Search results for "Adrenergic receptor"

showing 10 items of 62 documents

MiR-133 Modulates the β1Adrenergic Receptor Transduction Cascade.

2014

Rationale : The sympathetic nervous system plays a fundamental role in the regulation of myocardial function. During chronic pressure overload, overactivation of the sympathetic nervous system induces the release of catecholamines, which activate β-adrenergic receptors in cardiomyocytes and lead to increased heart rate and cardiac contractility. However, chronic stimulation of β-adrenergic receptors leads to impaired cardiac function, and β-blockers are widely used as therapeutic agents for the treatment of cardiac disease. MicroRNA-133 (miR-133) is highly expressed in the myocardium and is involved in controlling cardiac function through regulation of messenger RNA translation/stability. …

MalePhysiologyMessengerheart failureApoptosiscardiomyocytesInbred C57BLSecond Messenger SystemsTransgenicRats Sprague-DawleyBeta-1 adrenergic receptorMiceGenes ReporterReceptorsCyclic AMPGuanine Nucleotide Exchange FactorsMyocytes CardiacAlpha-1D adrenergic receptor3' Untranslated RegionsCells CulturedCulturedbiologyChemistryadrenergic beta-1 receptor antagonists; cardiac; cyclic AMP; heart failure; microRNAs; myocytes; 3' Untranslated Regions; Adenylyl Cyclases; Animals; Apoptosis; Cells Cultured; Cyclic AMP; Cyclic AMP-Dependent Protein Kinases; Disease Progression; Gene Expression Regulation; Genes Reporter; Guanine Nucleotide Exchange Factors; Male; Metoprolol; Mice; Mice Inbred C57BL; Mice Transgenic; MicroRNAs; Myocardium; Myocytes Cardiac; RNA Messenger; Rats; Rats Sprague-Dawley; Receptors Adrenergic beta-1; Recombinant Fusion Proteins; Second Messenger Systems; Physiology; Cardiology and Cardiovascular Medicine; Medicine (all)Medicine (all)Cell biologyAdrenergicadrenergic beta-1 receptor antagonistsDisease ProgressionCARDIAC HYPERTROPHYSignal transductionCardiology and Cardiovascular MedicineAdenylyl CyclasesMetoprololmedicine.medical_specialtyAdrenergic receptorcardiacCellsRecombinant Fusion ProteinsMice Transgenicbeta-1Alpha-1B adrenergic receptorInternal medicinecAMPmedicineAnimalsRNA MessengerReporterPressure overloadalpha and beta adrenoceptorsMyocytesMyocardiumBeta adrenergic receptor kinaseCyclic AMP-Dependent Protein KinasesAlpha-1A adrenergic receptorRatsMice Inbred C57BLMicroRNAsEndocrinologyGenesGene Expression Regulationbiology.proteinRNASprague-DawleyReceptors Adrenergic beta-1MicroRNAs; alpha and beta adrenoceptors; cardiomyocytes; CARDIAC HYPERTROPHY; cAMP
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Nitric oxide is formed in a subpopulation of rat pineal cells and acts as an intercellular messenger.

1998

In the rat pineal, formation of the second messenger cyclic GMP (cGMP) is under adrenergic control. Two important sequential steps mediate adrenergic signal transduction by cGMP, receptor-stimulated nitric oxide (NO) formation by the enzyme NO synthase I (NOS I), and NO-induced cGMP formation by the cytosolic enzyme guanylyl cyclase. With regard to the first step in cGMP transduction (i.e. NO formation) we found, by means of NOS I immunostaining and NADPH-diaphorase staining, that the presence of NOS I was restricted to a subpopulation of pineal cells, generally surrounded by NOS I-negative cells. Considering the fact that NO is able to permeate the cell membrane, the question arises whethe…

Malemedicine.medical_specialtyAdrenergic receptorEndocrinology Diabetes and MetabolismAdrenergicBiologyNitric OxidePineal GlandSecond Messenger SystemsNitric oxideCell membraneRats Sprague-DawleyCellular and Molecular Neurosciencechemistry.chemical_compoundPineal glandEndocrinologyInternal medicineReceptors Adrenergic betamedicineAnimalsCyclic GMPEndocrine and Autonomic SystemsNADPH DehydrogenaseReceptors Adrenergic alphaImmunohistochemistryRatsCytosolmedicine.anatomical_structureEndocrinologychemistryGuanylate CyclaseOxyhemoglobinsSecond messenger systemSignal transductionNitric Oxide SynthaseSignal TransductionNeuroendocrinology
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Alpha-adrenergic modulation of glutathione metabolism in isolated rat hepatocytes.

1988

Glutathione metabolism was studied in isolated hepatocytes from 48-h starved rats. Phenylephrine (10 microM, final concentration) was incubated in the presence of a mixture of L-glutamine, glycine, L-serine, and L-methionine (at 10 times their normal plasma concentration). Alpha-adrenergic stimulation provoked a decrease in glutathione (GSH) synthesis. This effect was accompanied by an enhanced efflux of glutathione from the cells. Phenylephrine stimulated the rate of glutathione disulfide (GSSG) formation; however, this effect was clearly insufficient to explain the disappearance of GSH. Our results suggest that the decrease in cellular GSH levels observed under conditions of shock, stress…

Malemedicine.medical_specialtyAdrenergic receptorPhysiologyEndocrinology Diabetes and MetabolismStimulationIn Vitro Techniqueschemistry.chemical_compoundPhenylephrineReference ValuesPhysiology (medical)Internal medicinemedicineAnimalsCysteineAmino AcidsPhenylephrineGlutathione DisulfideRats Inbred StrainsGlutathioneMetabolismGlutathioneRatsKineticsEndocrinologymedicine.anatomical_structurechemistryLiverHepatocyteGlutathione disulfideEffluxmedicine.drugThe American journal of physiology
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beta-Adrenoceptor and GRK3 expression in human lymphocytes is related to blood pressure and urinary albumin excretion.

2010

31 p., figuras, abreviaturas y bibliografía

Malemedicine.medical_specialtyAmbulatory blood pressureACE inhibitorsG-Protein-Coupled Receptor Kinase 3PhysiologySerum albuminAdrenergicBlood PressureGRK3ExcretionInternal medicineReceptors Adrenergic betaInternal MedicinemedicineAlbuminuriaHumansLymphocytesReceptorUrinary albumin excretionbiologyBeta-adrenoceptorsbusiness.industryReverse Transcriptase Polymerase Chain ReactionBeta adrenergic receptor kinaseMiddle AgedEndocrinologymedicine.anatomical_structureBlood pressureHypertensionbiology.proteinVascular resistanceFemaleCardiology and Cardiovascular MedicinebusinessJournal of hypertension
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Effects of cerivastatin on adrenergic pathways, hypertrophic growth and TGFbeta expression in adult ventricular cardiomyocytes.

2010

Abstract The effects of statin treatment in the setting of heart failure have already been shown. Nevertheless, there is little knowledge about its influence on adrenergic pathways in cardiomyocytes. Therefore, this study investigated the impact of cerivastatin on adrenoceptor-mediated signalling pathways in isolated adult ventricular cardiomyocytes. It focused on two endpoints: hypertrophic growth and TGFbeta expression. Cultured cardiomyocytes were used to study rac activation (analysed by its translocation into the membrane fraction), ROS formation (H 2 DCF fluorescence) and hypertrophic growth ( 14 C-phenylalanine incorporation). Alpha- and beta-adrenoceptor stimulation showed significa…

Malemedicine.medical_specialtyHistologyAdrenergic receptorMAP Kinase Signaling SystemPyridinesp38 mitogen-activated protein kinasesHeart VentriclesAdrenergicAlpha (ethology)StimulationPharmacologyp38 Mitogen-Activated Protein KinasesPathology and Forensic MedicineTransforming Growth Factor betaInternal medicineReceptors Adrenergic betamedicineAnimalsMyocytes CardiacRats WistarCells CulturedHeart FailurebiologyCerivastatinCell BiologyGeneral MedicineReceptors Adrenergic alphaRatsEnzyme ActivationEndocrinologyGene Expression RegulationNAD(P)H oxidaseMitogen-activated protein kinasebiology.proteinHydroxymethylglutaryl-CoA Reductase InhibitorsReactive Oxygen SpeciesProto-Oncogene Proteins c-aktmedicine.drugEuropean journal of cell biology
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Effects of antidepressants in adrenergic neurotransmission of human vas deferens

2000

Objectives. To evaluate the effects of sertraline, fluoxetine, and amitriptyline on the contractile responses of the human vas deferens muscle elicited by norepinephrine, electrical field stimulation, and KCl, because the therapeutic action of antidepressants may be accompanied by sexual dysfunction related to the contractility of the vas deferens smooth muscle. Methods. Ring segments of the epididymal part of the vas deferens were taken from 32 elective vasectomies and mounted in organ baths for isometric recording of tension. We then studied the effects of sertraline, fluoxetine, and amitriptyline on the neurogenic and agonist-induced contractile responses. Results. Amitriptyline caused c…

Malemedicine.medical_specialtyNifedipineAdrenergic receptorAmitriptylineUrologyAdrenergicSynaptic TransmissionNorepinephrine (medication)Vas DeferensNifedipineCulture TechniquesFluoxetineSertralineInternal medicinemedicineHumansAmitriptylineSertralineFluoxetineDose-Response Relationship Drugbusiness.industryVas deferensCalcium Channel BlockersAntidepressive AgentsReceptors AdrenergicEndocrinologymedicine.anatomical_structureCalciumbusinessMuscle Contractionmedicine.drugUrology
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Molecular connectivity to find β-blockers with low toxicity

1997

Abstract Molecular connectivity has been used to find new β-blocker drugs using linear discriminant analysis and connectivity functions with different topological descriptors. Among the selected compounds stands out the probucol and the β-carotene. Both of them interact with β adrenoceptors.

Molecular modelLow toxicityChemistryStereochemistryOrganic ChemistryClinical BiochemistryProbucolPharmaceutical ScienceLinear discriminant analysisBiochemistryβ adrenoceptorDrug DiscoverymedicineMolecular Medicineβ adrenergic receptorMolecular Biologymedicine.drugBioorganic & Medicinal Chemistry Letters
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Is the beta3-adrenoceptor (ADRB3) a potential target for uterorelaxant drugs?

2007

International audience; The management of premature birth still remains unsatisfactory. Since the relative lack of efficiency and/or safety of current tocolytic agents have been highlighted, it is necessary to develop new uterorelaxant drugs deprived of important maternal and foetal side effects. Our work reported in this review focuses on a potential new target for tocolytic drugs, the beta3-adrenoceptor (ADRB3). This third type of ADRB is shown to be present and functional in human myometrium. We demonstrated that ADRB3 agonists are able to inhibit in-vitro spontaneous contractions of myometrial strips, via a cyclic AMP-mediated pathway. Furthermore, we established that ADRB3 is the predo…

Muscle RelaxationMESH : Receptors Adrenergic beta-3MESH : Adrenergic beta-AgonistsUterusAdrenergic beta-3 Receptor AgonistsMESH: Adrenergic beta-AgonistsPharmacologyUterine contractionUterine Contraction0302 clinical medicineMESH: PregnancyMESH : UterusPregnancyObstetrics and GynaecologyMedicineMESH : FemaleMESH: Obstetric Labor Premature[ SDV.MHEP.GEO ] Life Sciences [q-bio]/Human health and pathology/Gynecology and obstetricsreproductive and urinary physiology0303 health sciencesMESH : MyometriumMyometriumMESH : Obstetric Labor PrematureObstetrics and GynecologyAdrenergic beta-Agonists3. Good healthmedicine.anatomical_structureMuscle relaxation030220 oncology & carcinogenesisTocolyticMESH: Uterine ContractionMyometriumMESH: MyometriumMESH: UterusFemalemedicine.symptomTocolytic agentmedicine.medical_specialtyAdrenergic receptorAdrenergic beta-3 Receptor Agonists[SDV.MHEP.GEO]Life Sciences [q-bio]/Human health and pathology/Gynecology and obstetricsMESH : Muscle Relaxation03 medical and health sciencesObstetric Labor PrematureInternal medicineHumans030304 developmental biologyMESH: Humansbusiness.industryMESH : HumansUterus[SDV.MHEP.GEO] Life Sciences [q-bio]/Human health and pathology/Gynecology and obstetricsMESH : PregnancyEndocrinologyProceedingsReceptors Adrenergic beta-3MESH: Muscle RelaxationbusinessMESH: Receptors Adrenergic beta-3MESH: FemaleMESH : Uterine Contraction
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Die Wirkung von ?-Receptoren blockierenden Substanzen auf die durch Adrenalin gesteigerte 42K-Abgabe des Meerschweinchenvorhofs

1966

An isolierten linken Meerschweinchenvorhofen wurde der Einflus des Adrenalins und der β-Receptoren blockierenden Substanz 1-(3-Methylphenoxy)-2-hydroxy-3-isopropylaminopropan (MHIP; Ko 592) auf den K-Umsatz und auf elektrophysiologische Mesgrosen, die mit Hilfe der Mikroelektrodentechnik bestimmt wurden, untersucht.

PharmacologyGuinea pigAtrium (architecture)Adrenergic receptorChemistryBlocking (radio)Pharmacology toxicologyGeneral MedicinePharmacologyNaunyn-Schmiedebergs Archiv f�r Experimentelle Pathologie und Pharmakologie
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8-NH2-Boldine, an Antagonist of α1Aand α1BAdrenoceptors without Affinity for the α1DSubtype: Structural Requirements for Aporphines at α1-Adrenocepto…

2005

Structure-activity analysis of 21 aporphine derivatives was performed by examining their affinities for cloned human alpha (1A), alpha (1B) and alpha (1D) adrenoceptors (AR) using membranes prepared from rat-1 fibroblasts stably expressing each alpha (1)-AR subtype. All the compounds tested competed for [ (125)I]-HEAT binding with steep and monophasic curves. The most interesting compound was 8-NH (2)-boldine, which retains the selective affinity for alpha(1A)-AR (pKi = 6.37 +/- 0.21) vs. alpha(1B)-AR (pKi = 5.53 +/- 0.11) exhibited by 1,2,9,10-tetraoxygenated aporphines, but shows low affinity for alpha(1D)-AR (pKi < 2.5). Binding studies on native adrenoceptors present in rat cerebral cor…

Pharmacologychemistry.chemical_classificationeducation.field_of_studyAdrenergic receptorStereochemistryOrganic ChemistryPopulationAntagonistPharmaceutical ScienceBiologyAnalytical Chemistrychemistry.chemical_compoundComplementary and alternative medicinechemistryDrug DiscoveryMolecular MedicineStructure–activity relationshipBoldineAporphineBinding siteInositol phosphateeducationPlanta Medica
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