Search results for " Linkage"

showing 10 items of 174 documents

Localization of non-specific X-linked mental retardation gene (MRX73) to Xp22.2.

2001

Clinical and molecular studies are reported on a family (MRX73) of five males with non-specific X-linked mental retardation (XLMR). A total of 33 microsatellite and RFLP markers was typed. The gene for this XLMR condition was been linked to DXS1195, with a lod score of 2.36 at theta = 0. The haplotype and multipoint linkage analyses suggest localization of the MRX73 locus to an interval of 2 cM defined by markers DXS8019 and DXS365, in Xp22.2. This interval contains the gene of Coffin-Lowry syndrome (RSK2), where a missense mutation has been associated with a form of non-specific mental retardation. Therefore, a search for RSK2 mutations was performed in the MRX73 family, but no causal muta…

Family HealthMaleGeneticscongenital hereditary and neonatal diseases and abnormalitiesCoffin–Lowry syndromeX ChromosomeGenetic LinkageHaplotypeChromosome MappingLocus (genetics)Biologymedicine.diseasePedigreeGenetic linkageIntellectual DisabilitymedicineHumansMissense mutationMicrosatelliteFemaleLod ScoreRestriction fragment length polymorphismGenetics (clinical)X chromosomeMicrosatellite Repeats
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Human cationic amino acid transporter gene hCAT-2 is assigned to 8p22 but is not the causative gene in lysinuric protein intolerance

1997

Lysinuric protein intolerance (LPI) is a recessively inherited amino acid disorder characterized by defective efflux of cationic amino acids at the basolateral membrane of the intestinal and renal tubular epithelium. Recently, cDNAs encoding the related proteins hCAT-2A and hCAT-2B have been cloned. These two carrier proteins are most likely the product of the same gene, hCAT-2. Using the hCAT-2B cDNA, we assigned the hCAT-2 gene to chromosome 8p22. Furthermore, by linkage analysis in Finnish LPI families, we ruled out that hCAT-2B is involved in LPI disease.

Genetic LinkageBiologyGene mappingGenetic linkageComplementary DNAGeneticsmedicineHumansAmino acid transporterAmino Acid Metabolism Inborn ErrorsGeneGenetics (clinical)chemistry.chemical_classificationLysineChromosome MappingMembrane Proteinsmedicine.diseaseLysinuric protein intoleranceAmino acidchemistryBiochemistryAmino Acid Transport Systems BasicEffluxCarrier ProteinsChromosomes Human Pair 8Microsatellite RepeatsHuman Genetics
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rDNA (18S-28S and 5S) co-localization and linkage between ribosomal genes and (TTAGGG)n telomeric sequence in the earthworm Octodrilus complanatus (A…

2002

Spermatogonial and metaphase I chromosomes of the lumbricid earthworm Octodrilus complanatus (Annelida: Oligochaeta) were examined using fluorescent in situ hybridization (FISH) with three repetitive DNA probes-5S rDNA, 18S-26S rDNA, and (TTAGGG)(n). Single-color FISH consistently mapped one chromosome pair per spread using either 5S rDNA or 18S-26S rDNA as probes. Simultaneous (18S-26S)-5S and (18S-26S)-(TTAGGG)(n) FISH demonstrated that repeated units of the two ribosomal families were overlapped and closely associated with telomeric sequences.

Genetic LinkageDNA Ribosomalchemistry.chemical_compoundbiology.animalGeneticsAnimalsLumbricidaeOligochaetaRepeated sequenceMolecular BiologyGeneIn Situ Hybridization FluorescenceGenetics (clinical)fishbiologyEcologyEarthwormTelomereRibosomal RNAbiology.organism_classificationMolecular biologyTelomerechemistryOligochaetaDNABiotechnology
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Polymerase chain reaction analysis of the Xba I polymorphism of the human complement C4 genes provides evidence for strong haplotype conservation.

1995

The genes coding for the two isotypes of the fourth component of human complement, C4A and C4B, are located between the HLA-B and -DR loci of the MHC. We studied the linkage relationship of the previously described XbaI RFLP to obtain further insight into the evolution of the tandemly arranged C4 genes. Using exon-specific PCR amplification followed by restriction analysis and direct DNA sequencing, the polymorphic site could be located in exon 40 of the C4 gene (cDNA position 5095). The polymorphism does not change an amino acid residue. Using nested PCR amplification with isotype-specific primers to amplify either C4A or C4B alleles the haplotype arrangement of the XbaI sites in both isot…

Genetic LinkageImmunologyMolecular Sequence DataBiologyPolymerase Chain Reactionlaw.inventionExonlawComplementary DNAImmunology and AllergyHumansDeoxyribonucleases Type II Site-SpecificGenePolymerase chain reactionGeneticsPolymorphism GeneticBase SequenceHaplotypeIntronChromosome MappingComplement C4General MedicineMolecular biologyRestriction siteHaplotypesRestriction fragment length polymorphismPolymorphism Restriction Fragment LengthHuman immunology
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Human type I cytokeratin genes are a compact cluster

1997

A YAC clone (211F11) containing approximately 0.5 Mb of human DNA was isolated from a human genomic library by PCR-based screening with cytokeratin (KRT) 13-specific primers. The YAC clone was mapped by FISH to the long arm of chromosome 17 (17q12→q21), a region to which several other type I KRT genes had been mapped previously. We now show by Southern blot hybridization and PFGE analyses that KRT13, 14, 15, and 16 are all contained within YAC clone 211F11. Long-range restriction mapping analysis of clone 211F11 and of two smaller YAC clones that were also isolated with KRT13-specific primers, suggests that KRT13, 14, 15, 16 and their linked type I genes KRT17 and 19, are contained in less …

Genetic LinkageLocus (genetics)BiologyPolymerase Chain ReactionRestriction mapGene mappingGene clusterGeneticsHumansGenomic libraryCloning MolecularChromosomes Artificial YeastMolecular BiologyIn Situ Hybridization FluorescenceGenetics (clinical)Southern blotGeneticsBase SequenceChromosome MappingMolecular biologyChromosome 17 (human)genomic DNAMultigene FamilyKeratinsDNA ProbesChromosomes Human Pair 17
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Sequence and evolution of the gene for the monomeric globin I and its linkage to genes coding for dimeric globins in the insect Chironomus thummi.

1995

We isolated genomic clones containing sequences encoding globins I and IA from a Chironomus thummi thummi genomic library. Three clones contain globin IA (ctt-1A) genes, while one contains a globin I (ctt-1) gene. The coding regions of the four genes are identical except for the single base substitution accounting for the globin I/IA polymorphism. The noncoding DNA flanking the coding region is more than 98% similar, confirming a previous hypothesis that the globin ctt-1 and ctt-1A genes are alleles. Hemoglobins I and IA are monomeric in the insect hemolymph. Earlier in situ hybridization studies suggested that monomeric and dimeric globin genes are clustered at different chromosomal loci. …

Genetic LinkageMolecular Sequence DataGenes InsectBiologyChironomidaechemistry.chemical_compoundMolecular evolutionhemic and lymphatic diseasesGeneticsCoding regionAnimalsGenomic libraryGlobinAmino Acid SequenceMolecular BiologyGeneEcology Evolution Behavior and SystematicsIn Situ HybridizationGeneticsPolytene chromosomeBase SequenceSequence Homology Amino AcidChromosome MappingMolecular biologyNoncoding DNABiological EvolutionGlobinschemistrySequence AlignmentDNAJournal of molecular evolution
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Development and implementation of high-throughput SNP genotyping in barley

2009

Abstract Background High density genetic maps of plants have, nearly without exception, made use of marker datasets containing missing or questionable genotype calls derived from a variety of genic and non-genic or anonymous markers, and been presented as a single linear order of genetic loci for each linkage group. The consequences of missing or erroneous data include falsely separated markers, expansion of cM distances and incorrect marker order. These imperfections are amplified in consensus maps and problematic when fine resolution is critical including comparative genome analyses and map-based cloning. Here we provide a new paradigm, a high-density consensus genetic map of barley based…

Genetic Markers0106 biological sciencesGenotypelcsh:QH426-470Genetic Linkagelcsh:BiotechnologyPopulationSingle-nucleotide polymorphismBiologyPolymorphism Single Nucleotide01 natural sciences03 medical and health sciencesGene mappinglcsh:TP248.13-248.65Research articleGeneticseducationAlleles030304 developmental biology2. Zero hungerGenetics0303 health scienceseducation.field_of_studyfood and beveragesHordeumSNP genotypingMinor allele frequencylcsh:GeneticsGenetic TechniquesGenetic distanceGenetic markerDoubled haploidy010606 plant biology & botanyBiotechnology
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Replication of linkage of familial hypobetalipoproteinemia to chromosome 3p in six kindreds

2002

Familial hypobetalipoproteinemia (FHBL) is a genetically heterogeneous condition characterized by very low apolipoprotein B (apoB) concentrations in plasma and/or low levels of LDL-cholesterol (LDL-C) with a propensity to developing fatty liver. In a minority of cases, truncation-specifying mutations of the apoB gene (APOB) are etiologic, but the genetic basis of most cases is unknown. We previously reported linkage of FHBL to a 10 cM region on 3p21.1-22 in one kindred. The objectives of the current study were to identify other FHBL families with linkage to 3p and to narrow the FHBL susceptibility region on 3p. Six additional FHBL kindreds unlinked to the APOB region on chromosome 2 were ge…

Genetic MarkersAdultMaleMeiosiSettore MED/09 - Medicina InternaApolipoprotein BGenotypeGenetic LinkageQD415-436BiologyBiochemistryChromosomal crossoverHypobetalipoproteinemiasEndocrinologyQuantitative Trait HeritableGenetic linkageGenetic MarkerHaplotypeHumanslinkage analysisCrossing Over GeneticChildAgedAdult; Aged; Aged 80 and over; Child; Chromosome Mapping; Chromosomes Human Pair 3; Crossing Over Genetic; Female; Genetic Linkage; Genetic Markers; Genotype; Haplotypes; Humans; Hypobetalipoproteinemias; Male; Meiosis; Middle Aged; Pedigree; Quantitative Trait HeritableGeneticsAged 80 and overGenetic heterogeneityHaplotypeChromosomeChromosome MappingCell BiologyoligogenicMiddle AgedPedigreeMeiosisMarkov chain Monte CarloChromosome 3HaplotypesGenetic markerbiology.proteinvariance componentslipids (amino acids peptides and proteins)FemaleChromosomes Human Pair 3geneticHypobetalipoproteinemiaHuman
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Fine mapping of the 2p11 dyslexia locus and exclusion of TACR1 as a candidate gene.

2003

Developmental dyslexia, or reading disability, is a multigenic complex disease for which at least five loci, i.e. DYX1-3 and DYX5-6, have been clearly identified from the human genome. To date, DYX1C1 is the only dyslexia candidate gene cloned. We have previously reported linkage to 2p11 and 7q32 in 11 Finnish pedigrees. Here, we report the fine mapping of the approximately 40-cM linked region from chromosome 2 as we increased marker density to one per 1.8 cM. Linkage was supported with the highest NPL score of 3.0 (P=0.001) for marker D2S2216. Association analysis using the six pedigrees showing linkage pointed to marker D2S286/rs3220265 (P value0.001) in the near vicinity of D2S2216. We w…

Genetic MarkersCandidate geneLocus (genetics)Quantitative trait locusBiologyPolymorphism Single NucleotideDyslexia03 medical and health sciences0302 clinical medicineGene mappingGenetic linkageGeneticsmedicineHumansGenetics (clinical)FinlandReceptors Tachykinin030304 developmental biologyGenetics0303 health sciencesGene Expression ProfilingHaplotypeDyslexiaChromosome Mappingmedicine.diseaseBlotting NorthernPedigreeGenetic markerChromosomes Human Pair 2030217 neurology & neurosurgeryMicrosatellite RepeatsHuman genetics
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HLA antigens in Sicilian patients affected by chronic myelogenous leukaemia.

1987

SUMMARY HLA antigens were investigated in Sicilian patients with chronic myelogenous leukaemia (CML) and in Sicilian healthy controls. The frequency of the HLA-DRw6 antigen was significantly decreased in the group of patients. These results suggest that DRw6 may be a marker for decreased susceptibility to the etiological or pathogenic mechanism(s) which produce CMLs.

Genetic MarkersGenetic LinkageImmunologyHLA-DR6 AntigenHuman leukocyte antigenImmunogeneticsHLA-DR AntigensBiologylanguage.human_languageAntigenGene FrequencyHaplotypesItalyHLA Antigenshemic and lymphatic diseasesLeukemia Myelogenous Chronic BCR-ABL PositiveImmunologyGeneticsEtiologylanguageHumansRisk factorChronic myelogenous leukaemiaSicilianJournal of immunogenetics
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