Search results for " trip"

showing 10 items of 535 documents

Possible reason for preferential damage to renal tubular epithelial cells evoked by amphotericin B

1996

An important determinant of nephrotoxicity, which is the major complication of long-term amphotericin B treatment, is dysfunction of distal tubular epithelial cells. The underlying cause for this rather selective damage to the cells is unknown. In the present investigation, it was shown that kidney epithelial cells were initially damaged by amphotericin B at concentrations of 2.5 to 10 micrograms/ml, as demonstrable by a dramatic drop in cellular K+ levels. Cells could recover from the initial toxic action of the polyene if they were kept in medium of neutral pH, and cellular K+ levels returned to normal after 6 h. However, the recovery mechanisms failed at lower pHs of 5.6 to 6.0. At low p…

medicine.medical_specialtyAntifungal AgentsLumen (anatomy)PharmacologyBiologyEpitheliumCell LineNephrotoxicitychemistry.chemical_compoundAdenosine TriphosphateAmphotericin BInternal medicineAmphotericin BLactate dehydrogenasemedicineAnimalsPharmacology (medical)Kidney Tubules DistalPharmacologyKidneyL-Lactate DehydrogenaseHydrogen-Ion ConcentrationMacaca mulattaIn vitroEpitheliumInfectious Diseasesmedicine.anatomical_structureEndocrinologychemistryToxicityPotassiumResearch Articlemedicine.drugAntimicrobial Agents and Chemotherapy
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Intestinal filtration as a consequence of increased mucosal hydraulic permeability

1980

Two mechanisms have been proposed to explain the secretory action of laxative compounds in the intestine: 1. increase of the intracellular amount of cyclic adenosine monophosphate due to stimulation of the adenylate cyclase system and 2. inhibition of intestinal transfer processes, in particular the Na,K-ATPase activated sodium absorption. In a set of in vivo and in vitro experiments in rat colon it could be demonstrated that dihydroxy bile acids (deoxycholate) and diphenolic laxatives (oxyphenisatin) enhance the hydraulic permeability of the mucosal tissue. The permeability changes take place--and there is good experimental evidence--at the zonulae occludentes which bind the epithelial cel…

medicine.medical_specialtyColonSodiummedicine.medical_treatmentHydrostatic pressureLaxativechemistry.chemical_elementStimulationPermeabilitychemistry.chemical_compoundInternal medicineDrug DiscoveryElectrochemistrymedicineStarling equationAnimalsCyclic adenosine monophosphateIntestinal MucosaGenetics (clinical)Adenosine TriphosphatasesTight junctionCatharticsSodiumOxyphenisatin AcetateGeneral MedicineRatsEndocrinologychemistryPotassiumBiophysicsMolecular MedicineFiltrationIntracellularDeoxycholic AcidKlinische Wochenschrift
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On the purinergic system in rat duodenum : existence of P1and P2receptors on the smooth muscle

1990

In rat duodenum, in vitro, in the presence of atropine and guanethidine, ATP administration caused a tetrodotoxin-insensitive relaxation followed by a rebound contraction. A similar response was obtained also after electrical field stimulation (EFS) of non-adrenergic, non-cholinergic (NANC) nerves. alpha, beta-methylene-TP and theophylline antagonized the response to ATP, but they failed to affect the noradrenaline- and EFS-induced relaxation. These results suggest that P1 and P2 receptors are present in rat duodenum, but their activation is not responsible for the inhibitor effects due to the NANC nerves.

medicine.medical_specialtyContraction (grammar)DuodenumPhysiologyMuscle RelaxationIn Vitro TechniquesBiologydigestive systemBiochemistryAdenosine TriphosphateTheophyllineInternal medicinemedicineAnimalsTheophyllineReceptorGuanethidinePurinergic receptorReceptors PurinergicMuscle SmoothElectric StimulationRatsAtropineElectrophysiologymedicine.anatomical_structureEndocrinologyDuodenummedicine.drugArchives Internationales de Physiologie et de Biochimie
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DNA-dependent DNA polymerase pattern in noninfected and herpesvirus infected rabbit kidney cells.

1973

In this paper we report on a DNA-dependent DNA polymerase produced in herpesvirus infected cells which is not present in virions. It differs from the polymerases of noninfected cells by its molecular weight as well as by its insensitivity to cytosine arabinoside triphosphate (ara-CTP).

medicine.medical_specialtyDNA polymerasevirusesDeoxyribonucleotidesKidneyTritiumchemistry.chemical_compoundMedical microbiologyAdenosine TriphosphateVirologyRabbit kidneymedicineAnimalsSimplexvirusheterocyclic compoundsPolymeraseCells CulturedbiologyCell-Free SystemCytarabineGeneral Medicinebiochemical phenomena metabolism and nutritionVirologyMolecular biologyMolecular WeightchemistryDNA Nucleotidyltransferasesbiology.proteinChromatography GelRabbitsArabinofuranosylcytosine triphosphateDNAArchiv fur die gesamte Virusforschung
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Adenosine triphosphate bioluminescence in intensive care units: Be careful with its use

2016

LETTER TO EDITOR

medicine.medical_specialtyEpidemiologyIntensive Care UnitMEDLINEInfection controlPilot Projects030501 epidemiologylaw.invention03 medical and health scienceschemistry.chemical_compoundAdenosine Triphosphate0302 clinical medicinelawIntensive careInfection controlMedicineBioluminescence030212 general & internal medicineIntensive care medicinebusiness.industryHealth PolicyPublic Health Environmental and Occupational HealthIntensive care unitDisinfectionIntensive Care UnitsInfectious Diseaseschemistry0305 other medical sciencebusinessAdenosine triphosphateAmerican Journal of Infection Control
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Inhibition of intracellular Ca2+ release by a Rho-kinase inhibitor for the treatment of ischemic damage in primary cultured rat hippocampal neurons.

2008

The effects of hydroxy fasudil, a specific Rho-kinase inhibitor, on behavior and brain neuronal activity in animal studies have been described previously. However, whether a Rho-kinase inhibitor can directly protect neurons against ischemic damage and the molecular mechanisms underlying these effects are poorly understood. The present work was designed to investigate the effect of hydroxy fasudil against oxygen-glucose deprivation (OGD) induced acute neuronal injury and the underlying mechanisms in vitro. Pretreatment with hydroxy fasudil at 5 and 10 microM could concentration-dependently improve cell viability and decrease Lactate dehydrogenase (LDH) level in extracellular solution of neur…

medicine.medical_specialtyExcitotoxicityIntracellular SpaceGlutamic AcidBiologymedicine.disease_causeNeuroprotectionHippocampusCalcium in biologyPotassium ChlorideRats Sprague-DawleyCalcium imagingAdenosine TriphosphateIschemiaInternal medicine1-(5-Isoquinolinesulfonyl)-2-MethylpiperazinemedicineAnimalsHypoxiaProtein Kinase InhibitorsCells CulturedPharmacologyNeuronsrho-Associated KinasesDose-Response Relationship DrugCalcium channelFasudilGlutamate receptorRatsEndocrinologyGlucoseRho kinase inhibitorCalciumEuropean journal of pharmacology
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Is there a critical tissue oxygen tension for bioenergetic status and cellular pH regulation in solid tumors?

1996

Bioenergetic and metabolic status have been correlated with tissue oxygenation in murine fibrosarcomas (FSaII) of varying sizes (44-600 mm3). Ratios of beta-nucleoside triphosphates to inorganic phosphate (beta NTP/P) and phosphocreatine to inorganic phosphate (PCr/P(i)) ratios derived from 31P nuclear magnetic resonance spectroscopy (NMR) were positively correlated to median tissue O2 tension (pO2) values using O2-sensitive needle electrodes. pH declined during growth with intracellular acidosis being evident in tumors350 mm3. Whereas lactic acid formation greatly contributed to this decline in small and medium-sized tumors, adenosine triphosphate (ATP) hydrolysis and slowing down of the a…

medicine.medical_specialtyMagnetic Resonance SpectroscopyAlkalosisBioenergeticsFibrosarcomaPartial PressureAdenylate kinasePhosphocreatineMiceCellular and Molecular Neurosciencechemistry.chemical_compoundInternal medicinemedicineAnimalsHomeostasisEnergy chargeMolecular BiologyPharmacologyAdenine NucleotidesCell BiologyHydrogen-Ion Concentrationmedicine.diseaseLactic acidOxygenEndocrinologychemistryMolecular MedicineEnergy MetabolismAdenosine triphosphateIntracellularExperientia
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Intracellular acidosis in murine fibrosarcomas coincides with ATP depletion, hypoxia, and high levels of lactate and total Pi

1994

Bioenergetic and metabolic status of murine FSaII tumours were evaluated using 31P MRS, acid extracts ('global' techniques) and quantitative bioluminescence ('microregional' assay). Data obtained from s.c. tumours of varying sizes (44-600 mm3) have been correlated with the oxygenation status evaluated using O2-sensitive needle electrodes. beta-NTP/Pi and phosphocreatine (PCr)/Pi ratios derived from 31P MRS were positively correlated to the median tissue pO2 values. pH declined during growth with intracellular acidosis being evident in tumours > 350 mm3. Whereas lactic acid formation greatly contributed to this decline in small- and medium-sized tumours, ATP hydrolysis and slowing down of th…

medicine.medical_specialtyMagnetic Resonance SpectroscopyAlkalosisPhosphocreatineBioenergeticsFibrosarcomaAdenylate kinaseBiologyPhosphatesPhosphocreatineMicechemistry.chemical_compoundAdenosine TriphosphateATP hydrolysisInternal medicinemedicineAnimalsRadiology Nuclear Medicine and imagingLactic AcidEnergy chargeSpectroscopyAcidosisMice Inbred C3HHydrogen-Ion Concentrationmedicine.diseaseCell HypoxiaOxygenGlucoseEndocrinologychemistryLactatesMolecular MedicineFemalemedicine.symptomAcidosisEnergy MetabolismIntracellularNMR in Biomedicine
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MUSCLE BIOENERGETICS IN OBESE ZUCKER RATS

1994

International audience; Abstract: The purpose of this study was to investigate the energetic metabolism in obese Zucker rats, using phosphorus nuclear magnetic resonance spectroscopy at rest and during a 2-Hz muscle stimulation and subsequent recovery. Animals were anesthetized with ketamine (150 mg/kg ip). Fed obese rats and 2-day-fasted obese rats were compared with their normally fed and 2-day-fasted lean litter mates. No differences were found between the two groups for ATP, total creatine, phosphocreatine (PCr), and intracellular pH. Starvation in lean rats resulted in a significant fall in inorganic phosphate (Pi), increased resting ADP level, and decreased PCr and ADP recovery after …

medicine.medical_specialtyMagnetic Resonance SpectroscopyPhysiologyEndocrinology Diabetes and MetabolismIntracellular pHPHOSPHOCREATINE[INFO.INFO-IM] Computer Science [cs]/Medical ImagingStimulation030204 cardiovascular system & hematologyBiologyPhosphatesPhosphocreatine03 medical and health scienceschemistry.chemical_compoundAdenosine Triphosphate0302 clinical medicineMITOCHONDRIAGENETIC OBESITYPhysiology (medical)Internal medicineMUSCLESmedicine[INFO.INFO-IM]Computer Science [cs]/Medical ImagingAnimalsNUCLEAR MAGNETIC RESONANCEObesityAcidosis2. Zero hungerAnalysis of Variance[ INFO.INFO-IM ] Computer Science [cs]/Medical ImagingADENOSINE 5'-TRIPHOSPHATEADENOSINE 5'-DIPHOSPHATEMetabolismElectric StimulationRatsRats ZuckerAdenosine DiphosphateAdenosine diphosphateEndocrinologychemistryENERGETIC METABOLISMINTRACELLULAR PHFemalemedicine.symptomEnergy MetabolismAdenosine triphosphate030217 neurology & neurosurgeryMuscle ContractionMuscle contraction
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Stabilization of Perivascular Mast Cells by Endothelial CNP (C-Type Natriuretic Peptide)

2020

Objective: Activated perivascular mast cells (MCs) participate in different cardiovascular diseases. Many factors provoking MC degranulation have been described, while physiological counterregulators are barely known. Endothelial CNP (C-type natriuretic peptide) participates in the maintenance of vascular barrier integrity, but the target cells and mechanisms are unclear. Here, we studied whether MCs are regulated by CNP. Approach and Results: In cultured human and murine MCs, CNP activated its specific GC (guanylyl cyclase)-B receptor and cyclic GMP signaling. This enhanced cyclic GMP–dependent phosphorylation of the cytoskeleton-associated VASP (vasodilator-stimulated phosphoprotein) and…

medicine.medical_specialtyMice 129 StrainMedizinMyocardial Reperfusion InjuryCell DegranulationCell LineMicrocirculationCapillary PermeabilityCyclic gmpAdenosine TriphosphateInternal medicineParacrine CommunicationmedicineAnimalsMast CellsPhosphorylationCyclic GMPMice KnockoutChemistryMicrofilament ProteinsDegranulationEndothelial CellsNatriuretic Peptide C-TypeThrombosisPhosphoproteinsMice Inbred C57BLDisease Models AnimalEndocrinologyNeutrophil InfiltrationC-type natriuretic peptideCardiology and Cardiovascular MedicineCell Adhesion MoleculesReceptors Atrial Natriuretic FactorSignal TransductionGuanylate cyclase
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