Search results for "610 Medicine & health"

showing 10 items of 627 documents

Consistent device simulation model describing perovskite solar cells in steady-state, transient, and frequency domain

2019

​This document is the Accepted Manuscript version of a Published Work that appeared in final form in ACS Applied Materials & Interfaces, copyright © American Chemical Society after peer review and technical editing by the publisher. To access the final edited and published work see https://pubs.acs.org/doi/10.1021/acsami.9b04991

Steady state (electronics)Materials scienceIMPSImpedance spectroscopy610 Medicine & health02 engineering and technology010402 general chemistrycomputer.software_genre01 natural sciencesChemical societyGeneral Materials ScienceTransient (computer programming)Device simulation10266 Clinic for Reconstructive SurgeryMaterials621.3: Elektrotechnik und ElektronikCèl·lules fotoelèctriquesTrapsPerovskite (structure)Drift-diffusion modelingProgramming languagePerovskite solar cellsHysteresis021001 nanoscience & nanotechnology2500 General Materials Science0104 chemical sciencesMobile ionsFrequency domainTransient photo-current0210 nano-technologycomputer
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Tonic T cell signalling and T cell tolerance as opposite effects of self-recognition on dendritic cells.

2010

Naive T cells spend most of their time scanning the surface of dendritic cells (DCs), indicating that self-MHC/T cell receptor (TCR) interactions between these immune cells occur routinely in peripheral organs during the steady state. Peripheral self-MHC recognition on DCs drives seemingly opposing effects in the absence of inflammatory stimuli such as deletion of certain self-reactive T cells as well as maintenance of the T cell responsiveness to antigen, both of which shape the T cell repertoire and regulate T cell responses. Here we review recent data on the role of self-MHC recognition on steady-state DCs in the periphery and propose that interactions between T cells and steady-state DC…

T cellT-LymphocytesImmunologyAntigen presentation610 Medicine & healthchemical and pharmacologic phenomenaBiologyLymphocyte ActivationMajor Histocompatibility ComplexmedicineImmunology and AllergyCytotoxic T cellAnimalsHumansIL-2 receptorAntigen-presenting cell2403 ImmunologyAntigen PresentationZAP70CD28Dendritic CellsNatural killer T cellCell biologymedicine.anatomical_structureSelf Tolerance10032 Clinic for Oncology and Hematology2723 Immunology and AllergySignal TransductionCurrent opinion in immunology
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Tolerance without clonal expansion: self-antigen-expressing B cells program self-reactive T cells for future deletion.

2008

Abstract B cells have been shown in various animal models to induce immunological tolerance leading to reduced immune responses and protection from autoimmunity. We show that interaction of B cells with naive T cells results in T cell triggering accompanied by the expression of negative costimulatory molecules such as PD-1, CTLA-4, B and T lymphocyte attenuator, and CD5. Following interaction with B cells, T cells were not induced to proliferate, in a process that was dependent on their expression of PD-1 and CTLA-4, but not CD5. In contrast, the T cells became sensitive to Ag-induced cell death. Our results demonstrate that B cells participate in the homeostasis of the immune system by abl…

T-LymphocytesProgrammed Cell Death 1 ReceptorAutoimmunityAntigens CD/biosynthesisAntigens CD5/geneticsAutoantigensInterleukin 21MiceImmunology and AllergyCytotoxic T cellHomeostasisCTLA-4 AntigenIL-2 receptorAntigens Differentiation/biosynthesisB-LymphocytesAntigens CD/geneticsB-Lymphocytes/immunologyT-Lymphocytes/metabolismNatural killer T cellCell biologymedicine.anatomical_structureHomeostasis/immunology2723 Immunology and AllergyAntigens CD5/biosynthesisAntigens Differentiation/geneticsAntigens CD5/immunologyT cellImmunologyAntigens CD/immunologyClonal Deletion610 Medicine & healthchemical and pharmacologic phenomenaMice TransgenicBiologyAutoantigens/biosynthesisCD5 AntigensAutoimmunity/physiologyAutoantigens/immunologyAntigens CDmedicineAnimalsB-Lymphocytes/metabolismAntigen-presenting cellCell Proliferation2403 ImmunologyAntigens Differentiation/immunologyGene Expression Regulation/immunologyCD40Clonal Deletion/physiologyT-Lymphocytes/immunologyAntigens Differentiation10040 Clinic for NeurologyB-1 cellGene Expression Regulationbiology.protein
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Predictor variables for half marathon race time in recreational female runners

2011

Import JabRef | WosArea General and Internal Medicine; International audience; INTRODUCTION: The relationship between skin-fold thickness and running performance has been investigated from 100 m to the marathon distance, except the half marathon distance. OBJECTIVE: To investigate whether anthropometry characteristics or training practices were related to race time in 42 recreational female half marathoners to determine the predictor variables of half-marathon race time and to inform future novice female half marathoners. METHODS: Observational field study at the 'Half Marathon Basel' in Switzerland. RESULTS: In the bivariate analysis, body mass (r = 0.60), body mass index (r = 0.48), body …

Time FactorsTRAINING CHARACTERISTICSPhysical fitnessLEVEL2700 General MedicineRunningEndurance0302 clinical medicineSkin fold030212 general & internal medicineSKINFOLD THICKNESSES2. Zero hungerlcsh:R5-920AnthropometryGeneral MedicineClinical ScienceCircumference3. Good healthSkinfold ThicknessSkinfold thicknessCIRCUMFERENCEBody fat[ SCCO.NEUR ] Cognitive science/NeuroscienceDISTANCE RUNNING PERFORMANCEFemalelcsh:Medicine (General)Adult11035 Institute of General Practicemedicine.medical_specialtyULTRAMARATHONERSeducationECONOMY610 Medicine & healthPredictor variablesAthletic PerformanceCAPACITY03 medical and health sciencesAthletemedicineHumansbusiness.industryUpper body[SCCO.NEUR]Cognitive science/NeuroscienceGender030229 sport sciencesAnthropometryBODY-MASSPhysical FitnessPhysical therapyRecreationUPPER ARMEpidemiologic MethodsbusinessBody mass indexhuman activities
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D2R striatopallidal neurons inhibit both locomotor and drug reward processes.

2009

The specific functions of dopamine D(2) receptor-positive (D(2)R) striatopallidal neurons remain poorly understood. Using a genetic mouse model, we found that ablation of D(2)R neurons in the entire striatum induced hyperlocomotion, whereas ablation in the ventral striatum increased amphetamine conditioned place preference. Thus D(2)R striatopallidal neurons limit both locomotion and, unexpectedly, drug reinforcement.

Time FactorsstriatumParkinson's diseaseStriatumNeurons -- drug effectsEnkephalins -- metabolism10263 Institute of Experimental ImmunologyMiceDopamine Uptake InhibitorsTyrosine 3-Monooxygenase -- geneticsCorpus Striatum -- cytologyDiphtheria ToxinGlutamate Decarboxylase -- metabolismstriatum; indirect opathway; A2A receptors; D2 receptors; locomotion; amphetamine addiction; Parkinson's diseaseNeuronsamphetamine addictionGlutamate DecarboxylaseGeneral NeuroscienceAmphetamine -- pharmacologyNeurodegeneration2800 General NeuroscienceEnkephalinsSciences bio-médicales et agricoleslocomotionmedicine.anatomical_structureA2A receptorsIntercellular Signaling Peptides and ProteinsReceptors Dopamine D2 -- metabolismPsychologyLocomotionmedicine.drugHeparin-binding EGF-like Growth FactorProtein BindingGlobus Pallidus -- cytologyReceptors Dopamine D2 -- deficiencyReinforcement ScheduleTyrosine 3-MonooxygenaseGlutamate Decarboxylase -- geneticsLocomotion -- geneticsIntercellular Signaling Peptides and Proteins -- genetics610 Medicine & healthMice TransgenicNerve Tissue ProteinsDiphtheria Toxin -- pharmacologyGlobus PallidusNeurons -- physiologyLocomotion -- drug effectsRewardDopamineDopamine receptor D2medicineNerve Tissue Proteins -- metabolismAnimalsGene Expression Regulation -- geneticsAmphetamineD2 receptorsReceptors Adenosine A2Receptors Dopamine D2indirect opathwayVentral striatumReceptors Adenosine A2 -- geneticsDopamine Uptake Inhibitors -- pharmacologymedicine.diseaseConditioned place preferenceCorpus StriatumMice Inbred C57BLGene Expression Regulation -- drug effectsAmphetaminenervous systemGene Expression RegulationProtein Binding -- drug effectsTyrosine 3-Monooxygenase -- metabolism570 Life sciences; biologyAutoradiographyConditioning OperantNeuronConditioning Operant -- physiologyNeuroscienceEnkephalins -- geneticsNature neuroscience
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Efficacy and cost-effectiveness of guided and unguided internet- and mobile-based indicated transdiagnostic prevention of depression and anxiety (ICa…

2019

Background Depression and anxiety are highly prevalent and often co-occur. Several studies indicate the potential of disorder-specific psychological interventions for the prevention of each of these disorders. To treat comorbidity, transdiagnostic treatment concepts seem to be a promising approach, however, evidence for transdiagnostic concepts of prevention remains inconclusive. Internet- and mobile-based interventions (IMIs) may be an effective means to deliver psychological interventions on a large scale for the prevention of common mental disorders (CMDs) such as depression and anxiety. IMIs have been shown to be effective in treating CMDs, e.g. in reducing symptoms of depression and an…

Transdiagnosticlcsh:T58.5-58.64lcsh:Information technologyDepressionPreventionlcsh:BF1-990610 Medicine & healthInternet-basedAnxietyArticlelcsh:PsychologyQA76 Computer softwareSDG 3 - Good Health and Well-beingRandomized controlled trial/dk/atira/pure/core/keywords/600089002/dk/atira/pure/sustainabledevelopmentgoals/good_health_and_well_beingPsychologyddc:158150 PsychologyPrevention Transdiagnostic Depression Anxiety Internet-based Randomized controlled trialRC0321 Neuroscience. Biological psychiatry. Neuropsychiatry
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TBVAC2020: Advancing Tuberculosis Vaccines from Discovery to Clinical Development

2017

International audience; TBVAC2020 is a research project supported by the Horizon 2020 program of the European Commission (EC). It aims at the discovery and development of novel tuberculosis (TB) vaccines from preclinical research projects to early clinical assessment. The project builds on previous collaborations from 1998 onwards funded through the EC framework programs FP5, FP6, and FP7. It has succeeded in attracting new partners from outstanding laboratories from all over the world, now totaling 40 institutions. Next to the development of novel vaccines, TB biomarker development is also considered an important asset to facilitate rational vaccine selection and development. In addition, …

TuberculosiImmunologybacille Calmette–Guérin610 Medicine & healthReview[SDV.MHEP.PSR]Life Sciences [q-bio]/Human health and pathology/Pulmonology and respiratory tract[SDV.MHEP.MI]Life Sciences [q-bio]/Human health and pathology/Infectious diseasesTuberculosis; Bacille Calmette-Guérin; Vaccination; Biomarker; Clinical trial; Portfolio management; Discovery[SDV.BC.IC]Life Sciences [q-bio]/Cellular Biology/Cell Behavior [q-bio.CB]Immunology and AllergyBacille Calmette-Guérinbacille Calmette-Guérinbacille Calmette-Guerin2403 Immunology10179 Institute of Medical MicrobiologyBacille Calmette-Guérin; Biomarker; Clinical trial; Discovery; Portfolio management; Tuberculosis; Vaccination; Immunology and Allergy; Immunologyclinical trialvaccination[SDV.MP.BAC]Life Sciences [q-bio]/Microbiology and Parasitology/Bacteriology[SDV.GEN.GH]Life Sciences [q-bio]/Genetics/Human geneticstuberculosis2723 Immunology and Allergy570 Life sciences; biologybiomarkerportfolio managementdiscovery
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ADHD and DAT1: Further evidence of paternal over-transmission of risk alleles and haplotype

2010

Contains fulltext : 87259.pdf (Publisher’s version ) (Closed access) We [Hawi et al. (2005); Am J Hum Genet 77:958-965] reported paternal over-transmission of risk alleles in some ADHD-associated genes. This was particularly clear in the case of the DAT1 3'-UTR VNTR. In the current investigation, we analyzed three new sample comprising of 1,248 ADHD nuclear families to examine the allelic over-transmission of DAT1 in ADHD. The IMAGE sample, the largest of the three-replication samples, provides strong support for a parent of origin effect for allele 6 and the 10 repeat allele (intron 8 and 3'-UTR VNTR, respectively) of DAT1. In addition, a similar pattern of over-transmission of paternal ri…

Untranslated region2716 Genetics (clinical)Candidate gene2804 Cellular and Molecular NeuroscienceMedizin610 Medicine & healthMinisatellite RepeatsBiology2738 Psychiatry and Mental HealthGenomic Imprinting03 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicineSDG 3 - Good Health and Well-beingmental disordersPerception and Action [DCN 1]HumansGenetics(clinical)ddc:610Medizinische Fakultät » Universitätsklinikum Essen » LVR-Klinikum Essen » Klinik für Psychiatrie Psychosomatik und Psychotherapie des Kindes- und JugendaltersRisk factorAllele3' Untranslated RegionsNuclear familyGeneAllelesGenetics (clinical)GeneticsMental Health [NCEBP 9]Dopamine Plasma Membrane Transport ProteinsHaplotypeIntron10058 Department of Child and Adolescent Psychiatry030227 psychiatryPsychiatry and Mental healthHaplotypesAttention Deficit Disorder with Hyperactivity/dk/atira/pure/sustainabledevelopmentgoals/good_health_and_well_being030217 neurology & neurosurgeryAmerican Journal of Medical Genetics Part B: Neuropsychiatric Genetics
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Involvement of KSRP in the post-transcriptional regulation of human iNOS expression–complex interplay of KSRP with TTP and HuR

2005

We purified the KH-type splicing regulatory protein (KSRP) as a protein interacting with the 3'-untranslated region (3'-UTR) of the human inducible nitric oxide (iNOS) mRNA. Immunodepletion of KSRP enhanced iNOS 3'-UTR RNA stability in in vitro-degradation assays. In DLD-1 cells overexpressing KSRP cytokine-induced iNOS expression was markedly reduced. In accordance, downregulation of KSRP expression increases iNOS expression by stabilizing iNOS mRNA. Co-immunoprecipitations showed interaction of KSRP with the exosome and tristetraprolin (TTP). To analyze the role of KSRP binding to the 3'-UTR we studied iNOS expression in DLD-1 cells overexpressing a non-binding mutant of KSRP. In these ce…

Untranslated regionRNA StabilityTristetraprolinNitric Oxide Synthase Type II610 Medicine & healthRNA-binding proteinBiologyImmediate early proteinArticleGene Expression Regulation EnzymologicELAV-Like Protein 1Immediate-Early ProteinsTristetraprolinCell Line TumorGeneticsHumansRNA Messenger610 Medicine & healthPost-transcriptional regulation3' Untranslated RegionsRegulation of gene expressionMessenger RNAThree prime untranslated regionRNA-Binding ProteinsMolecular biologyDNA-Binding ProteinsELAV ProteinsAntigens SurfaceMutationTrans-ActivatorsCytokinesNitric Oxide SynthaseNucleic Acids Research
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VEGF-B-induced vascular growth leads to metabolic reprogramming and ischemia resistance in the heart

2014

Abstract Angiogenic growth factors have recently been linked to tissue metabolism. We have used genetic gain‐ and loss‐of function models to elucidate the effects and mechanisms of action of vascular endothelial growth factor‐B (VEGF‐B) in the heart. A cardiomyocyte‐specific VEGF‐B transgene induced an expanded coronary arterial tree and reprogramming of cardiomyocyte metabolism. This was associated with protection against myocardial infarction and preservation of mitochondrial complex I function upon ischemia‐reperfusion. VEGF‐B increased VEGF signals via VEGF receptor‐2 to activate Erk1/2, which resulted in vascular growth. Akt and mTORC1 pathways were upregulated and AMPK downregulated, …

VEGF‐Bmedicine.medical_specialtyMedicine (General)AngiogenesiseducationMOUSE HEARTIschemiaVEGF-B610 Medicine & healthmTORC1ischemiaBiologyQH426-470CONTRIBUTESchemistry.chemical_compoundangiogenesisR5-920CARDIAC-FUNCTIONInternal medicinemedicineGeneticsFAILUREta318Myocardial infarctionFATTY-ACID UPTAKEREPERFUSION INJURY610 Medicine & healthProtein kinase BMYOCARDIAL HYPERTROPHYAMPKta3121medicine.diseaseCell biologyARTERIOGENESISVascular endothelial growth factorMICEEndocrinologychemistry3121 General medicine internal medicine and other clinical medicineendothelial cellMolecular Medicine3111 BiomedicineReperfusion injurymetabolism
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