Search results for " leptin"

showing 10 items of 56 documents

Increased expression of leptin and the leptin receptor as a marker of breast cancer progression: possible role of obesity-related stimuli.

2006

Abstract Purpose: Recent in vitro studies suggested that the autocrine leptin loop might contribute to breast cancer development by enhancing cell growth and survival. To evaluate whether the leptin system could become a target in breast cancer therapy, we examined the expression of leptin and its receptor (ObR) in primary and metastatic breast cancer and noncancer mammary epithelium. We also studied whether the expression of leptin/ObR in breast cancer can be induced by obesity-related stimuli, such as elevated levels of insulin, insulin-like growth factor-I (IGF-I), estradiol, or hypoxic conditions. Experimental Design: The expression of leptin and ObR was examined by immunohistochemistry…

LeptinCancer ResearchER-BETAmedicine.medical_treatmentHYPOXIA-INDUCIBLE FACTOR-1NeoplasmsTumor Cells CulturedBreastInsulin-Like Growth Factor Iskin and connective tissue diseasesReceptorAged 80 and overEstradiolIGF-I RECEPTORCELL-LINEReverse Transcriptase Polymerase Chain ReactionLeptindigestive oral and skin physiologyCarcinoma Ductal BreastMiddle AgedMetastatic breast cancerINSULINCell HypoxiaESTROGENOncologyDisease ProgressionIGF-I RECEPTOR; HYPOXIA-INDUCIBLE FACTOR-1; OB GENE; GROWTH-FACTOR; CELL-LINE; ER-BETA; ESTROGEN; ALPHA; INSULIN; MCF-7Receptors LeptinFemaleOB GENEhormones hormone substitutes and hormone antagonistsAdultmedicine.medical_specialtyGROWTH-FACTORBreast NeoplasmsReceptors Cell SurfaceBreast cancerInternal medicinemedicineBiomarkers TumorHumansObesityAutocrine signallingAgedLeptin receptorbusiness.industryInsulinmedicine.diseaseALPHAEndocrinologyTumor progressionCase-Control StudiesMCF-7business
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Effect of 2,3,7,8-Tetrachlorodibenzo-p-dioxin (TCDD) on Hormones of Energy Balance in a TCDD-Sensitive and a TCDD-Resistant Rat Strain

2014

One of the hallmarks of the acute toxicity of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) is a drastically reduced feed intake by an unknown mechanism. To further elucidate this wasting syndrome, we followed the effects of a single large dose (100 μg/kg) of TCDD on the serum levels of several energy balance-influencing hormones, clinical chemistry variables, and hepatic aryl hydrocarbon receptor (AHR) expression in two rat strains that differ widely in their TCDD sensitivities, for up to 10 days. TCDD affected most of the analytes in sensitive Long-Evans rats, while there were few alterations in the resistant Han/Wistar strain. However, analyses of feed-restricted unexposed Long-Evans rats i…

LeptinFOOD-INTAKETCDDFGF21Polychlorinated Dibenzodioxinsmedicine.medical_treatmentAHRwasting syndromeacute toxicity413 Veterinary science8-tetrachlorodibenzo-p-dioxinlcsh:Chemistry2378-tetrachlorodibenzo-<i>p</i>-dioxin; TCDD; wasting syndrome; energy balance; hormones; acute toxicity; strain differences; AHRPPAR-ALPHAInsulinMESSENGER-RNA EXPRESSIONInsulin-Like Growth Factor Ita315Receptorlcsh:QH301-705.5AH RECEPTORSpectroscopyenergiatasebiologyChemistryLeptinGeneral MedicineCENTRAL LEPTIN INFUSIONstrain differencesComputer Science ApplicationsLiverGhrelinAdiponectinARYL-HYDROCARBON RECEPTOR7medicine.medical_specialty3education2GlucagonCatalysisArticleInorganic ChemistrySpecies SpecificityInternal medicinemedicineAnimals2378-tetrachlorodibenzo-p-dioxinRats Long-EvansRNA MessengerPhysical and Theoretical ChemistryRats WistarCARBOXYKINASE PEPCK ACTIVITYMolecular BiologyI IGF-IhormonesGrowth factorOrganic ChemistryBody WeightAryl hydrocarbon receptorGlucagonenergy balancehormonitRatsFibroblast Growth FactorsEndocrinologylcsh:Biology (General)lcsh:QD1-999Receptors Aryl Hydrocarbonbiology.proteinGROWTH-FACTOR 21Energy MetabolismHormoneInternational Journal of Molecular Sciences
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Immunohistochemical expression and distribution of orexin, orphanin and leptin in the major salivary glands of some mammals

2012

Abstract: The aim of the study was to assess the involvement of apoptotic factors, cytokeratins and metalloproteinase- 9 in the histogenesis of both Epithelialized Gingival Lesions (EGL) and Periapical Lesions (PAL). 55 consecutive patients, 30 with PAL and 25 with EGL, were selected for the study after clinical and radiological examinations. The PAL patients had severe periapical lesions and tooth decay with exposure of the pulp chamber. All PAL and EGL biopsies were surgically extracted, fixed in 10% buffered formalin, and processed for routine light microscopy. Ten biopsies of each category were processed for immunohistochemistry (IHC). Serial paraffin sections were stained by IHC with a…

LeptinKey words: cytokeratins MMP-9 caspase-3 caspase-9 perapical lesions epithelial gingival lesions apoptosis IHC PCNA TUNELSettore BIO/17 - Istologiamedicine.medical_specialtyHistologySubmandibular Glandcytokeratins MMP-9 caspase-3 caspase-9 perapical lesions epithelial gingival lesions apoptosis IHC PCNA TUNEL [Key words]major salivary glands orphanin FQ nociception orexin leptin IHC rat sheep cowBiologySalivary GlandsPathology and Forensic MedicineOrexin-ASublingual Glandstomatognathic systemInternal medicineMajor Salivary GlandOrexigenicmedicineEndocrine systemAnimalsParotid GlandMammalsOrexinsSheepSalivary glandNeuropeptidesConnective tissue stromaIntracellular Signaling Peptides and ProteinsGeneral MedicineImmunohistochemistryEpitheliumOrexinRatsmedicine.anatomical_structureEndocrinologyOpioid PeptidesCattlemedicine.drug
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In a randomized trial in prostate cancer patients, dietary protein restriction modifies markers of leptin and insulin signaling in plasma extracellul…

2017

Obesity, metabolic syndrome, and hyperleptinemia are associated with aging and age-associated diseases including prostate cancer. One experimental approach to inhibit tumor growth is to reduce dietary protein intake and hence levels of circulating amino acids. Dietary protein restriction (PR) increases insulin sensitivity and suppresses prostate cancer cell tumor growth in animal models, providing a rationale for clinical trials. We sought to demonstrate that biomarkers derived from plasma extracellular vesicles (EVs) reflect systemic leptin and insulin signaling and respond to dietary interventions. We studied plasma samples from men with prostate cancer awaiting prostatectomy who particip…

LeptinMale0301 basic medicineAgingmedicine.medical_specialtyhyperleptinemiamedicine.medical_treatmentexosomesBiologymetabolic syndrome03 medical and health sciencesProstate cancer0302 clinical medicineInternal medicineDiet Protein-RestrictedmedicineHumansInsulinprotein restrictionexosomes; extracellular vesicles; IRS-1; leptin receptor; prostate cancer; protein restriction; Aging; Cell BiologyObesityIRS-1leptin receptorIRS-1; exosomes; extracellular vesicles; leptin receptor; prostate cancer; protein restrictionIRS‐1Caloric RestrictionShort TakesLeptin receptorLeptinInsulinProstatic NeoplasmsShort TakeCell BiologyMiddle Agedprostate cancermedicine.diseaseObesity3. Good healthIRS1Insulin receptor030104 developmental biologyEndocrinologybiology.proteinMetabolic syndromeEnergy Metabolismextracellular vesicles030217 neurology & neurosurgeryage-associated disease
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Aerobic Fitness Does Not Modify the Effect of FTO Variation on Body Composition Traits

2012

Purpose Poor physical fitness and obesity are risk factors for all cause morbidity and mortality. We aimed to clarify whether common genetic variants of key energy intake determinants in leptin (LEP), leptin receptor (LEPR), and fat mass and obesity-associated (FTO) are associated with aerobic and neuromuscular performance, and whether aerobic fitness can alter the effect of these genotypes on body composition. Methods 846 healthy Finnish males of Caucasian origin were genotyped for FTO (rs8050136), LEP (rs7799039) and LEPR (rs8179183 and rs1137101) single nucleotide polymorphisms (SNPs), and studied for associations with maximal oxygen consumption, body fat percent, serum leptin levels, wa…

LeptinMaleAnatomy and PhysiologyLiikuntatiede - Sport and fitness sciencesMuscle FunctionsEpidemiologyRespiratory SystemPhysical fitnesslcsh:MedicineCardiovascular SystemBody Mass Indexlcsh:ScienceMusculoskeletal SystemMultidisciplinaryIGF1VO2 maxAdipose TissueGenetic EpidemiologyBody CompositionCirculatory PhysiologyReceptors LeptinMuscleMedicinePublic HealthWaist CircumferenceBehavioral and Social Aspects of HealthResearch ArticleAdultmedicine.medical_specialtyWaistAlpha-Ketoglutarate-Dependent Dioxygenase FTOBiologyPolymorphism Single NucleotideOxygen ConsumptionInternal medicineGeneticsmedicineHumansAerobic exerciseRespiratory PhysiologySports and Exercise MedicineMuscle SkeletalExerciseBiologyGenetic Association StudiesAerobic capacityLeptin receptorbusiness.industrylcsh:RProteinsnutritional and metabolic diseasesHuman Geneticsmedicine.diseaseObesityOikeuslääketiede ja muut lääketieteet - Forensic science and other medical sciencesEndocrinologylcsh:QPreventive MedicinebusinessBody mass indexPLoS ONE
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Clusterin and LRP2 are critical components of the hypothalamic feeding regulatory pathway.

2012

Hypothalamic feeding circuits are essential for the maintenance of energy balance. There have been intensive efforts to discover new biological molecules involved in these pathways. Here we report that central administration of clusterin, also called apolipoprotein J, causes anorexia, weight loss and activation of hypothalamic signal transduction-activated transcript-3 in mice. In contrast, inhibition of hypothalamic clusterin action results in increased food intake and body weight, leading to adiposity. These effects are likely mediated through the mutual actions of the low-density lipoprotein receptor-related protein-2, a potential receptor for clusterin, and the long-form leptin receptor…

LeptinMaleSTAT3 Transcription Factormedicine.medical_specialtyApolipoprotein BHypothalamusGeneral Physics and AstronomyGeneral Biochemistry Genetics and Molecular BiologyCell LineMiceInternal medicinemedicineAnimalsHumansObesityPhosphorylationReceptorInjections IntraventricularEpididymisMultidisciplinaryLeptin receptorbiologyClusterinLeptinBody WeightGeneral ChemistryFeeding BehaviorLRP2ImmunohistochemistryHedgehog signaling pathwayAnorexiaRatsLow Density Lipoprotein Receptor-Related Protein-2EndocrinologyClusterinStarvationbiology.proteinReceptors LeptinLipoproteinProtein BindingSignal TransductionNature communications
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Serum leptin and interleukin-6 levels in pediatric patients with HIV.

2003

Recent therapeutic approaches have improved the prognosis of children with HIV. Many new efforts could be involved in their quality of life and therefore could need additional diagnostic strategies. Leptin regulates pubertal development; furthermore a continuous immune stimulus, as in chronic infectious diseases, can enhance leptin's secretion by the action of cytokines such as interleukin (IL)-6. To clarify this role in patients infected with HIV, we assayed leptin and IL-6 and evaluated the influence of HIV severity on its secretion. IL-6 (380.5 +/- 257.6 pg/ml; range: 22-900 pg/ml) showed a significant correlation with leptinemia, HIV-1 RNA, and viremia related to the stage of HIV diseas…

LeptinMalemedicine.medical_specialtyAnti-HIV AgentsEndocrinology Diabetes and MetabolismViremiaEnzyme-Linked Immunosorbent AssayHIV InfectionsPubertal stageEndocrinologyImmune systemStatistical significanceInternal medicinemedicineHumansHIV InfectionSecretionSexual MaturationInterleukin 6ChildPediatric HIVbiologybusiness.industryInterleukin-6Reverse Transcriptase Polymerase Chain ReactionCD4 AntigenLeptinPubertyAnti-HIV AgentInterleukinInfantmedicine.diseaseEndocrinologyChild PreschoolPediatrics Perinatology and Child HealthImmunologyCD4 Antigensbiology.proteinHIV-1Receptors LeptinFemalebusinessHumanJournal of pediatric endocrinologymetabolism : JPEM
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Genetic variations of leptin and leptin receptor are associated with body composition changes in response to physical training

2010

Leptin regulates body weight, metabolism, and tissue adaptations to environmental stressors. We examined the association of single nucleotide polymorphism (SNP) of leptin promoter G-2548A (rs7799039) and leptin receptor Gln223Arg (rs1137101) with body composition, plasma leptin levels, and peak oxygen uptake (VO(2)peak) in response to 8 weeks of physical training in 48 male military conscripts. AA homozygotes of leptin promoter SNP-2548 showed higher body fat and BMI values than G allele carriers. Acute exercise decreased leptin levels in G allele carriers, but increased in AA homozygotes. Physical training significantly decreased BMI values and also a tendency for decreased plasma leptin l…

LeptinMalemedicine.medical_specialtyGenotypeClinical BiochemistrySingle-nucleotide polymorphismBiologyPolymorphism Single NucleotideBiochemistryBody Mass IndexYoung AdultOxygen ConsumptionInternal medicinemedicineHumansAllelePromoter Regions GeneticExerciseAllelesLeptin receptorLeptinHomozygotedigestive oral and skin physiologyVO2 maxCell BiologyGeneral MedicineEndocrinologyBlood chemistryHormone receptorBody CompositionReceptors LeptinBody mass indexhormones hormone substitutes and hormone antagonistsCell Biochemistry and Function
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Leptin and the OB-receptor as anti-obesity target: recent in silico advances in the comprehension of the protein-protein interaction and rational dru…

2014

The OB-receptor or leptin receptor (LR) is crucial for energy homeostasis and regulation of food uptake. Leptin is a 16 kDa hormone that is mainly secreted by fat cells into the bloodstream. Under normal circumstances, circulating leptin levels are proportionate to the fat body mass. Sensing of elevated leptin levels by the hypothalamic neuro-circuitry activates a negative feedback loop resulting in reduced food intake and increased energy expenditure. Decreased leptin concentrations lead to opposite effects. Therefore, rational design of leptin agonists/antagonists could be an appealing challenge in the battle against obesity. The Leptin/LR interactions have been studied in several works b…

LeptinModels Molecularmedicine.medical_specialtyIn silicoAdipose tissueDrug designBiologyEnergy homeostasisRisk FactorsInternal medicineDrug DiscoverymedicineHumansObesityReceptorleptin receptorPharmacologyleptin receptor agonists/antagonistLeptin receptorLeptindigestive oral and skin physiologyBody WeightRational designInfant Newbornprotein/protein dockingSettore CHIM/08 - Chimica Farmaceuticamolecular modellingEndocrinologyDrug DesignReceptors LeptinHomology modellingAnti-Obesity Agentshormones hormone substitutes and hormone antagonistsProtein BindingSignal TransductionCurrent pharmaceutical design
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Expression of angiogenic regulators, VEGF and leptin, is regulated by the EGF/PI3K/STAT3 pathway in colorectal cancer cells.

2009

Both leptin and vascular endothelial growth factor (VEGF) are growth and angiogenic cytokines that are upregulated in different types of cancer and have been implicated in neoplastic progression. Here we investigated the molecular mechanism by which leptin and VEGF expression are regulated in colon cancer by epidermal growth factor (EGF). In colon cancer cell line HT-29, EGF induced the binding of signal transducer and activator transcription 3 (STAT3) to STAT3 consensus motifs within the VEGF and leptin promoters and stimulated leptin and VEGF mRNA and protein synthesis. All these EGF effects were significantly blocked when HT-29 cells were treated with an inhibitor of the phosphoinositide…

LeptinSTAT3 Transcription FactorVascular Endothelial Growth Factor ASmall interfering RNAPhysiologyColorectal cancerClinical BiochemistryNeovascularization PhysiologicEGF/PI3K/STAT3colorectal cancerchemistry.chemical_compoundPhosphatidylinositol 3-KinasesEpidermal growth factormedicineHumansLY294002Gene SilencingRNA MessengerSTAT3Promoter Regions GeneticPI3K/AKT/mTOR pathwayCell NucleusbiologyEpidermal Growth FactorChemistryLeptinangiogenic regulators VEGF leptinCell Biologymedicine.diseaseUp-RegulationVascular endothelial growth factorGene Expression Regulation NeoplasticCancer researchbiology.proteinColorectal NeoplasmsHT29 Cellshormones hormone substitutes and hormone antagonistsProtein Binding
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