Search results for "58"

showing 10 items of 2229 documents

Editorial: Understanding Gamma Delta T Cell Multifunctionality - Towards Immunotherapeutic Applications.

2020

Introduction: gd T cells have been characterized by the expression of a gd T cell receptor (TCR).When the gd TCR and the corresponding ab TCR were first discovered it was assumed that the corresponding cell types were likely to be functionally very similar. However, some 30 years later, we have realized that they are not. Unlike ab T cells, gd T cells (i) sense target antigens independent of MHC molecules; (ii) display NK-cell like innate reactivities, including killing of infected cells as well as microbes; (iii) are able to take up large particulates, including bacteria, and (iv) can act as professional antigen presenting cells. The “stress sensing” abilities of gd T cells have led to a g…

lcsh:Immunologic diseases. Allergy0301 basic medicineCell typeT cellImmunologygd T cells gd T cell receptor antigen recognition killing mechanisms infectious diseases tumor immunology.Major histocompatibility complexLigandsinfectious diseasesCommunicable DiseasesImmunotherapy Adoptiveγδ T cellsγδ T cell receptor03 medical and health sciences0302 clinical medicineAntineoplastic Agents ImmunologicalLymphocytes Tumor-InfiltratingAntigenAnti-Infective AgentsT-Lymphocyte SubsetsNeoplasmsmedicineImmunology and AllergyAnimalsHumanstumor immunologyGamma delta T cellAntigen-presenting cellSettore MED/04 - Patologia GeneralebiologyT-cell receptorReceptors Antigen T-Cell gamma-deltakilling mechanismsAcquired immune systemCell biologyantigen recognition030104 developmental biologymedicine.anatomical_structurePhenotypeEditorialbiology.proteinlcsh:RC581-607030215 immunologySignal TransductionFrontiers in immunology
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Role of Type I and II Interferons in Colorectal Cancer and Melanoma

2017

Cancer can be considered an aberrant organ with a hierarchical composition of different cell populations. The tumor microenvironment, including the immune cells and related cytokines, is crucial during all the steps of tumor development. In particular, type I and II interferons are involved in a plethora of mechanisms that regulate immune responses in cancer, thus balancing immune escape versus immune surveillance. Interferons are involved in both the direct and indirect regulation of cancer cell proliferation and metastatic potential. The mutational background of genes involved in interferons signaling could serve as a prognostic biomarker and a powerful tool to screen cancer patients elig…

lcsh:Immunologic diseases. Allergy0301 basic medicineColorectal cancerCellImmunologyContext (language use)ReviewBiology03 medical and health sciencesImmune systemInterferonmedicineAnti-cancer therapy; Cancer; Cancer progression; Colon; Interferon; Melanoma; Tumor immunology; Immunology and Allergy; ImmunologymelanomaImmunology and Allergycancertumor immunologyTumor microenvironmentcolonMelanomaCancerinterferonmedicine.diseasecancer progression030104 developmental biologymedicine.anatomical_structureImmunologyCancer researchlcsh:RC581-607anti-cancer therapymedicine.drugFrontiers in Immunology
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Reciprocal influence of B cells and tumor macro and microenvironments in the ApcMin/+ model of colorectal cancer

2017

One of the most fascinating aspects of the immune system is its dynamism, meant as the ability to change and readapt according to the organism needs. Following an insult, we assist to the spontaneous organization of different immune cells which cooperate, locally and at distance, to build up an appropriate response. Throughout tumor progression, adaptations within the systemic tumor environment, or macroenvironment, result in the promotion of tumor growth, tumor invasion and metastasis to distal organs, but also to dramatic changes in the activity and composition of the immune system. In this work, we show the changes of the B-cell arm of the immune system following tumor progression in the…

lcsh:Immunologic diseases. Allergy0301 basic medicineColorectal cancerImmunologySpleenintestinal cancerBiologylcsh:RC254-282Metastasis03 medical and health sciencesImmune systemPeritoneummedicineImmunology and Allergyapcmin/+ miceApcMin/+mice; B lymphocytes; IgA; IL-10; intestinal cancer; Immunology and Allergy; Immunology; OncologyB lymphocyteApcMin/+micelcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensmedicine.diseasePhenotypeInterleukin 10030104 developmental biologymedicine.anatomical_structureOncologyTumor progressionIL-10Immunologyb lymphocyteslcsh:RC581-607IgAOncoImmunology
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Complement Protein C1q Binds to Hyaluronic Acid in the Malignant Pleural Mesothelioma Microenvironment and Promotes Tumor Growth

2017

C1q is the first recognition subcomponent of the complement classical pathway, which acts towards the clearance of pathogens and apoptotic cells. C1q is also known to modulate a range of functions of immune and non-immune cells, including their involvement in placental development and sensorial synaptic pruning. We have recently shown that C1q can promote tumour by encouraging their adhesion, migration and proliferation in addition to angiogenesis and metastasis. In this study, we have examined the role of C1q in the microenvironment of malignant pleuric mesothelioma (MPM), a rare form of cancer commonly associated with exposure to asbestos. We found that C1q was highly expressed in all MPM…

lcsh:Immunologic diseases. Allergy0301 basic medicineComplement system; Malignant pleural mesothelioma; Hyaluronic acid; Mesothelioma cells; C1q; CancerAngiogenesisMPMp38 mitogen-activated protein kinasesImmunologyHAchemical and pharmacologic phenomenaBiologyMetastasisMesothelioma cell03 medical and health sciencesClassical complement pathwaychemistry.chemical_compound0302 clinical medicineImmune systemhyaluronic acidHyaluronic acidmedicinemalignant pleural mesotheliomacancerImmunology and AllergyCell adhesioncomplement systemC1qcomplement system; MPM; HA; Mesothelioma cells; C1q and cancerOriginal ResearchC1q and cancermedicine.diseaseComplement system030104 developmental biologyC1q; Cancer; Complement system; Hyaluronic acid; Malignant pleural mesothelioma; Mesothelioma cells; Immunology and Allergy; Immunologychemistrymesothelioma cells030220 oncology & carcinogenesisImmunologyCancer researchlcsh:RC581-607Frontiers in Immunology
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Macrophages as an Emerging Source of Wnt Ligands: Relevance in Mucosal Integrity

2019

The Wnt signaling pathway is a conserved pathway involved in important cellular processes such as the control of embryonic development, cellular polarity, cellular migration, and cell proliferation. In addition to playing a central role during embryogenesis, this pathway is also an essential part of adult homeostasis. Indeed, it controls the proliferation of epithelial cells in different organs such as intestine, lung, and kidney, and guarantees the maintenance of the mucosa in physiological conditions. The origin of this molecular pathway is the binding between Wnt ligands (belonging to a family of 19 different homologous secreted glycoproteins) and their specific membrane receptors, from …

lcsh:Immunologic diseases. Allergy0301 basic medicineFrizzledCellular polarityImmunologyReviewmacrophageBiologyLigandsProinflammatory cytokine03 medical and health sciencesParacrine signalling0302 clinical medicineNeoplasmsWnt ligandsmucosal homeostasisHumanscancerImmunology and AllergyAutocrine signallingWnt Signaling PathwayInflammationMucous MembraneInnate immune systemMacrophagesfibrosisWnt signaling pathwayCell migrationImmunity InnateCell biologyWnt Proteins030104 developmental biologyregenerationlcsh:RC581-607Protein Binding030215 immunologyFrontiers in Immunology
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F-Type Lectins: A highly diversified family of fucose-binding proteins with a unique sequence motif and structural fold, involved in self/non-self-re…

2017

The F-type lectin (FTL) family is one of the most recent to be identified and structurally characterized. Members of the FTL family are characterized by a fucose recognition domain [F-type lectin domain (FTLD)] that displays a novel jellyroll fold (“F-type” fold) and unique carbohydrate- and calcium-binding sequence motifs. This novel lectin family comprises widely distributed proteins exhibiting single, double, or greater multiples of the FTLD, either tandemly arrayed or combined with other structurally and functionally distinct domains, yielding lectin subunits of pleiotropic properties even within a single species. Furthermore, the extraordinary variability of FTL sequences (isoforms) th…

lcsh:Immunologic diseases. Allergy0301 basic medicineGene isoformImmunologySettore BIO/05 - ZoologiaFucose bindingReviewFucoseF-type lectinsSelf/non-self-recognitionKelch motif03 medical and health scienceschemistry.chemical_compoundGene duplicationImmunology and AllergyStructural modelingGeneticsInnate immunitybiologyPhylogenetic treefucolectinsLectinGlycan recognition030104 developmental biologychemistrybiology.proteinFucose-bindingFucolectinlcsh:RC581-607Sequence motifF-type lectinF-type lectins; Fucolectins; Fucose-binding; Glycan recognition; Innate immunity; Self/non-self-recognition; Structural modeling; Immunology and Allergy; Immunology
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Pathogenic Role of Complement in Antiphospholipid Syndrome and Therapeutic Implications

2018

Antiphospholipid syndrome (APS) is an acquired autoimmune disease characterized by thromboembolic events, pregnancy morbidity, and the presence of antiphospholipid (aPL) antibodies. There is sound evidence that aPL act as pathogenic autoantibodies being responsible for vascular clots and miscarriages. However, the exact mechanisms involved in the clinical manifestations of the syndrome are still a matter of investigation. In particular, while vascular thrombosis is apparently not associated with inflammation, the pathogenesis of miscarriages can be explained only in part by the aPL-mediated hypercoagulable state and additional non-thrombotic effects, including placental inflammation, have b…

lcsh:Immunologic diseases. Allergy0301 basic medicineImmunologyComplementMiscarriagesAnti-beta2 glycoprotein I antibodieInflammationMiscarriagePathogenesis03 medical and health sciences0302 clinical medicineImmune systemAntiphospholipid syndromeimmune system diseasesAntiphospholipid syndromeMedicineImmunology and AllergyAnimal model030203 arthritis & rheumatologyAutoimmune diseaseInflammationbusiness.industryAutoantibodyThrombosismedicine.diseaseComplement (complexity)Complement systemAnimal models030104 developmental biologyAnti-beta2 glycoprotein I antibodiesPerspectiveThrombosiImmunologyAnimal models; Anti-beta2 glycoprotein I antibodies; Antiphospholipid syndrome; Complement; Inflammation; Miscarriages; Therapy; Thrombosis; Immunology and Allergy; ImmunologyTherapymedicine.symptomlcsh:RC581-607businessFrontiers in Immunology
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Genome-Wide Inhibition of Pro-atherogenic Gene Expression by Multi-STAT Targeting Compounds as a Novel Treatment Strategy of CVDs.

2018

Cardiovascular diseases (CVDs), including atherosclerosis, are globally the leading cause of death. Key factors contributing to onset and progression of atherosclerosis include the pro-inflammatory cytokines Interferon (IFN)a and IFN? and the Pattern Recognition Receptor (PRR) Toll-like receptor 4 (TLR4). Together, they trigger activation of Signal Transducer and Activator of Transcription (STAT)s. Searches for compounds targeting the pTyr-SH2 interaction area of STAT3, yielded many small molecules, including STATTIC and STX-0119. However, many of these inhibitors do not seem STAT3-specific. We hypothesized that multi-STAT-inhibitors that simultaneously block STAT1, STAT2, and STAT3 activit…

lcsh:Immunologic diseases. Allergy0301 basic medicineMaleIn silicoImmunologyGene ExpressionBiologystatIn silico dockingCell LineSmall Molecule Librariessrc Homology Domains03 medical and health sciencesCVDs treatment strategyImmunology and AllergyAnimalsHumansvascular inflammationSTAT1STAT2STAT3Vascular inflammationCells CulturedOriginal ResearchOxadiazolesGene Expression ProfilingSTATPattern recognition receptorin silico dockingFarmaciaAtherosclerosisCyclic S-OxidesMice Inbred C57BLSTAT Transcription Factors030104 developmental biologyCardiovascular DiseasesTLR4biology.proteinSTAT proteinCancer researchQuinolinesmulti-STAT inhibitorsMulti-STAT inhibitorslcsh:RC581-607Genome-Wide Association StudySignal TransductionFrontiers in immunology
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Serological Levels of Anti-clathrin Antibodies Are Decreased in Patients With Pseudoexfoliation Glaucoma

2021

Evidence for immunologic contribution to glaucoma pathophysiology is steadily increasing in ophthalmic research. Particularly, an altered abundance of circulating autoantibodies to ocular antigens is frequently observed. Here, we report an analysis of autoantibody abundancies to selected antigens in sera of open-angle glaucoma patients, subdivided into normal-tension glaucoma (N = 31), primary open-angle glaucoma (N = 43) and pseudoexfoliation glaucoma (N = 45), vs. a non-glaucomatous control group (N = 46). Serum samples were analyzed by protein microarray, including 38 antigens. Differences in antibody levels were assessed by ANOVA. Five serological antibodies showed significantly altered…

lcsh:Immunologic diseases. Allergy0301 basic medicineMicroarraygenetic structuresautoantibodiesImmunologyProtein Array AnalysisGlaucomamedicine.disease_causeExfoliation SyndromeAutoantigensSerologyAutoimmunityPathogenesis03 medical and health sciences0302 clinical medicineAntigenmedicineHumansImmunology and AllergyOriginal Researchbiologybusiness.industryautoimmunityAutoantibodyComputational Biologybioinformaticsmedicine.diseaseimmunoproteomicseye diseasesClathrin030104 developmental biologyglaucomaCase-Control StudiesImmunology030221 ophthalmology & optometrybiology.proteinsense organsDisease SusceptibilityAntibodylcsh:RC581-607businessmicroarrayBiomarkersGlaucoma Open-AngleFrontiers in Immunology
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Tumor lymphocyte immune response to preoperative radiotherapy in locally advanced rectal cancer: The LYMPHOREC study

2017

IF 7.719; International audience; Introduction: Some studies have suggested that baseline tumor-infiltrating-lymphocytes (TILs), such as CD8+ and FoxP3+ T-cells, may be associated with a better prognosis in colorectal cancer. We sought to investigate modulation of the immune response by preoperative radiotherapy (preopRT) and its impact on survival in locally advanced rectal cancer (LARC). Materials & Methods: We analyzed data for 237 patients with LARC who received RT. Density of TILS (CD8+ and FoxP3+) in intraepithelial (iTILs) and stromal compartments (sTILs) were evaluated from surgery pathological specimens and biopsies performed at baseline. The primary endpoint was to assess the impa…

lcsh:Immunologic diseases. Allergy0301 basic medicineOncologymedicine.medical_specialtyColorectal cancermedicine.medical_treatmentImmunology[SDV.CAN]Life Sciences [q-bio]/Cancerchemical and pharmacologic phenomenalcsh:RC254-282radiation therapy[ SDV.CAN ] Life Sciences [q-bio]/Cancer03 medical and health sciences0302 clinical medicineInternal medicinemedicineClinical endpointImmunology and Allergyrectal cancertranslational studyOriginal ResearchUnivariate analysisbusiness.industryTumor-infiltrating lymphocytesDose fractionationFOXP3hemic and immune systemslcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensmedicine.disease3. Good healthRadiation therapy030104 developmental biologyOncology030220 oncology & carcinogenesistumor-infiltrating lymphocyteslcsh:RC581-607businessAdjuvant
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