Search results for " Small"

showing 10 items of 806 documents

A phase II study of induction chemotherapy followed by concurrent chemoradiotherapy in elderly patients with locally advanced non-small-cell lung can…

2007

The optimal management of unresectable locally advanced non-small-cell lung cancer in older patients has not been defined to date. The present phase II study was planned to evaluate the activity and safety of platinum-based induction chemotherapy followed by concurrent chemoradiotherapy in elderly patients with locally advanced non-small-cell lung cancer. Patients received two cycles of paclitaxel (175 mg/m) and carboplatin (area under the curve: 5) day 1, every 3 weeks. Chemoradiotherapy (thoracic radiation therapy) was initiated on day 42 and consisted of 1.8 Gy daily, five times per week over 5 weeks (45.0 Gy target dose) followed by 10 2.0 Gy daily fractions. Concomitant chemotherapy wa…

MaleOncologyCancer Researchmedicine.medical_specialtyLung NeoplasmsSettore MED/06 - Oncologia MedicaLocally advancedPhases of clinical researchDisease-Free SurvivalDrug Administration ScheduleOlder patientsCarcinoma Non-Small-Cell LungInternal medicineAntineoplastic Combined Chemotherapy ProtocolsmedicineHumansPharmacology (medical)Lung cancerAgedNeoplasm StagingPharmacologybusiness.industryInduction chemotherapymedicine.diseaseCombined Modality TherapyNeoadjuvant TherapyOptimal managementConcurrent chemoradiotherapynon-small-cell lung cancerchemoradiotherapyOncologyFemaleNon small cellbusiness
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Prognostic Role of Mismatch Repair Status, Histotype and High-Risk Pathologic Features in Stage II Small Bowel Adenocarcinomas

2020

Abstract Background Small bowel adenocarcinoma is a relatively rare cancer, often diagnosed in an advanced stage. In localized and resectable disease, surgery alone or in combination with adjuvant chemotherapy is the mainstay of treatment. In the recently published National Comprehensive Cancer Network Clinical Practice guidelines, criteria for selecting patients with stage II small bowel adenocarcinoma to receive adjuvant chemotherapy are provided, and they are mainly extrapolated from studies on colorectal cancer. Patients and Methods In the present study, we aimed to verify whether mismatch repair deficiency phenotype, high-risk pathologic features (including T4, positive resection margi…

MaleOncologyColorectal cancerDNA Mismatch RepairCOLORECTAL-CANCERSettore MED/120302 clinical medicinePMS2small bowel adenocarcinomaMismatch Repair Endonuclease PMS20303 health sciencesPrognosisMMRMutS Homolog 2 ProteinOncologyCARCINOMAS030220 oncology & carcinogenesisimmunohistochemistryMismatch Repair Status small bowel adenocarcinomaFemaleMicrosatellite InstabilityDNA mismatch repairMutL Protein Homolog 1Colorectal Neoplasmsstage IImedicine.medical_specialtyhigh-risk pathologic featuresDNA Mismatch Repair; Female; Humans; Male; Microsatellite Instability; Mismatch Repair Endonuclease PMS2; MutL Protein Homolog 1; MutS Homolog 2 Protein; Prognosis; Adenocarcinoma; Colorectal Neoplasmssmall bowel adenocarcinoma; mismatch repair statusAdenocarcinomaNO03 medical and health sciencessmall bowel carcinomahistotypeInternal medicineTranslational ResearchmedicineHumansmismatch repair status030304 developmental biologysmall bowel adenocarcinomasbusiness.industryCancerMicrosatellite instabilityMismatch Repair ProteinAdenocarcinoma IBD Cancermedicine.diseasedigestive system diseasesMSH6COLORECTAL-CANCER; CARCINOMAS; CONSENSUSsmall bowel carcinoma MMR immunohistochemistryMismatch repair Small bowel AdenocarcinomaMSH2Mismatch repair status; stage II; small bowel adenocarcinomas; histotype; high-risk pathologic featuresSurgeryCONSENSUSbusiness
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Cetuximab in small bowel adenocarcinoma: a new friend?

2010

Sir, Small bowel adenocarcinoma (SBA) is a rare and aggressive tumour. SBA in the United States increased from 5.7 cases per million in 1973 to 7.3 cases per million in 2004 (Surveillance Epidemiology and End Results (SEER), 1973–2004 database; Jemal et al (2009). Surgery is the mainstay of treatment, even if chemotherapy in advanced disease has been associated with an increased survival. The most effective agents include 5-FU, irinotecan, platinum agents and gemcitabine (Fishman et al, 2006; Speranza et al, 2010). The molecular characterisation of this cancer could help to improve prognosis. Specifically, the frequency of KRAS gene mutations is similar than in colorectal cancer (Ari et al,…

MaleOncologyIntestinal NeoplasmCancer Researchmedicine.medical_specialtySurvivalSettore MED/06 - Oncologia MedicaColorectal cancerAdenocarcinoma; Antibodies Monoclonal; Antibodies Monoclonal Humanized; Antineoplastic Agents; Antineoplastic Combined Chemotherapy Protocols; Camptothecin; Cetuximab; Combined Modality Therapy; Female; Humans; Immunotherapy; Intestinal Neoplasms; Intestine Small; Male; Middle Aged; Survival; Oncology; Cancer ResearchCetuximabAdenocarcinomaGene mutationAntibodies Monoclonal Humanizedmedicine.disease_causeAntineoplastic AgentInternal medicineIntestine SmallmedicineAntineoplastic Combined Chemotherapy ProtocolCetuximabbusiness.industryAntibodies MonoclonalCancerMiddle Agedmedicine.diseaseCombined Modality TherapyGemcitabineSurgeryIrinotecanOncologyFOLFIRICamptothecinFemaleImmunotherapyKRASbusinessHumanmedicine.drug
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Long-term quality of life in inoperable non-small cell lung cancer patients treated with conventionally fractionated compared to hyperfractionated ac…

2017

Abstract Background and purpose To evaluate the quality of life (QoL) of patients with inoperable non-small cell lung cancer treated with conventionally fractionated radiotherapy (CF) vs. continuous hyperfractionated accelerated radiotherapy weekend-less (CHARTWEL). Material and methods The largest monocentric subgroup of the phase III CHARTWEL trial was analyzed up to three years after randomization. QoL was assessed with the European Organization for Research and Treatment of Cancer QoL Core Questionnaire (QLQ-C30) and lung cancer module (QLQ-LC13) and compared using linear mixed models. QoL interrelations with recurrence, metastasis, and death were explored by multi-state modeling. Resul…

MaleOncologymedicine.medical_specialtyLung NeoplasmsRandomizationMetastasis03 medical and health sciences0302 clinical medicineQuality of lifeCarcinoma Non-Small-Cell LungInternal medicinemedicineHumansRadiology Nuclear Medicine and imaging030212 general & internal medicineLung cancerHyperfractionated accelerated radiotherapyAgedAged 80 and overbusiness.industryCancerHematologyMiddle Agedmedicine.diseaseDysphagiahumanitiesTreatment OutcomeOncology030220 oncology & carcinogenesisQuality of LifeFemaleDose Fractionation RadiationNon small cellNeoplasm Recurrence Localmedicine.symptombusinessRadiotherapy and Oncology
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Italian clinical research in non-small-cell lung cancer

2005

ABSTRACT Lung cancer is the most common cause of cancer deaths in both men and women worldwide and has a poor prognosis. Non-small-cell lung cancer (NSCLC) represents approximately 80% of all lung cancers. Surgery is the only curative treatment of NSCLC but only 15–20% of tumours can be radically resected with a survival of about 40% at 5 years. Considering these disappointing results NSCLC is one of the most frequent subjects of clinical research worldwide. Italy is playing an important role in the clinical research of NSCLC performing phase I, II and III trials, prevalently by cooperative groups, and achieving important results that contributed to define the standard treatment for NSCLC p…

MaleOncologymedicine.medical_specialtyPoor prognosisLung NeoplasmsNSCLCCarcinoma Non-Small-Cell LungInternal medicineAntineoplastic Combined Chemotherapy ProtocolsmedicineHumansLung cancerneoplasmsAgedClinical Trials as TopicLungbusiness.industryStandard treatmentCancerHematologymedicine.diseaseItalian clinical researchrespiratory tract diseasesSurvival RateClinical trialTreatment OutcomeClinical researchmedicine.anatomical_structureItalyOncologyChemotherapy AdjuvantFemaleRadiotherapy AdjuvantNon small cellCisplatinLung cancerbusiness
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Difference in the expression of IL-9 and IL-17 correlates with different histological pattern of vascular wall injury in giant cell arteritis

2015

OBJECTIVE: GCA is a large- and medium-vessel arteritis characterized by a range of histological patterns of vascular wall injury. The aim of this study was to immunologically characterize the various histological patterns of GCA. METHODS: Thirty-five consecutive patients with biopsy-proven GCA and 15 normal controls were studied. IL-8, IL-9, IL-9R, IL-17, IL-4, TGF-β and thymic stromal lymphopoietin expression was evaluated by RT-PCR and immunohistochemistry on artery biopsy specimens. Confocal microscopy was used to characterize the phenotypes of IL-9-producing and IL-9R-expressing cells. Five additional patients who had received prednisone when the temporal artery biopsy was performed wer…

MalePathologyBiopsyT-LymphocytesSettore BIO/13 - Biologia ApplicataTransforming Growth Factor betaTh9Pharmacology (medical)Aged 80 and overMicroscopy Confocalmedicine.diagnostic_testSmall vessel vasculitisVasa vasorum vasculitiInterleukin-17vasa vasorum vasculitis Giant cell arteritiMiddle AgedTemporal Arteriesmedicine.anatomical_structurePhenotypeVasa vasorum vasculitisSmall vessel vasculitiCytokinesFemaleTh17medicine.symptomVasculitisgiant cell arteritimedicine.medical_specialtyThymic stromal lymphopoietinGiant Cell ArteritisInflammationThymic Stromal LymphopoietinRheumatologyBiopsyTh17; Th9; giant cell arteritis; small vessel vasculitis; vasa vasorum vasculitismedicineHumansInterleukin 9ArteritisGlucocorticoidsAgedbusiness.industryInterleukin-9Vascular System Injuriesmedicine.diseaseGiant cell arteritisSettore MED/16 - ReumatologiaVasa vasorumCase-Control StudiesImmunologyPrednisonebusinessBiomarkers
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Unusual B cell morphology in inflammatory bowel disease.

2012

B lymphocytes express various different types of surface immunoglobulins that are largely unrelated to other hematological lines, although some reports have described a relationship between malignant B cells and other cells such as macrophages. Multiple genes of hematopoietic lineage, including transcription factors, are co-expressed in hematopoietic stem cells and progenitors, a phenomenon referred to as "lineage priming". Changes in the expression levels and timing of transcription factors can induce the lineage conversion of committed cells, which indicates that the regulation of transcription factors might be particularly critical for maintaining hierarchical hematopoietic development. …

MalePathologyCD79BiopsyUlcerativeSmallInflammatory bowel diseaseInflammatory bowel diseaseMucosal immunityCrohn DiseaseIntestine SmallLymphocytesMicroscopyB-Lymphocytesmedicine.diagnostic_testMiddle AgedColitisFucosyltransferasesIntestineSurfaceHaematopoiesismedicine.anatomical_structureAntigens SurfaceFemaleStem cellB-1 B cellsAdultmedicine.medical_specialtyLymphocyte homingColonLewis X AntigenBiologyFluorescencePathology and Forensic MedicineAntigeninflammatory bowel diseaseBiopsymedicineHumansCell LineageProgenitor cellAntigensB cellInflammatory bowel disease; Inflammation; Mucosal immunity; Lymphocytes; B-1 B cells; Lymphocyte homing; CD15+cells; Adult; Antigens Surface; B-Lymphocytes; Biomarkers; Biopsy; Cell Lineage; Cell Nucleus; Colitis Ulcerative; Colon; Crohn Disease; Female; Fucosyltransferases; Humans; Immunoglobulin M; Inflammatory Bowel Diseases; Intestine Small; Lewis X Antigen; Male; Microscopy Fluorescence; Middle Aged; RectumInflammationCell NucleusRectumCell Biologymedicine.diseaseInflammatory Bowel DiseasesImmunoglobulin MMicroscopy FluorescenceImmunologyColitis UlcerativeCD15+cellsBiomarkersPathology, research and practice
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The mechanism of epithelial shedding after ischemic damage to the small intestinal mucosa

1979

The intestinal mucosa of the rat was examined by light and electron microscopy 15, 30, 60 and 120 min after complete ligation of the vessel arcades of the proximal jejunum. The characteristic sign of ischemic damage to the small intestinal mucosa and the reason for epithelial shedding is the appearance of membrane enclosed cytoplasmic blebs which arise at the cell base of the enterocytes and detach the epithelium from the basement membrane. This process begins at the tip of the villi before the enterocytes display signs of irreversible damage and progress to the base of the villi with continuation of the ischemia.

MalePathologymedicine.medical_specialtyCellIschemiaBiologyBasement MembranePathology and Forensic Medicinelaw.inventionIntestinal mucosaIschemialawIntestine SmallmedicineAnimalsRegenerationIntestinal MucosaBasement membraneAnatomymedicine.diseaseEpitheliumRatsMicroscopy Electronmedicine.anatomical_structureCytoplasmElectron microscopeLigationVirchows Archiv B Cell Pathology
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Morphology and chronology of ischemic mucosal changes in the small intestine

1983

The morphology and chronology of an absolute ischemia was investigated on the small intestine of man and rat. It could be shown that the morphogenesis of ischemic damage to the intestinal mucosa in man does not differ from that in the rat. However, there are crucial and in therapeutic terms significant differences in the time course of the ischemic damage to the intestinal mucosa. Whereas in the small intestine of the rat, the first appreciable alterations - detachment and disruption of the lining epithelium at the villous tip - are already to be observed after absolute ischemia lasting 15 min, in the human small intestine the same lesions only occur after 4 h. A comparable complete necrosi…

MalePathologymedicine.medical_specialtyNecrosisIschemiaMorphogenesislaw.inventionNecrosisIntestinal mucosaIschemialawIntestine SmallDrug DiscoverymedicineAnimalsHumansIntestinal MucosaGenetics (clinical)ChemistryRats Inbred StrainsGeneral Medicinemedicine.diseaseMolecular medicineEpitheliumSmall intestineRatsMicroscopy Electronmedicine.anatomical_structureMicroscopy Electron ScanningMolecular MedicineElectron microscopemedicine.symptomKlinische Wochenschrift
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Ischemia and post-ischemic regeneration of the small intestinal mucosa

1979

After reversible ligation of the arcade vessels of the proximal jejunum, the intestinal mucosa was investigated by light microscopic and autoradiographic methods after 15, 30, 60, 120 and 300 min of ischemia. Early ischemic damage to the mucosa (after about 15 min) is characterized by shedding of not yet irreversibly damaged enterocytes from the tips of the villi into the intestinal lumen and bleb formation starting at the base of the epithelia. This process advances from the tips to the bases of the villi with increasing duration of ischemia, and the villi are completely denuded of epithelium after ischemia lasting 2 h. Remains of the small intestinal crypts are still present at this time.…

MalePathologymedicine.medical_specialtyTime FactorsIschemiadigestive systemIntestinal mucosaIschemiaIntestine SmallmedicineAnimalsRegenerationBleb (cell biology)Intestinal MucosaChemistryRegeneration (biology)digestive oral and skin physiologyHistologyGeneral Medicinemedicine.diseaseEpitheliumRatsSmall intestinal mucosamedicine.anatomical_structureAutoradiographyLigationVirchows Archiv B Cell Pathology Including Molecular Pathology
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