Search results for " intravenous"

showing 10 items of 331 documents

In vitro evaluation of poloxamer in situ forming gels for bedaquiline fumarate salt and pharmacokinetics following intramuscular injection in rats

2019

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In situPO Propylene oxideIV IntravenousP338 Poloxamer 338lcsh:RS1-441Pharmaceutical Sciencechemistry.chemical_compoundn Sample sizeSD Standard deviationIM Intramuscularchemistry.chemical_classificationC0 Analyte plasma concentration at time zeroDoE Design of experimentsUV UltravioletPharmacology. TherapyK2.EDTA Potassium ethylenediaminetetraacetic acidLC–MS/MS Liquid chromatography-tandem mass spectrometryH&E Hematoxylin and eosintmax Sampling time to reach the maximum observed analyte plasma concentrationIn situ forming gelsCMC Critical micellar concentrationCmax Maximum observed analyte plasma concentrationIntramuscular injectionDN Dose normalizedGPT Gel point temperaturePLGA Poly-(DL-lactic-co-glycolic acid)TFA Trifluoroacetic acidCAN AcetonitrileATP Adenosine 5′ triphosphateSalt (chemistry)Polyethylene glycolPoloxamerArticlelcsh:Pharmacy and materia medicaPharmacokineticsIn vivoUHPLC Ultra-high performance liquid chromatographyPharmacokineticsAUClast Area under the analyte concentration versus time curve from time zero to the time of the last measurable (non-below quantification level) concentrationEO Ethylene oxideNMP N-methyl-2-pyrrolidoneComputingMethodologies_COMPUTERGRAPHICSAUC∞ Area under the analyte concentration vs time curve from time zero to infinite timeP407 Poloxamer 407In vitro releasePoloxamerCMT Critical micellar temperatureGel erosionIn vitrot1/2 Apparent terminal elimination half-lifechemistryMDR-TB Multi-drug resistant tuberculosisAUC80h Area under the analyte concentration versus time curve from time zero to 80 htlast Sampling time until the last measurable (non-below quantification level) analyte plasma concentrationMRM Multiple reaction monitoringNuclear chemistrySustained releaseInternational Journal of Pharmaceutics: X
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Drug-induced expansion and differentiation of Vγ9Vδ2 T cells in vivo: The role of exogenous IL-2

2005

Human Vgamma9Vdelta2 T cells recognize nonpeptidic Ags generated by the 1-deoxy-d-xylulose 5-phosphate (many eubacteria, algae, plants, and Apicomplexa) and mevalonate (eukaryotes, archaebacteria, and certain eubacteria) pathways of isoprenoid synthesis. The potent Vgamma9Vdelta2 T cell reactivity 1) against certain cancer cells or 2) induced by infectious agents indicates that therapeutic augmentations of Vgamma9Vdelta2 T cell activities may be clinically beneficial. The functional characteristics of Vgamma9Vdelta2 T cells from Macaca fascicularis (cynomolgus monkey) are very similar to those from Homo sapiens. We have found that the i.v. administration of nitrogen-containing bisphosphonat…

Injections SubcutaneousT cellImmunologyCD4-CD8 RatioPamidronateBiologyPharmacologyInterferon-gammaInterleukin 21HemiterpenesOrganophosphorus CompoundsT-Lymphocyte SubsetsmedicineAnimalsImmunology and AllergyCytotoxic T cellIL-2 receptorAntigensAntigen-presenting cellCells CulturedCell ProliferationInterleukin 323-DiphosphoglycerateDiphosphonatesZAP70Cell DifferentiationReceptors Antigen T-Cell gamma-deltaTh1 CellsNatural killer T cellDiphosphatesMacaca fascicularismedicine.anatomical_structureInjections IntravenousImmunologyEpoxy CompoundsInterleukin-2Immunologic Memory
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Effect of diazoxide on left ventricular performance in hypertension.

1975

The effect of diazoxide on left ventricular performance during rest and isometric exercise (handgrip) was examined in 16 unselected hypertensive patients, 6 of whom had been pretreated with the beta-adrenergic blocking agent pindolol. Diazoxide regularly and promptly produced a fall in left ventricular systolic and end diastolic pressures, and an increase in heart rate and left ventricular dp/dtmax. Haemodynamic changes were maximal 2 minutes after injection of the drug and decreased little over the next 8 minutes. After beta-adrenergic blockade, diazoxide caused a more pronounced reduction in left ventricular systolic pressure and a less marked fall in end-diastolic pressure, whilst the di…

InotropeAdultMalemedicine.medical_specialtyPhysical ExertionDiastoleHemodynamicsBlood PressureIsometric exerciseHeart RateInternal medicineHeart rateDiazoxideMedicineHumansPharmacology (medical)Drug InteractionsPharmacologybusiness.industryDiazoxideHemodynamicsGeneral MedicineMiddle AgedMyocardial ContractionBlood pressureAnesthesiaPindololHypertensionInjections Intravenouscardiovascular systemVentricular pressureCardiologyFemalebusinessmedicine.drugEuropean journal of clinical pharmacology
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Intestinal Involvement in Kawasaki Disease

2018

Objectives To describe a case of Kawasaki disease with intestinal involvement and to analyze other published reports to define clinical characteristics, diagnostic issues, and therapeutic approaches of gastrointestinal involvement in Kawasaki disease. Study design A computerized search without language restriction was conducted using PubMed and SCOPUS. An article was considered eligible for inclusion in the systematic review if it reported data on patient(s) with intestinal involvement in Kawasaki disease. Our case was also included in the analysis. Results Thirty-three articles reporting 48 cases of Kawasaki disease with intestinal involvement were considered. Fever, abdominal pain, and vo…

Intestinal pseudo-obstructionMalemedicine.medical_specialtyPediatricsAbdominal painSettore MED/17 - Malattie InfettiveAdolescentFeverMucocutaneous Lymph Node SyndromeDiagnosis Differential03 medical and health sciences0302 clinical medicinepediatric gastroenterology030225 pediatricsmedicineintestinal pseudo-obstructionHumans030212 general & internal medicineHematologic TestPediatric gastroenterologyCoronary artery aneurysmGangreneAspirinHematologic TestsIntestinal Diseasebusiness.industryabdominal painImmunoglobulins Intravenousmedicine.diseasecoronary artery aneurysmHospitalizationIntestinal DiseasesImmunoglobulins IntravenouPediatrics Perinatology and Child HealthKawasakiSplenomegalyVomitingKawasaki diseasemedicine.symptombusinessTomography X-Ray ComputedBowel diseaseIntestinal Obstructionmedicine.drugHepatomegalyHuman
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Positive Iron Balance in Chronic Kidney Disease: How Much is Too Much and How to Tell?

2017

<b><i>Background:</i></b> Regulation of body iron occurs at cellular, tissue, and systemic levels. In healthy individuals, iron absorption and losses are minimal, creating a virtually closed system. In the setting of chronic kidney disease and hemodialysis (HD), increased iron losses, reduced iron absorption, and limited iron availability lead to iron deficiency. Intravenous (IV) iron therapy is frequently prescribed to replace lost iron, but determining an individual’s iron balance and stores can be challenging and imprecise, contributing to uncertainty about the long-term safety of IV iron therapy. <b><i>Summary:</i></b> Patients on HD recei…

Iron030232 urology & nephrologyPhysiology030204 cardiovascular system & hematologyDirect reduced iron03 medical and health sciences0302 clinical medicineHepcidinmedicineHomeostasisHumansErythropoiesisRenal Insufficiency ChronicHemochromatosischemistry.chemical_classificationbiologybusiness.industryIron deficiencymedicine.diseaseTrace ElementschemistryNephrologyTransferrinToxicitybiology.proteinErythropoiesisAdministration IntravenousbusinessKidney diseaseAmerican journal of nephrology
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High dose infusion of activated protein C (rhAPC) fails to improve neuronal damage and cognitive deficit after global cerebral ischemia in rats

2013

Abstract Purpose : Recent studies demonstrated anticoagulatory, antiinflammatory, antiapoptotic, and neuroprotective properties of activated protein C (APC) in rodent models of acute neurodegenerative diseases, suggesting APC as promising broad acting therapeutic agent. Unfortunately, continuous infusion of recombinant human APC (rhAPC) failed to improve brain damage following cardiac arrest in rats. The present study was designed to investigate the neuroprotective effect after global cerebral ischemia (GI) with an optimized infusion protocol. Methods : Rats were subjected to bilateral clip occlusion of the common carotid arteries (BCAO) and controlled hemorrhagic hypotension to 40 mmHg for…

IschemiaInflammationBrain damagePharmacologyNeuroprotectionBrain IschemiaRats Sprague-DawleyBrain ischemiamedicineAnimalsHumansCerebral perfusion pressureInfusions IntravenousCell Deathbusiness.industryGeneral NeuroscienceDrotrecogin alfaBrainmedicine.diseaseRecombinant ProteinsRatsAnesthesiamedicine.symptombusinessProtein CProtein Cmedicine.drugNeuroscience Letters
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Risk of laryngeal edema and facial swellings after tooth extraction in patients with hereditary angioedema with and without prophylaxis with C1 inhib…

2010

Objective Tooth extractions may trigger clinical symptoms of hereditary angioedema due to C1 inhibitor deficiency (HAE-C1-INH). The aim of this study was to determine how many tooth extractions were followed by symptoms of HAE-C1-INH in patients with and without preoperative short-term prophylaxis with C1 inhibitor concentrate. Study design Tooth extractions and clinical symptoms of HAE-C1-INH were determined from clinical record files of 171 patients with HAE-C1-INH. Results Facial swelling or potentially life-threatening laryngeal edema, or both, occurred in 124/577 tooth extractions (21.5%) without prophylaxis. Similar symptoms occurred in a fewer proportion of patients undergoing extrac…

LarynxAdultMalemedicine.medical_specialtyTime FactorsPremedicationComplement C1 Inactivator ProteinsLaryngeal EdemaChemopreventionC1-inhibitorRisk FactorsEdemamedicineEdemaHumansRisk factorGeneral DentistryRetrospective StudiesbiologyDose-Response Relationship Drugbusiness.industryAngioedemas HereditaryRetrospective cohort studyLaryngeal Edemamedicine.diseaseSurgerymedicine.anatomical_structureTreatment OutcomeOtorhinolaryngologyFaceHereditary angioedemaInjections IntravenousTooth Extractionbiology.proteinSurgeryPremedicationFemaleOral Surgerymedicine.symptombusinessComplement C1 Inhibitor ProteinFollow-Up StudiesOral surgery, oral medicine, oral pathology, oral radiology, and endodontics
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Neuronal activity triggers uptake of hematopoietic extracellular vesicles in vivo

2019

Communication with the hematopoietic system is a vital component of regulating brain function in health and disease. Traditionally, the major routes considered for this neuroimmune communication are by individual molecules such as cytokines carried by blood, by neural transmission, or, in more severe pathologies, by the entry of peripheral immune cells into the brain. In addition, functional mRNA from peripheral blood can be directly transferred to neurons via extracellular vesicles (EVs), but the parameters that determine their uptake are unknown. Using varied animal models that stimulate neuronal activity by peripheral inflammation, optogenetics, and selective proteasome inhibition of dop…

LipopolysaccharidesMaleGene ExpressionStimulationHippocampusBiochemistryStereotaxic Techniques0302 clinical medicineShort ReportsAnimal CellsMedicine and Health SciencesPremovement neuronal activityBiology (General)Routes of AdministrationNeurons0303 health sciencesBrain MappingKainic AcidBrainAnimal ModelsPeripheralCell biologyHaematopoiesisBioassays and Physiological AnalysisExperimental Organism SystemsHippocampus ; Yellow flourescent protein ; Intravenous injections ; Marker genes ; Gene expression ; Neurons ; Microglial cells ; OptogeneticsFemaleCellular TypesSignal TransductionProteasome Endopeptidase ComplexQH301-705.5Yellow Fluorescent ProteinMice TransgenicGlial CellsMouse ModelsStimulus (physiology)BiologyResearch and Analysis Methods03 medical and health sciencesExtracellular VesiclesImmune systemModel OrganismsIn vivoIntravenous InjectionsGeneticsAnimalsddc:610Molecular Biology TechniquesMolecular BiologyMicroglial Cells030304 developmental biologyInflammationPharmacologyMessenger RNABlood CellsUbiquitinDopaminergic NeuronsBiology and Life SciencesProteinsMarker GenesCell BiologyNeurophysiological AnalysisOptogeneticsLuminescent ProteinsCellular NeuroscienceAnimal Studies030217 neurology & neurosurgeryNeuroscience
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Involvement of neuronal processes and nitric oxide in the inhibition by endotoxin of pentagastrin-stimulated sastric acid secretion

1994

Administration of E. coli endotoxin (1 mg kg-1, i.v.) abolished the acid response induced by the i.v. infusion of pentagastrin (8 micrograms kg-1 h-1) in the continuously perfused stomach of the anaesthetized rat. Local serosal application of tetrodotoxin (36 ng per rat) completely restored acid responses to pentagastrin in endotoxin-treated rats. However, pretreatment with atropine (0.5 mg kg-1, s.c.), capsaicin (20, 30, and 50 mg kg-1, s.c. 2 weeks before the study) or guanethidine (16 mg kg-1, s.c. 3 and 16h before) did not influence the inhibitory effects of endotoxin. Continuous i.v. infusion with NG-nitro arginine methyl ester (L-NAME, 10 mg kg-1 h-1) restored the secretory responses …

LipopolysaccharidesMalemedicine.medical_specialtyTetrodotoxinPeptide hormoneBiologyArginineNitric OxideNitric oxideGastric AcidPhenylephrinechemistry.chemical_compoundInternal medicineEscherichia colimedicineAnimalsDrug InteractionsRats WistarInfusions IntravenousGuanethidineNeuronsPharmacologyStomachGeneral MedicineRatsEndotoxinsPentagastrinNG-Nitroarginine Methyl EsterEndocrinologyMechanism of actionchemistryGastrointestinal hormoneGastric MucosaCapsaicinTetrodotoxinFemalePentagastrinmedicine.symptommedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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Intravenous high-dose furosemide and hypertonic saline solutions for refractory heart failure and ascites.

2011

Several studies have shown the efficacy of hypertonic saline solution infusion in conditions in which regional organ blood flow is impaired. Our group has shown that treatment of patients with diuretic-resistant heart failure with high-dose furosemide plus hypertonic saline is effective and well tolerated, improving symptoms of congestion, reducing plasma levels of markers of neurohormonal and inflammatory activation, decreasing hospital readmission rates, and reducing long-term mortality. The same regimen was shown to be better than repeated paracentesis in patients with cirrhosis and refractory ascites, yielding better control of ascites, pleural effusions, and/or leg edema without an inc…

Liver CirrhosisCirrhosisFurosemideAscitesmedicineParacentesisHumansAdverse effectDiureticsInfusions IntravenousHepatic encephalopathyHeart FailureSaline Solution Hypertonicmedicine.diagnostic_testCardio-Renal Syndromebusiness.industryFurosemideAscitesmedicine.diseaseHypertonic salineNephrologyAnesthesiaHeart failuremedicine.symptombusinessmedicine.drugSeminars in nephrology
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