Search results for "Allergy"

showing 10 items of 3181 documents

How the knowledge of interactions between meningococcus and the human immune system has been used to prepare effective Neisseria meningitidis vaccines

2015

In the last decades, tremendous advancement in dissecting the mechanisms of pathogenicity ofNeisseria meningitidisat a molecular level has been achieved, exploiting converging approaches of different disciplines, ranging from pathology to microbiology, immunology, and omics sciences (such as genomics and proteomics). Here, we review the molecular biology of the infectious agent and, in particular, its interactions with the immune system, focusing on both the innate and the adaptive responses. Meningococci exploit different mechanisms and complex machineries in order to subvert the immune system and to avoid being killed. Capsular polysaccharide and lipooligosaccharide glycan composition, in…

lcsh:Immunologic diseases. AllergyImmunologyGenomicsMeningococcal VaccinesMeningococcal vaccineReview ArticleBiologyMeningitis MeningococcalNeisseria meningitidisProteomicsmedicine.disease_causeImmune systemAntigenConjugate vaccineImmunityAnimals; Gram-Negative Bacteria; Host-Pathogen Interactions; Humans; Immunity; Meningitis Meningococcal; Meningococcal Vaccines; Neisseria meningitidis; Immune System; Immunology and Allergy; ImmunologyGram-Negative BacteriamedicineAnimalsHumansImmunology; Immunology and AllergyImmunology and AllergyNeisseria meningitidisImmunityGeneral MedicineVirologyImmune SystemHost-Pathogen Interactionslcsh:RC581-607
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THE ROLE OF EXTRACELLULAR VESICLES IN MODULATING THE HOST IMMUNE RESPONSE DURING PARASITIC INFECTIONS

2014

Parasites are the cause of major diseases affecting billions of people. As the inflictions caused by these parasites affect mainly developing countries, they are considered as neglected diseases. These parasitic infections are often chronic and lead to significant immunomodulation of the host immune response by the parasite, which could benefit both the parasite and the host and are the result of millions of years of co-evolution. The description of parasite extracellular vesicles (EVs) in protozoa and helminths suggests that they may play an important role in host–parasite communication. In this review, recent studies on parasitic (protozoa and helminths) EVs are presented and their potent…

lcsh:Immunologic diseases. AllergyImmunologyReview ArticleimmunomodulationExtracellular vesiclesprotozoaImmune systemProtozoan infectionHelminthsImmunodulationparasitic diseasesmedicineImmunology and AllergyParasite hostingHelminthshelminthProtozoan InfectionsbiologyHost (biology)biology.organism_classificationmedicine.diseaseImmunologyparasiteProtozoaextracellular vesicleslcsh:RC581-607Frontiers in Immunology
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Tumor-Infiltrating γδ T Lymphocytes: Pathogenic Role, Clinical Significance, and Differential Programing in the Tumor Microenvironment.

2014

There is increasing clinical evidence indicating that the immune system may either promote or inhibit tumor progression. Several studies have demonstrated that tumors undergoing remission are largely infiltrated by T lymphocytes [tumor-infiltrating lymphocytes (TILs)], but on the other hand, several studies have shown that tumors may be infiltrated by TILs endowed with suppressive features, suggesting that TILs are rather associated with tumor progression and unfavorable prognosis. γδ T lymphocytes are an important component of TILs that may contribute to tumor immunosurveillance, as also suggested by promising reports from several small phase-I clinical trials. Typically, γδ T lymphocytes …

lcsh:Immunologic diseases. AllergyImmunologyReview Articleγδ T cellspolarization.Immune systemImmunesuppressionImmunology and AllergyMedicinetumor microenvironmentCytotoxicityTumor microenvironmentimmunosuppressionbusiness.industryCancerDendritic cellT lymphocyteTILmedicine.diseasegamma delta cellIL-17Tumor progressionImmunologygd T cellsInterleukin 17businesslcsh:RC581-607Frontiers in immunology
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Interleukin 10 restores lipopolysaccharide-induced alterations in synaptic plasticity probed by repetitive magnetic stimulation

2020

Systemic inflammation is associated with alterations in complex brain functions such as learning and memory. However, diagnostic approaches to functionally assess and quantify inflammation-associated alterations in synaptic plasticity are not well-established. In previous work, we demonstrated that bacterial lipopolysaccharide (LPS)-induced systemic inflammation alters the ability of hippocampal neurons to express synaptic plasticity, i.e., the long-term potentiation (LTP) of excitatory neurotransmission. Here, we tested whether synaptic plasticity induced by repetitive magnetic stimulation (rMS), a non-invasive brain stimulation technique used in clinical practice, is affected by LPS-induc…

lcsh:Immunologic diseases. AllergyLipopolysaccharides0301 basic medicinenon-invasive brain stimulationInterleukin-1betaImmunologyTNFα-reporter mouseMice TransgenicStimulationNeurotransmissionHippocampusSynaptic TransmissionneuroinflammationInterferon-gammaMice03 medical and health sciences0302 clinical medicineGenes Reportertranscranial magnetic stimulationAnimalsImmunology and Allergyddc:610NeuroinflammationOriginal ResearchInflammationNeuronsNeuronal Plasticitysynaptic plasticityInterleukin-6Tumor Necrosis Factor-alphaChemistryLong-term potentiationInterleukin-10Mice Inbred C57BLOrganoids030104 developmental biologyBrain stimulationSynaptic plasticityExcitatory postsynaptic potentialTumor necrosis factor alphaMicrogliainterleukin 10lcsh:RC581-607Neuroscience030217 neurology & neurosurgery
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eIF2α confers cellular tolerance to S. aureus α-toxin

2015

We report on the role of conserved stress-response pathways for cellular tolerance to a pore forming toxin. First, we observed that small molecular weight inhibitors including of eIF2α-phosphatase, jun-N-terminal kinase (JNK), and PI3-kinase sensitized normal mouse embryonal fibroblasts (MEFs) to the small pore forming S. aureus α-toxin. Sensitization depended on expression of mADAM10, the murine ortholog of a proposed high-affinity receptor for α-toxin in human cells. Similarly, eIF2α (S51A/S51A) MEFs, which harbor an Ala knock-in mutation at the regulated Ser51 phosphorylation site of eukaryotic translation initiation factor 2α, were hyper-sensitive to α-toxin. Inhibition of translation w…

lcsh:Immunologic diseases. AllergyMAPK/ERK pathwayImmunologyeIF2αBiologyCycloheximide03 medical and health scienceschemistry.chemical_compoundCellular toleranceImmunology and AllergyInitiation factorpore forming toxinsReceptorOriginal Research030304 developmental biologyGenetics0303 health sciencesKinase030302 biochemistry & molecular biologyJNK Mitogen-Activated Protein KinasesADAM10Translation (biology)MAPKCell biologyEIF2AK4chemistryPhosphorylationCytolysinS. aureus α-toxinlcsh:RC581-607Frontiers in Immunology
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Centenarians and diet: what they eat in the Western part of Sicily.

2012

Abstract This paper pays attention to the modifiable lifestyle factors such as diet and nutrition that might influence life extension and successful ageing. Previous data reported that in Sicily, the biggest Mediterranean island, there are some places where there is a high frequency of male centenarians with respect to the Italian average. The present data show that in Sicani Mountain zone there are more centenarians with respect to the Italian average. In fact, in five villages of Sicani Mountains, there were 19 people with an age range of 100–107 years old from a total population of 18,328 inhabitants. So, the centenarian number was 4.32-fold higher than the national average (10.37 vs. 2.…

lcsh:Immunologic diseases. AllergyMediterranean climateGerontologyAgingmedicine.medical_specialtyMediterranean dietSaturated fatmedia_common.quotation_subjectImmunologyLongevityShort ReportClinical nutritionlcsh:Geriatricsageing Mediterranean dietMediterranean dietMedicineCentenarianmedia_commonSettore MED/04 - Patologia Generalebusiness.industryPublic healthLongevityAgeing Centenarian Longevity Mediterranean dietlcsh:RC952-954.6AgeingAgeingCentenarianlcsh:RC581-607businessDemographyImmunityageing : IA
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Control of Murine Cytomegalovirus Infection by γδ T Cells

2015

Infections with cytomegalovirus (CMV) can cause severe disease in immunosuppressed patients and infected newborns. Innate as well as cellular and humoral adaptive immune effector functions contribute to the control of CMV in immunocompetent individuals. None of the innate or adaptive immune functions are essential for virus control, however. Expansion of γδ T cells has been observed during human CMV (HCMV) infection in the fetus and in transplant patients with HCMV reactivation but the protective function of γδ T cells under these conditions remains unclear. Here we show for murine CMV (MCMV) infections that mice that lack CD8 and CD4 αβ-T cells as well as B lymphocytes can control a MCMV i…

lcsh:Immunologic diseases. AllergyMuromegalovirusAdoptive cell transferCD3 ComplexT cellImmunologyPopulation-MicrobiologyMiceImmune systemT-Lymphocyte SubsetsMedizinische FakultätVirologyGeneticsmedicineAnimalsCytotoxic T cellddc:610educationlcsh:QH301-705.5Molecular BiologyMice Knockouteducation.field_of_studybiologyvirus diseasesHerpesviridae InfectionsFlow CytometryAdoptive TransferVirologyHigh-Throughput Screening AssaysMice Inbred C57BLmedicine.anatomical_structurelcsh:Biology (General)Immunologybiology.proteinParasitologyAntibodyStem celllcsh:RC581-607CD8Research ArticlePLOS Pathogens
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Inhibition of Arginase 1 Liberates Potent T Cell Immunostimulatory Activity of Human Neutrophil Granulocytes

2021

Myeloid cell arginase-mediated arginine depletion with consecutive inhibition of T cell functions is a key component of tumor immune escape. Both, granulocytic myeloid-derived suppressor cells (G-MDSC) and conventional mature human polymorphonuclear neutrophil granulocytes (PMN) express high levels of arginase 1 and can act as suppressor cells of adaptive anti-cancer immunity. Here we demonstrate that pharmacological inhibition of PMN-derived arginase 1 not only prevents the suppression of T cell functions but rather leads to a strong hyperactivation of T cells. Human PMN were incubated in cell culture medium in the absence or presence of an arginase inhibitor. T cells from healthy donors w…

lcsh:Immunologic diseases. AllergyMyeloidArginineNeutrophilsT cellT-LymphocytesCellImmunologyGranulocyteLymphocyte ActivationProinflammatory cytokineDownregulation and upregulationmedicineImmunology and AllergyHumanshumanCells CulturedOriginal ResearchCell Proliferationarginase 1ArginaseChemistryT cellMolecular biologyArginasemedicine.anatomical_structuregranulocyteactivationTumor Escapelcsh:RC581-607Multiple MyelomaFrontiers in Immunology
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Antigen-specific T cells and cytokines detection as useful tool for understanding immunity against zoonotic infections.

2012

Zoonoses include a broad range of diseases, that are becoming of great interest, due to the climate changing, that cause the adaptation of vectors to new niches and environments. Host immune responses play a crucial role in determining the outcome of infections, as documented by expansion of antigen-specific T cells during several zoonotic infections. Thus, understanding of the contribution of antigen-specific T-cell subsets in the host immune response is a powerful tool to evaluate the different immunological mechanisms involved in zoonotic infections and for the development of effective vaccines. In this paper we discuss the role of T cells in some eukaryotic and prokaryotic infectious mo…

lcsh:Immunologic diseases. AllergyNematodaT-LymphocytesImmunologyReview ArticleAdaptive ImmunityBiologyHost-Parasite InteractionsImmune systemT-Lymphocyte SubsetsAntigen specificImmunityZoonosesAnimalsHumansImmunology and AllergyAntigensTh1-Th2 BalanceZoonoses antigen specific T-cells animal immunology.VaccinesBacteriaZoonotic InfectionGeneral MedicineAcquired immune systemVirologyImmunity InnateHost-Pathogen InteractionsImmunologyCytokinesAdaptationlcsh:RC581-607
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Neuro-endocrine networks controlling immune system in health and disease

2014

The nervous and immune systems have long been considered as compartments that perform separate and different functions. However, recent clinical, epidemiological, and experimental data have suggested that the pathogenesis of several immune-mediated disorders, such as multiple sclerosis (MS), might involve factors, hormones, and neural mediators that link the immune and nervous system. These molecules are members of the same superfamily, which allow the mutual and bi-directional neural–immune interaction. More recently, the discovery of leptin, one of the most abundant adipocyte-derived hormones that control food intake and metabolism, has suggested that nutritional/metabolic status, acting …

lcsh:Immunologic diseases. AllergyNervous systemLeptinMultiple sclerosisExperimental autoimmune encephalomyelitisCentral nervous systemImmunologyAutoimmunityReview ArticleDiseaseMSBiologymedicine.diseaseBioinformatics3. Good healthImmune tolerancemedicine.anatomical_structureImmune systemMetabolismImmunopathologymedicineNeuro-immune modulationImmunology and Allergylcsh:RC581-607MS; autoimmunity; leptin; metabolism; neuro-immune modulation
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