Search results for " Smooth"

showing 10 items of 455 documents

Isolated human chorionic vascular reactivity: Technical considerations for fresh preparations

1998

1. 1. Sixty chorionic vascular rings from normal term placentas were immersed in an organ bath for isometric tension recording to study (A) the contractile response to 120 mM of potassium chloride (KCI) after adjustment and equilibration to 1–5 g of passive tension; and (B) the concentration-response curve to KCI after adjustment and equilibration to the optimal passive tension. 2. 2. Adjustment to 4 g of passive tension elicited the maximal (P<0.007) and the latest (P<0.006) KCl-induced contraction among arterial rings. Venous rings showed the greatest contraction when adjusted to 3 g, but the differences were not significant except when compared to 1 g of passive tension (P<0.03). 3. 3. T…

medicine.medical_specialtyContraction (grammar)PlacentaPotassiumchemistry.chemical_elementIsometric exerciseIn Vitro TechniquesMuscle Smooth VascularPotassium ChlorideVeinsPregnancyIsometric ContractionPlacentaInternal medicinemedicineHumansPharmacologyArteriesChorionKineticsmedicine.anatomical_structureEndocrinologychemistryCirculatory systemFemalemedicine.symptomVasoconstrictionBlood vesselArteryGeneral Pharmacology: The Vascular System
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Effects of nicotine receptor agonists on acetylcholine release from the isolated motor nerve, small intestine and trachea of rats and guinea-pigs

1992

The effects of nicotine receptor agonists on the release of [3H]acetylcholine from the phrenic nerve, the small intestine and the trachea were investigated to characterize neuronal nicotine receptors within the peripheral nervous system. Contraction of the indirectly-stimulated hemidiaphragm was recorded to investigate desensitization of the postsynaptic muscular nicotine receptors. Nicotine, cytisine, 1,1-dimethyl-4-phenylpiperazinium and 2-(4-aminophenyl)-ethyl-trimethyl-ammoniumiodide caused a concentration-dependent (0.1-30 microM) increase in evoked [3H]acetylcholine release from the phrenic nerve, whereby bell-shaped concentration-response curves were obtained. The rank order of decre…

medicine.medical_specialtyDiaphragmGuinea PigsMyenteric PlexusMotor nerveReceptors NicotinicMotor EndplateNicotineCytisinechemistry.chemical_compoundPostsynaptic potentialInternal medicineIntestine SmallDrug DiscoverymedicineAnimalsGenetics (clinical)Phrenic nerveDose-Response Relationship DrugMuscle SmoothGeneral MedicineAcetylcholineStimulation ChemicalRatsPhrenic NerveTracheaEndocrinologymedicine.anatomical_structureParasympathomimeticschemistryPeripheral nervous systemMolecular Medicinemedicine.symptomSecretory RateAcetylcholineMuscle Contractionmedicine.drugMuscle contractionThe Clinical Investigator
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Antihistaminic and anticholinergic activities of mequitazine in comparison with clemizole

1988

Abstract The antihistamine and anticholinergic properties of mequitazine have been investigated and compared with those of clemizole. Both mequitazine and clemizole antagonized the effect of histamine in guinea-pig ileum competitively, the pA2 values calculated by Schild plot were 9.95 ± 0.44 for mequitazine and 10.54 ± 0.44 for clemizole. Mequitazine at 10−7 M produced a parallel shift of the dose-response curve to acetylcholine in the rat duodenum, clemizole and the lower doses of mequitazine failed to modify the effect of acetylcholine. The potency of mequitazine and clemizole as H1-histamine blockers is similar, but only mequitazine at highest concentration used showed anticholinergic a…

medicine.medical_specialtyDuodenummedicine.drug_classmedicine.medical_treatmentGuinea PigsPharmaceutical ScienceIn Vitro TechniquesBiologyParasympatholyticchemistry.chemical_compoundPhenothiazinesInternal medicinemedicineAnticholinergicAnimalsPotencyMequitazinePharmacologyParasympatholyticsMuscle SmoothRats Inbred StrainsAcetylcholineRatsClemizoleSchild regressionEndocrinologychemistryHistamine H1 AntagonistsBenzimidazolesAntihistamineHistamineHistamineMuscle Contractionmedicine.drugJournal of Pharmacy and Pharmacology
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Effects of endothelin-1 on the cerebrovascular bed of the goat.

1991

Abstract The aim of the present study was to analyze the effects of endothelin-1 (ET-1) in the cerebrovascular bed of the goat, the importance of endothelial integrity and the contribution of extracellular Ca2+ to these responses. We measured isometric tension and 45Ca2+ uptake in isolated middle cerebral arteries, and cerebral blood flow (CBF) in unanesthetized animals. ET-1 elicited concentration-dependent contractions which were potentiated in arteries without endothelium. Ca2+-free medium and nicardipine inhibited, and Bay K 8644 potentiated the ET-1-induced contractions. ET-1 enhanced 45Ca2+ uptake in isolated arteries. Injections of ET-1 directly into the cerebral circulation decrease…

medicine.medical_specialtyEndotheliumCerebral arteriesNicardipineIn Vitro TechniquesMuscle Smooth VascularCerebral circulationNicardipineInternal medicinemedicine.arteryIsometric ContractionmedicineAnimalsPharmacologyChemistryCalcium RadioisotopesEndothelinsGoatsAnatomy3-Pyridinecarboxylic acid 14-dihydro-26-dimethyl-5-nitro-4-(2-(trifluoromethyl)phenyl)- Methyl esterEndothelin 1Acetylcholinemedicine.anatomical_structureEndocrinologyCerebral blood flowCerebrovascular CirculationMiddle cerebral arteryPotassiumCalciumFemaleBlood vesselmedicine.drugEuropean journal of pharmacology
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Isoflavones and cardiovascular disease

2010

The specific profile of estrogens on cardiovascular risk, with limiting action on atherogenesis but a less clear protection on cardiovascular episodes, might be improved by other agonists of the estrogen receptor, such as isoflavones. By using a systematic search based on the electronic Medline database plus a hand-search of reference lists of selected review papers, we reviewed the rapidly growing body of experimental and clinical data that, on average, follow a pattern of benefit rather similar to estrogens. Experimental models have used endothelial and vascular smooth muscle cells, isolated arteries, and live animals, including monkeys. The clinical evidence arises from studies on the li…

medicine.medical_specialtyEndotheliumPhysiologyProstacyclinMuscle Smooth VascularGeneral Biochemistry Genetics and Molecular Biologychemistry.chemical_compoundInsulin resistanceLipid oxidationInternal medicineAnimalsHumansMedicineEvidence-Based Medicinemedicine.diagnostic_testbusiness.industrySoy FoodsObstetrics and GynecologyIsoflavonesLipid Metabolismmedicine.diseaseIsoflavonesPostmenopauseMenopauseEndocrinologymedicine.anatomical_structurechemistryCardiovascular DiseasesHemostasisSoybean ProteinsWomen's HealthFemaleEndothelium VascularbusinessLipid profilemedicine.drugMaturitas
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Functional relevance of presynaptic muscarinic autoreceptors.

1993

Pre- and postsynaptic muscarinic receptors have been characterized in the isolated trachea and ileal circular muscle of the guinea pig. The muscarinic autoreceptors mediating inhibition of acetylcholine release in the circular muscle belong to the M1 subtype, whereas those inhibiting acetylcholine release in the trachea are M2 or M4 receptors. In both tissues the postsynaptic muscarinic receptors are M3 receptors. Blockade of the autoreceptors by selective M1 and M2/M4 receptor antagonists leads to facilitation of cholinergic neurotransmission.

medicine.medical_specialtyGeneral NeuroscienceGuinea PigsPresynaptic TerminalsMuscarinic acetylcholine receptor M3Muscarinic acetylcholine receptor M2Muscle SmoothMuscarinic acetylcholine receptor M1PharmacologyBiologyIn Vitro TechniquesReceptors MuscarinicTracheaEndocrinologyPostsynaptic potentialIleumPhysiology (medical)Internal medicineMuscarinic acetylcholine receptorMuscarinic acetylcholine receptor M5medicineMuscarinic acetylcholine receptor M4AnimalsAcetylcholinemedicine.drugJournal of physiology, Paris
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gamma-Aminobutyric acid and cholinergic transmission in the guinea-pig ileum.

1983

1. The effects of GABA on release of acetylcholine and on contractility of the smooth muscle were studied in the myenteric plexus-longitudinal muscle preparation of the guinea pig. Acetylcholine was determined as radiolabelled transmitter from strips preloaded with 3H-choline. 2. GABA (1–300 μM) caused an increase in resting tension of smooth muscle as well as an increase in release of acetylcholine that was considerably reduced by tetrodotoxin. The facilitation by GABA of acetylcholine release exhibited a marked tachyphylaxis. The increase in muscle tension was clearly related to the increase in acetylcholine release. Muscimol (0.1–10 μM) also enhanced the release of acetylcholine. The eff…

medicine.medical_specialtyGuinea PigsIn Vitro TechniquesBicucullineTritiumSynaptic Transmissiongamma-Aminobutyric acidchemistry.chemical_compoundIleumInternal medicineMuscle tensionMuscarinic acetylcholine receptormedicineAnimalsPicrotoxingamma-Aminobutyric AcidPharmacologyMuscimolMuscle SmoothGeneral MedicineBicucullineAcetylcholineElectric StimulationEndocrinologynervous systemchemistryMuscimolCholinergicAcetylcholinemedicine.drugPicrotoxinMuscle ContractionNaunyn-Schmiedeberg's archives of pharmacology
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Characterization of prejunctional muscarinic autoreceptors in the guinea-pig trachea

1991

1. The effects of ten muscarinic antagonists on electrically evoked [3H]-acetylcholine release and muscle contraction were compared in an epithelium-free preparation of the guinea-pig trachea that had been preincubated with [3H]-choline. 2. The M3-selective antagonists UH-AH 37, 4-diphenyl-acetoxy-N-piperidine methobromide and para-fluorohexahydrosiladiphenidol were more potent in reducing the contractile response than in facilitating the evoked [3H]-acetylcholine release. Hexahydrosiladiphenidol did not discriminate between pre- and postjunctional effects. The rank order of the postjunctional potencies of the ten antagonists as well as the postjunctional pA2 values obtained for hexahydrosi…

medicine.medical_specialtyGuinea PigsIn Vitro TechniquesBiologyCholinechemistry.chemical_compoundInternal medicineMuscarinic acetylcholine receptormedicineMethoctramineAnimalsReceptorPharmacologyParasympatholyticsMuscarinic acetylcholine receptor M3Muscle SmoothMuscarinic acetylcholine receptor M2Receptors MuscarinicAcetylcholineElectric StimulationTracheaEndocrinologychemistryAutoreceptormedicine.symptomAcetylcholineMuscle ContractionResearch Articlemedicine.drugMuscle contractionBritish Journal of Pharmacology
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Inhibitory and excitatory muscarinic receptors modulating the release of acetylcholine from the postganglionic parasympathetic neuron of the chicken …

1992

The effects of muscarinic receptor antagonists on ACh release were studied in the absence or presence of cholinesterase (ChE) inhibition using the isolated perfused chicken heart. Presynaptic inhibitory muscarinic autoreceptor were characterized by determining the potency of various antagonists to enhance [3H]-ACh release evoked by field stimulation (3 Hz, 1 min). The order of potencies was: (±)-telenzepine > atropine > 4-DAMP > silahexocyclium > pirenzepine > hexahydro-siladifenidol > AF-DX 116. The comparison with known pA2 values for M1-, M2- and M3-receptors revealed that the presynaptic autoreceptor meets the criteria of an M1-receptor. Basal, not electrically evoked overflow of unlabe…

medicine.medical_specialtyGuinea PigsMuscarinic AntagonistsInhibitory postsynaptic potentialchemistry.chemical_compoundHeart RateInternal medicineMuscarinic acetylcholine receptormedicineMuscarinic acetylcholine receptor M4AnimalsPharmacologyChemistryMyocardiumHeartMuscle SmoothGeneral MedicinePirenzepineMyocardial ContractionAcetylcholineElectric StimulationAtropineEndocrinologyTelenzepineAutoreceptorCholinesterase InhibitorsChickensAcetylcholinemedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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Increase by NO synthase inhibitors of acetylcholine release from guinea-pig myenteric plexus

1994

The effects of nitric oxide (NO) synthase inhibitors on the electrically evoked release of [3H]acetylcholine were studied in guinea-pig myenteric plexus preparations preincubated with [3H]choline. NG-monomethyl-L-arginine (EC50 5.3 mumol l-1) and NG-nitro-L-arginine (EC50 1.3 mumol l-1) concentration-dependently increased the evoked release of [3H]acetylcholine without affecting the basal outflow. The facilitatory effect of NG-mono-methyl-L-arginine was prevented by L-arginine but not by D-arginine. The results suggest that endogenous NO inhibits the depolarisation-evoked release of acetylcholine.

medicine.medical_specialtyGuinea PigsMyenteric PlexusArginineNitric OxideNitroarginineNitric oxideGuinea pigchemistry.chemical_compoundInternal medicinemedicineAnimalsCholineEvoked PotentialsMyenteric plexusPharmacologyomega-N-MethylargininebiologyMuscle SmoothGeneral MedicineAcetylcholineElectric StimulationNitric oxide synthaseEndocrinologychemistryEnzyme inhibitorbiology.proteinLiberationAmino Acid OxidoreductasesNitric Oxide SynthaseAcetylcholinemedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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