmunosenescencebiochemical phenomena metabolism and nutritionmedicine.diseaseAcquired immune systemAgeingAgeingChemokineImmunologybacteriaLymphocytesense organsmedicine.symptombusinessAlzheimer’s disease
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What is the physiological function of mast cells?

2003

Under physiological conditions, skin mast cells preferentially localize around nerves, blood vessels and hair follicles. This observation, which dates back to Paul Ehrlich, intuitively suggests that these enigmatic, multifacetted protagonists of natural immunity are functionally relevant to many more aspects of tissue physiology than just to the generation of inflammatory and vasodilatory responses to IgE-dependent environmental antigens. And yet, for decades, mainstream-mast cell research has been dominated by a focus on the -undisputedly prominent and important - mast cell functions in type I immune responses and in the pathogenesis and management of allergic diseases. Certainly, it is ha…

InflammationAllergyWound HealingInnate immune systemCellInflammationDermatologyBiologymedicine.diseaseMast cellBiochemistryModels Biologicalmedicine.anatomical_structureImmune systemAntigenImmune SystemImmunoglobulin GImmunologymedicineAnimalsHumansMast Cellsmedicine.symptomWound healingMolecular BiologyExperimental dermatology
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Exploring a regulatory role for mast cells: 'MCregs'?

2010

Regulatory cells can mould the fate of the immune response by direct suppression of specific subsets of effector cells, or by redirecting effectors against invading pathogens and infected or neoplastic cells. These functions have been classically, although not exclusively, ascribed to different subsets of T cells. Recently, mast cells have been shown to regulate physiological and pathological immune responses, and thus to act at the interface between innate and adaptive immunity assuming different functions and behaviors at discrete stages of the immune response. Here, we focus on these poorly defined, and sometimes apparently conflicting, functions of mast cells.

InflammationEffectorMast cell; Regulatory cells; cell-cell crosstalkImmunologyRegulatory cellModels ImmunologicalAutoimmunityAdaptive ImmunityBiologybiochemical phenomena metabolism and nutritionAcquired immune systemT-Lymphocytes RegulatoryImmunity InnateClassical complement pathwaycell-cell crosstalkImmune systemRegulatory cellsNeoplasmsImmunologyImmune ToleranceMAST CELLAnimalsHumansImmunology and AllergyMast Cells
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