Search results for " glycoprotein"

showing 10 items of 430 documents

Stromal Interaction Molecule 1 (STIM1) Is Involved in the Regulation of Mitochondrial Shape and Bioenergetics and Plays a Role in Oxidative Stress

2012

Calcium ions are involved in a plethora of cellular functions including cell death and mitochondrial energy metabolism. Store-operated Ca(2+) entry over the plasma membrane is activated by depletion of intracellular Ca(2+) stores and is mediated by the sensor STIM1 and the channel ORAI1. We compared cell death susceptibility to oxidative stress in STIM1 knock-out and ORAI1 knockdown mouse embryonic fibroblasts and in knock-out cells with reconstituted wild type and dominant active STIM1. We show that STIM1 and ORAI1 deficiency renders cells more susceptible to oxidative stress, which can be rescued by STIM1 and ORAI1 overexpression. STIM1 knock-out mitochondria are tubular, have a higher Ca…

inorganic chemicalsProgrammed cell deathORAI1 ProteinEukaryotic Initiation Factor-2Active Transport Cell NucleusApoptosisMitochondrionBiologymedicine.disease_causeBiochemistryMiceeIF-2 KinasemedicineAnimalsStromal Interaction Molecule 1PhosphorylationMolecular BiologyTranscription factorCells CulturedMice KnockoutEIF-2 kinaseMembrane GlycoproteinsEndoplasmic reticulumMolecular Bases of DiseaseSTIM1Cell BiologyFibroblastsEmbryo MammalianMitochondriaCell biologyOxidative Stressbiology.proteinCalciumCalcium ChannelsEnergy MetabolismIntracellularOxidative stressJournal of Biological Chemistry
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Pathogenic Role of Complement in Antiphospholipid Syndrome and Therapeutic Implications

2018

Antiphospholipid syndrome (APS) is an acquired autoimmune disease characterized by thromboembolic events, pregnancy morbidity, and the presence of antiphospholipid (aPL) antibodies. There is sound evidence that aPL act as pathogenic autoantibodies being responsible for vascular clots and miscarriages. However, the exact mechanisms involved in the clinical manifestations of the syndrome are still a matter of investigation. In particular, while vascular thrombosis is apparently not associated with inflammation, the pathogenesis of miscarriages can be explained only in part by the aPL-mediated hypercoagulable state and additional non-thrombotic effects, including placental inflammation, have b…

lcsh:Immunologic diseases. Allergy0301 basic medicineImmunologyComplementMiscarriagesAnti-beta2 glycoprotein I antibodieInflammationMiscarriagePathogenesis03 medical and health sciences0302 clinical medicineImmune systemAntiphospholipid syndromeimmune system diseasesAntiphospholipid syndromeMedicineImmunology and AllergyAnimal model030203 arthritis & rheumatologyAutoimmune diseaseInflammationbusiness.industryAutoantibodyThrombosismedicine.diseaseComplement (complexity)Complement systemAnimal models030104 developmental biologyAnti-beta2 glycoprotein I antibodiesPerspectiveThrombosiImmunologyAnimal models; Anti-beta2 glycoprotein I antibodies; Antiphospholipid syndrome; Complement; Inflammation; Miscarriages; Therapy; Thrombosis; Immunology and Allergy; ImmunologyTherapymedicine.symptomlcsh:RC581-607businessFrontiers in Immunology
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Plasma membrane glycoproteins covalently bound to silica beads as a model for molecular studies of cell-cell interactions in culture.

1987

Abstract In previous studies, we have shown that plasma membrane glycoproteins are of major importance in the density-dependent regulation of growth of normal diploid fibroblasts. Due to the hydrophobic portions of these molecules, functional studies in cell culture are often diffucult to perform and to interpret. Specially, the addition of these molecules in soluble form to cell culture, after depletion of detergents needed for their solubilization, leads to aggregation and internalization. Therefore, we developed a method for the covalent immobilization of the solubilized plasma membrane proteins to derivatized silica beads for further investigations on the molecular nature of the active …

media_common.quotation_subjectCellBiophysicsBiochemistryModels BiologicalmedicineHumansCentrifugationInternalizationCells Culturedmedia_commonMembrane GlycoproteinsbiologyChemistryCell growthContact InhibitionFibroblastsSilicon DioxideMembrane glycoproteinsmedicine.anatomical_structureBiochemistryMembrane proteinCell cultureCovalent bondbiology.proteinCell DivisionProtein BindingJournal of biochemical and biophysical methods
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The C-terminal antibody binding domain ofCandida albicansmp58 represents a protective epitope during candidiasis

2003

The 58-kDa surface mannoprotein of Candida albicans (mp58) elicits strong antibody responses during infection. Epitope mapping with sera from patients with candidiasis and control individuals indicated the presence of multiple IgG-reactive continuous epitopes on the protein, expanding both the amino- and carboxy-terminal domains and several internal regions. These immunoreactive regions were similar to the ones previously identified using sera from immunized animals. Two of the epitopic regions (including the C-terminal domain) showed increased reactivity with antibodies present in sera from patients with candidiasis as compared to control individuals. Patients who survived the infection di…

medicine.drug_classEnzyme-Linked Immunosorbent AssayMonoclonal antibodyMicrobiologyEpitopeImmunoglobulin GFungal ProteinsEpitopesMiceCandida albicansGeneticsmedicineAnimalsAmino Acid SequenceCandida albicansMolecular BiologyMice Inbred BALB CMembrane GlycoproteinsbiologyCandidiasisAntibodies Monoclonalbiology.organism_classificationDisseminated CandidiasisVirologyCorpus albicansProtein Structure TertiaryEpitope mappingbiology.proteinFemaleAntibodyEpitope MappingFEMS Microbiology Letters
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Macrolides May Prevent Severe Acute Respiratory Syndrome Coronavirus 2 Entry into Cells: A Quantitative Structure Activity Relationship Study and Exp…

2021

The global pandemic caused by the emerging severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) is threatening the health and economic systems worldwide. Despite the enormous efforts of scientists and clinicians around the world, there is still no drug or vaccine available worldwide for the treatment and prevention of the infection. A rapid strategy for the identification of new treatments is based on repurposing existing clinically approved drugs that show antiviral activity against SARS-CoV-2 infection. In this study, after developing a quantitative structure activity relationship analysis based on molecular topology, several macrolide antibiotics are identified as promising SARS-…

medicine.drug_classGeneral Chemical EngineeringvirusesQuantitative Structure-Activity RelationshipDiseaseLibrary and Information Sciencesmedicine.disease_causeAzithromycin01 natural sciencesAntiviral AgentsVirusArticleMacrolide AntibioticsViral life cycleClarithromycin0103 physical sciencesPandemicmedicineHumansCoronavirus010304 chemical physicsbusiness.industrySARS-CoV-2COVID-19General ChemistryVirology3. Good health0104 chemical sciencesComputer Science ApplicationsAnti-Bacterial Agents010404 medicinal & biomolecular chemistryPharmaceutical PreparationsSpike Glycoprotein CoronavirusMacrolidesbusinessmedicine.drugJournal of Chemical Information and Modeling
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Fab fragments from a monoclonal antibody against a germ tube mannoprotein block the yeast-to-mycelium transition in Candida albicans.

1990

Fab fragments prepared from the immunoglobulin G monoclonal antibody (MAb) 4C12, which reacts with a determinant expressed on the hyphal extension of germ tubes of Candida albicans, inhibited germ tube formation, but intact MAb 4C12 did not. Indirect immunofluorescence showed a punctate binding pattern on cells incubated with Fab fragments but a confluent binding on cells incubated with intact MAb 4C12.

medicine.drug_classImmunologyGerm tubeFluorescent Antibody TechniqueMonoclonal antibodyMicrobiologyImmunoglobulin GMicrobiologyImmunoglobulin Fab FragmentsCell WallCandida albicansmedicineAscitic FluidHumansCandida albicansMyceliumMembrane GlycoproteinsbiologyImmunoglobulin Fab FragmentsAntibodies Monoclonalbiology.organism_classificationMolecular biologyYeastInfectious DiseasesMicroscopy FluorescenceImmunoglobulin Gbiology.proteinParasitologyAntibodyResearch ArticleInfection and immunity
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Antibodies to cell surface ganglioside GD3 perturb inductive epithelial-mesenchymal interactions

1988

Abstract Most epithelial sheets emerge during embryogenesis by a branching and growth of the epithelium. The surrounding mesenchyme is crucial for this process. We report that branching morphogenesis and the formation of a new epithelium from the mesenchyme in the embryonic kidney can be blocked by a monoclonal antibody reacting with a surface glycolipid, disialoganglioside G D3 . In contrast, a more than 10-fold excess of antibodies to adhesive glycoproteins (N-CAM, L -CAM, fibronectin) fails to inhibit morphogenesis. Although the anti-G D3 antibody affected epithelial development, the disialoganglioside G D3 was expressed not in the epithelium, but in the mesenchyme surrounding the develo…

medicine.drug_classMesenchymeMorphogenesisFluorescent Antibody TechniqueBiologyKidneyMonoclonal antibodyEpitheliumGeneral Biochemistry Genetics and Molecular BiologyMesodermMiceOrgan Culture TechniquesCell–cell interactionGangliosidesMorphogenesismedicineAnimalsGanglioside GD3Embryonic InductionMembrane GlycoproteinsAntibodies MonoclonalEmbryonic stem cellEpitheliumFibronectinsCell biologyFibronectinmedicine.anatomical_structureBiochemistryAntigens Surfacebiology.proteinUreterCell Adhesion MoleculesCell
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Decidual endothelial cells express surface-bound C1q as a molecular bridge between endovascular trophoblast and decidual endothelium.

2008

This study was prompted by the observation that decidual endothelial cells (DECs), unlike endothelial cells (ECs) of blood vessels in normal skin, kidney glomeruli and brain, express surface-bound C1q in physiologic pregnancy. This finding was unexpected, because deposits of C1q are usually observed in pathologic conditions and are associated with complement activation. In the case of DECs, we failed to detect immunoglobulins and C4 co-localized with C1q on the cell surface. Surprisingly, DECs expressed mRNA for the three chains of C1q and secreted detectable level of this component in serum-free medium. The ability to synthesize C1q is acquired by DECs during pregnancy and is not shared by…

medicine.medical_specialtyC1q; Trophoblast; Endothelial cells; GlycosaminoglycansEndotheliumBlood VesselEndothelial cellsCellImmunologychemical and pharmacologic phenomenaBiologyurologic and male genital diseasesArticleEndothelial cellimmune system diseasesPregnancyInternal medicineparasitic diseasesmedicineCell AdhesionDeciduaHumansReceptorCell adhesionskin and connective tissue diseasesMolecular BiologyC1qGlycosaminoglycansC1q; Endothelial cells; Glycosaminoglycans; Trophoblast; Blood Vessels; Cell Adhesion; Complement C1q; Decidua; Endothelial Cells; Female; Humans; Membrane Glycoproteins; Pregnancy; Receptors Complement; Trophoblasts; Molecular Biology; ImmunologyEndothelial CellMembrane GlycoproteinsComplement C1qDeciduaTrophoblastTrophoblastComplement systemCell biologyTrophoblastsReceptors Complementmedicine.anatomical_structureEndocrinologyGlycosaminoglycanBlood VesselsFemaleMembrane GlycoproteinIntracellularHuman
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Potential involvement of fas and its ligand in the pathogenesis of Hashimoto's thyroiditis

1997

The mechanisms responsible for thyrocyte destruction in Hashimoto's thyroiditis (HT) are poorly understood. Thyrocytes from HT glands, but not from nonautoimmune thyroids, expressed Fas. Interleukin-1β (IL-1β), abundantly produced in HT glands, induced Fas expression in normal thyrocytes, and cross-linking of Fas resulted in massive thyrocyte apoptosis. The ligand for Fas (FasL) was shown to be constitutively expressed both in normal and HT thyrocytes and was able to kill Fas-sensitive targets. Exposure to IL-1β induced thyrocyte apoptosis, which was prevented by antibodies that block Fas, suggesting that IL-1β-induced Fas expression serves as a limiting factor for thyrocyte destruction. Th…

medicine.medical_specialtyFas Ligand Proteinmedicine.medical_treatmentThyroid GlandApoptosisPolymerase Chain ReactionThyroiditisFas ligandPathogenesisImmunoenzyme TechniquesInternal medicinemedicineTumor Cells CulturedHumansRNA Messengerfas ReceptorCells CulturedNucleic Acid Synthesis InhibitorsProtein Synthesis InhibitorsMultidisciplinaryMembrane GlycoproteinsChemistryThyroidThyroiditis AutoimmuneInterleukinAntibodies Monoclonalmedicine.diseaseFas receptorRecombinant ProteinsCytokinemedicine.anatomical_structureEndocrinologyApoptosisCytokinesInterleukin-1
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Placenta-derived CD95 ligand causes liver damage in hemolysis, elevated liver enzymes, and low platelet count syndrome.

2004

Background & Aims: The HELLP (hemolysis, elevated liver enzymes, low platelets) syndrome is a life-threatening complication during pregnancy. The associated liver disease may be severe, and maternal hepatic complications may progress to the point that transplantation becomes necessary. CD95 (APO-1, Fas)-mediated apoptosis of liver cells is one of the major pathogenic mechanisms during liver disease. The interaction of CD95 with its ligand, CD95L(FasL), induces apoptosis and thus the source of the death-inducing ligand is critical for understanding the pathomechanism of liver damage involving the CD95-system. Methods: Sera from HELLP patients were analyzed and used in cell culture experiment…

medicine.medical_specialtyHELLP SyndromeFas Ligand ProteinHELLP syndromePlacentaApoptosisBiologyHepatic ComplicationFas ligandAcute fatty liver of pregnancyLiver diseaseJurkat CellsMicePregnancyInternal medicinemedicineAnimalsHumansCells CulturedTransaminasesMembrane GlycoproteinsHepatologymedicine.diagnostic_testLiver cellGastroenterologymedicine.diseaseHemolysisMolecular WeightEndocrinologyLiverCancer researchHepatocytesFemaleLiver function testsGastroenterology
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