Search results for " hepatocellular"

showing 10 items of 592 documents

Adaptive immunity suppresses formation and progression of diethylnitrosamine-induced liver cancer

2012

Background Hepatocellular carcinoma (HCC) is a typical inflammation-associated cancer, but may also provoke antitumour immune responses whose significance and underlying mechanisms are incompletely understood. Objective To characterise immune responses in the diethylnitrosamine (DEN)-liver cancer mouse model. Design Tumour development and immune cell functions upon DEN treatment were compared between C57BL/6 wild-type (WT), chemokine scavenging receptor D6-deficient, B cell- (Igh6), CD4 T cell- (MHC-II) and T-/B cell-deficient (Rag1) mice. Relevance for human HCC was tested by comparing gene array results from 139 HCC tissues. Results The induction of premalignant lesions after 24 weeks and…

MalePathologymedicine.medical_specialtyCarcinoma HepatocellularAdaptive ImmunityBiologyMajor histocompatibility complexChemokine CXCL9ArticleCCL5MiceLiver Neoplasms ExperimentalImmune systemAntigenLeukocytesmedicineAnimalsHumansCytotoxic T cellDiethylnitrosamineChemokine CCL5Chemokine CCL2B cellOligonucleotide Array Sequence AnalysisMice KnockoutB-LymphocytesMacrophagesLiver NeoplasmsGastroenterologyAcquired immune systemSurvival Analysisdigestive system diseasesMice Inbred C57BLmedicine.anatomical_structureCarcinogensDisease ProgressionCancer researchbiology.proteinPrecancerous ConditionsBiomarkersCD8T-Lymphocytes CytotoxicGut
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Liver transplantation for hepatocellular carcinoma with intrahepatic lymphatic invasion: case reports.

2008

Multiple studies addressing liver transplantation (OLT) for hepatocellular carcinoma (HCC) have identified various prognostic determinants of tumor recurrence and decreased patient survival. However, little information is available on the impact of intrahepatic lymphatic invasion on tumor recurrence and survival after OLT for HCC. Intrahepatic lymphatic invasion was observed in 1.4% (n = 2) of liver explants with HCC in our series. Both recipients are alive without tumor recurrence at 16 and 39 months post-OLT, respectively. Intrahepatic lymphatic invasion may not be an absolute adverse prognostic factor in cases of HCC with no hilar lymph node involvement at the time of OLT.

MalePathologymedicine.medical_specialtyCarcinoma HepatocellularLymphovascular invasionmedicine.medical_treatmentLiver transplantationGastroenterologyMetastasisInternal medicinemedicineHumansNeoplasm InvasivenessLymph nodeTransplantationbusiness.industryLiver NeoplasmsCancerMiddle Agedmedicine.diseasedigestive system diseasesLiver Transplantationsurgical procedures operativeLymphatic systemmedicine.anatomical_structureTreatment OutcomeHepatocellular carcinomaSurgeryFemaleLiver cancerbusinessTransplantation proceedings
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Magnetic resonance pharmacokinetic imaging clusterization of hepatocellular carcinomas as a means to grade tumor aggressiveness.

2012

Hepatocellular carcinoma (HCC) management takes into account clinical and radiological findings, such as tumor stage, hepatic functional status and clinical symptoms. It is necessary to evaluate the number, size and location of the lesions. However, lesion aggressiveness is not considered in this therapeutic workflow, although the biology and the growth rate of the lesions have an important impact on survival. The aim of this work was to establish if the quantitative pharmacokinetic assessment of dynamic contrast-enhanced magnetic resonance images of HCC can separate lesions with different microvascular properties and biological evolution. Forty five patients with HCC and dynamic contrast-e…

MalePathologymedicine.medical_specialtyCarcinoma HepatocellularPharmacokinetic modelingContrast MediaKaplan-Meier EstimateLesionCapillary PermeabilityPharmacokineticsTumor stagemedicineBiomarkers TumorCluster AnalysisHumansAgedHepatologymedicine.diagnostic_testbusiness.industryLiver NeoplasmsGastroenterologyMagnetic resonance imagingBiological evolutionMiddle Agedmedicine.diseaseImage EnhancementMagnetic Resonance ImagingHepatocellular carcinomaDisease ProgressionFunctional statusFemalemedicine.symptomNeoplasm GradingbusinessExpert review of gastroenterologyhepatology
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Immune escape mechanism: defective resting and stimulated leukocyte-endothelium interaction in hepatocellular carcinoma of the rat.

2004

Hepatocellular carcinoma represents an increasing therapeutic challenge due to its high incidence and early metastasis. Numerous studies have demonstrated the influence of the vascular system on tumor growth and development. In addition, the role of leukocyte-endothelium interaction in tumor vessels is of particular significance with regard to immunological tumor therapy. In this study we used an experimental in vivo animal model that allows a quantitative analysis on vessel morphology, microcirculation, and leukocyte-endothelium interaction. The vessel architecture in tumor tissue was found to be extremely heterogeneous, with a consecutively variable blood flow velocity. Following superfus…

MalePathologymedicine.medical_specialtyCell signalingCarcinoma HepatocellularEndotheliumPhysiologyCell CommunicationBiologyMetastasisMicrocirculationNeovascularizationmedicineLeukocytesAnimalsChemotactic FactorsNeovascularization PathologicCell adhesion moleculeMicrocirculationLiver NeoplasmsGastroenterologyHemodynamicsChemotaxismedicine.diseaseRatsRats Inbred ACImedicine.anatomical_structureLiverModels AnimalEndothelium Vascularmedicine.symptomLiver cancerCell Adhesion MoleculesBlood Flow VelocityDigestive diseases and sciences
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Metastatic hepatocellular carcinoma of the parotid region without lung metastasis: a case report

2009

Parotid gland metastatic tumours are rare, usually arising from a primary mucosal or cutaneous cancer located in the ipsilateral head and neck region, although metastases from a primary cancer outside the head and neck region are possible. Hepatocellular carcinoma (HCC) rarely metastasizes to the head and neck region and the parotid glands are an even more unusual site. The authors describe the case of an 82-year-old male who presented with a right parotid mass. Percutaneous incisional biopsy with histological and immunohistochemical studies suggested metastatic HCC. Radiological investigations excluded other metastatic lesions. This case illustrates the difficulties that may be encountered…

MalePathologymedicine.medical_specialtyPercutaneousCarcinoma HepatocellularLung NeoplasmsLung metastasisDiagnosis Differentialhepatocellular carcinoma/ parotid region/ lung metastasisFatal Outcomestomatognathic systemmedicineHumansAged 80 and overbusiness.industryRespiratory diseaseLiver NeoplasmsCancermedicine.diseaseParotid glandParotid Neoplasmsstomatognathic diseasesParotid Regionmedicine.anatomical_structureOtorhinolaryngologyHepatocellular carcinomaImmunohistochemistrySurgeryOral Surgerybusiness
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Predictors of survival in patients with advanced hepatocellular carcinoma who permanently discontinued sorafenib

2015

Treatment with sorafenib of patients with advanced hepatocellular carcinoma is challenged by anticipated discontinuation due to tumor progression, liver decompensation, or adverse effects. While postprogression survival is clearly determined by the pattern of tumor progression, understanding the factors that drive prognosis in patients who discontinued sorafenib for any reason may help to improve patient management and second-line trial design. Patients consecutively admitted to three referral centers who were receiving best supportive care following permanent discontinuation of sorafenib for any reason were included. Postsorafenib survival (PSS) was calculated from the last day of treatmen…

MalePredictive Value of TestGastroenterologyCohort StudiesAntineoplastic AgentProspective StudiesProspective cohort studyMultivariate AnalysiLiver NeoplasmsHepatitis CMiddle AgedSorafenibItalyLiver NeoplasmHepatocellular carcinomaFemaleSurvival Analysimedicine.drugHumanSorafenibNiacinamidePhenylurea Compoundmedicine.medical_specialtyCarcinoma HepatocellularDrug-Related Side Effects and Adverse ReactionsSettore MED/12 - GASTROENTEROLOGIAReproducibility of ResultAntineoplastic AgentsRisk AssessmentPredictive Value of TestsInternal medicinemedicineHumansNeoplasm InvasivenessSurvival analysisAgedNeoplasm StagingNeoplasm InvasiveneAnalysis of VarianceHepatologyPerformance statusbusiness.industryPhenylurea CompoundsPatient SelectionCarcinomaSettore MED/09 - MEDICINA INTERNAReproducibility of ResultsHepatocellularHepatologymedicine.diseaseSurvival AnalysisSurgeryDiscontinuationProspective StudieWithholding TreatmentMultivariate AnalysisCohort StudiebusinessDrug-Related Side Effects and Adverse Reaction
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Cytotoxic activity of the novel small molecule AKT inhibitor SC66 in hepatocellular carcinoma cells

2014

Hepatocellular carcinoma (HCC) is characterized by limited response to current drug therapies. Here, we report that SC66, a novel AKT inhibitor, reduced cell viability in a dose- and time-dependent manner, inhibited colony formation and induced apoptosis in HCC cells. SC66 treatment led to a reduction in total and phospho-AKT levels. This was associated with alterations in cytoskeleton organization, a reduction in expression levels of E-cadherin, β-catenin and phospho-FAK, together with up-regulation of Snail protein levels. All these alterations were coupled with anoikis cell death induction. In addition, SC66 induced the production of reactive oxygen species (ROS) and DNA damage. Pre-trea…

MaleProgrammed cell deathCarcinoma HepatocellularCytoskeleton organizationPyridinesMice NudeApoptosisBiologyMice03 medical and health sciences0302 clinical medicineanoikisCell Line TumorAnimalsHumansAnoikisViability assayHCCProtein Kinase InhibitorsProtein kinase BPI3K/AKT/mTOR pathwayCell Proliferation030304 developmental biologySC660303 health sciencesCyclohexanonesCell growthAKTLiver NeoplasmsXenograft Model Antitumor AssaysMolecular biology3. Good healthOncologyApoptosis030220 oncology & carcinogenesismTORCancer researchHCC AKT mTOR SC66 anoikisProto-Oncogene Proteins c-aktResearch Paper
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Preliminary experience on safety of regorafenib after sorafenib failure in recurrent hepatocellular carcinoma after liver transplantation

2019

Regorafenib is one option for second-line treatment of hepatocellular carcinoma (HCC), improving overall survival (OS) of sorafenib-tolerant patients who develop progression. We aim to evaluate the safety and outcomes of regorafenib as second-line treatment for HCC recurrence after liver transplantation (LT). This is a retrospective, multicenter, international study including regorafenib-treated LT patients (2015-2018), with analysis of baseline characteristics and evolutionary events during sorafenib/regorafenib treatment. Twenty-eight LT patients (57 years, 7% cirrhotics, 54% performance status 1) were included. Median time from LT to regorafenib initiation was 3.9 (1.1-18.5) years; media…

MalePyridinesmedicine.medical_treatment030230 surgeryLiver transplantationchemotherapyGastroenterologychemistry.chemical_compound0302 clinical medicineAntineoplastic Combined Chemotherapy ProtocolsImmunology and AllergyPharmacology (medical)Liver NeoplasmsMiddle AgedSorafenibPrognosisRecurrent Hepatocellular Carcinomaside effectsHepatocellular carcinomaFemalemedicine.drugSorafenibAdultmedicine.medical_specialtyCarcinoma Hepatocellularcancer/malignancy/neoplasiaclinical research/practice03 medical and health sciencesYoung AdultInternal medicineRegorafenibmedicineHumansAdverse effectAgedRetrospective StudiesTransplantationdrug interactionPerformance statusbusiness.industryPhenylurea Compoundsmedicine.diseaseDiscontinuationLiver TransplantationchemistryDrug Resistance NeoplasmNeoplasm Recurrence Localpharmacologybusinessliver transplantation/hepatologyFollow-Up Studies
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Circulating IL-6 and sIL-6R in patients with hepatocellular carcinoma

2002

Interleukin-6 plays a central role in regulating the immune system, hematopoiesis, and acute phase reaction. It interacts with a receptor complex consisting of a specific ligand-binding protein (IL-6R, gp80) and a signal transduction protein (gp130). In this report, serum levels of IL-6 and a soluble form of the interleukin-6 receptor (sIL-6R) were evaluated in patients with hepatocellular carcinoma. The correlation between IL-6 and sIL-6R values, the stage of hepatocellular carcinoma, and main liver function tests was also studied.

MaleReceptor complexCarcinoma HepatocellularHepatocellular carcinomaGeneral Biochemistry Genetics and Molecular BiologySignal transduction proteingp130History and Philosophy of SciencemedicineCarcinomaHumansReceptorBiochemistry Genetics and Molecular Biology (all)medicine.diagnostic_testbusiness.industryInterleukin-6General NeuroscienceLiver NeoplasmsAcute-phase proteinMiddle Agedmedicine.diseaseGlycoprotein 130Receptors Interleukin-6Hepatocellular carcinomaLiver function testCancer researchFemaleSignal transductionLiver function testsbusinessReceptor
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Dissemination of hepatocellular carcinoma is mediated via chemokine receptor CXCR4

2006

In different tumour entities, expression of the chemokine receptor 4 (CXCR4) has been linked to tumour dissemination and poor prognosis. Therefore, we evaluated, if the expression of CXCR4 exerts similar effects in human hepatocellular carcinoma (HCC). Expression analysis and functional assays were performed in vitro to elucidate the impact of CXCL12 on human hepatoma cells lines. In addition, expression of CXCR4 was evaluated in 39 patients with HCC semiquantitatively and correlated with both, tumour and patients characteristics. Human HCC and hepatoma cell lines displayed variable intensities of CXCR4 expression. Loss of p53 function did not impact on CXCR4 expression. Exposure to CXCL12 …

MaleReceptors CXCR4Cancer ResearchChemokinePathologymedicine.medical_specialtyCarcinoma HepatocellularActive Transport Cell NucleusliverSensitivity and SpecificityCXCR4MetastasisChemokine receptorhepatocellularCell MovementPredictive Value of TestsTumor Cells CulturedCarcinomamedicinemetastasisHumansNeoplasm InvasivenessReceptorMolecular DiagnosticsCell ProliferationCXCR4biologychemokineLiver NeoplasmsMiddle AgedFlow Cytometrymedicine.diseaseImmunohistochemistryChemokine CXCL12digestive system diseasesSurvival RateOncologyHepatocellular carcinomaDisease ProgressionCancer researchbiology.proteinImmunohistochemistryFemaleChemokines CXCBritish Journal of Cancer
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