Search results for "Aging"

showing 10 items of 10496 documents

Telomere Length and Frailty: The Helsinki Birth Cohort Study

2018

Objectives: Telomere length is associated with aging-related pathologies. Although the association between telomere length and frailty has been studied previously, only a few studies assessing longitudinal changes in telomere length and frailty exist. Design: Longitudinal cohort study. Setting and participants: A subpopulation of the Helsinki Birth Cohort Study consisting of 1078 older adults aged 67 to 79 years born in Helsinki, Finland, between 1934 and 1944. Measures: Relative leukocyte telomere length (LTL) was measured using quantitative real-time polymerase chain reaction at the average ages of 61 and 71 years, and at the latter the participants were assessed for frailty according to …

0301 basic medicineMaleAgingBODY-WATERbiomarkkeritvanheneminenPHENOTYPECohort StudiesRisk FactorsElectric ImpedanceMedicineLongitudinal StudiesLongitudinal cohortOXIDATIVE STRESSGeneral NursingFinlandTelomere ShorteningtelomeregerasteniaFrailtyHealth PolicyConfoundingAge FactorsRISK FACTORta3142General MedicineASSOCIATIONMiddle Aged3. Good healthCohortBody CompositionbiomarkerFemaleBirth cohortLongevityfrailtyta311103 medical and health sciencesSex FactorsINFLAMMATIONHumansELDERLY CHINESERisk factorOLDER-ADULTSAgedbusiness.industryMORTALITYBiomarkerConfidence intervalTelomere030104 developmental biologyPHYSICAL-ACTIVITYCross-Sectional StudiesSocioeconomic FactorsmarkkeritRelative risk3121 General medicine internal medicine and other clinical medicinetelomeeritGeriatrics and Gerontologybusinesshauraus-raihnausoireyhtymäDemographyJournal of the American Medical Directors Association
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Declining Physical Performance Associates with Serum FasL, miR-21, and miR-146a in Aging Sprinters.

2016

Aging is associated with systemic inflammation and cellular apoptosis accelerating physiological dysfunctions. Whether physically active way of life affects these associations is unclear. This study measured the levels of serum inflammatory and apoptotic molecules, their change over 10 years, and their associations with physical performance in sprint-trained male athletes. HsCRP, cell counts, HGB, FasL, miR-21, and miR-146a were measured cross-sectionally (n=67, 18–90 yrs) and serum FasL, miR-21, and miR-146a and their aging-related associations with physical performance were assessed over a 10-year follow-up (n=49, 50–90 yrs). The cross-sectional study showed positive age correlations for …

0301 basic medicineMaleAgingCelllcsh:MedicineSystemic inflammationBench pressFas ligandRunning0302 clinical medicineYoung adultpikajuoksijatAged 80 and overta3141General Medicineinflammatory responseMiddle Agedmedicine.anatomical_structuremedicine.symptomInflammation MediatorsResearch ArticleAdultmedicine.medical_specialtyFas Ligand ProteinArticle SubjectAdolescentGeneral Biochemistry Genetics and Molecular Biologysprinters03 medical and health sciencesYoung AdultInternal medicinemedicineHumansAgedGeneral Immunology and Microbiologybusiness.industryaginglcsh:R030229 sport sciencesphysical performanceCirculating MicroRNAMicroRNAs030104 developmental biologyEndocrinologyCross-Sectional StudiesPhysical performanceApoptosisPhysical FitnessImmunologybusinessBiomarkersFollow-Up StudiesBioMed research international
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Mild exacerbation of obesity- and age-dependent liver disease progression by senolytic cocktail dasatinib + quercetin.

2021

Abstract Background Nonalcoholic fatty liver disease (NAFLD) is increasingly prevalent and represents a growing challenge in terms of prevention and treatment. A minority of affected patients develops inflammation, subsequently fibrosis, cirrhosis and hepatocellular carcinoma (HCC). HCC is a leading cause of cancer-related death. An increased number of senescent cells correlate with age-related tissue degeneration during NAFLD-induced HCC. Senolytics are promising agents that target selectively senescent cells. Previous studies showed that whereas a combination of the senolytic drugs dasatinib and quercetin (D + Q) reduced NAFLD in mice, D + Q lacked efficacy in removing doxorubicin-induced…

0301 basic medicineMaleAgingCirrhosisDasatiniblcsh:MedicineBiochemistrySenolytics.Liver disease0302 clinical medicineFibrosisNon-alcoholic Fatty Liver DiseaseSenotherapeuticsNonalcoholic fatty liver diseaseDiethylnitrosamineCancerlcsh:CytologyLiver Diseases3. Good healthDasatinib030220 oncology & carcinogenesisHepatocellular carcinomaDisease ProgressionQuercetinmedicine.symptomLiver diseasemedicine.drugShort ReportInflammationDiet High-Fat03 medical and health sciencesmedicineAnimalsObesitylcsh:QH573-671SenolyticMolecular BiologyInflammationbusiness.industrySenolyticslcsh:RCell Biologymedicine.diseasedigestive system diseasesMice Inbred C57BLDisease Models Animal030104 developmental biologyGene Expression RegulationCancer researchbusinessCell communication and signaling : CCS
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Modulation of brain PUFA content in different experimental models of mice.

2016

International audience; The relative amounts of arachidonic acid (AA) and docosahexaenoic acid (DHA) govern the different functions of the brain. Their brain levels depend on structures considered, on fatty acid dietary supply and the age of animals. To have a better overview of the different models available in the literature we here compared the brain fatty acid composition in various mice models (C57BL/6J, CD1, Fat-1, SAMP8 mice) fed with different n-3 PUFA diets (deficient, balanced, enriched) in adults and aged animals. Our results demonstrated that brain AA and DHA content is 1) structure-dependent; 2) strain-specific; 3) differently affected by dietary approaches when compared to gen…

0301 basic medicineMaleAgingClinical Biochemistryfat-1 miceHippocampuschemistry.chemical_compoundMice0302 clinical medicineCerebellumDocosahexaenoic acid (DHA)fatty-acid-compositionFood science2. Zero hungerchemistry.chemical_classificationCerebral CortexArachidonic Acidanxiety-like behaviordocosahexaenoic acidaccelerated mouse samBiochemistryDocosahexaenoic acidArachidonic acid (AA)Arachidonic acidFemaleFatty acid compositionSAMP8 miceBrain regionsPolyunsaturated fatty acidN-3 PUFAdiet-induced obesityDocosahexaenoic AcidsHypothalamusPrefrontal CortexBiology03 medical and health sciencesrat-brainDietary Fats UnsaturatedGenetic modelAnimals[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular Biology[ SDV.BBM ] Life Sciences [q-bio]/Biochemistry Molecular BiologyN 3 pufaBrain Chemistryage-related-changesFatty acidCell BiologyModels Theoreticalgene-expressiondepressive-like behaviorMice Inbred C57BL030104 developmental biologychemistry030217 neurology & neurosurgeryBrain StemProstaglandins, leukotrienes, and essential fatty acids
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The Older Finnish Twin Cohort : 45 Years of Follow-up

2019

AbstractThe older Finnish Twin Cohort (FTC) was established in 1974. The baseline survey was in 1975, with two follow-up health surveys in 1981 and 1990. The fourth wave of assessments was done in three parts, with a questionnaire study of twins born during 1945–1957 in 2011–2012, while older twins were interviewed and screened for dementia in two time periods, between 1999 and 2007 for twins born before 1938 and between 2013 and 2017 for twins born in 1938–1944. The content of these wave 4 assessments is described and some initial results are described. In addition, we have invited twin-pairs, based on response to the cohortwide surveys, to participate in detailed in-person studies; these …

0301 basic medicineMaleAgingHORMONE-REPLACEMENT THERAPYphysical activityBLOOD-PRESSURECohort Studies0302 clinical medicineSurveys and QuestionnairesTwins Dizygotickohonnut verenpaineMedicinekohorttitutkimusGenetics (clinical)FinlandBiological Specimen BanksAged 80 and overalcohol1184 Genetics developmental biology physiologyObstetrics and GynecologytwinsMiddle AgedBiobankPOPULATION-BASED TWINepigenetiikkaDEPRESSIVE SYMPTOMSCohortSKELETAL-MUSCLEFemalefyysinen aktiivisuusCohort studyAdulthypertensionAlcohol DrinkinglongitudinalPhysical activityreviewPAIRS DISCORDANTpitkittäistutkimussmokingENVIRONMENTAL-INFLUENCES03 medical and health sciencesTIME PHYSICAL-ACTIVITYtupakointiDiseases in Twinscohort studyDementiaHumansGENOME-WIDE ASSOCIATIONalkoholi (päihteet)Depressive symptomsQuestionnaire studyAgedkaksostutkimusepigeneticsbusiness.industryagingBaseline surveyTwins Monozygoticmedicine.diseasekaksoset030104 developmental biologyikääntyminenPediatrics Perinatology and Child HealthTELOMERE LENGTHbusiness030217 neurology & neurosurgeryDemographyFollow-Up Studiesdementia
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Lifespan Changes of the human brain in Alzheimer's disease

2018

[EN] Brain imaging studies have shown that slow and progressive cerebral atrophy characterized the development of Alzheimer's Disease (AD). Despite a large number of studies dedicated to AD, key questions about the lifespan evolution of AD biomarkers remain open. When does the AD model diverge from the normal aging model? What is the lifespan trajectory of imaging biomarkers for AD? How do the trajectories of biomarkers in AD differ from normal aging? To answer these questions, we proposed an innovative way by inferring brain structure model across the entire lifespan using a massive number of MRI (N = 4329). We compared the normal model based on 2944 control subjects with the pathological …

0301 basic medicineMaleAgingLongevityHippocampuslcsh:MedicineTrastorns de la cognició en la vellesaAmygdalaArticle03 medical and health sciencesLateral ventricles0302 clinical medicineNeuroimagingmedicine[INFO.INFO-IM]Computer Science [cs]/Medical ImagingHumanslcsh:ScienceAgedCerebral atrophyAged 80 and overMultidisciplinarybusiness.industryNeurodegenerationlcsh:RBrainTrastorns de la memòriaHuman brainMiddle Agedmedicine.disease3. Good health030104 developmental biologymedicine.anatomical_structureFISICA APLICADADisease Progressionlcsh:QFemaleAbnormalitybusinessNeuroscience030217 neurology & neurosurgery
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Aging-associated genes and let-7 microRNAs: a contribution to myogenic program dysregulation in oculopharyngeal muscular dystrophy

2019

Oculopharyngeal muscular dystrophy (OPMD) is a late-onset muscle disease caused by an abnormal (GCN) triplet expansion within the polyadenylate-binding protein nuclear 1 gene and consequent mRNA pr...

0301 basic medicineMaleAgingOculopharyngealMuscle DevelopmentBiochemistryMyoblasts0302 clinical medicine80 and overMuscular DystrophyHMGB1 ProteinPAX7 Transcription FactorCell DifferentiationdifferentiationMiddle AgedCell biologymedicine.anatomical_structureFemaleMyogeninMitogen-Activated Protein KinasesBiotechnologyDifferentiation regeneration skeletal muscleAdultBiologyInclusion BodyOculopharyngeal muscular dystrophy03 medical and health sciencesmicroRNAGeneticsmedicineHumansGenetic Predisposition to Diseasedifferentiation; regeneration; skeletal muscle; Adult; Aged; Aged 80 and over; Aging; Antigens Neoplasm; Cell Differentiation; Female; Gene Expression Regulation; HMGB1 Protein; Humans; Male; MicroRNAs; Middle Aged; Mitogen-Activated Protein Kinases; Muscle Development; Muscular Dystrophy Oculopharyngeal; Myoblasts; Myogenin; Myositis Inclusion Body; PAX7 Transcription Factor; Genetic Predisposition to Diseaseskeletal muscleAntigensMolecular BiologyGeneAgedMessenger RNAMyositisRegeneration (biology)Skeletal musclemedicine.diseaseMicroRNAs030104 developmental biologyMuscle diseaseGene Expression RegulationregenerationNeoplasm030217 neurology & neurosurgery
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p38α regulates actin cytoskeleton and cytokinesis in hepatocytes during development and aging.

2017

[Background]: Hepatocyte poliploidization is an age-dependent process, being cytokinesis failure the main mechanism of polyploid hepatocyte formation. Our aim was to study the role of p38α MAPK in the regulation of actin cytoskeleton and cytokinesis in hepatocytes during development and aging. [Methods]: Wild type and p38α liver-specific knock out mice at different ages (after weaning, adults and old) were used. [Results]: We show that p38α MAPK deficiency induces actin disassembly upon aging and also cytokinesis failure leading to enhanced binucleation. Although the steady state levels of cyclin D1 in wild type and p38α knock out old livers remained unaffected, cyclin B1- a marker for G2/M…

0301 basic medicineMaleAgingRHOAPhysiologylcsh:MedicineArp2/3 complexBiochemistryMitogen-Activated Protein Kinase 14Gene Knockout TechniquesMice0302 clinical medicineContractile ProteinsAnimal CellsMedicine and Health SciencesSmall interfering RNAsCell Cycle and Cell DivisionPost-Translational ModificationPhosphorylationlcsh:ScienceCytoskeletonCyclin B1Cells CulturedCellular SenescenceCytoskeletonMice KnockoutMultidisciplinarybiologyChemistryImmunohistochemistry3. Good healthCell biologyNucleic acidsLiverCell Processes030220 oncology & carcinogenesisCellular TypesAnatomyCellular Structures and OrganellesProtein BindingResearch ArticleMitosismacromolecular substancesProtein Serine-Threonine Kinases03 medical and health sciencesHsp27CyclinsGeneticsAnimalsNon-coding RNAActinCytokinesislcsh:RBiology and Life SciencesProteinsCell BiologyActin cytoskeletonActinsGene regulationCytoskeletal Proteins030104 developmental biologybiology.proteinHepatocytesRNAlcsh:QGene expressionProtein MultimerizationPhysiological ProcessesOrganism DevelopmentCytokinesisBiomarkersDevelopmental BiologyPloS one
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Adolescent and adult mice display differential sensitivity to the effects of bupropion on the acquisition of a water maze task.

2017

Abstract Background Adolescence is characterized by major neurobiological changes, and the effects of some psychoactive drugs seem to differ between adolescents and adults. Bupropion, an antidepressant that is also used to treat nicotine addiction, induces behavioral actions in both adolescent and adult rodents. However, the effects of this drug on spatial ability have not been compared in animals at different stages of their development. The present study was conducted to assess the effects of bupropion on spatial learning and memory in adolescent and adult mice. Methods Adolescent (post-natal day: PND35-36) and adult (PND >65) NMRI mice received bupropion (10, 20 and 40 mg/kg) or saline d…

0301 basic medicineMaleAgingSpatial abilityPhysiologyMorris water navigation taskWater mazePharmacologyAffect (psychology)Task (project management)03 medical and health sciencesMice0302 clinical medicineEscape ReactionMemoryStatistical significancemental disordersmedicineAnimalsMaze LearningBupropionPharmacologyBupropionDose-Response Relationship DrugAge FactorsWaterGeneral Medicine030104 developmental biologyAntidepressantAntidepressive Agents Second-GenerationPsychologypsychological phenomena and processes030217 neurology & neurosurgerymedicine.drugPharmacological reports : PR
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NT3/TrkC Pathway Modulates the Expression of UCP-1 and Adipocyte Size in Human and Rodent Adipose Tissue

2021

Neurotrophin-3 (NT3), through activation of its tropomyosin-related kinase receptor C (TrkC), modulates neuronal survival and neural stem cell differentiation. It is widely distributed in peripheral tissues (especially vessels and pancreas) and this ubiquitous pattern suggests a role for NT3, outside the nervous system and related to metabolic functions. The presence of the NT3/TrkC pathway in the adipose tissue (AT) has never been investigated. Present work studies in human and murine adipose tissue (AT) the presence of elements of the NT3/TrkC pathway and its role on lipolysis and adipocyte differentiation. qRT-PCR and immunoblot indicate that NT3 (encoded by NTF3) was present in human re…

0301 basic medicineMaleAgingSympathetic Nervous SystemEndocrinology Diabetes and Metabolismbeta-adrenoceptorsAdipose tissueWhite adipose tissueTropomyosin receptor kinase Clcsh:Diseases of the endocrine glands. Clinical endocrinologychemistry.chemical_compound0302 clinical medicineEndocrinologyAdipocyteBrown adipose tissueUncoupling Protein 1Original ResearchbiologyChemistryCell Differentiationtropomyosin-related kinase receptor CCell biologymedicine.anatomical_structureAdipose Tissueembryonic structuresFemaleSignal Transductionanimal structuresadipocytesLipolysisUCP-1Mice TransgenicNeurotrophin-303 medical and health scienceswhite adipose tissueneurotrophin-3Receptors Adrenergic betamedicineLipolysisAnimalsHumansReceptor trkCRats WistarAgedCell Sizelcsh:RC648-665Body Weightbrown adipose tissue030104 developmental biologybiology.proteinBlood VesselsThermogenesis030217 neurology & neurosurgeryBiomarkersFrontiers in Endocrinology
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