Search results for "AGLA"

showing 10 items of 272 documents

Gathering of aging and estrogen withdrawal in vascular dysfunction of senescent accelerated mice.

2010

The aim of this work was to characterize a mouse model of experimental menopause and cardiovascular aging that closely reflects menopause in women. Senescence accelerated mouse (SAM)-Resistant type 1 (SAMR1, n=30) and SAM-Prone type 8 (SAMP8, n=30) were separated at 5months of age into three groups: 1) sham-operated (Sham); 2) ovariectomized (Ovx); and 3) ovariectomized chronically-treated with estrogen (Ovx+E2). Contractile responses to KCl (60mM) and thromboxane A(2) were greater in aorta from SAMP8 mice compared with SAMR1 in all groups. Neither ovariectomy nor estrogen replacement modified the contractile responses from SAMR1 mice. Conversely, in Ovx SAMP8 the increased maximal contract…

SenescenceAgingmedicine.medical_specialtyEndotheliummedicine.drug_classThromboxaneOvariectomyVasodilator AgentsDown-RegulationIn Vitro TechniquesNitric OxideBiochemistryReceptors Thromboxane A2 Prostaglandin H2Thromboxane A2chemistry.chemical_compoundMiceEndocrinologyInternal medicineGeneticsmedicineAnimalsVasoconstrictor AgentsEnzyme InhibitorsMolecular BiologyAortaEstradiolChemistryEstrogen Replacement TherapyAging PrematureEstrogensCell Biologymedicine.diseaseAcetylcholineMenopauseVasodilationEndocrinologymedicine.anatomical_structureNG-Nitroarginine Methyl EsterEstrogenVasoconstriction15-Hydroxy-11 alpha9 alpha-(epoxymethano)prosta-513-dienoic AcidOvariectomized ratBlood VesselsFemalehormones hormone substitutes and hormone antagonistsAcetylcholinemedicine.drugExperimental gerontology
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Haem oxygenase-1 counteracts the effects of interleukin-1β on inflammatory and senescence markers in cartilage-subchondral bone explants from osteoar…

2011

IL (interleukin)-1β plays an important role in cartilage extracellular matrix degradation and bone resorption in OA (osteoarthritis) through the induction of degradative enzymes and pro-inflammatory mediators. In the present study, we have determined the consequences of HO-1 (haem oxygenase-1) induction on markers of inflammation and senescence in the functional unit cartilage–subchondral bone stimulated with IL-1β. Cartilage–subchondral bone specimens were obtained from the knees of osteoarthritic patients. Treatment with the HO-1 inducer CoPP (cobalt protoporphyrin IX) counteracted the stimulatory effects of IL-1β on IL-6, nitrite, PGE2 (prostaglandin E2), TGF (transforming growth factor)…

SenescenceCartilage Articularmedicine.medical_treatmentInterleukin-1betaDown-RegulationNitric Oxide Synthase Type IIProtoporphyrinsBone resorptionDinoprostoneOsteoarthritismedicineHumansTelomerase reverse transcriptaseProstaglandin E2Bone ResorptionRNA Small InterferingCellular SenescenceProstaglandin-E SynthasesbiologyInterleukin-6InterleukinGeneral MedicineCOPPMolecular biologyIntramolecular OxidoreductasesCyclooxygenase 2ImmunologyHeme Oxygenase (Decyclizing)Osteocalcinbiology.proteinBiomarkersmedicine.drugProstaglandin EClinical science (London, England : 1979)
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Inhibition of neuronally mediated secretion in rat colonic mucosa by prostaglandin D2

1992

Abstract The effect of prostaglandin D 2 (PGD 2 ) on ion transport across the mucosa of the descending colon was studied in rats. PGD 2 dose-dependently decreased baseline short-circuit current of mucosa-submucosal preparations mounted either in the Ussing chamber or mounted as an everted sac. However, with the everted sac technique, the tissue was about 1000 times more sensitive to PGD 2 . Concomitant with the decrease in short-circuit current, PGD 2 increased the mucosal-to-serosal fluxes of sodium and chloride and decreased the serosal-tomucosal flux of chloride. PGD 2 inhibited the secretory action of the PGI 2 analogue iloprost, PGD 2α , and neurotensin. The action of these secretagogu…

Serotoninmedicine.medical_specialtyColonProstaglandinSecretomotorSubstance PSubstance PBiologyDinoprostMembrane Potentialschemistry.chemical_compoundBody WaterChloridesInternal medicinemedicineAnimalsIloprostIntestinal MucosaProstaglandin E2NeurotensinHepatologyUssing chamberProstaglandin D2SodiumGastroenterologyRats Inbred Strainsrespiratory systemEpoprostenolRatsEndocrinologychemistryDiffusion Chambers CultureFemalelipids (amino acids peptides and proteins)SerotoninProstaglandin D2Neurotensinmedicine.drugGastroenterology
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Mycoplasma fermentans-derived lipid inhibits class II major histocompatibility complex expression without mediation by interleukin-6, interleukin-10,…

1996

Mycoplasma cause several diseases in man and animals. Some strains can chronically infect humans, leading to fever or inflammatory syndromes such as arthritis, particularly in immunosuppressed patients. A set of pathogenicity factors shared by many mollicutes may be membrane components that activate macrophages to secrete cytokines and other inflammatory mediators. Mycoplasma-derived high molecular weight material (MDHM) is a macrophage-activating amphiphilic lipid which was purified from Mycoplasma fermentans. We studied the influence of MDHM on the expression of major histocompatibility complex (MHC) class II molecules by mouse resident peritoneal macrophages with an ELISA. Highly purifie…

T cellImmunologyAntigen presentationBiologyNitric OxideMajor histocompatibility complexMicrobiologyMiceAntigenTransforming Growth Factor betaInterferonMHC class ImedicineAnimalsImmunology and AllergyMycoplasma fermentansCells CulturedMycoplasma fermentansMice Inbred C3HInterleukin-6Tumor Necrosis Factor-alphaHistocompatibility Antigens Class IIInterleukinbiology.organism_classificationLipidsInterleukin-10Molecular WeightKineticsmedicine.anatomical_structureInterferon Type IImmunologyProstaglandinsbiology.proteinCytokinesFemaleImmunosuppressive Agentsmedicine.drugEuropean Journal of Immunology
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Discovery and validation of small-molecule heat-shock protein 90 inhibitors through multimodality molecular imaging in living subjects.

2012

Up-regulation of the folding machinery of the heat-shock protein 90 (Hsp90) chaperone protein is crucial for cancer progression. The two Hsp90 isoforms (α and β) play different roles in response to chemotherapy. To identify isoform-selective inhibitors of Hsp90(α/β)/cochaperone p23 interactions, we developed a dual-luciferase (Renilla and Firefly) reporter system for high-throughput screening (HTS) and monitoring the efficacy of Hsp90 inhibitors in cell culture and live mice. HTS of a 30,176 small-molecule chemical library in cell culture identified a compound, N -(5-methylisoxazol-3-yl)-2-[4-(thiophen-2-yl)-6-(trifluoromethyl)pyrimidin-2-ylthio]acetamide (CP9), that binds to Hsp90(α/β) an…

Thymidine kinase activityProtein FoldingImmunoprecipitationLactams MacrocyclicBlotting WesternMice NudeThiophenesBiologyThioacetamideTritiumSmall Molecule LibrariesMiceco-chaperone p23Luciferases FireflyHeat shock proteinCell Line TumorNeoplasmsAcetamidesDrug DiscoveryBenzoquinonesAnimalsHumansImmunoprecipitationProtein IsoformsLuciferaseHSP90 Heat-Shock ProteinsLuciferases RenillaProstaglandin-E SynthasesMultidisciplinaryCell growthImidazolesbioluminescence imagingHsp90Small moleculeMolecular biologydrug developmentHigh-Throughput Screening Assayssmall-molecule inhibitorsIntramolecular OxidoreductasesLeadPNAS PlusCell culturePositron-Emission TomographyPyrazinesbiology.proteinPET/computed tomography imagingTomography X-Ray ComputedProceedings of the National Academy of Sciences of the United States of America
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Co-regulation between cyclo-oxygenase-2 and inducible nitric oxide synthase expression in the time-course of murine inflammation.

2000

Many in vitro studies have used cell cultures to focus on the relationships between cyclo-oxygenase-2 (COX-2) and inducible nitric oxide synthase (iNOS) isoforms. We have investigated the time-course of regulation and the role of COX-2 and iNOS in a model of experimental inflammation in mice, the air pouch injected with zymosan. This study demonstrates that there is an early acute phase (4 h) mediated mainly by eicosanoids, with high levels of prostaglandin E2 (PGE2) produced by cyclo-oxygenase-1. In addition, in the later phase (from 12 h) there is a participation of nitric oxide (NO) and PGE2 accompanied by co-induction of both iNOS and COX-2. These enzymes were detected in migrating leuk…

Time FactorsBlotting WesternAnti-Inflammatory AgentsFluorescent Antibody TechniqueNitric Oxide Synthase Type IIInflammationPharmacologyDexamethasoneDinoprostoneNitric oxidechemistry.chemical_compoundMiceIn vivomedicineLeukocytesAnimalsCyclooxygenase InhibitorsProstaglandin E2NitritePharmacologyInflammationbiologyCyclooxygenase 2 InhibitorsZymosanZymosanGeneral MedicineExudates and TransudatesNitric oxide synthaseIsoenzymeschemistryBiochemistryCell cultureCyclooxygenase 2Prostaglandin-Endoperoxide SynthasesEnzyme Inductionbiology.proteinEicosanoidsFemalemedicine.symptomNitric Oxide SynthaseColchicinemedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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Effects of cyclooxygenase-1/cyclooxygenase-2 inhibition on leukocyte/endothelial cell interactions in the rat mesentery.

2002

Nonsteroidal anti-inflammatory drugs (NSAID) inhibit cyclooxygenase activity and cause gastrointestinal damage in part by promoting leukocyte accumulation in the mucosa. Our aim was to evaluate the effects of selective blockade of the isoenzymes cyclooxygenase-1 and cyclooxygenase-2 on leukocyte adhesion in vivo. Leukocyte/endothelial cell interactions were examined in rat mesenteric venules before and after treatment with indomethacin, SC-560 (5-(4-chlorophenyl)-1-(4-methoxyphenyl)-3-trifluoromethylpyrazole, cyclooxygenase-1 inhibitor), DFP (5,5-dimethyl-3-(2-propoxy)-4-(4-methanesulfonyl)-2(5H)-furanone, cyclooxygenase-2 inhibitor), or SC-560 plus DFP (20 mg/kg, i.v. each). Indomethacin i…

Time FactorsEndotheliumIndomethacinCell CommunicationPharmacologyRats Sprague-DawleyIn vivomedicineBenzene DerivativesCell AdhesionLeukocytesTumor Cells CulturedAnimalsHumansCyclooxygenase InhibitorsMesenteryFuransPharmacologybiologyCyclooxygenase 2 InhibitorsChemistryAnti-Inflammatory Agents Non-SteroidalMembrane ProteinsBiological activityDrug SynergismRatsEndothelial stem cellIsoenzymesmedicine.anatomical_structureMechanism of actionEnzyme inhibitorCyclooxygenase 2Prostaglandin-Endoperoxide SynthasesImmunologybiology.proteinCyclooxygenase 1PyrazolesCyclooxygenaseEndothelium Vascularmedicine.symptomBlood vesselEuropean journal of pharmacology
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Prostaglandin E(2)-loaded microspheres as strategy to inhibit phagocytosis and modulate inflammatory mediators release.

2008

PGE(2), an arachidonic acid metabolite produced by various type of cells regulates a broad range of physiological activities in the endocrine, cardiovascular, gastrointestinal, and immune systems, and is involved in maintaining the local homeostasis. In the immune system, PGE(2) is mainly produced by APCs and it can suppress the Th1-mediated immune responses. The aim of this study was to develop PGE(2)-loaded biodegradable MS that prolong and sustain the in vivo release of this mediator. An o/w emulsion solvent extraction-evaporation method was chosen to prepare the MS. We determined their diameters, evaluated the in vitro release of PGE(2), using enzyme immunoassay and MS uptake by periton…

Time Factorsmedicine.medical_treatmentPhagocytosisChemistry PharmaceuticalDrug CompoundingPharmaceutical ScienceInflammationPharmacologyBiologyNitric OxideDinoprostonechemistry.chemical_compoundMiceImmune systemPhagocytosisIn vivoSepsismedicineAnimalsHumansImmunologic FactorsProstaglandin E2Particle SizeCells CulturedChemokine CCL2Tumor Necrosis Factor-alphaEndothelial CellsWaterGeneral MedicineMicrospheresDisease Models AnimalchemistryBiochemistrySolubilityDelayed-Action PreparationsMacrophages PeritonealLiberationlipids (amino acids peptides and proteins)Arachidonic acidEmulsionsmedicine.symptomInflammation MediatorsOilsBiotechnologyProstaglandin Emedicine.drugEuropean journal of pharmaceutics and biopharmaceutics : official journal of Arbeitsgemeinschaft fur Pharmazeutische Verfahrenstechnik e.V
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Lipid and fatty acid variations inCiona intestinalis ovary after tri-n-butyltin(IV)chloride exposure

2005

Reduction of total lipids (TL) content and significant variations of triglyceride (TG) and phospholipid (PL) fractions were observed as a consequence of exposure of Ciona intestinalis ovaries to tributyltin chloride (TBTCL) solutions. In particular, an evident TG decrease and a PL increase were observed, which probably provoked an increment in membrane fluidity, because of the high concentration of long chain fatty acids and, as a consequence, PL. This could be a cell adaptive standing mechanism towards the pollutants, as observed in Saccharomyces cerevisiae. Also the increase in the content of the polyunsaturated fatty acids (PUFA), important in the synthesis of compounds such as prostagla…

Total lipidsCiona intestinaliChlorideFluiditytributyltinstresschemistry.chemical_compoundSettore BIO/10 - BiochimicaMembrane fluiditychemistry.chemical_classificationbiologymembrane fluidityarticlepolyunsaturated fatty acidBiological membranemedicine.anatomical_structureBiochemistrySettore CHIM/03 - Chimica Generale E InorganicaSynthesis (chemical)pollutantprostaglandintriacylglyceroladaptive behaviorPolyunsaturated fatty acidmedicine.drugSettore BIO/07 - EcologiaPhospholipidOvarylipid compositionSaccharomyces cerevisiaelong chain fatty acidOvarieInorganic ChemistrylipidmedicineCiona intestinalisdefense mechanismPolyunsaturated fatty acids (PUFA)TriglyceridesphospholipidnonhumanTriglycerideProstaglandineOvaryFatty acidGeneral ChemistryTBTClFatty acidbiology.organism_classificationprostaglandin synthesichemistryCellApplied Organometallic Chemistry
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Heme oxygenase-1 induction modulates microsomal prostaglandin E synthase-1 expression and prostaglandin E2 production in osteoarthritic chondrocytes

2009

Pro-inflammatory cytokines such as interleukin-1beta (IL-1beta) may participate in the pathogenesis of cartilage damage in osteoarthritis (OA) through the production of catabolic enzymes and inflammatory mediators. Induction of heme oxygenase-1 (HO-1) has previously been shown to exert anti-inflammatory effects in different cell types. We have investigated whether HO-1 induction may modify chondrocyte viability and the production of relevant mediators such as oxidative stress and prostaglandin E(2) (PGE(2)) elicited by IL-1beta in OA chondrocytes. Chondrocytes were isolated from OA cartilage and used in primary culture. Cells were stimulated with IL-1beta in the absence or presence of the H…

Transcriptional Activationmedicine.medical_specialtyCell Survivalmedicine.medical_treatmentBiologymedicine.disease_causeProstaglandin E synthaseBiochemistryDinoprostoneChondrocyteChondrocytesMicrosomesInternal medicineOsteoarthritismedicineHumansProstaglandin E2Cells CulturedAggrecanProstaglandin-E SynthasesPharmacologyCOPPMolecular biologyIntramolecular OxidoreductasesHeme oxygenasemedicine.anatomical_structureEndocrinologybiology.proteinHeme Oxygenase-1Oxidative stressProstaglandin Emedicine.drugBiochemical Pharmacology
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