Search results for " diabetes"

showing 10 items of 2906 documents

Insulin resistance and diabetes in hyperthyroidism: a possible role for oxygen and nitrogen reactive species.

2019

In addition to insulin, glycemic control involves thyroid hormones. However, an excess of thyroid hormone can disturb the blood glucose equilibrium, leading to alterations of carbohydrate metabolism and, eventually, diabetes. Indeed, experimental and clinical hyperthyroidism is often accompanied by abnormal glucose tolerance. A common characteristic of hyperthyroidism and type 2 diabetes is the altered mitochondrial efficiency caused by the enhanced production of reactive oxygen and nitrogen species. It is known that an excess of thyroid hormone leads to increased oxidant production and mitochondrial oxidative damage. It can be hypothesised that these species represent the link between hype…

0301 basic medicinemedicine.medical_specialtyendocrine systemendocrine system diseasesmedicine.medical_treatmentDiabetes hyperthyroidism insulin resistance insulin secretion reactive nitrogen species (RNS) reactive oxygen species (ROS)Type 2 diabetesCarbohydrate metabolismBiochemistryHyperthyroidism03 medical and health sciencesInsulin resistanceDiabetes mellitusInternal medicinemedicineHumansGlycemic030102 biochemistry & molecular biologyChemistryInsulinThyroidGeneral Medicinemedicine.diseaseReactive Nitrogen SpeciesOxygen030104 developmental biologymedicine.anatomical_structureEndocrinologyDiabetes Mellitus Type 2Insulin ResistanceReactive Oxygen SpeciesHormoneFree radical research
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The neuropeptide 26RFa in the human gut and pancreas: potential involvement in glucose homeostasis

2019

Objective Recent studies performed in mice revealed that the neuropeptide 26RFa regulates glucose homeostasis by acting as an incretin and by increasing insulin sensitivity. However, in humans, an association between 26RFa and the regulation of glucose homeostasis is poorly documented. In this study, we have thus investigated in detail the distribution of 26RFa and its receptor, GPR103, in the gut and the pancreas, and determined the response of this peptidergic system to an oral glucose challenge in obese patients. Design and methods Distribution of 26RFa and GPR103 was examined by immunohistochemistry using gut and pancreas tissue sections. Circulating 26RFa was determined using a specif…

0301 basic medicinemedicine.medical_specialtyinsulinobesityEndocrinology Diabetes and Metabolismmedicine.medical_treatment[SDV]Life Sciences [q-bio]Incretinpancréas030209 endocrinology & metabolismlcsh:Diseases of the endocrine glands. Clinical endocrinology03 medical and health sciences0302 clinical medicineEndocrinologyInsulin resistanceintestinGastric glandsInternal medicineInternal MedicineMedicineGlucose homeostasisglucose homeostasisFood and Nutritiongut;pancreas;glucose homeostasis;insulin;incretin;obesitypancreasglucoseComputingMilieux_MISCELLANEOUSinsulinehoméostasielcsh:RC648-665business.industryResearchStomachPancreatic isletsInsulindigestive oral and skin physiologyNeurosciencesmedicine.diseaseincretin[SDV] Life Sciences [q-bio]obésité030104 developmental biologymedicine.anatomical_structureEndocrinologyNeurons and CognitionAlimentation et NutritiongutbusinessPancreas
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Cutaneous manifestations of polycystic ovary syndrome

2020

Abstract Hirsutism, acne, and/or female pattern hair loss are common cutaneous manifestations of polycystic ovary syndrome (PCOS). However, while hirsutism is mainly related to androgen excess, both acne and female pattern hair loss may often depend on cutaneous alterations that are not linked to hyperandrogenism or increased androgen sensitivity. Because both acne and female pattern hair loss cannot be considered a sign of hyperandrogenism, neither should be included in the diagnostic process of PCOS without demonstration of increased levels of circulating androgens. In addition, in patients with androgen-dependent acne, the concomitant proliferation of Propionibacterium acnes in skin may …

0301 basic medicinemedicine.medical_specialtyintegumentary systembiologybusiness.industrymedicine.drug_classEndocrinology Diabetes and MetabolismHyperandrogenism030209 endocrinology & metabolismmedicine.diseaseAndrogen ExcessAntiandrogenbiology.organism_classificationPolycystic ovaryDermatology03 medical and health sciencesPropionibacterium acnes030104 developmental biology0302 clinical medicineHair lossmedicinebusinessAcnehirsutismCurrent Opinion in Endocrine and Metabolic Research
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Does high sugar consumption exacerbate cardiometabolic risk factors and increase the risk of type 2 diabetes and cardiovascular disease?

2012

Consumption of sugar has been relatively high in the Nordic countries; the impact of sugar intake onmetabolic risk factors and related diseases has been debated. The objectives were to assess the effect of sugarintake (sugar-sweetened beverages, sucrose and fructose) on association with type 2 diabetes, cardiovasculardisease and related metabolic risk factors (impaired glucose tolerance, insulin sensitivity, dyslipidemia, bloodpressure, uric acid, inflammation markers), and on all-cause mortality, through a systematic review ofprospective cohort studies and randomised controlled intervention studies published between January 2000and search dates. The methods adopted were as follows: the fir…

0301 basic medicinemedicine.medical_specialtylcsh:TX341-641030209 endocrinology & metabolismType 2 diabetesDiseaseReview ArticleVDP::Medical disciplines: 700::Health sciences: 800::Nutrition: 811fructoseImpaired glucose tolerance03 medical and health scienceschemistry.chemical_compound0302 clinical medicineNordic nutrition recommendationssystematic reviewInternal medicinemedicineProspective cohort study030109 nutrition & dieteticsNutrition and Dieteticsbusiness.industryPublic Health Environmental and Occupational Healthmedicine.diseaseBlood pressurechemistrysugarUric acidbusinesssugar-sweetened beverageslcsh:Nutrition. Foods and food supplyDyslipidemiaFood ScienceCohort studyFoodnutrition research
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Management of Ponatinib in Patients with Chronic Myeloid Leukemia with Cardiovascular Risk Factors

2019

Cardiovascular (CV) adverse events are considered common complications of ponatinib treatment. Recently, it has been demonstrated that ponatinib dose reductions in definite settings can obtain optimal responses and lower ponatinib-related CV events. In this study, we describe the management of 5 patients with chronic myeloid leukemia treated with ponatinib, from second to fourth line of tyrosine kinase inhibitor therapy, carrying high pre-ponatinib CV risk, who obtained optimal molecular response and developed no CV adverse event during follow-up. Among these 5 patients, 2 had diagnosis of ischemic heart disease and underwent percutaneous angioplasty, 2 had type 2 diabetes and arterial hype…

0301 basic medicinemedicine.medical_specialtymedicine.drug_class030106 microbiologyCardiovascular risk factorsType 2 diabetesDiseaseTyrosine-kinase inhibitorSettore MED/15 - Malattie Del Sangue03 medical and health scienceschemistry.chemical_compound0302 clinical medicineInternal medicineDrug DiscoverymedicinePharmacology (medical)In patientAdverse effectPharmacologybusiness.industryPonatinibChronic myeloid leukemiaMyeloid leukemiaGeneral Medicinemedicine.diseaseCardiovascular riskInfectious DiseasesOncologychemistry030220 oncology & carcinogenesisPonatinibbusiness
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Srebf2 Locus Overexpression Reduces Body Weight, Total Cholesterol and Glucose Levels in Mice Fed with Two Different Diets

2020

Macronutrients represent risk factors for hyperlipidemia or diabetes. Lipid alterations and type 2 diabetes mellitus are global health problems. Overexpression of sterol regulatory element-binding factor (Srebf2) in transgenic animals is linked to elevated cholesterol levels and diabetes development. We investigated the impact of increased Srebf2 locus expression and the effects of control and high-fat, high-sucrose (HFHS) diets on body weight, glucose and lipid metabolisms in transgenic mice (S-mice). Wild type (WT) and S-mice were fed with both diets for 16 weeks. Plasma glucose, insulin and lipids were assessed (n = 25). Immunostainings were performed in liver, pancreas and fat (N = 10).…

0301 basic medicinemedicine.medical_specialtymedicine.medical_treatment030209 endocrinology & metabolismlcsh:TX341-641Carbohydrate metabolismtransgenic miceArticle03 medical and health scienceschemistry.chemical_compound0302 clinical medicineInternal medicineAdipocyteDiabetes mellitusHyperlipidemialipid metabolismmedicinecarbohydrate metabolismhigh-sucrose diethigh-fatNutrition and DieteticsCholesterolInsulinType 2 Diabetes MellituscholesterolLipid metabolismmedicine.diseaselipoproteins030104 developmental biologyEndocrinologychemistrylipids (amino acids peptides and proteins)atherosclerosissterol regulatory element-binding protein 2 (SREBP-2)lcsh:Nutrition. Foods and food supplyFood ScienceNutrients
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Branched-Chain Amino Acid Levels Are Related with Surrogates of Disturbed Lipid Metabolism among Older Men.

2016

Aims/hypothesis: Existing studies suggest that decreased branched-chain amino acid (BCAA) catabolism and thus elevated levels in blood are associated with metabolic disturbances. Based on such information, we have developed a hypothesis how BCAA degradation mechanistically connects to tricarboxylic acid cycle, intramyocellular lipid storage, and oxidation, thus allowing more efficient mitochondrial energy production from lipids as well as providing better metabolic health. We analyzed whether data from aged Finnish men are in line with our mechanistic hypothesis linking BCAA catabolism and metabolic disturbances. Methods: Older Finnish men enriched with individuals having been athletes in y…

0301 basic medicinemedicine.medical_specialtymitokondriotBranched-chain amino acideducationType 2 diabetesBiologyBody fat percentage03 medical and health scienceschemistry.chemical_compoundLipid oxidationlipid oxidationValineInternal medicineDiabetes mellitusenergy metabolismmedicinebranched-chain amino acidsta315Original Researchlcsh:R5-920Catabolismta3141General Medicinemedicine.diseasemetabolic disease3142 Public health care science environmental and occupational healthmitochondria030104 developmental biologyEndocrinologychemistry3121 General medicine internal medicine and other clinical medicinetricarboxylic acid cycleMedicineBlood sugar regulationlcsh:Medicine (General)Frontiers in Medicine
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Does sex hormone-binding globulin cause insulin resistance during pubertal growth?

2019

Background The directional influences between serum sex hormone-binding globulin (SHBG), adiposity and insulin resistance during pubertal growth remain unclear. The aim of this study was to investigate bidirectional associations between SHBG and insulin resistance (HOMA-IR) and adiposity from childhood to early adulthood. Methods Participants were 396 healthy girls measured at baseline (age 11.2 years) and at 1, 2, 4 and 7.5 years. Serum concentrations of estradiol, testosterone and SHBG were determined by ELISA, glucose and insulin by enzymatic photometry, insulin-like growth factor 1 (IGF-1) by time-resolved fluoroimmunoassays, whole-body fat mass by dual-energy X-ray absorptiometry and …

0301 basic medicinemedicine.medical_specialtypubertyGlobulinEndocrinology Diabetes and Metabolismmedicine.medical_treatment030209 endocrinology & metabolismlcsh:Diseases of the endocrine glands. Clinical endocrinology03 medical and health sciences0302 clinical medicineEndocrinologyInsulin resistanceSex hormone-binding globulinInternal medicineinsulin resistanceInternal Medicinemedicinesex hormone-binding globulinkehonkoostumussukupuolihormonitadipositylcsh:RC648-665biologybusiness.industryResearchInsulinmenarcheConfoundinginsuliiniresistenssimurrosikämedicine.diseasetytöt030104 developmental biologyEndocrinologyglobuliinitHomeostatic model assessmentMenarchebiology.proteinbusinesshormones hormone substitutes and hormone antagonistsEarly pubertyEndocrine Connections
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Systemic Oxidative Stress and Visceral Adipose Tissue Mediators of NLRP3 Inflammasome and Autophagy Are Reduced in Obese Type 2 Diabetic Patients Tre…

2020

Obesity is a low-grade inflammatory condition affecting a range of individuals, from metabolically healthy obese (MHO) subjects to type 2 diabetes (T2D) patients. Metformin has been shown to display anti-inflammatory properties, though the underlying molecular mechanisms are unclear. To study whether the effects of metformin are mediated by changes in the inflammasome complex and autophagy in visceral adipose tissue (VAT) of obese patients, a biopsy of VAT was obtained from a total of 68 obese patients undergoing gastric bypass surgery. The patients were clustered into two groups: MHO patients and T2D patients treated with metformin. Patients treated with metformin showed decreased levels o…

0301 basic medicinemedicine.medical_specialtyvisceral adipose tissue (VAT)obesityautophagyendocrine system diseasesPhysiologyinflammatory cytokinesClinical BiochemistryATG5Adipose tissue030209 endocrinology & metabolismLeukocyte homeostasisType 2 diabetesBiochemistryArticleProinflammatory cytokine03 medical and health sciences0302 clinical medicineInternal medicinemedicineoxidative stressMolecular Biologytype 2 diabetes (T2D)business.industrylcsh:RM1-950nutritional and metabolic diseasesInflammasomeCell Biologymedicine.diseaseMetforminlcsh:Therapeutics. Pharmacology030104 developmental biologyEndocrinologybusinessmetforminInflammasome complexmedicine.drugAntioxidants (Basel, Switzerland)
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Lack of Hypothalamus Polysialylation Inducibility Correlates With Maladaptive Eating Behaviors and Predisposition to Obesity

2018

This original research article (6 p.) is part of the research topic . Specialty section: This article was submitted to Neuroenergetics, Nutrition and Brain Health, a section of the journal Frontiers in Nutrition.; International audience; High variability exists in individual susceptibility to develop overweight in an obesogenic environment and the biological underpinnings of this heterogeneity are poorly understood. In this brief report, we show in mice that the vulnerability to diet-induced obesity is associated with low level of polysialic acid-neural cell adhesion molecule (PSA-NCAM), a factor of neural plasticity, in the hypothalamus. As we previously shown that reduction of hypothalami…

0301 basic medicineobesityfood intakePSA-NCAMEndocrinology Diabetes and Metabolismmedia_common.quotation_subject[SDV]Life Sciences [q-bio][SDV.NEU.NB]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/Neurobiologybrainmaladaptive eating behaviorlcsh:TX341-641BiologyOverweightEnergy homeostasis03 medical and health sciencesNeuroplasticitymedicineFood and Nutritionhypothalamusmedia_commonNutritionOriginal ResearchNutrition and Dieteticssynaptic plasticitycomportement alimentairepolysialylation[SDV.NEU.NB] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/NeurobiologyNeurosciencesAppetitemedicine.diseaseprise alimentaireObesity3. Good health[SDV] Life Sciences [q-bio]food intake;obesity;maladaptive eating behavior;synaptic plasticity;PSA-NCAM;polysialylation;brain;hypothalamusEating disordersobésité030104 developmental biologynervous systemHypothalamusNeurons and CognitionSynaptic plasticityAlimentation et Nutritionplasticité synaptiquecerveaumedicine.symptomlcsh:Nutrition. Foods and food supplyNeuroscienceFood ScienceFrontiers in Nutrition
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