Search results for " VAS"

showing 10 items of 1524 documents

Alkaline phosphatase dual-binding sites for collagen dictate cell migration and microvessel assembly in vitro

2020

Interactions between cell types, growth factors, and extracellular matrix components involved in angiogenesis are crucial for new vessel formation leading to tissue regeneration. This study investigated whether cocultures of fibroblasts and endothelial cells (ECs; from macro- or microvasculature) play a role in the formation of microvessel-like structures by ECs, as well as modulate fibroblast differentiation and growth factors production (vascular endothelial cell growth factor, basic fibroblast growth factor, active transforming growth factor-beta 1, and interleukin-8), which are important for vessel sprouting and maturation. Data obtained revealed that in vitro coculture systems of fibro…

0301 basic medicineCell typeAngiogenesisProtein ConformationBasic fibroblast growth factorNeovascularization PhysiologicIn Vitro TechniquesBiochemistryExtracellular matrix03 medical and health scienceschemistry.chemical_compound0302 clinical medicineCell MovementmedicineHumansFibroblastMolecular BiologyMicrovesselCells CulturedCell ProliferationBinding SitesChemistryHealth sciences Medical and Health sciencesCiências médicas e da saúdeCell migrationCell DifferentiationCell BiologyFibroblastsAlkaline PhosphataseCell biology030104 developmental biologymedicine.anatomical_structure030220 oncology & carcinogenesisMicrovesselsMedical and Health sciencesAlkaline phosphataseCollagenEndothelium VascularCiências da Saúde Ciências médicas e da saúde
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Novel Immune Features of the Systemic Inflammation Associated with Primary Hypercholesterolemia: Changes in Cytokine/Chemokine Profile, Increased Pla…

2018

Primary hypercholesterolemia (PH) is associated with a low grade systemic inflammation that is likely the main driver of premature atherosclerosis. Accordingly, we characterized the immune cell behaviour in PH and its potential consequences. Whole blood from 22 PH patients and 21 age-matched controls was analysed by flow cytometry to determine the percentage of leukocyte immunophenotypes, activation, and platelet-leukocyte aggregates. Plasma markers were determined by Enzyme-Linked ImmunoSorbent Assay (ELISA). The adhesion of platelet-leukocyte aggregates to tumor necrosis factor-&alpha

0301 basic medicineChemokinemedicine.medical_treatmentlcsh:Medicinechemokines030204 cardiovascular system & hematologySystemic inflammationArticleendothelial dysfunction03 medical and health sciences0302 clinical medicineleukocyte activationResposta immunitàriaprimary hypercholesterolemiaplatelet activationMedicinePlateletPlatelet activationEndothelial dysfunctionSistema cardiovascularsystemic inflammationbiologybusiness.industryMonocytelcsh:REndoteli vascularGeneral Medicinemedicine.diseasecytokines030104 developmental biologyCytokinemedicine.anatomical_structureImmunologybiology.proteinTumor necrosis factor alphamedicine.symptombusinessJournal of clinical medicine
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A rare association of chronic lymphocytic leukemia with c-ANCA-positive Wegener’s granulomatosis: a case report

2016

Background Wegener’s granulomatosis is a systemic vasculitis of the small- and medium-sized vessels, produced by the action of ANCA, which involves the respiratory tract, kidneys, and eyes, with a potential for lethal evolution in the first year after diagnosis. Its association with chronic lymphocytic leukemia is rarely described in the literature, and it may be difficult to diagnose and to treat this association. Case presentation We present the case of a 73-year-old Caucasian patient, a rare case in which Wegener’s granulomatosis is associated in a patient with chronic lymphocytic leukemia, who is admitted in the Infectious Disease Department for fever, diplopia, headache, purulent and h…

0301 basic medicineChemosisMalePathologymedicine.medical_specialtyChronic lymphocytic leukemiaMucous membrane of noseCase ReportAntibodies Antineutrophil Cytoplasmic03 medical and health sciences0302 clinical medicineBiopsymedicinec-ANCA positiveHumansAgedmedicine.diagnostic_testbusiness.industryGranulomatosis with PolyangiitisWegener’s granulomatosismedicine.diseasePrognosisLeukemia Lymphocytic Chronic B-Cell030104 developmental biologyOncology030220 oncology & carcinogenesisChronic lymphocytic leukemiaSurgerymedicine.symptomGranulomatosis with polyangiitisVasculitisbusinessScleritisSystemic vasculitisWorld Journal of Surgical Oncology
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Effects of gaseous and solid constituents of air pollution on endothelial function

2018

Abstract Ambient air pollution is a leading cause of non-communicable disease globally. The largest proportion of deaths and morbidity due to air pollution is now known to be due to cardiovascular disorders. Several particulate and gaseous air pollutants can trigger acute events (e.g. myocardial infarction, stroke, heart failure). While the mechanisms by which air pollutants cause cardiovascular events is undergoing continual refinement, the preponderant evidence support rapid effects of a diversity of pollutants including all particulate pollutants (e.g. course, fine, ultrafine particles) and gaseous pollutants such as ozone, on vascular function. Indeed alterations in endothelial function…

0301 basic medicineClinical ReviewFine particulateAir pollutionAir pollution030204 cardiovascular system & hematologymedicine.disease_cause03 medical and health sciencesGreenhouse Gases0302 clinical medicineOzoneRisk FactorsEnvironmental healthMedicineAnimalsHumansEndothelial dysfunctionNitrogen dioxidePollutantInflammationAir PollutantsHuman studiesAmbient air pollutionbusiness.industryEnvironmental ExposureParticulates3. Good healthEditor's Choice030104 developmental biology13. Climate actionCardiovascular DiseasesOxidative stressEndothelium VascularCurrent (fluid)Cardiology and Cardiovascular MedicinebusinessVascular functionParticulate matterTranslational MedicineEuropean Heart Journal
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Visualizing the spatiotemporal map of Rac activation in bovine aortic endothelial cells under laminar and disturbed flows.

2017

Disturbed flow can eliminate the alignment of endothelial cells in the direction of laminar flow, and significantly impacts on atherosclerosis in collateral arteries near the bifurcation and high curvature regions. While shear stress induced Rac polarity has been shown to play crucial roles in cell polarity and migration, little is known about the spatiotemporal map of Rac under disturbed flow, and the mechanism of flow-induced cell polarity still needs to be elucidated. In this paper, disturbed flow or laminar flow with 15 dyn/cm2 of average shear stress was applied on bovine aortic endothelial cells (BAECs) for 30 minutes. A genetically-encoded PAK-PBD-GFP reporter was transfected into BA…

0301 basic medicineFluorescence-lifetime imaging microscopyCell Membraneslcsh:MedicineMicrotubulesCell membraneLaminar Flow0302 clinical medicineCell polarityFluorescence microscopeMembrane fluidityCytoskeletonlcsh:ScienceShear StressesCytoskeletonAortaMultidisciplinaryChemistryPhysicsClassical MechanicsCell Polarityrac GTP-Binding Proteinsmedicine.anatomical_structurePhysical SciencesMechanical StressCellular Structures and OrganellesResearch ArticleCell PhysiologyImaging TechniquesMembrane FluidityFluid MechanicsResearch and Analysis MethodsContinuum Mechanics03 medical and health sciencesFluorescence ImagingShear stressmedicineAnimalsFluid Flowlcsh:RBiology and Life SciencesFluid DynamicsLaminar flowCell Biology030104 developmental biologyBiophysicsCattlelcsh:QEndothelium Vascular030217 neurology & neurosurgeryPLoS ONE
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IL-17A mediated endothelial breach promotes metastasis formation

2015

Abstract The role of the IL23/IL17A axis in tumor–immune interactions is a matter of controversy. Although some suggest that IL17A-producing T cells (TH17) can suppress tumor growth, others report that IL17A and IL23 accelerate tumor growth. Here, we systematically assessed the impact of IL17A-secreting lymphocytes in several murine models of tumor lung metastasis. Genetic fate mapping revealed that IL17A was secreted within lung metastases predominantly by γδ T cells, whereas TH17 cells were virtually absent. Using different tumor models, we found Il17a−/− mice to consistently develop fewer pulmonary tumor colonies. IL17A specifically increased blood vessel permeability and the expression …

0301 basic medicineGenetically modified mouseCancer ResearchPathologymedicine.medical_specialtyLung NeoplasmsEndotheliumImmunologyMelanoma ExperimentalVascular permeability610 Medicine & healthBiology10263 Institute of Experimental ImmunologyCapillary Permeability03 medical and health sciencesCarcinoma Lewis LungCell Line TumormedicineCell AdhesionAnimals1306 Cancer ResearchCell adhesionMice Knockout2403 ImmunologyLungMelanomaInterleukin-17Transendothelial and Transepithelial MigrationEndothelial Cellsmedicine.diseaseMice Inbred C57BL030104 developmental biologymedicine.anatomical_structureCell culture570 Life sciences; biologyInterleukin 17Endothelium VascularNeoplasm Transplantation
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Wnt-Dependent Oligodendroglial-Endothelial Interactions Regulate White Matter Vascularization and Attenuate Injury

2020

Recent studies have indicated oligodendroglial-vascular crosstalk during brain development, but the underlying mechanisms are incompletely understood. We report that oligodendrocyte precursor cells (OPCs) contact sprouting endothelial tip cells in mouse, ferret and human neonatal white matter. Using transgenic mice, we show that increased or decreased OPC density results in cognate changes in white matter vascular investment. Hypoxia promoted both increased OPC numbers and higher white matter vessel density, and endothelial cell expression of the Wnt pathway targets Apcdd1 and Axin2, suggesting paracrine OPC-endothelial signaling. Conditional knockout of OPC Wntless resulted in diminished w…

0301 basic medicineGenetically modified mouseoligodendrocytesMice TransgenicBiologyArticleWhite matter03 medical and health sciencesParacrine signallingMice0302 clinical medicinetip cell angiogenesisAxin ProteinConditional gene knockoutmedicineAXIN2AnimalsHumanshypoxic-ischemic encephalopathyHypoxiaWnt Signaling PathwayGeneral NeuroscienceWnt signaling pathwayFerretsIntracellular Signaling Peptides and ProteinsEndothelial CellsMembrane ProteinsCell DifferentiationHypoxia (medical)Wnt signalingWhite Matter3. Good healthCell biologyEndothelial stem cellstomatognathic diseasesOligodendroglia030104 developmental biologymedicine.anatomical_structurenervous systemEndothelium Vascularmedicine.symptom030217 neurology & neurosurgeryNeuron
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Hieracium hypochoeroides subsp. cilentanum (Asteraceae), a new taxon from S Italy

2016

A new hawkweed taxon from southern Italy, Hieracium hypochoeroides subsp. cilentanum, is described and illustrated. It is endemic to Monte Cervati (Parco Nazionale del Cilento e Vallo di Diano, S Campania). Information on its ecology and taxonomic relationships is provided.

0301 basic medicineHieraciumEcologySettore BIO/02 - Botanica SistematicaPlant ScienceBiologyAsteraceaebiology.organism_classification03 medical and health sciences030104 developmental biologyTaxonBotanyTaxonomy (biology)EndemismEudicotsCampania endemism morphology relict taxon taxonomy vascular floraEcology Evolution Behavior and SystematicsPhytotaxa
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Targeting Angiogenesis in Biliary Tract Cancers: An Open Option

2017

Abstract: Biliary tract cancers (BTCs) are characterized by a bad prognosis and the armamentarium of drugs for their treatment is very poor. Although the inflammatory status of biliary tract represents the first step in the cancerogenesis, the microenvironment also plays a key role in the pathogenesis of BTCs, promoting tumor angiogenesis, invasion and metastasis. Several molecules, such as vascular endothelial growth factor (VEGF) and fibroblast growth factor (FGF), are involved in the angiogenesis process and their expression on tumor samples has been explored as prognostic marker in both cholangiocarcinoma and gallbladder cancer. Recent studies evaluated the genomic landscape of BTCs and…

0301 basic medicineMAPK/ERK pathwayVascular Endothelial Growth Factor AAngiogenesisDrug Evaluation PreclinicalTyrosine kinase inhibitorAngiogenesis InhibitorsReviewFibroblast growth factorCatalysiMetastasisAntineoplastic Agentlcsh:Chemistrychemistry.chemical_compoundangiogenesis0302 clinical medicinetyrosine kinase inhibitorsMolecular Targeted Therapylcsh:QH301-705.5SpectroscopyClinical Trials as TopicMonoclonal antibodieNeovascularization Pathologicvascular endothelial growth factorComputer Science Applications1707 Computer Vision and Pattern RecognitionGeneral MedicineComputer Science ApplicationsVascular endothelial growth factorGene Expression Regulation NeoplasticAngiogenesiChemistryBiliary Tract NeoplasmsTreatment OutcomeBiliary Tract Neoplasm030220 oncology & carcinogenesismonoclonal antibodiesTyrosine kinaseAngiogenesis InhibitorHumanSignal TransductionProtein Kinase InhibitorAntineoplastic Agentsbiliary tract cancersBiologyModels BiologicalAngiogenesis; Biliary tract cancers; Monoclonal antibodies; Tyrosine kinase inhibitors; Vascular endothelial growth factor; Angiogenesis Inhibitors; Animals; Antineoplastic Agents; Biliary Tract Neoplasms; Clinical Trials as Topic; Drug Evaluation Preclinical; Gene Expression Regulation Neoplastic; Genetic Variation; Humans; Models Biological; Neovascularization Pathologic; Protein Kinase Inhibitors; Signal Transduction; Treatment Outcome; Vascular Endothelial Growth Factor A; Molecular Targeted Therapy; Catalysis; Molecular Biology; Spectroscopy; Computer Science Applications1707 Computer Vision and Pattern Recognition; Physical and Theoretical Chemistry; Organic Chemistry; Inorganic ChemistryCatalysisInorganic Chemistry03 medical and health sciencesIn vivomedicineAnimalsHumansPhysical and Theoretical ChemistryGallbladder cancerMolecular BiologyProtein Kinase InhibitorsBiologyAnimalOrganic ChemistryGenetic Variationmedicine.disease030104 developmental biologychemistrylcsh:Biology (General)lcsh:QD1-999Immunologyangiogenesis; biliary tract cancers; monoclonal antibodies; tyrosine kinase inhibitors; vascular endothelial growth factorCancer researchBiliary tract cancerInternational Journal of Molecular Sciences
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Endothelial Dab1 signaling orchestrates neuro-glia-vessel communication in the central nervous system.

2018

Developing the bloodbrain barrier During development, signals need to be dynamically integrated by endothelial cells, neurons, and glia to achieve functional neuro-glia-vascular units in the central nervous system. During cortical development, neuronal Dab1 and ApoER2 receptors respond to a guidance cue called reelin. Studying mice, Segarra et al. found that Dab1 and ApoER2 are also expressed in endothelial cells (see the Perspective by Thomas). The integration of reelin signaling in endothelial cells and neurons facilitates the communication between vessels, glia, and neurons that is necessary for the correct positioning of neurons during cortical development. This integration is also impo…

0301 basic medicineMaleCell signalingLow-density lipoprotein receptor-related protein 8EndotheliumCell Adhesion Molecules NeuronalCentral nervous systemNeovascularization PhysiologicNerve Tissue ProteinsCell Communication03 medical and health sciencesMiceCell MovementmedicineAnimalsReelinLDL-Receptor Related ProteinsCerebral CortexMice KnockoutNeuronsRetinaExtracellular Matrix ProteinsMultidisciplinarybiologyIntegrin beta1Serine EndopeptidasesRetinal VesselsDAB1Reelin Protein030104 developmental biologymedicine.anatomical_structurenervous systemCerebral cortexBlood-Brain Barrierbiology.proteinFemaleEndothelium VascularLamininNeuroscienceNeurogliaGene DeletionSignal TransductionScience (New York, N.Y.)
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