Search results for " Inflammation"

showing 10 items of 634 documents

Facilitated neurogenic inflammation in unaffected limbs of patients with complex regional pain syndrome.

2003

Pain, edema, increased skin temperature, reddening and trophic changes characterize complex regional pain syndrome (CRPS). Recently, we have been able to show facilitated neurogenic inflammation on the affected limb. In the current study unaffected limbs were examined after resolution of the CRPS symptoms to assess possible generalized changes predisposing to CRPS. In 12 patients and in 12 healthy volunteers dermal microdialysis in combination with electrical C-fiber stimulation was employed to induce neuropeptide release. Dialysate protein concentration and axon reflex vasodilation were measured. Neither in patients nor in controls did electrical stimulation lead to protein extravasation, …

AdultMicrodialysisVasodilationStimulationEdemamedicineHumansNeurogenic inflammationbusiness.industryGeneral NeuroscienceNeuropeptidesExtremitiesMiddle Agedmedicine.diseaseExtravasationElectric StimulationVasodilationComplex regional pain syndromeAnesthesiaAxon reflexFemaleDisease Susceptibilitymedicine.symptomNeurogenic InflammationbusinessComplex Regional Pain SyndromesNeuroscience letters
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Exhaled nitric oxide is associated with cyclic changes in sexual hormones

2013

Abstract Background We hypothesized that changes in the levels of sexual hormones during the menstrual cycle influence the concentration of nitric oxide in the exhaled air (FeNO) and alveolar exhaled nitric oxide (CANO). Methods Twelve healthy, non allergic women in their reproductive age (age range 25–37 years) were recruited. Subjects were studied, on alternate days, over the course of their menstrual cycle. At each visit, measurements of FeNO and CANO were performed. Progesterone and 17-β-estradiol concentrations were measured in salivary samples. Results Eight subjects completed the study. The levels of FeNO and CANO were 13 ± 4.7 pbb and 3.5 ± 1.9 pbb, respectively (mean ± SD). The mea…

AdultPulmonary and Respiratory Medicinemedicine.medical_specialtymedia_common.quotation_subjectReproductive ageBronchiSettore MED/10 - Malattie Dell'Apparato RespiratorioNitric OxideNitric oxidechemistry.chemical_compoundsexual hormonesInternal medicinemedicineHumansPharmacology (medical)SalivaGeneralized estimating equationMenstrual cycleMenstrual CycleProgesteronemedia_commonEstradiolbusiness.industryBiochemistry (medical)GenderBiomarkerHormonesSexual hormonesPulmonary AlveoliEndocrinologychemistryBreath TestsNon allergicExhaled nitric oxideAirway inflammation; Biomarker; Gender; Hormones; Adult; Breath Tests; Bronchi; Estradiol; Female; Humans; Menstrual Cycle; Nitric Oxide; Progesterone; Pulmonary Alveoli; Saliva; Pulmonary and Respiratory Medicine; Pharmacology (medical); Biochemistry (medical)FemalebusinessHormoneAirway inflammation
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Double Negative (CD19+IgG+IgD-CD27-) B Lymphocytes: A New Insight from Telomerase in Healthy Elderly, in Centenarian Offspring, and in Alzheimer’s Di…

2014

Background: We have previously reported the increase of IgD-CD27- (Double Negative, DN) B cell population in the aged. These memory B cells have short telomeres and poor abilities to proliferate in vitro. Here, we investigated whether the low ability of DN B cells to proliferate depends on the expression levels of the CD307d and CD22 inhibitory receptors or whether DN B cells can proliferate and reactivate telomerase by the engagement of both innate and adaptive immune receptors. Methods: Phenotypic analyses were made by using flow cytometry. Quantitative analysis of telomerase activity was made by using a TRAP and a photometric enzyme immunoassay in young, healthy elderly, centenarian offs…

AdultTelomeraseAgingImmunologyPopulationNaive B cellB-Lymphocyte SubsetsReceptors Antigen B-CellCentenarian offspringLymphocyte ActivationSeverity of Illness IndexCD19ImmunophenotypingYoung AdultAlzheimer DiseasemedicineIgD-CD27- (Double Negative DN) B cell population in the aged DN B cell telomerase activity in young elderly CO and AD patientsImmunology and AllergySettore MED/05 - Patologia ClinicaHumanseducationTelomeraseB cellCellular SenescenceAgedInflammationSettore MED/04 - Patologia GeneraleAged 80 and overeducation.field_of_studyCD40biologyB lymphocyteAge FactorsTLR9ImmunosenescenceMiddle Agedmedicine.anatomical_structurePhenotypeImmunologyAntigens Surfacebiology.proteinAlzheimerAging; Telomerase; B lymphocytes; Alzheimer; Centenarian offspring; InflammationSettore MED/26 - NeurologiaImmunologic Memory
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Menopause and ovariectomy cause a low grade of systemic inflammation that may be prevented by chronic treatment with low doses of estrogen or losarta…

2009

Abstract The incidence of cardiovascular diseases in premenopausal women is lower than in men or postmenopausal women. This study reports the discovery of a low grade of systemic inflammation, including monocyte adhesion to arterial endothelium, elicited by menopause or estrogen depletion. Chronic treatment with low dose of 17-β-estradiol or inhibition of the renin-angiotensin system reduced this inflammation. Using an in vitro flow chamber system with human arterial and venous endothelial cells, we found that leukocytes from healthy postmenopausal women were more adhesive to the arterial endothelium than those from premenopausal women regardless of the stimulus used on endothelial cells. I…

Adultmedicine.medical_specialtyEndotheliummedicine.drug_classOvariectomyImmunologyInflammationSystemic inflammationLosartanRats Sprague-DawleyInternal medicinemedicineCell AdhesionLeukocytesImmunology and AllergyAnimalsHumansCells CulturedInflammationCell adhesion moleculebusiness.industryEndothelial CellsEstrogensBenzazepinesMiddle Agedmedicine.diseaseRatsMenopausemedicine.anatomical_structureEndocrinologyLosartanEstrogenCase-Control StudiesOvariectomized ratFemaleEndothelium Vascularmedicine.symptomChemokinesMenopausebusinessAngiotensin II Type 1 Receptor BlockersCell Adhesion Moleculesmedicine.drugJournal of immunology (Baltimore, Md. : 1950)
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Oxidant mechanisms in childhood obesity: the link between inflammation and oxidative stress.

2011

Evidence of obesity-induced oxidative stress in adults has emerged in the past several years, and similar evidence has been demonstrated in children more recently. The reactive species of oxygen or nitrogen can chemically alter all major classes of biomolecules by modifying their structure and function. Organisms have developed mechanisms to protect biomolecules from the deleterious effects of free radicals. These include the enzymes superoxide dismutase, catalase, and glutathione peroxidase, as well as water and lipid-soluble antioxidants, such as glutathione, ascorbate (vitamin C), α-tocopherol (vitamin E), and β-carotene. Obesity creates oxidant conditions that favor the development of c…

Adultmedicine.medical_specialtyOxidative phosphorylationSystemic inflammationmedicine.disease_causeSuperoxide dismutasechemistry.chemical_compoundRisk FactorsPhysiology (medical)Internal medicinemedicineHumansObesityChildchemistry.chemical_classificationInflammationReactive oxygen speciesbiologybusiness.industryGlutathione peroxidaseBiochemistry (medical)Public Health Environmental and Occupational HealthGeneral MedicineGlutathioneMalondialdehydeOxidantsOxidative StressEndocrinologychemistrybiology.proteinmedicine.symptomMorbiditybusinessOxidative stressTranslational research : the journal of laboratory and clinical medicine
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Association of Inflammation Markers with Impaired Insulin Sensitivity and Coagulative Activation in Obese Healthy Women

2003

Abstract Insulin resistance is associated with a low chronic inflammatory state. In this study we investigated the relationship between impaired insulin sensitivity and selected markers of inflammation and thrombin generation in obese healthy women. We examined 32 healthy obese women (body mass index ≥ 28), with normal insulin sensitivity (NIS, n = 14) or impaired insulin sensitivity (n = 18), and 10 nonobese women (body mass index < 25). Impaired insulin sensitivity patients had significantly higher levels of C-reactive protein (CRP), TGF-β1, plasminogen activator inhibitor-1 (PAI-1), activated factor VII (VIIa), and prothrombin fragment 1 + 2 (F1 + 2) compared with either control s…

Adultmedicine.medical_specialtyobesityEndocrinology Diabetes and Metabolismmedicine.medical_treatmentClinical BiochemistryPopulationInflammationFactor VIIaBiologyBiochemistryEndocrinologyInsulin resistanceTransforming Growth Factor betaInternal medicineinsulin resistancePlasminogen Activator Inhibitor 1medicineHumansProtein PrecursorseducationBlood CoagulationPancreatic hormoneeducation.field_of_studyInsulinBiochemistry (medical)ThrombinMiddle Agedmedicine.diseaseObesityPeptide FragmentsC-Reactive ProteinEndocrinologyhemostasis; inflammation; insulin resistance; obesityinflammationhemostasisFemaleProthrombinmedicine.symptomBody mass indexPlasminogen activatorBiomarkers
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Pro-inflammatory status is not a limit for longevity: case report of a Sicilian centenarian

2020

Most studies on centenarians represent them as the best model of ageing. They are defined “delayers”, if they exhibit age-related diseases between 80 and 99 years, “survivors” if they show clinically demonstrable diseases before the age of 80 years, and “escapers” when they attain their 100th year of life without any common age-associated pathologies.

Aged 80 and overGerontologySettore MED/04 - Patologia GeneraleAgingGeriatrics gerontologymedia_common.quotation_subjectLongevityLongevityBiologylanguage.human_languageCase-Control StudieslanguageHumansLimit (mathematics)Geriatrics and GerontologyCentenarianSicilianCentenarian inflammation miRNAmedia_common
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Pro-inflammatory genetic background and zinc status in old atherosclerotic subjects.

2008

Inflammation and genetics are prominent mechanisms in the pathogenesis of atherosclerosis (AT) and its complications. In this review we discuss the possible impact on AT development of several genetic determinants involved in inflammation, oxidative stress and cytoprotection (IL-6, TNF-alpha, IL-10, CD14, TLR4, MT, HSP70). Genetic polymorphisms of these genes may affect a differential inflammatory response predisposing to AT. However, allelic polymorphisms of genes which increase the risk of AT frequently occur in the general population but, only adequate gene-environment-polymorphism interactions promote the onset of the disease. Zinc deficiency has been suggested as an environmental risk …

AgingCandidate genePopulationInflammationDiseaseBiologymedicine.disease_causeBiochemistryPathogenesisAgeing atherosclerosis inflammation geneticsmedicineSettore MED/05 - Patologia ClinicaHomeostasisHumansAlleleeducationMolecular BiologyAgedSettore MED/04 - Patologia GeneraleAged 80 and overInflammationeducation.field_of_studymedicine.diseaseAtherosclerosisOxidative StressZincNeurologyImmunologyZinc deficiencyMetallothioneinmedicine.symptomOxidative stressBiotechnologyAgeing research reviews
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Immunological and immunogenetic markers in sporadic Alzheimer’s disease

2006

Background: Common polymorphisms of genes controlling inflammation-modulating cytokines and acute-phase proteins which play important roles in the pathogenesis of Alzheimer''s disease (AD) have been shown to be associated with AD. Aims: The immunological and immunogenetic markers potentially useful for the AD risk evaluation and diagnosis are briefly reviewed. Conclusion: The state-of-the-art of immunological and immunogenetic markers of AD indicates that new tools and strategies are necessary to identify gene products useful as diagnostic tools.

AgingDiseaseImmunogeneticsDiagnostic toolsProteomicsPathogenesisApolipoproteins EAlzheimer DiseaseHumansMedicineOligonucleotide Array Sequence AnalysisInflammationAlzheimer’s disease cytokines immunogenetics inflammation proteomicsPolymorphism GeneticGeriatrics gerontologybusiness.industryDNARisk evaluationGene Expression RegulationPositron-Emission TomographyImmunologyCytokinesMicrogliaGeriatrics and GerontologybusinessBiomarkersAcute-Phase ProteinsAging Clinical and Experimental Research
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Diagnosis and management of asthma in the elderly.

2013

Bronchial asthma is one of the most common chronic diseases worldwide, and by definition not expected to recover with aging. However, the concept that asthma can affect older individuals has been largely denied in the past. In clinical practice, asthma that occurs in the most advanced ages is often diagnosed as COPD, thus leading to undertreatment or improper treatment. The heterogeneity of clinical and functional presentation of geriatric asthma, including the partial loss of reversibility and of the allergic component, contributes to this misconception. A large body of evidence has accumulated demonstrating that the prevalence of asthma in the most advanced ages is similar to that in youn…

Agingmedicine.medical_specialtyComorbiditySettore MED/10 - Malattie Dell'Apparato RespiratorioAffect (psychology)Human lungMedication AdherenceOlder patientsPartial lossInternal MedicinemedicineHumansAnti-Asthmatic AgentsIntensive care medicineAsthmaAgedCOPDbusiness.industryAsthma treatmentAge Factorsmedicine.diseaseComorbidityLung functionAsthmarespiratory tract diseasesClinical Practicemedicine.anatomical_structurePhysical therapybusinessAirway inflammationEuropean journal of internal medicine
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