Search results for " neurosurgery"

showing 10 items of 7727 documents

Translation of HTT mRNA with expanded CAG repeats is regulated by the MID1-PP2A protein complex.

2012

Expansion of CAG repeats is a common feature of various neurodegenerative disorders, including Huntington's disease. Here we show that expanded CAG repeats bind to a translation regulatory protein complex containing MID1, protein phosphatase 2A and 40S ribosomal S6 kinase. Binding of the MID1-protein phosphatase 2A protein complex increases with CAG repeat size and stimulates translation of the CAG repeat expansion containing messenger RNA in a MID1-, protein phosphatase 2A- and mammalian target of rapamycin-dependent manner. Our data indicate that pathological CAG repeat expansions upregulate protein translation leading to an overproduction of aberrant protein and suggest that the MID1-com…

metabolism [Microtubule Proteins]General Physics and AstronomyHTT protein humanRibosomal s6 kinaseMice0302 clinical medicinemetabolism [Transcription Factors]Protein Phosphatase 2Luciferasesgenetics [Nerve Tissue Proteins]genetics [Protein Biosynthesis]0303 health sciencesHuntingtin ProteinMultidisciplinarybiologyTOR Serine-Threonine KinasesNuclear ProteinsTranslation (biology)3. Good healthmetabolism [Luciferases]Microtubule Proteinsddc:500metabolism [Nuclear Proteins]genetics [Trinucleotide Repeat Expansion]Protein Bindingcongenital hereditary and neonatal diseases and abnormalitiesMTOR protein humanUbiquitin-Protein LigasesBlotting WesternNerve Tissue Proteinsmetabolism [TOR Serine-Threonine Kinases]metabolism [RNA Messenger]General Biochemistry Genetics and Molecular Biology03 medical and health sciencesgenetics [RNA Messenger]mental disordersHuntingtin ProteinAnimalsHumansEukaryotic Small Ribosomal SubunitRNA MessengerNucleotide Motifs030304 developmental biologyMessenger RNAmetabolism [Nerve Tissue Proteins]RNAmetabolism [Protein Phosphatase 2]General ChemistryProtein phosphatase 2Molecular biologynervous system diseasesProtein Biosynthesisbiology.proteinTrinucleotide repeat expansionTrinucleotide Repeat Expansion030217 neurology & neurosurgeryMid1 protein humanHeLa CellsTranscription FactorsNature communications
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Isolation and Culture of Oligodendrocytes

2019

Primary cultures of brain-derived rodent cells are widely used to study molecular and cellular mechanisms in neurobiology. In this chapter, we describe methods of purifying and culturing oligodendroglial cells from mouse perinatal brains. In addition, we describe methods of coculturing the purified oligodendrocytes with neurons. When prepared and cultured according to these protocols, many essential aspects of the biology of oligodendrocytes, such as their proliferation, differentiation, and myelination, can be studied in culture.

methods [Cell Separation]0303 health sciencesMagnetic-activated cell sortingMagnetic PhenomenaOligodendrocyte differentiationOligodendrocyte progenitorCell DifferentiationBiologyFlow CytometryIsolation (microbiology)Coculture TechniquesCell biologyMice03 medical and health sciences0302 clinical medicineAnimals Newbornddc:570cytology [Neurons]methods [Cell Culture Techniques]Animalscytology [Oligodendroglia]Cells Cultured030217 neurology & neurosurgeryCell Proliferation030304 developmental biology
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Amplicon-based high-throughput pooled sequencing identifies mutations in CYP7B1 and SPG7 in sporadic spastic paraplegia patients

2011

Hereditary spastic paraplegia (HSP) is a neurodegenerative disorder defined clinically by progressive lower limb spasticity and weakness. HSP is a genetically highly heterogeneous condition with at least 46 gene loci identified so far, involving X-linked, autosomal recessive (AR) and autosomal dominant inheritance. For correct diagnosis, molecular testing is essential because clinical parameters by themselves are not reliable to differentiate HSP forms. The purpose of this study was to establish amplicon-based high-throughput genotyping for AR-HSP. A sample of 187 index cases with apparently sporadic or recessive spastic paraplegia were analyzed by applying an array-based amplification stra…

methods [High-Throughput Nucleotide Sequencing]GenotypeHereditary spastic paraplegiaDNA Mutational AnalysisMolecular Sequence DataSPG7 protein humanCytochrome P450 Family 7diagnosis [Paraplegia]Biologymedicine.disease_causegenetics [Paraplegia]Cohort Studiesgenetics [Metalloendopeptidases]03 medical and health sciences0302 clinical medicineGenetic variationGenotypeGeneticsmedicineHumansddc:610Genetic TestingGenotypingGenetics (clinical)CYP7B1 protein human030304 developmental biologyGenetic testingParaplegiaGenetics0303 health sciencesMutationBase SequenceParapleginmedicine.diagnostic_testgenetics [Steroid Hydroxylases]Genetic VariationHigh-Throughput Nucleotide SequencingMetalloendopeptidasesmethods [DNA Mutational Analysis]Ampliconmedicine.diseasegenetics [Genetic Variation]3. Good healthMutationSteroid HydroxylasesATPases Associated with Diverse Cellular Activities030217 neurology & neurosurgeryClinical Genetics
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Infective Endocarditis: Preliminary Results of a Cohort Study in the Southern Italian Population.

2020

Background Infective endocarditis (IE) is an uncommon disease with an involved interplay of clinical and surgical team management. We aimed to define diagnosis parameters and delineate in-hospital management in patients with IE admitted in a tertiary hospital of Southern Italian. Materials and methods Fifty-six consecutive patients (42 males, 14 females; age range: 34-85 years) admitted for IE in the Infectious Diseases, Cardiac Surgery, and Cardiology units, between January 2011 and August 2017, were enrolled. Demographic data, mortality, comorbidities, specimen type, microscopy results, special histological staining performed, and antimicrobial therapy were collected and analyzed. Any com…

microorganismmedicine.medical_specialtyArterial embolismcomplicationsCardiologyunivariate analysicandida endocarditi030204 cardiovascular system & hematologyendocarditi03 medical and health sciences0302 clinical medicinematlabInternal medicinemedicinegenderEndocarditisBlood cultureadult cardiac surgerymicroorganismsSurgical teamUnivariate analysismedicine.diagnostic_testbusiness.industryGeneral Engineeringmedicine.diseaseCardiac surgeryunivariate analysiscandida endocarditismultivariate analysisInfective endocarditismulti-drug resistant bacteriaendocarditisbusiness030217 neurology & neurosurgeryCohort studyCureus
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Walking Stroop carpet: an innovative dual-task concept for detecting cognitive impairment

2013

A Perrochon,1,2,5 G Kemoun,1,2 E Watelain,3,4 A Berthoz51ISIS, Research Institute on Handicap and Aging, Paris; 2Université de Poitiers, Laboratoire Mobilité, Vieillissement et Exercice (MOVE), EA 6314, 3Université de Valenciennes, LAMIH, UMR CNRS 8201, 4Université Sud Toulon Var, HandiBio, EA 4322, La Garde, 5LPPA, UMR CNRS 7152, Collège de France, Paris, FranceBackground: Several studies have reported the potential value of the dual-task concept during locomotion in clinical evaluation because cognitive decline is strongly associated with gait abnormalities. However, current dual-task tests appear to be insufficient for early diagnosi…

mild cognitive impairment.AdultMalemedicine.medical_specialty[SDV]Life Sciences [q-bio]Neuropsychological TestsSensitivity and Specificitybehavioral disciplines and activities050105 experimental psychologyDevelopmental psychologyTask (project management)03 medical and health scienceswalking0302 clinical medicineGait (human)Physical medicine and rehabilitationmild cognitive impairmentmedicineCluster AnalysisHumans0501 psychology and cognitive sciencesCognitive declineCognitive impairmentdual-taskGait Disorders NeurologicComputingMilieux_MISCELLANEOUSAgedOriginal ResearchAged 80 and overAnalysis of Variancestroop[SCCO.NEUR]Cognitive science/Neuroscience05 social sciencesRC952-954.6[SDV.NEU.SC]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/Cognitive SciencesGeneral MedicineDUAL (cognitive architecture)Middle AgedGeriatricsClinical Interventions in AgingFemaleGeriatrics and GerontologyPsychologyCognition DisordersClinical evaluation030217 neurology & neurosurgeryPsychomotor Performancepsychological phenomena and processesStroop effect
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How amniotic fluid shapes early odor-guided responses to colostrum and milk (and more)

2016

Revue; Among the multiple transitions that characterize mammalian development, birth certainly is the most abrupt and spectacular. It is also the most challenging for newborns when one considers their high mortality rates in any mammalian species. Although evolution has selected newborn organisms that possess the physiological and behavioral keys leading to adapt to this harsh psychobiological challenge, their capabilities cannot be considered separately from the maternal organism and the environment she creates. Mammalian females provide indeed passive (physiological) and/or active (behavioral) assistance to complement and boost their offspring’s capacities. One notable strategy of mammali…

milkAmniotic fluidflavor learningOffspring[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionPhysiologyamniotic fluidContext (language use)Sensory system[ SCCO.PSYC ] Cognitive science/PsychologyOlfactionBiology03 medical and health sciences0302 clinical medicineOdorcolostrum030225 pediatricsImmunology[SCCO.PSYC]Cognitive science/PsychologyColostrumtransnatal[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition030217 neurology & neurosurgeryOrganismperinatal
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Brain Opioid Activity and Oxidative Injury: Different Molecular Scenarios Connecting Celiac Disease and Autistic Spectrum Disorder

2020

Celiac Disease (CD) is an immune-mediated disease triggered by the ingestion of wheat gliadin and related prolamins from other cereals, such as barley and rye. Immunity against these cereal-derived proteins is mediated by pro-inflammatory cytokines produced by both innate and adaptive system response in individuals unable to adequately digest them. Peptides generated in this condition are absorbed across the gut barrier, which in these patients is characterized by the deregulation of its permeability. Here, we discuss a possible correlation between CD and Autistic Spectrum Disorder (ASD) pathogenesis. ASD can be induced by an excessive and inappropriate brain opioid activity during the neon…

mitochondrial damageDiseaseReviewNeurotransmissionmedicine.disease_causelcsh:RC321-571Pathogenesis03 medical and health sciences0302 clinical medicineAutistic Spectrum Disordermedicineoxidative stressReceptorlcsh:Neurosciences. Biological psychiatry. Neuropsychiatry030304 developmental biologyEndogenous opioid0303 health sciencesbiologybusiness.industryGeneral Neurosciencefood and beveragesopioidsOpioidImmunologybiology.proteinGliadinbusiness030217 neurology & neurosurgeryOxidative stressceliac diseasemedicine.drugBrain Sciences
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Effects of Transcranial Direct Current Stimulation on Baseline and Slope of Prefrontal Cortex Hemodynamics During a Spatial Working Memory Task

2020

Background: Transcranial direct current stimulation (tDCS) has been shown to be an inexpensive, safe, and effective way of augmenting a variety of cognitive abilities. Relatively recent advances in neuroimaging technology have provided the ability to measure brain activity concurrently during active brain stimulation rather than after stimulation. The effects on brain activity elicited by tDCS during active tDCS reported by initial studies have been somewhat conflicted and seemingly dependent on whether a behavioral improvement was observed. Objective: The current study set out to address questions regarding behavioral change, within and between-participant designs as well as differentiatin…

mixed modelsBrain activity and meditationmedicine.medical_treatmentfNIRSSpatial memory050105 experimental psychologytDCSworking memorylcsh:RC321-57103 medical and health sciencesBehavioral Neuroscience0302 clinical medicineMedicine0501 psychology and cognitive sciencesPrefrontal cortexlcsh:Neurosciences. Biological psychiatry. NeuropsychiatryBiological PsychiatryOriginal ResearchTranscranial direct-current stimulationbusiness.industryWorking memory05 social sciencesHuman NeuroscienceDorsolateral prefrontal cortexPsychiatry and Mental healthNeuropsychology and Physiological Psychologymedicine.anatomical_structureNeurologyBrain stimulationFunctional near-infrared spectroscopybusinessneural efficiencyNeuroscience030217 neurology & neurosurgeryFrontiers in Human Neuroscience
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Myostatin/activin blocking combined with exercise reconditions skeletal muscle expression profile of mdx mice

2015

Duchenne Muscular Dystrophy is characterized by muscle wasting and decreased aerobic metabolism. Exercise and blocking of myostatin/activin signaling may independently or combined counteract muscle wasting and dystrophies. The effects of myostatin/activin blocking using soluble activin receptor-Fc (sActRIIB-Fc) administration and wheel running were tested alone or in combination for seven weeks in dystrophic mdx mice. Expression microarray analysis revealed decreased aerobic metabolism in the gastrocnemius muscle of mdx mice compared to healthy mice. This was not due to reduced home-cage physical activity, and was further downregulated upon sActRIIB-Fc treatment in enlarged muscles. However…

muscular dystrophymedicine.medical_specialtyDuchenne muscular dystrophyActivin Receptors Type IIRecombinant Fusion Proteinsphysical activityMyostatinBiologyta3111BiochemistryMuscle hypertrophy03 medical and health sciencesGastrocnemius muscleMice0302 clinical medicineEndocrinologyoxidative metabolismInternal medicinePhysical Conditioning AnimalGene expressionmedicineSTAT5 Transcription FactorAnimalsmuscle hypertrophyMuscular dystrophyPhosphorylationta315Muscle SkeletalMolecular BiologyWasting030304 developmental biologyInhibin-beta Subunits0303 health sciencesPhysical activitySkeletal muscleMyostatinmusculoskeletal systemmedicine.diseaseMuscular dystrophymRNA profilingEndocrinologymedicine.anatomical_structurebiology.proteinMice Inbred mdxOxidative metabolismMuscle hypertrophymedicine.symptom030217 neurology & neurosurgery
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Posttraumatic Propofol Neurotoxicity Is Mediated via the Pro–Brain-Derived Neurotrophic Factor-p75 Neurotrophin Receptor Pathway in Adult Mice*

2016

Objectives:The gamma-aminobutyric acid modulator propofol induces neuronal cell death in healthy immature brains by unbalancing neurotrophin homeostasis via p75 neurotrophin receptor signaling. In adulthood, p75 neurotrophin receptor becomes down-regulated and propofol loses its neurotoxic effect. H

musculoskeletal diseases0301 basic medicineBrain-derived neurotrophic factorProgrammed cell deathbiologybusiness.industryNeurotoxicityCaspase 3PharmacologyCritical Care and Intensive Care Medicinemedicine.disease03 medical and health sciences030104 developmental biology0302 clinical medicinenervous systemAnesthesiamedicinebiology.proteinLow-affinity nerve growth factor receptorReceptorbusiness030217 neurology & neurosurgeryHomeostasisNeurotrophinCritical Care Medicine
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