Search results for " Simplex"

showing 10 items of 123 documents

Clinical practice guidelines: Oral health care for children and adults living with epidermolysis bullosa.

2020

Background Inherited epidermolysis bullosa (EB) is a genetic disorder characterized by skin fragility and unique oral features. Aims To provide (a) a complete review of the oral manifestations in those living with each type of inherited EB, (b) the current best practices for managing oral health care of people living with EB, (c) the current best practices on dental implant-based oral rehabilitation for patients with recessive dystrophic EB (RDEB), and (d) the current best practice for managing local anesthesia, principles of sedation, and general anesthesia for children and adults with EB undergoing dental treatment. Methods Systematic literature search, panel discussion including clinical…

AdultSpecial Issue Articlesmedicine.medical_specialtymedicine.medical_treatmentBest practiceAnesthesia DentalOral Healthepidermolysis bullosa simplexrecessive dystrophic epidermolysis bullosadystrophic epidermolysis bullosa03 medical and health sciencesWound care0302 clinical medicineQuality of life (healthcare)junctional epidermolysis bullosadental treatmentdental implantsmedicineHumansLocal anesthesia030212 general & internal medicineepidermolysis bullosaChildGeneral DentistryRehabilitationbusiness.industryoral care030206 dentistryGuidelinekindler epidermolysis bullosamedicine.diseasegeneral anesthesiaEpidermolysis Bullosa Dystrophicastomatognathic diseasesSystematic reviewsedationoral rehabilitationFamily medicinePractice Guidelines as TopicQuality of LifeEpidermolysis bullosabusinessclinical practice guidelineSpecial care in dentistry : official publication of the American Association of Hospital Dentists, the Academy of Dentistry for the Handicapped, and the American Society for Geriatric Dentistry
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Attributional style in a case of Cotard delusion.

2007

Young and colleagues (e.g. Young, A. W., & Leafhead, K. M. (1996). Betwixt life and death: case studies of the Cotard delusion. In P. W. Halligan & J. C. Marshall (Eds.), Method in madness: Case studies in cognitive neuropsychiatry. Mahway, NJ: Lawrence Erlbaum Associates.) have suggested that cases of the Cotard delusion (the belief that one is dead) result when a particular perceptual anomaly (caused by a disruption to the affective component of visual recognition) occurs in the context of an internalising attributional style. This hypothesis has not previously been tested directly. We report here an investigation of attributional style in a 24-year-old woman with Cotard delusion ("LU"). …

Adultmedia_common.quotation_subjectExperimental and Cognitive PsychologyContext (language use)Cotard delusionDelusionsDevelopmental psychologyArts and Humanities (miscellaneous)DelusionSocial cognitionDevelopmental and Educational PsychologymedicinePersonalityHumansmedia_commonEgoEpilepsyBrainRecognition Psychologymedicine.diseaseDeathCapgras SyndromeSocial PerceptionCase-Control StudiesFaceCognitive neuropsychiatryFemaleEncephalitis Herpes Simplexmedicine.symptomPsychologyCognitive styleCapgras SyndromePersonalityConsciousness and cognition
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Combined therapy with acyclovir and adenosine arabinoside in herpes simplex encephalitis.

1987

We report a case of herpes simplex encephalitis (HSE) with initial coma and severe left-sided hemiparesis in which combined treatment with adenine arabinoside and acyclovir was followed by complete recovery. This favorable result is discussed in view of the literature of HSE treatment including experimental studies on antiviral activity of both drugs. Combined treatment may be useful in severe cases of HSE.

Adultmedicine.medical_treatmentAcyclovirmedicineHumansAciclovirVidarabineComaChemotherapybusiness.industryHerpes Simplexmedicine.diseaseVirologyAdenosineHemiparesisNeurologyEncephalitisDrug Therapy CombinationFemaleNeurology (clinical)Viral diseasemedicine.symptombusinessTomography X-Ray ComputedEncephalitisVidarabinemedicine.drugEuropean neurology
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Factors influencing the interaction of herpes simplex virus glycoprotein C with the third component of complement.

1992

The factors influencing the interaction of herpes simplex virus (HSV) glycoprotein C (gC) with the third component of complement (C3) were investigated in this study. The ability of gC of HSV type 1 (gC-1) to bind to the C3b fragment of C3 was found to be influenced by cell specific processing of gC-1 in a different manner, binding being remarkably enhanced in some cell lines following removal of sialic acid residues. Testing several intertypic recombinants of HSV we found that only strains expressing gC-1 exhibited binding to C3b, even though their genome consisted mainly of HSV-2 sequences in some recombinants. Expression of type-2 glycoproteins gB, gD, gE, gG, gH, and gI did not alter th…

AnionsRosette FormationMolecular Sequence DataBiologyIn Vitro Techniquesmedicine.disease_causeViruschemistry.chemical_compoundStructure-Activity RelationshipViral Envelope ProteinsVirologymedicineTumor Cells CulturedAnimalsHumansSimplexvirusAmino Acid SequenceVero Cellschemistry.chemical_classificationHeparinTemperatureGeneral MedicineNeomycinHerpesvirus glycoprotein BVirologySialic acidHerpes simplex viruschemistryComplement C3bVero cellbiology.proteinAntibodyGlycoproteinmedicine.drugProtein BindingArchives of virology
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Synthesis and biological evaluation of pyridinebetaine A and B

2009

The synthesis of the marine natural products pyridinebetaine A and B is reported. The biological evaluation of pyridinebetaine A and B and several analogues as cytotoxic, antifungal and antiviral agents is also described. Unfortunately, none of the compounds tested showed relevant antifungal or cytotoxic activity. Only pyridinebetaine B reduced the Herpes simplex virus type 1 virus replication, though only weakly.

AntifungalAntifungal Agentsmedicine.drug_classHerpesvirus 1 HumanPlant ScienceBiologyVirus Replicationmedicine.disease_causeAntiviral AgentsBiochemistryAnalytical ChemistryMicrobiologyStructure-Activity RelationshipAlkaloidsChlorocebus aethiopsmedicineAnimalsHumansCytotoxic T cellVero CellsBiological evaluationMolecular StructureOrganic ChemistryFungiVirologyBetaineHerpes simplex virusViral replicationHeLa CellsNatural Product Research
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Impact of infectious burden on progression of carotid atherosclerosis.

2002

Background and Purpose— Recent findings suggest a causative role of infections in the pathogenesis of atherosclerosis. The extent of atherosclerosis and the prognosis of patients with atherosclerosis seem to be increased by the number of infections to which an individual has been exposed. In a prospective study, we evaluated the effect of 8 pathogens and the aggregate pathogen burden on the progression of carotid atherosclerosis. Methods— In 504 patients (74.9% men; age, 62.9±10 years), we measured intima-media thickness and prevalence of carotid artery stenosis. Follow-up measurements after a mean of 2.5 years were available in 427 patients (85%). Blood samples were taken, and IgG or IgA …

Carotid Artery DiseasesMalemedicine.medical_specialtyMycoplasma pneumoniaePathologyEpstein-Barr Virus InfectionsStatinHaemophilus Infectionsmedicine.drug_classComorbiditymedicine.disease_causeAntibodies ViralInfectionsGastroenterologyVirusHelicobacter InfectionsSeroepidemiologic StudiesInternal medicineGermanymedicineOdds RatioPrevalenceHumansMycoplasma InfectionsProspective StudiesProspective cohort studyChlamydophila InfectionsUltrasonographyAdvanced and Specialized NursingChlamydiaVascular diseasebusiness.industryHerpes SimplexOdds ratioMiddle Agedmedicine.diseaseAntibodies BacterialImmunoglobulin AHerpes simplex virusCarotid ArteriesImmunoglobulin GCytomegalovirus InfectionsDisease ProgressionFemaleNeurology (clinical)Cardiology and Cardiovascular MedicinebusinessTunica IntimaTunica MediaStroke
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Differentiation of herpes simplex virus-induced fusion from without and fusion from within by cyclosporin A and compound 48/80.

1991

Treating strains of herpes simplex virus (HSV) in culture with either cyclosporin A or compound 48/80, allowed the strains to be divided into two groups. Group 1 contains the strains ANG and HFEM of HSV-1 and Lux syn (HSV-2) producing fusion from within (FFWI) and fusion from without (FFWO). Cyclosporin A fails to inhibit both types of fusion at concentrations up to 100 microM. Strains ANG and HFEM belong to the syn 3 marker locus group identified for HSV-1. Group 2 contains all other fusion-producing strains of HSV tested so far. Cyclosporin A inhibits FFWI at concentrations as low as 10 to 20 microM. These strains belong to the syn locus marker groups 1, 2, 4 and 5. From the fact that mut…

Cell fusionbiologyCyclosporinsCompound 48/80biology.organism_classificationmedicine.disease_causeVirus ReplicationVirologyVirusCell Fusionchemistry.chemical_compoundStructure-Activity RelationshipHerpes simplex viruschemistryCell cultureVirologyCyclosporin aAlphaherpesvirinaemedicineAnimalsSimplexvirusp-Methoxy-N-methylphenethylamineVero CellsCyclophilinThe Journal of general virology
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Two mutations in gB-1 and gD-1 of herpes simplex virus type 1 are involved in the "fusion from without" phenotype in different cell types.

1996

Previous studies have shown that certain strains of herpes simplex viruses type 1 (HSV-1) are able to induce “fusion from without” (FFWO) which means no transcription or translation of the viral genome happens. The main determinants for FFWO in BHK cells are mutations in the C-terminal part of gB-1. But single mutations in this part of the genome are not sufficient to transfer the FFWO phenotype also to Vero cells. Here, we report that FFWO of HSV strains indeed need additional mutations in the N-terminal part of gD in order to produce the FFWO phenotype in BHK and Vero cells. By marker transfer we are able to show that loss of mutations in the N-terminal part of gD influences the ability t…

Cell typevirusesCellMolecular Sequence DataGenome ViralHerpesvirus 1 HumanBiologymedicine.disease_causeTransfectionGiant CellsVirusCell LineViral Envelope ProteinsTranscription (biology)VirologyCricetinaeChlorocebus aethiopsGeneticsmedicineBaby hamster kidney cellAnimalsHumansAmino Acid SequenceMolecular BiologyVero CellsBase SequenceGeneral MedicineVirologyPhenotypemedicine.anatomical_structureHerpes simplex virusPhenotypeDNA ViralMutationVero cellVirus genes
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Structural and regulatory functions of keratins.

2007

The diversity of epithelial functions is reflected by the expression of distinct keratin pairs that are responsible to protect epithelial cells against mechanical stress and to act as signaling platforms. The keratin cytoskeleton integrates these functions by forming a supracellular scaffold that connects at desmosomal cell-cell adhesions. Multiple human diseases and murine knockouts in which the integrity of this system is destroyed testify to its importance as a mechanical stabilizer in certain epithelia. Yet, surprisingly little is known about the precise mechanisms responsible for assembly and disease pathology. In addition to these structural aspects of keratin function, experimental e…

CellIntermediate Filamentsmacromolecular substancesBiologyFocal adhesionEpidermolysis bullosa simplexMicrotubuleOrganelleKeratinmedicineAnimalsHumansCytoskeletonCytoskeletonchemistry.chemical_classificationCell PolarityEpithelial CellsCell BiologyDesmosomesmedicine.diseaseCell biologymedicine.anatomical_structureCell Transformation NeoplasticchemistryKeratinsStress MechanicalFunction (biology)Signal TransductionExperimental cell research
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Loss of surface fibronectin after infection of cultured cells by HSV-1 and 2

1985

Fibronectin is lost from the surface of HSV infected cells during cell rounding. In order to investigate also the fate of fibronectin during the process of HSV-induced cell-fusion, BHK, Vero as well as primary or secondary rabbit kidney cells were infected with HSV-1 strains producing cell-fusion. By immunofluorescence and immunoelectron microscopy a considerable loss of fibronectin after HSV infection could be demonstrated leaving only irregular clumps of fibronectin containing virus particles on the cell surface. Decrease and disarrangement of fibronectin was similar during cell rounding and cell fusion. Loss of Fibronectin was closely connected with the two types of the cytopathic effect…

Cytochalasin BvirusesImmunoelectron microscopyBiologyKidneyVirus ReplicationCell FusionCell membranechemistry.chemical_compoundCricetinaeVirologymedicineAnimalsSimplexvirusProtease InhibitorsCytochalasin BCells CulturedCytopathic effectCell fusionHerpes SimplexGeneral MedicineActin cytoskeletonVirologyFibronectinsFibronectinActin Cytoskeletonmedicine.anatomical_structurechemistryCell culturebiology.proteinRabbitsArchives of Virology
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