Search results for " trip"

showing 10 items of 535 documents

Eel ATPase activity as biomarker of thiobencarb exposure

2003

Abstract European eels ( Anguilla anguilla ) were exposed to a sublethal thiobencarb concentration of 0.22 mg/L in a flow-through system for 96 h. Mg 2+ and Na + –K + adenosine triphosphatase (ATPase) activities were evaluated in gill and muscle tissues at 2, 12, 24, 48, 72, and 96 h of thiobencarb exposure. Gill ATPase activities were rapidly inhibited from 2 h of contact onward. Highest inhibition was registered for Na + , K + -ATPase (85%) from 2 to 12 h. Both Mg 2+ and total ATPase were inhibited (>73%) during the first hours of toxicant exposure. At the end of the exposure period (96 h) ATPase activities were still different from those of the controls (>50%). Significant inhibition was…

GillsMuscle tissueGillmedicine.medical_specialtyHealth Toxicology and MutagenesisATPasechemistry.chemical_compoundThiocarbamatesAnguillidaeInternal medicinemedicineAnimalsTissue DistributionMuscle SkeletalAdenosine Triphosphataseschemistry.chemical_classificationbiologyHerbicidesPublic Health Environmental and Occupational HealthEnvironmental ExposureGeneral MedicineAnguillabiology.organism_classificationPollutionmedicine.anatomical_structureEnzymeEndocrinologychemistryBiochemistryEnzyme inhibitorToxicitybiology.proteinBiomarkersWater Pollutants ChemicalToxicantEcotoxicology and Environmental Safety
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Effects of primary- and secondary-treated bleached kraft mill effluents on the immune system and physiological parameters of roach.

2000

The present study was designed to examine, whether, effluents from a modern pulp and paper mill using elemental chlorine-free/total chlorine-free (ECF/TCF) bleaching, exert effects on the immune system of fish and, in addition, to relate these findings to physiological parameters known to be affected by bleached kraft-mill effluents (BKME). Roach (Rutilus rutilus) were exposed in laboratory conditions to primary- or secondary-treated effluent from a pulp and paper mill. In order to study their capability to respond to foreign antigens they were immunised with bovine gamma-globulin (BGG) prior to exposure. The number of anti-BGG antibody-secreting cells (ASC) and the number of immunoglobulin…

GillsPaperHydrocortisoneNeutrophilsHealth Toxicology and MutagenesisIndustrial WasteSpleenEnzyme-Linked Immunosorbent AssayFresh WaterAquatic Scienceengineering.materialAndrologyImmune systemAntigenCell MovementmedicineCytochrome P-450 CYP1A1AnimalsLymphocytesRespiratory BurstAdenosine Triphosphatasesbiologybusiness.industryPulp (paper)FishesPaper millWater-Electrolyte Balancebiology.organism_classificationLiver Glycogenmedicine.anatomical_structureImmunoglobulin MImmune SystemImmunologyengineeringOsmoregulationbiology.proteinCarbohydrate MetabolismRutilusAntibodyChlorinebusinessWater Pollutants ChemicalAquatic toxicology (Amsterdam, Netherlands)
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The adenine nucleotide content of rat liver during infusions of carbohydrates and polyols

1972

Injection of large doses of fructose, sorbitol, or a mixture of glucose, fructose and xylitol in rats causes a drop of liver ATP, total adenine nucleotides and Pi and a rise of AMP, which is in agreement with data from the literature. These changes are considered as a transient disturbance of homeostasis by compounds which are rapidly phosporylated in the liver. This is confirmed by the fact that during continuous infusion of these and other compounds at doses of 1,5 g · kg−1 · h−1 there was no such change. It is concluded that infusions of fructose or of the other carbohydrates tested with rates not exceeding those recommended for parenteral nutrition (0,5 g · kg−1 · h−1) are not likely to…

GlycerolMaleParenteral NutritionTime FactorsMedicine (miscellaneous)FructoseXylitolBiochemistryPhosphateschemistry.chemical_compoundAdenosine TriphosphateAdenine nucleotidePiAnimalsHomeostasisSorbitolXylitolAdenine NucleotidesRats Inbred StrainsFructoseAdenosine MonophosphateRatsAdenosine DiphosphateDrug CombinationsGlucoseParenteral nutritionLiverchemistryBiochemistryRat liverInjections IntravenousSorbitolHomeostasisFood ScienceZeitschrift für Ernährungswissenschaft
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Inhibition of gluconeogenesis by extracellular ATP in isolated rat hepatocytes.

1991

The aim of this study was to determine the effect of externally added ATP on gluconeogenesis by isolated hepatocytes from starved rats. High concentrations of extracellular ATP inhibited gluconeogenesis from lactate and pyruvate but not from glycerol or fructose. This inhibition was associated with an increase in intracellular adenosine contents. ADP, AMP, or adenosine but not guanosine 5'triphosphate, inosine 5' triphosphate, or adenine also inhibited gluconeogenesis. alpha, beta-Methylene-ATP, a nonmetabolizable structural analogue of ATP, did not affect the rate of gluconeogenesis. Intracellular ATP levels were increased by externally added ATP or adenosine, but ATP-to-ADP ratios in the…

GlycerolMalePhysiologyFructoseBiologyAdenosine TriphosphateAdenine nucleotidePhysiology (medical)Pyruvic AcidmedicineExtracellularAnimalsGlycolysisLactic AcidPyruvatesChemiosmosisGluconeogenesisRats Inbred StrainsMetabolismAdenosineRatsAdenosine DiphosphateBiochemistryGluconeogenesisLiverLactatesPhosphoenolpyruvate carboxykinasemedicine.drugThe American journal of physiology
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Design, Synthesis and Biological Evaluation of 7-Chloro-9

2019

Glycogen synthase kinase-3β (GSK-3β) represents a relevant drug target for the treatment of neurodegenerative pathologies including Alzheimer’s disease. We herein report on the optimization of a novel class of GSK-3β inhibitors based on the tofacitinib-derived screen hit 3-((3R,4R)-3-((7-chloro-9H-pyrimido[4,5-b]indol-4-yl)(methyl)amino)-4-methylpiperidin-1-yl)-3-oxopropanenitrile (1). We synthesized a series of 19 novel 7-chloro-9H-pyrimido[4,5-b]indole-based derivatives and studied their structure–activity relationships with focus on the cyanoacetyl piperidine moiety. We unveiled the crucial role of the nitrile group and its importance for the activity of this compound series. A successfu…

Glycogen synthase kinase-3βBinding SitesGlycogen Synthase Kinase 3 betatofacitinibDose-Response Relationship DrugMolecular Structurekinase inhibitorMolecular Conformationprotein kinaseChemistry Techniques SyntheticMolecular Dynamics SimulationArticle7-chloro-9H-pyrimido[45-b]indoleEnzyme ActivationMolecular Docking SimulationStructure-Activity RelationshipAdenosine TriphosphateDrug DesignHumansEnzyme InhibitorsHydrophobic and Hydrophilic InteractionsProtein Kinase InhibitorsProtein BindingMolecules (Basel, Switzerland)
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Pivotal Advance: Up-regulation of acetylcholine synthesis and paracrine cholinergic signaling in intravascular transplant leukocytes during rejection…

2009

Abstract A new role and source of the old mediator acetylcholine is described, which is produced by graft monocytes and attenuates monocytic ATP-signaling. During acute rejection, large numbers of leukocytes accumulate in the blood vessels of experimental renal allografts. About 70% of them are activated, cytotoxic monocytes that appear to be involved in allograft destruction. ACh exerts anti-inflammatory effects upon monocytes/macrophages and has been proposed to be a key player in neuroimmunological interactions. Its short half-life, however, makes it unlikely that neuronal ACh affects blood leukocytes. Renal transplantation was performed in the allogeneic DA to LEW and in the isogeneic L…

Graft RejectionPathologymedicine.medical_specialtyIsograftImmunologyBiologyReceptors NicotinicParacrine signallingAdenosine TriphosphateIn vivoParacrine CommunicationmedicineImmunology and AllergyCytotoxic T cellAnimalsTransplantation HomologousLymphocytesCation Transport ProteinsMonocyteCell BiologyKidney TransplantationAcetylcholineRatsUp-RegulationTransplantationTransplantation Isogeneicmedicine.anatomical_structureRats Inbred LewImmunohistochemistryCholinergicSignal TransductionJournal of leukocyte biology
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Co-transmitter mediated facilitation by sympathetic nerve stimulation of evoked acetylcholine release from the rabbit perfused atria preparation.

1995

Rabbit atria were isolated with the extrinsic right sympathetic and vagus nerves attached and perfused with Tyrode solution. Acetylcholine overflow was determined after labelling of the transmitter stores with [14C]choline and fractionation of the radioactivity on cation exchange columns. Sympathetic nerve stimulation (SNS, 2 Hz, 3 min) carried out together with vagus nerve stimulation (VNS, 2 Hz, 3 min), but each SNS pulse preceding a vagal one by 19 ms, caused a facilitation of acetylcholine overflow of about 60% versus independent controls in the absence of SNS. Antagonists of putative neurotransmitters were tested to find out the prejunctional mediator involved in the facilitation. The …

Guanethidinemedicine.medical_specialtyanimal structuresAdenosineReserpineSympathetic Nervous SystemPurinergic Antagonistsmedicine.medical_treatmentStimulationSympathetic nerveIn Vitro TechniquesSynaptic TransmissionCholinechemistry.chemical_compoundAdenosine TriphosphateInternal medicinemedicineCholineAnimalsPharmacologyNeurotransmitter AgentsPulse (signal processing)MyocardiumHeartGeneral MedicineCo transmitterAcetylcholineElectric StimulationPerfusionEndocrinologynervous systemchemistryFacilitationRabbitsAcetylcholineVagus nerve stimulationmedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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Closer to nature: an ATP-driven bioinspired catalytic oxidation process

2013

The capability of DNA to acquire enzyme-like properties has led to the emergence of the so-called DNAzyme field; herein, we take a further leap along this nature-inspired road, demonstrating that a template assembled synthetic G-quartet (TASQ) can act as a pre-catalyst for catalytic peroxidase-mimicking oxidation reactions, whatever its nature (guanine or guanosine-based G-quartets), in an ATP-dependent manner, thereby bringing this bioinspired TASQzyme process even closer to nature.

GuanineDeoxyribozymeGuanosineNanotechnology010402 general chemistry01 natural sciencesRedox[ CHIM ] Chemical SciencesCatalysisCatalysischemistry.chemical_compoundAdenosine TriphosphateMaterials Chemistry[CHIM]Chemical SciencesComputingMilieux_MISCELLANEOUS010405 organic chemistryMetals and AlloysDNA CatalyticGeneral Chemistry[CHIM.CATA]Chemical Sciences/Catalysis0104 chemical sciencesSurfaces Coatings and FilmsElectronic Optical and Magnetic MaterialsG-QuadruplexeschemistryCatalytic oxidationScientific methodCeramics and CompositesOxidation-Reduction
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H89 enhances the sensitivity of cancer cells to glyceryl trinitrate through a purinergic receptor-dependent pathway

2014

// Marion Cortier 1, 2, 3 , Rahamata Boina-Ali 1, 2, 3 , Cindy Racoeur 1, 2, 3 , Catherine Paul 1, 2, 3 , Eric Solary 2, 4, 5 , Jean-Francois Jeannin 1, 2, 3 , Ali Bettaieb 1, 2, 3 1 EPHE, Tumor Immunology and Immunotherapy Laboratory, Dijon, F-21000, France 2 Inserm U866, Dijon, F-21000, France 3 EA7269, University of Burgundy, Dijon, F-21000, France 4 Inserm UMR1009, Gustave Roussy Institute, Villejuif F-94805, France 5 University Paris-Sud, Faculty of Medicine, Le Kremlin-Bicetre, F-94800, France Correspondence to: Ali Bettaieb, e-mail: ali.bettaieb@u-bourgogne.fr Keywords: H89, GTN, cancer, purinergic receptors, cGMP Received: October 08, 2014      Accepted: January 09, 2015      Publis…

H89SuraminApoptosisPharmacologyBiologyNitric OxideTransfectionNitric oxideMiceNitroglycerinReceptors Purinergic P2Y1chemistry.chemical_compoundAdenosine TriphosphateCell Line TumorNeoplasmspurinergic receptorsmedicineAnimalsHumanscancerCytotoxic T cellReceptorProtein Kinase InhibitorsMembrane Potential MitochondrialSulfonamidesReceptors Purinergic P2Gene Expression ProfilingPurinergic receptorReceptors PurinergicDrug SynergismOligonucleotides AntisenseIsoquinolinescGMPOncologychemistryApoptosisColonic NeoplasmsCancer cellcardiovascular systemSignal transductionReactive Oxygen SpeciesGTNReceptors Purinergic P2X3circulatory and respiratory physiologySignal TransductionResearch Papermedicine.drugOncotarget
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Hepatitis B virus maturation is sensitive to functional inhibition of ESCRT-III, Vps4, and gamma 2-adaptin.

2007

ABSTRACT Hepatitis B virus (HBV) is an enveloped DNA virus that presumably buds at intracellular membranes of infected cells. HBV budding involves two endocytic host proteins, the ubiquitin-interacting adaptor γ2-adaptin and the Nedd4 ubiquitin ligase. Here, we demonstrate that HBV release also requires the cellular machinery that generates internal vesicles of multivesicular bodies (MVBs). In order to perturb the MVB machinery in HBV-replicating liver cells, we used ectopic expression of dominant-negative mutants of different MVB components, like the ESCRT-III complex-forming CHMP proteins and the Vps4 ATPases. Upon coexpression of mutated CHMP3, CHMP4B, or CHMP4C forms, as well as of ATPa…

Hepatitis B virusVacuolar Proton-Translocating ATPasesEndosomeImmunologyEndocytic cycleVesicular Transport Proteinsmacromolecular substancesEndosomesmedicine.disease_causeMicrobiologyESCRTVirusCell LineViral ProteinsVirologymedicineHumansAdaptor Protein Complex gamma SubunitsHepatitis B virusAdenosine TriphosphatasesMicroscopy ConfocalbiologyEndosomal Sorting Complexes Required for TransportVirus AssemblyDNA virusMolecular biologyUbiquitin ligaseCell biologyGenome Replication and Regulation of Viral Gene ExpressionMicroscopy FluorescenceInsect Sciencebiology.proteinHepatocytesATPases Associated with Diverse Cellular ActivitiesEctopic expressionJournal of virology
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