Search results for " protein"

showing 10 items of 12147 documents

Molecular Mechanisms of the Blockage of Glioblastoma Motility

2021

Glioblastoma (GBM) is the most common and lethal brain tumor. GBM has a remarkable degree of motility and is able to infiltrate the healthy brain. In order to perform a rationale-based drug-repositioning study, we have used known inhibitors of two small Rho GTPases, Rac1 and Cdc42, which are upregulated in GBM and are involved in the signaling processes underlying the orchestration of the cytoskeleton and cellular motility. The selected inhibitors (R-ketorolac and ML141 for Cdc42 and R-ketorolac and EHT 1864 for Rac1) have been successfully employed to reduce the infiltration propensity of GBM in live cell imaging studies. Complementarily, all-atom simulations have elucidated the molecular …

rac1 GTP-Binding Proteinrho GTP-Binding ProteinsGeneral Chemical EngineeringBrain tumorMotilityRAC1CDC42Library and Information SciencesBiologySettore BIO/09 - FisiologiaMicrotubules01 natural sciencesDownregulation and upregulationLive cell imaging0103 physical sciencesmedicineHumanscdc42 GTP-Binding Protein010304 chemical physicsDrug discoveryCancerGeneral Chemistrymedicine.disease0104 chemical sciencesComputer Science Applications010404 medicinal & biomolecular chemistrySettore CHIM/03 - Chimica Generale E InorganicaCancer researchGlioblastomaJournal of Chemical Information and Modeling
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Antitumor Effect of Cabozantinib in Bone Metastatic Models of Renal Cell Carcinoma

2021

Background: The presence of bone metastases in renal cell carcinoma (RCC) negatively affects patients’ survival. Data from clinical trials has highlighted a significant benefit of cabozantinib in bone metastatic RCC patients. Here, we evaluated the antitumor effect of cabozantinib in coculture models of renal cell carcinoma (RCC) and osteoblasts (OBs) to investigate whether and how its antiproliferative activity is influenced by OBs. Methods: Bone/RCC models were generated, coculturing green fluorescent protein (GFP)-tagged Caki-1 and 786-O cells with human primary OBs in a “cell–cell contact” system. RCC proliferation and the OB molecular profile were evaluated after the cabozantinib treat…

renal cell carcinomaCabozantinibQH301-705.5Biologyurologic and male genital diseasesGeneral Biochemistry Genetics and Molecular BiologyArticleGreen fluorescent proteinchemistry.chemical_compoundCabozantinib; Osteoblasts; Renal cell carcinomaRenal cell carcinomacabozantinibmedicineBiology (General)Antitumor activityGeneral Immunology and MicrobiologyCell growthOsteoblastosteoblastsmedicine.diseasefemale genital diseases and pregnancy complicationsInhibitory potencychemistryCancer researchMolecular ProfileGeneral Agricultural and Biological SciencesBiology
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Photoinduced lethal and sublethal toxicity of retene, a polycyclic aromatic hydrocarbon derived from resin acid, to coregonid larvae

2004

A comparative investigation on the acute phototoxicity of retene to vendace (Coregonus albula) and whitefish (C. lavaretus), both having pelagial larvae in spring, was conducted. To test the concept of early warning of sublethal biomarkers in relation to lethality to posthatch stages, we examined the effects of ultraviolet-B (UV-B) and retene on the levels of cytochrome P4501A (CYP1A) and heat shock protein 70 (HSP70) by exposing the animals to elevated levels of these factors for 48 and 72 h, respectively. Whereas UV-B and retene on their own were not lethal, simultaneous retene and UV-B exposure caused very high mortality to both species. The median lethal concentration (LC50; i.e., the c…

reteneanimal structuresPhotochemistryUltraviolet RaysHealth Toxicology and MutagenesisBiologyMedian lethal doseToxicologyLethal Dose 50chemistry.chemical_compoundphototoxicityfoodCoregonus lavaretusEnvironmental ChemistryCoregonus albulaAnimalsHSP70 Heat-Shock Proteinsheat shock protein 70SalmonidaeRetenefood.dishUV-BPhenanthrenesbiology.organism_classificationMolecular biologyHsp70cytochrome P4501AchemistryLarvaToxicityPhototoxicityBiomarkersSalmonidaeWater Pollutants Chemical
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Regulation of the HTRA2 Protease Activity by an Inhibitory Antibody-Derived Peptide Ligand and the Influence on HTRA2-Specific Protein Interaction Ne…

2021

The mitochondrial serine protease HTRA2 has many versatile biological functions ranging from being an important regulator of apoptosis to being an essential component for neuronal cell survival and mitochondrial homeostasis. Loss of HTRA2 protease function is known to cause neurodegeneration, whereas overactivation of its proteolytic function is associated with cell death and inflammation. In accordance with this, our group verified in a recent study that the synthetic peptide ASGYTFTNYGLSWVR, encoding the hypervariable sequence part of an antibody, showed a high affinity for the target protein HTRA2 and triggered neuroprotection in an in vitro organ culture model for glaucoma. To unravel t…

retinaImmunoprecipitationQH301-705.5medicine.medical_treatmentMedicine (miscellaneous)PeptideAggrephagyNeuroprotectioninteraction partnersGeneral Biochemistry Genetics and Molecular BiologyArticlemedicineBiology (General)mass spectrometrychemistry.chemical_classificationProteaseHTRA2Neurodegenerationco-immunoprecipitationmedicine.diseaseProtease inhibitor (biology)Cell biologychemistryneuroprotectionTarget proteinmedicine.drugBiomedicines
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Dietary fatty acids in the retina: beyond DHA, is EPA the underestimate intermediate?

2016

Dietary fatty acids in the retina: beyond DHA, is EPA the underestimate intermediate?. 12. congress of the international society for the study of fatty acids and lipids

retina[SDV.MHEP] Life Sciences [q-bio]/Human health and pathology[ SDV.AEN ] Life Sciences [q-bio]/Food and Nutritioneducationagingsocial sciences[SDV.AEN] Life Sciences [q-bio]/Food and Nutrition[SDV.MHEP.OS] Life Sciences [q-bio]/Human health and pathology/Sensory Organs[ SDV.MHEP ] Life Sciences [q-bio]/Human health and pathology[ SDV.MHEP.OS ] Life Sciences [q-bio]/Human health and pathology/Sensory Organslipids (amino acids peptides and proteins)fatty acid[SDV.MHEP.OS]Life Sciences [q-bio]/Human health and pathology/Sensory Organsdiet[SDV.AEN]Life Sciences [q-bio]/Food and Nutritiongeographic locationshealth care economics and organizations[SDV.MHEP]Life Sciences [q-bio]/Human health and pathology
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Effect of dietary omega-3 and omega-6 fatty acids on IOP elevation, electroretinographic changes and retinal ganglion cell loss in a laser-induced ra…

2008

International audience; Purpose:To test the efficacy of dietary omega-3 and omega-6 fatty acids in a rat model of glaucoma induced by laser photocoagulation.Methods:Male Sprague Dawley rats were fed for 3 months with a diet containing either: 1) 17% of omega-3 fatty acids (10% EPA + 7% DHA), 2) 10% of omega-6 fatty acids (as GLA), or 3) a combination of both omega-3 and omega-6 fatty acids (10% EPA + 7% DHA + 10% GLA), by comparison with a control group of animals fed with a standard diet deprived of EPA, DHA and GLA (n=10 in each group). After 3 months of diet, glaucoma was induced in one eye of each animal by laser photocoagulation (532nm) of the episcleral veins, the trabeculum and the l…

retinagenetic structures[SPI.GPROC] Engineering Sciences [physics]/Chemical and Process Engineering[SDV.IDA] Life Sciences [q-bio]/Food engineeringPOLYUNSATURED FATTY ACIDeye diseasesGANGLION CELLSglial fibrillary acid proteinELECTRORETINOGRAPHYNON CLINICAL[SDV.IDA]Life Sciences [q-bio]/Food engineeringarachidonic acid[SPI.GPROC]Engineering Sciences [physics]/Chemical and Process EngineeringPGE2sense organs[SDV.MHEP.OS]Life Sciences [q-bio]/Human health and pathology/Sensory Organs[SDV.AEN]Life Sciences [q-bio]/Food and NutritionComputingMilieux_MISCELLANEOUSintraocular pressureLIPIDS
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Low levels of both xanthine dehydrogenase and of cellular retinol binding protein are responsible for retinoic acid deficiency in malignant human mam…

2009

The seeming impairment of retinoid metabolism in human breast tumor cells has been attributed to the lower expression of cellular retinol binding proteins (CRBPs), of alcohol/retinol dehydrogenases, or aldehyde/retinaldehyde dehydrogenases. In a previous study we indicated that xanthine dehydrogenase (XDH) is able to oxidize actively both all-trans-retinol (t-ROL) bound to the CRBP (holo-CRBP) and all-trans-retinaldehyde (t-RAL) to all-trans-retinoic acid (t-RA) in human mammary epithelial cells (HMEC). Since both XDH and CRBP are required for the biosynthesis of t-RA, we have inspected their bioavailability in both estrogen-responsive and nonresponsive human mammary epithelial cancer cells…

retinoic acid biosynthesis tumor mammary cellsXanthine DehydrogenaseCellular differentiationRetinoic acidBreast NeoplasmsTretinoinBiologyGeneral Biochemistry Genetics and Molecular BiologyCell Linechemistry.chemical_compoundHistory and Philosophy of ScienceBiosynthesisCell Line TumorSettore BIO/10 - BiochimicaHumansMammary Glands HumanRadiometryChromatography High Pressure LiquidGeneral NeuroscienceRetinolRetinol-Binding Proteins CellularMolecular biologyRetinol binding proteinBiochemistrychemistryXanthine dehydrogenaseCell cultureCancer cell
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The Clinical Value of Autoantibodies in Rheumatoid Arthritis

2018

Rheumatoid arthritis (RA) is a highly heterogeneous syndrome in terms of clinical presentation, progression, and response to therapy. In such a complicated context, the identification of disease-related biomarkers would be undoubtedly helpful in assisting tailored approaches for every patient. Despite remarkable efforts, however, progress in new biomarker development and validation is dramatically slow. At present, none of the candidate genetic, cellular, or molecular biomarker has yet surpassed the clinical value of RA-specific autoantibodies, including rheumatoid factor (RF) and anti-citrullinated protein autoantibodies (ACPA). Rather, recent years have witnessed significant advancements …

rheumatoid arthritis0301 basic medicineResponse to therapyautoantibodiesMini ReviewContext (language use)Bioinformaticsrheumatoid factor03 medical and health sciencesremission0302 clinical medicinemedicineRheumatoid factoranti-citrullinated protein antibodies030203 arthritis & rheumatologylcsh:R5-920biologybusiness.industryAutoantibodyAnti–citrullinated protein antibodyGeneral Medicinemedicine.disease030104 developmental biologyRheumatoid arthritisbiology.proteinClinical valueMedicineBiomarker (medicine)lcsh:Medicine (General)businessFrontiers in Medicine
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Etanercept maintains the clinical benefit achieved by infliximab in patients with rheumatoid arthritis who discontinued infliximab because of side ef…

2007

Objective: To evaluate the efficacy of switching to etanercept treatment in patients with rheumatoid arthritis who already responded to infliximab, but presented side effects. Methods: Charts of 553 patients with rheumatoid arthritis were retrospectively reviewed to select patients who responded to the treatment with infliximab and switched to etanercept because of occurrence of adverse effects. Clinical data were gathered during 24 weeks of etanercept treatment and for the same period of infliximab treatment before infliximab was stopped. Disease Activity Score computed on 44 joints (DAS-44), erythrocyte sedimentation rate (ESR) 1st hour, Visual Analogue Scale (VAS) of pain, Health Assessm…

rheumatoid arthritisMaleConcise ReportArthritisGastroenterologyReceptors Tumor Necrosis FactorEtanerceptEtanerceptArthritis Rheumatoidimmune system diseasesImmunology and AllergyHealth Status Indicatorsskin and connective tissue diseasesmedicine.diagnostic_testbiologyAntibodies MonoclonalMiddle AgedC-Reactive ProteinTreatment OutcomeBiologic agents; etanercept; infliximab; infusion reactionRheumatoid arthritisErythrocyte sedimentation rateAntirheumatic AgentsFemalemedicine.drugmusculoskeletal diseasesAdultmedicine.medical_specialtyVisual analogue scaleImmunologyBlood Sedimentationinfusion reactionGeneral Biochemistry Genetics and Molecular BiologyRheumatologyInternal medicinemedicineHumansAdverse effectRetrospective StudiesChi-Square Distributionbusiness.industryTumor Necrosis Factor-alphaC-reactive proteinetanercept; infliximab; rheumatoid arthritismedicine.diseaseInfliximabInfliximabSurgeryBiologic agentsstomatognathic diseasesImmunoglobulin Gbiology.proteinbusinessinfliximabetanercept
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Rho GTPases Are Involved in the Regulation of NF-κB by Genotoxic Stress

2001

A common cellular response to genotoxic agents and inflammatory cytokines is the activation of NF-kappaB. Here, we addressed the question of whether small GTPases of the Rho family are involved in the stimulation of NF-kappaB signaling by genotoxic agents or TNFalpha in HeLa cells. Inhibition of isoprenylation of Rho proteins by use of the HMG-CoA reductase inhibitor lovastatin attenuated UV-, doxorubicin-, and TNFalpha-induced degradation of IkappaBalpha as well as drug-stimulated DNA binding activity of NF-kappaB. Furthermore, NF-kappaB-regulated gene expression stimulated by either UV irradiation or treatment with TNFalpha was abrogated by lovastatin pretreatment. This indicates that iso…

rho GTP-Binding ProteinsBacterial ToxinsClostridium difficile toxin BGenotoxic StressGTPaseBiologyProinflammatory cytokinechemistry.chemical_compoundBacterial ProteinsNF-KappaB Inhibitor alphamedicineHumansLovastatinTumor Necrosis Factor-alphaNF-kappa BNF-kappa B p50 SubunitNF-κBCell BiologyCell biologyDNA-Binding ProteinsIκBαchemistryDoxorubicinI-kappa B ProteinsTumor necrosis factor alphaLovastatinHeLa CellsSignal Transductionmedicine.drugExperimental Cell Research
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